ライフサイエンスコーパス: membranoproliferative

1 revealed endocapillary proliferative (35%), membranoproliferative (29%) and membranous (29%) pattern

2 Histologic patterns were predominantly membranoproliferative (57%) or endocapillary proliferati

3 noninvolved portion of the kidney revealed a membranoproliferative glomerular lesion, a lesion usuall

4 d premature death due to rapidly progressing membranoproliferative glomerular nephritis.

5 athological diagnoses on renal biopsies were membranoproliferative glomerulonephritis (23%) followed

6 ciated with hemolytic uremic syndrome (HUS), membranoproliferative glomerulonephritis (dense deposit

7 (FSGS), 57; immunoglobulin A nephritis, 22; membranoproliferative glomerulonephritis (GN), 18; and m

8 C3 glomerulopathy (C3G) and immune-complex membranoproliferative glomerulonephritis (IC-MPGN) are r

9 We also evaluated the risk of membranoproliferative glomerulonephritis (MPGN) and cryo

10 Membranoproliferative glomerulonephritis (MPGN) is seen

11 Membranoproliferative glomerulonephritis (MPGN) occurs i

12 Membranoproliferative glomerulonephritis (MPGN) recently

13 opathies, such as IgA nephropathy (IgAN) and membranoproliferative glomerulonephritis (MPGN) remains

14 Membranoproliferative glomerulonephritis (MPGN) type II

15 The same trend appeared with membranoproliferative glomerulonephritis (MPGN), diabeti

16 , a model of cryoglobulinemia and associated membranoproliferative glomerulonephritis (MPGN), in whic

17 TSLPtg) mice, which develop cryoglobulinemic membranoproliferative glomerulonephritis (MPGN).

18 y progressive glomerulonephritis (RPGN), and membranoproliferative glomerulonephritis (MPGN).

19 as been associated with de novo or recurrent membranoproliferative glomerulonephritis and acute trans

20 d-stage renal disease in 12; one patient had membranoproliferative glomerulonephritis and another pat

21 een reported to be typically associated with membranoproliferative glomerulonephritis and less freque

22 This included membranoproliferative glomerulonephritis and relapses in

23 nty-one patients with biopsy-proven, diffuse membranoproliferative glomerulonephritis and significant

24 By 6 months of age, ABCA1-/- animals develop membranoproliferative glomerulonephritis due to depositi

25 malities in factor H have been implicated in membranoproliferative glomerulonephritis in both humans

26 ch was before the development of spontaneous membranoproliferative glomerulonephritis in some Cfh(-/-

27 in a mouse model of cryoglobulin-associated membranoproliferative glomerulonephritis induced by over

28 Membranoproliferative glomerulonephritis recurred in eig

29 ients had immune-complex glomerulonephritis: membranoproliferative glomerulonephritis type 1 (n = 12)

30 The predominant renal biopsy findings were membranoproliferative glomerulonephritis type 1 or type

31 ceased donor transplants were not present in membranoproliferative glomerulonephritis type I (adjuste

32 Membranoproliferative glomerulonephritis type I [24 of 2

33 reviously published studies of patients with membranoproliferative glomerulonephritis type I are smal

34 allograft for end-stage renal disease due to membranoproliferative glomerulonephritis type I is prese

35 Membranoproliferative glomerulonephritis type II (MPGN I

36 Membranoproliferative glomerulonephritis type II (MPGN I

37 ypical hemolytic uremic syndrome (aHUS), and membranoproliferative glomerulonephritis type II (MPGN2)

38 Dense deposit disease (DDD; also known as membranoproliferative glomerulonephritis type II) is a p

39 eration, atypical hemolytic uremic syndrome, membranoproliferative glomerulonephritis type II, and pa

40 nephropathy to 2.09 (95% CI, 1.56-2.78) for membranoproliferative glomerulonephritis type II.

41 innate immunity, and CFH deficiency leads to membranoproliferative glomerulonephritis type II.

42 plant FSGS was 2.25 (1.6-3.1), P<0.0001, for membranoproliferative glomerulonephritis was 2.37 (1.3-4

43 Renal involvement usually manifests as a membranoproliferative glomerulonephritis with marked mon

44 enal (membranous glomerulonephritis [GN] and membranoproliferative glomerulonephritis); hematologic (

45 nephritis, specifically cryoglobulinemic and membranoproliferative glomerulonephritis, and has also b

46 al was seen with recurrent and de novo FSGS, membranoproliferative glomerulonephritis, and HUS/TTP.

47 hey developed splenomegaly, lymphadenopathy, membranoproliferative glomerulonephritis, and lymphocyti

48 nephritides (e.g. Alport's Syndrome, type II membranoproliferative glomerulonephritis, and membranous

49 vere kidney disorders including mesangial or membranoproliferative glomerulonephritis, and/or Henoch

50 gy, whereas overexpression of TSLP induced a membranoproliferative glomerulonephritis, as previously

51 ulting from other glomerular diseases (FSGS, membranoproliferative glomerulonephritis, IgA nephropath

52 riably demonstrating severe neonatal anemia, membranoproliferative glomerulonephritis, liver fibrosis

53 tic nephropathy, membranous lupus nephritis, membranoproliferative glomerulonephritis, postinfectious

54 3 glomerulopathies/immune complex-associated membranoproliferative glomerulonephritis, serum-induced

55 on with hepatitis C virus (HCV) infection is membranoproliferative glomerulonephritis, with or withou

56 nemia and severe cryoglobulinemia-associated membranoproliferative glomerulonephritis.

57 recurrent bacterial infections and to type 1 membranoproliferative glomerulonephritis.

58 tis, IgA nephropathy, Alport's syndrome, and membranoproliferative glomerulonephritis.

59 ed with diseases such as IgA nephropathy and membranoproliferative glomerulonephritis.

60 Two patients developed membranoproliferative glomerulonephritis.

61 s nephropathy (HR, 0.88; 95% CI, 0.83-0.93), membranoproliferative GN (HR, 0.84; 95% CI, 0.76-0.92),

62 erulopathy (C3G) and immune complex-mediated membranoproliferative GN (IC-MPGN).

63 t of 141 patients with C3G and Ig-associated membranoproliferative GN (Ig-MPGN) for anti-FB and anti-

64 in C3 glomerulopathy and primary Ig-mediated membranoproliferative GN (Ig-MPGN).

65 hropathy (DN), FSGS, IgA nephropathy (IgAN), membranoproliferative GN (MPGN) (n=19-23 for each diseas

66 Renal microangiopathies and membranoproliferative GN (MPGN) can manifest similar cli

67 Membranoproliferative GN (MPGN) was recently reclassifie

68 C3 activation is a characteristic finding in membranoproliferative GN (MPGN).

69 There was a greater prevalence of membranoproliferative GN among patients with HCV but not

70 sis of HLA serotypes in 338 individuals with membranoproliferative GN and 15,614 individuals with non

71 ent gene mutations typically causing primary membranoproliferative GN and implicate an underlying aut

72 immune complex GNs such as membranous GN and membranoproliferative GN are particularly common renal m

73 whole-genome sequence data from 165 primary membranoproliferative GN cases and 10,250 individuals wi

74 Our analysis included 146 primary membranoproliferative GN cases and 6442 controls who wer

75 ngial proliferative GN in eight patients and membranoproliferative GN in six patients.

76 +/-eGFR was 39.5+/-20.4 ml/min per 1.73 m(2) Membranoproliferative GN was the predominant histologic

77 iated crescentic GN, lupus nephritis, type I membranoproliferative GN), and nephrotic syndrome (minim

78 , lichen planus, vitiligo, cryoglobulinemia, membranoproliferative GN, and NHL.

79 Primary membranoproliferative GN, including complement 3 (C3) gl

80 AN (referent), FSGS, membranous nephropathy, membranoproliferative GN, lupus nephritis, and vasculiti

81 A nephropathy and one specimen of sclerosing membranoproliferative GN, showed bright (2-3+) C4d stain

82 ort of 142 unrelated patients diagnosed with membranoproliferative GN.

83 s in complement genes in nonfamilial primary membranoproliferative GN.

84 t gene variation, is associated with primary membranoproliferative GN.

85 of C3 glomerulopathy and primary Ig-mediated membranoproliferative GN.

86 ntified DGKE gene variants as the cause of a membranoproliferative-like glomerular microangiopathy.

87 Renal biopsy revealed a membranoproliferative pattern of glomerular disease in f

88 linemia, which enables detailed studies of a membranoproliferative pattern of glomerular injury.

89 way and frequently deviates from the classic membranoproliferative pattern of injury on light microsc

90 Renal involvement was of a membranoproliferative type demonstrating thickened capil