ライフサイエンスコーパス: memory

1 daily basis, relatively few are committed to memory.

2 lesions suffer profound failures in episodic memory.

3 knockin (APP-KI) mice with impaired spatial memory.

4 eurological function, including learning and memory.

5 infection, a phenomenon called innate immune memory.

6 We referred to this phenomenon as EMT memory.

7 ng a delayed decision task requiring working memory.

8 s efficient cortical activity during working memory.

9 ng a delayed decision task requiring working memory.

10 cise is a powerful modulator of learning and memory.

11 ovide insight into the maladaptive nature of memory.

12 s indispensable for the establishment of EMT memory.

13 ral Ability, Speed/Flexibility, and Learning/Memory.

14 ophagy in neuronal function, plasticity, and memory.

15 o internal representations in visual working memory.

16 ient for the behavioural expression of odour memory.

17 ur understanding of the neural substrates of memory.

18 siological processes, including learning and memory.

19 s considered a neuronal correlate of working memory.

20 o form sleep-dependent and sleep-independent memory.

21 ential for long-term synaptic plasticity and memory.

22 to facilitate associative fear learning and memory.

23 ltifaceted SARS-CoV-2-specific immunological memory.

24 after training did not require sleep to form memories.

25 and are resistant to the extinction of fear memories.

26 ant for stable encoding of specific types of memories.

27 olecular switching systems and storage-class memories.

28 n of water maze, as well as contextual fear, memories.

29 n in the human brain needed to form episodic memories.

30 to describe the neural substrate for storing memories.

31 electric tetragonality logic and nonvolatile memories(1,2).

32 ng high-quality-factor resonators as quantum memories(3,9).

33 directly related elements of the overlapping memories (A and C images) in isolation.

34 yses to measure reactivation of initial pair memories (A items) during overlapping pair (BC) learning

35 By contrast, trisomic mice exhibited poor memory abilities and disordered prefrontal-hippocampal f

36 chanisms necessary to generate immunological memory able to induce long-term protection against disea

37 In a behavioral study, we found that memory accuracy is enhanced selectively for unexpected h

38 encode space at a finer scale following fear memory acquisition.

39 To assay spatial working memory, all animals performed a reinforced T-maze altern

40 ICANCE STATEMENT The flexibility of episodic memory allows us to remember both the details that diffe

41 m building blocks for magnetic random access memories and magnetic sensors.

42 lidation can weaken maladaptive context-drug memories and reduce the propensity for drug relapse.

43 two tasks: one where attention was guided by memory and another in which attention was explicitly ins

44 cterized by deficits in executive functions, memory and attention.

45 multisensory processes predict higher-order memory and cognition already during childhood, even if s

46 general executive processes, such as working memory and cognitive control, have long been implicated

47 heds light on the pathophysiology of altered memory and cognitive function in BBSOAS.

48 tive tests that may depend on verbal working memory and encoding.

49 cal translation of key mRNAs in learning and memory and expand on the notion of dynamic functional RN

50 We find that contemporaneously developing memory and GC B cells differ in their affinity for antig

51 the CRN to the temporal dynamics of episodic memory and highlight the role of alpha rhythms in reveal

52 Memory and Naive B cells are good potential predictors o

53 r to impairment in cognitive domains such as memory and perceptual reasoning, and act as intermediate

54 ng memory subnetwork relates to both working memory and reasoning performance whereas disruption to t

55 imaging unit with a combination of in-built memory and signal processing capability is imperative to

56 a crucial functional substrate for episodic memory and spatial representation.

57 two covert factors: strength of recognition memory and the criterion for deciding what memory streng

58 has previously been attributed to molecular memory and, more specifically, to the maintenance of adh

59 emory, executive function, language/semantic memory, and global composite) using z-scores for neurops

60 eye movement (REM) sleep regulates emotional memory, and persistent REM sleep impairment after cocain

61 ental changes in behaviors such as learning, memory, and social interactions.

62 directed influences compete for selection in memory, and that the balance of this competition-as refl

63 indicate that THC does impair visual working memory, and that this impairment may be related to both

64 resentations has provided deep insights into memory architecture and its neural underpinnings.

65 revealing when and where different types of memories are retrieved.SIGNIFICANCE STATEMENT Our abilit

66 Immune memories are stored in the form of 'spacers' which are s

67 ppocampal synaptic plasticity, learning, and memory are impaired in these mutant mice.

68 al systems aimed at testing various sorts of memory are therefore also central.

69 elp buffer stress or the effects of negative memories, as well as extinguish maladaptive behaviors.

70 bal action by selectively promoting specific memories associated with local sleep oscillations.

71 rsal hippocampus (CA1), selectively disrupts memories associated with methamphetamine (METH) days aft

72 ment and in five cognitive domains (episodic memory, attention/working memory, executive function, la

73 n (IgG) antibodies, neutralizing plasma, and memory B and memory T cells that persisted for at least

74 center reaction, limiting the generation of memory B cell and long-lived plasma cell responses.

75 ants with B cells led to greater tissue like memory B cell frequencies.

76 te the presence of concurrent donor-specific memory B cell-derived HLA antibodies (DSA-M) in renal al

77 +) T cells (CD3(+)CD8(+)CD161(+)PD1(+)), and memory B cells (CD3(-)CD19(+)CD20(+)CD24(+)CD27(+)) were

78 Memory B cells (MBCs) expressing the transcription facto

79 he generation of long-lived plasma cells and memory B cells and highlight the challenges for successf

80 de evidence that primary FCRL4-bearing human memory B cells are constitutively bound to IgA.

81 expanded upon antigen re-encounter, whereas memory B cells expressed receptors capable of neutralizi

82 Memory B cells had reemerged in 2 of 10 (20%) relapses i

83 h-affinity germinal center (GC) B cells into memory B cells versus plasma cells is a major quest of a

84 Alternatively, subsets of memory B cells were lower in abundance in cirrhotic rela

85 es from ~2,200 IgG-secreting activated human memory B cells, activated ex vivo, demonstrating its ver

86 chemokine receptors were up-regulated in CSF memory B cells.

87 s can perform comparably to the more complex memory-based exemplar models dominant in laboratory sett

88 1] argue that boundary extension (BE), false memory beyond a view, is an artifact of stimulus selecti

89 These associative memory biases were especially pronounced for stimuli rat

90 ecurrent neural network with long short-term memory (BLSTM) to capture the long-range interaction pat

91 opamine replacement on the prioritization of memories by reward and the time-dependence of this effec

92 T cell epigenetic memory can persist long-term, contributing to long-lasti

93 Parkinson's disease have focused on overall memory capacity rather than what is versus what isn't re

94 gulatory effects at eight time points during memory CD4(+) T cell activation with high-depth RNA-seq

95 lop a higher frequency of antiviral effector memory CD4(+) T(EM) cells specific to two immunodominant

96 n pancreatic tumors and increases numbers of memory CD4+ and CD8+ T cells, eradicating all detectable

97 R sequencing, to dissect the human naive and memory CD4+ T cell repertoire against the influenza pand

98 Memory CD8 T cells provide durable protection against di

99 We demonstrate this by functionally altering memory CD8 T cells using CRISPR/Cas9-mediated targeted g

100 cess in genetic alteration of Ag-experienced memory CD8 T cells.

101 SARS-CoV-2-specific memory CD8(+) T cells exhibited functional characteristi

102 he transcriptome, phenotype, and function of memory CD8(+) T cells, sharing the same HSV-1 epitope-sp

103 cells can sometimes acquire properties of a memory cell without encountering foreign antigen.

104 g at a defined effector checkpoint to become memory cells.

105 in the tumor-bearing host, and persisted as memory cells.

106 n rodents and humans suggests that long-term memory consolidation can be enhanced by the exploration

107 y proposes a synaptic-level mechanism of how memory consolidation is affected by sensory stimulation

108 ke and active(4), the ability to switch to a memory consolidation mechanism that is not contingent on

109 Thus, we hypothesized that impaired memory consolidation mechanisms in hippocampal-cortical

110 affecting existing oligodendrocytes impaired memory consolidation of water maze, as well as contextua

111 pus and that SETD6 knockdown interferes with memory consolidation, alters gene expression patterns, a

112 tive conditioning needed increased sleep for memory consolidation, but flies starved after training d

113 vidence that glucocorticoid hormones enhance memory consolidation, helping to ensure that emotionally

114 failed to enhance sleep-dependent procedural memory consolidation.

115 e model, only AT FTP significantly predicted memory decline.

116 urthermore, ISRIB treatment reverses spatial memory deficits and ameliorates working memory in old mi

117 e long term effects, including cognitive and memory deficits.

118 -cortical networks could account for spatial memory deficits.

119 ecision of context and visual discrimination memories depend on interactions between the hippocampus

120 uencing and computational modelling to track memory development during Plasmodium infection and treat

121 insic role of Batf3 in regulating CD8 T cell memory development.

122 quently, the mechanism(s) that contribute to memory development.

123 hence this opens avenues for magnetooptical memory devices.

124 Memory disruption in mild cognitive impairment (MCI) and

125 Strong memories do not destabilize, for instance, although why

126 These data show that reactivation of related memories during new learning leads to dissociable coding

127 opaminergic neuron DAN-i1 can both establish memory during training and acutely terminate learned sea

128 Due to a memory effect, changes of NH protection during antibody

129 This study reveals how memory effects stymied the locomotion of a diversity of

130 presentational change for indirectly related memory elements in hippocampal subfields.

131 ucocorticoid synthesis inhibitor blocked the memory enhancement as well as the potentiated corticoste

132 e response, indicating the dependence of the memory enhancement on glucocorticoid release during the

133 sights into the neural mechanisms underlying memory enhancement, which has value for educational lear

134 This memory enhancing effect (behavioral tagging) is caused b

135 domains (episodic memory, attention/working memory, executive function, language/semantic memory, an

136 ring with memory reconsolidation or inducing memory extinction are two approaches for weakening malad

137 The formation of memory for a novel experience is a critical cognitive ca

138 Instead, our memory for a scene may be largely driven by its visual c

139 LE and juv-adol ELE formed lasting long-term memory for an object location memory task, whereas seden

140 as foraging orientation and navigation, time-memory for food sources, sleep, and learning/memory proc

141 ndscape of engram cells over the lifespan of memory formation and recall.

142 on, trafficking, expansion, persistence, and memory formation by strategic selection of substrate cel

143 findings provide a new mechanism for social memory formation, through regulating synaptic receptor t

144 kinase 3beta (GSK3beta) plays a key role in memory formation, yet its role in mood regulation remain

145 tested a hypothesis that sleep protects old memories from being forgotten after new learning.

146 ll as the evolution, of altered awareness of memory function across the preclinical and prodromal sta

147 Incorporating indexing into frameworks of memory function opens new avenues of study and even ther

148 esis plays an important role in learning and memory function throughout life.

149 having unique predictive value for long-term memory function, hippocampal volume and training benefit

150 al attention-predicts the quality of ensuing memory-guided behavior.

151 and mPFC, two regions that are important for memory-guided behaviors, in two tasks: one where attenti

152 indicating that the hippocampus coordinates memory-guided eye movements.

153 Together, our results show that memory-guided identity templates proactively impact perc

154 ' theta state of the brain are important for memory-guided navigation and rely on visual inputs.

155 hereas DG's function in spatial learning and memory has been extensively investigated, its role in re

156 ffects of ablation of the molecular clock on memory have been studied in many systems, little has bee

157 However, the effects of minocycline on human memory have not previously been investigated.

158 the lateral striatum supported both types of memories if they were formed in the same environment, ev

159 f synaptic connectivity is linked to working memory impairment and is specific to repeated exposure t

160 get for the treatment of disorders involving memory impairments.SIGNIFICANCE STATEMENT The dentate gy

161 are two approaches for weakening maladaptive memories in disorders such as addiction and post-traumat

162 Human cultures store memories in large distributed assemblies composed of ind

163 fferentiated representations for overlapping memories in the dentate gyrus/CA(2,3) and subiculum subf

164 We studied T cell memory in 42 patients following recovery from COVID-19 (

165 environmental geometry affects human spatial memory in a similar manner to rodent grid-cell activity

166 cers characterizes the acquisition of innate memory in activated NK cells and macrophages.

167 ronal excitability, synaptic plasticity, and memory in adulthood, despite the lack of requirement for

168 mote resurrection of exhausted CD4(+) T-cell memory in chronic infection.

169 or the first time that olfactory associative memory in Drosophila requires signaling by Pigment-dispe

170 ted with lower brain activity during working memory in extensive areas in the default mode network (i

171 n many processes fundamental to learning and memory in health and are implicated in Alzheimer's patho

172 ies and suggests the maintenance of semantic memory in healthy ageing.

173 tial memory deficits and ameliorates working memory in old mice.

174 Meanwhile, studies of memory in patients with Parkinson's disease have focused

175 velopmental milestones and spatial olfactory memory in Ts1Cje neonates.

176 neuroanatomical substrates of g and working memory include the arcuate fasciculus.

177 sting expectation violation does not enhance memory indiscriminately, but specifically aids the disam

178 y task (during magnetoencephalography) and a memory integration task.

179 mpus are critical for the integration of new memories into stable generalized representations in neoc

180 This direct conversion to memory is associated with a selective increase in TCR se

181 A powerful feature of adaptive memory is its inherent flexibility.

182 y simple educational manipulation to improve memory, learning, and transfer of theory-rich content.

183 ological processes that facilitate long-term memories (LTM) but also the suppression of inhibitory pr

184 Additionally, SARS-CoV-2-specific memory lymphocytes exhibited characteristics associated

185 regions involved in multisensory and spatial memory mediate the aftereffect following both trial-wise

186 proposed neural substrate of the integrative memory model supporting the core and attribution functio

187 th higher density of color names are held in memory more accurately.

188 For long-running jobs or large memory needs we provide detailed guidance on high-perfor

189 erolateral EC and the anterior-temporal (AT) memory network appears to drive higher tau deposition in

190 tion in AT than in the posterior-medial (PM) memory network.

191 st memristor for applications in ultra-dense memory, neuromorphic computing and radio-frequency commu

192 ark is possible in networks of noisy, finite-memory neurons, and shows that Hick's law may be a sympt

193 ophila dopaminergic neurons (DANs) reinforce memories of unique valence and provide state-dependent m

194 Concurrently, the concept of "memory of chirality" has been proven as a powerful tool

195 CD8(+) T cell immunological memory of past antigen exposure can confer long-lived pr

196 aracteristics of PPN such as pain threshold, memory of prior injury, and pain sensitization/desensiti

197 nally acquired histone methylation acts as a memory of prior transcription.

198 mpaired the consolidation of fear extinction memory of rats trained in contextual fear conditioning.

199 n conductance response (SCR) and declarative memory of stimulus-outcome contingencies during a differ

200 The resulting short-term memory of the membrane potential allows to generate pers

201 Materials possessing memory of thermal history hold promise for applications

202 Memory-one strategies are a set of Iterated Prisoner's D

203 omplexity informs our understanding of false memories or of the development of recollection and famil

204 Here we used Magnetoencephalography in a memory paradigm assessing correct rejection (CR) of lure

205 entate gyrus (DG) is important for learning, memory, pattern separation, and spatial navigation, and

206 task is sensitive to subtle deterioration in memory performance across ageing.

207 ons, and disrupted the relationships between memory performance and hippocampal connectivity.

208 voluntary and involuntary factors influence memory performance but do so in distinct ways.

209 We show that working memory performance depends on the strength of functional

210 TSD significantly reduced memory performance in a scene recognition task, impaired

211 ep duration was positively related to source memory performance.

212 ease in signal-to-noise ratio during working memory periods as well as an enhancement of the neurons'

213 studying prototypical telluride nonvolatile-memory, "phase-change" materials (PCMs), and related cha

214 Moreover, based on the enhanced memory phenotype of Tiam1 KO mice, Tiam1 may be a potent

215 lasting, pp65-specific T cells with effector memory phenotype were significantly higher in Triplex th

216 Here, a novel magnetic shape memory polymer composite is reported to achieve this.

217 y distinct Blimp1(lo)Id3(hi) tissue-resident memory population that subsequently accumulated at later

218 ntract to a stable activated tissue-resident memory population.

219 ion to contribute to a substantially reduced memory population.

220 To assess which memories predicted drinking, real-world behavior was ass

221 while down-weighting retrieval of erroneous memory predictions to promote an updated representation

222 elevant biological signals with learning and memory processes to regulate feeding.

223 recent decades as general neuromodulators of memory processes, sex steroid hormones such as the poten

224 memory for food sources, sleep, and learning/memory processes.

225 significance of the EC as a major player in memory processing, along with other associated structure

226 system in synaptic plasticity and emotional memory processing.

227 he wrong breadcrumb trail of transcriptional memory provides a framework for understanding how hetero

228 elated events in subfield CA(1) Furthermore, memory reactivation and subiculum representation predict

229 We found that memory reactivation during learning promoted formation o

230 Hence, targeted interference with memory reconsolidation can weaken maladaptive context-dr

231 Interfering with memory reconsolidation or inducing memory extinction are

232 We hypothesized that cocaine-memory reconsolidation requires cannabinoid type 1 recep

233 ings demonstrate that SETD6 is necessary for memory-related nuclear factor-kappaB RELA methylation at

234 eural dynamics, sub-serving auditory working memory, remains largely unexplored.

235 heir current environment and their long-term memory representations.

236 Long-lasting, consolidated memories require not only positive biological processes

237 that people strategically allocated working memory resources by ignoring information that appeared i

238 ogeneous magnitude of CD4(+) T cell-mediated memory responses was observed in regard to lymphoblast e

239 Filamentary resistive random access memory (RRAM) suffers from stochastic switching due to t

240 Memory stability is essential for animal survival when e

241 ocampal complex (HC) is central to long-term memory storage and retrieval as well as spatial navigati

242 ions between the hippocampus (HPC) and other memory storage networks.

243 n memory and the criterion for deciding what memory strength is sufficient for identification.

244 d is asymmetrical: disruption to the working memory subnetwork relates to both working memory and rea

245 iction emphasize the importance of different memory systems for understanding maladaptive use, clinic

246 olfactory, thermosensory, hygrosensory, and memory systems in the fly Drosophila melanogaster.

247 of memory T cells and screening of naive and memory T cell libraries, combined with T cell cloning an

248 differentiation programs in the human CD8(+) memory T cell pool, with potentially broad implications

249 T(RM) cells align closely with conventional memory T cell populations, bearing little resemblance to

250 Here, we used ex vivo stimulation of memory T cells and screening of naive and memory T cell

251 Memory T cells are unable to differentiate into the Th2

252 The fact that CD8(+) memory T cells can have features of both naive and effec

253 doptive transfer studies indicate that these memory T cells develop in a cell-intrinsic manner follow

254 s associated with potent antiviral function: memory T cells secreted cytokines and expanded upon anti

255 odies, neutralizing plasma, and memory B and memory T cells that persisted for at least 3 months.

256 Human skin contains a population of memory T cells that supports tissue homeostasis and prov

257 causal variants, implicating CD4 + effector memory T cells, as well as monocytes, B cells and stroma

258 on depletion of circulating and perivascular memory T cells, this brain signature was enriched and th

259 okine release that resembled those of normal memory T cells.

260 Total memory T-cell responses were measured after anti-CD3 or

261 companied by increased frequency of effector memory T-cells.

262 ocampal-prefrontal theta coupling: a spatial memory task (during magnetoencephalography) and a memory

263 We designed a flexible spatial working memory task that required rats to navigate - after distr

264 ting long-term memory for an object location memory task, whereas sedentary and adol ELE mice did not

265 ocampal function was assessed using a verbal memory task.

266 ngle-trial EEG data from a Sternberg working memory task.

267 nd performance on easy vs. difficult working memory tasks with emotional stimuli contributes to discr

268 Following two hippocampus-dependent memory tasks, DSW occurrence rates, ripple frequencies,

269 ells to the T effector (TEFF), T circulating memory (TCIRCM), and TRM pools by lineage-tracing and si

270 to empower transformative, energy-efficient memory technology.

271 th AUD at baseline and for 4 weeks following memory tests.

272 n contrast, relative frequencies of CXCR5(-) memory Th cells as well as regulatory T and B cells were

273 Selectively targeting memory Th17 cells may be a viable therapeutic approach i

274 ta suggest that age-related increases in the memory Th17 compartment predispose aged mice toward the

275 possible without storing images of shapes in memory that are accessible to more than one sensory moda

276 ometric correspondence between g and working memory - the ability to maintain and control mental info

277 experience and embed critical cue-associated memory traces that promote cocaine relapse.

278 al mechanism for the restructuring of neural memory traces.

279 mation exchange between the HPC and mPFC for memory transfer/consolidation, is not known.

280 as associated with a higher proliferation of memory Tregs.

281 Tissue-resident memory (Trm) CD8(+) T cells mediate protective immunity

282 hich people incorporate their trial-to-trial memory uncertainty with potential rewards and prior beli

283 stimate of a stimulus color and a measure of memory uncertainty, obtained through a rewarded decision

284 S is presented), and how fear and extinction memory undergo consolidation one day after the original

285 an effective, sensitive method of assessing memory updating in rodents.

286 , these results demonstrate that hippocampal memory updating is impaired with aging and establish tha

287 -cell variation and over 50% of CD8+ central memory variation.

288 A first MI-induced bone marrow "memory" via a circulating signal, reducing hematopoietic

289 In contrast, visual memory was worse with greater bilateral inferomedial hip

290 osphorus (BP) is exploited to achieve visual memory, wavelength-selective multibit programming, and e

291 day depend on their ability to induce B cell memory, we have not yet succeeded in developing vaccines

292 ting the importance of sleep in learning and memory, we tested a hypothesis that sleep protects old m

293 d with potentiated recall of fear extinction memory when tested 24 hours after extinction training.

294 ) are biased to differentiate highly similar memories, whereas CA(1) may integrate related events by

295 The fragile and jammed states exhibit memory, while the elastic and pinning dominated regimes

296 thstanding, it is likely that future DBS for memory will employ closed-loop, nuanced approaches that

297 s appear able to encode stimuli into working memory with little, if any, conscious experience of them

298 timescales, and areas important for working memory (WM) contain neurons capable of integrating infor

299 Theoretically, working memory (WM) representations are encoded by population ac

300 aintenance of sensory information in working memory (WM).