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1  novo protein synthesis is not essential for memory reconsolidation.
2 eral amygdala (BLA) is necessary for cocaine-memory reconsolidation.
3 tivating this signaling axis to disrupt fear memory reconsolidation.
4 s critical for regulation of contextual fear memory reconsolidation.
5 nidine-induced disruption of contextual fear memory reconsolidation.
6 tivity, and requisite involvement in cocaine-memory reconsolidation.
7 C) frequency in BLA principal neurons during memory reconsolidation.
8 ing match those that are required to trigger memory reconsolidation.
9 earning occurs within the temporal window of memory reconsolidation.
10 MMP-9 inhibition also interferes with threat memory reconsolidation.
11  the lateral neocortex is necessary for fear memory reconsolidation.
12 e trafficking and synaptic plasticity during memory reconsolidation.
13 facilitated extinction or disrupted drug-cue memory reconsolidation.
14  (DH) critically controls contextual cocaine-memory reconsolidation.
15 ful psychotherapy exerts its effects through memory reconsolidation.
16 uroscience and translational aspects of fear memory reconsolidation.
17 zing exposure therapy based on principles of memory reconsolidation.
18 ral pathways that may underlie therapy-based memory reconsolidation.
19 ion provides a more plausible alternative to memory reconsolidation.
20 ct on which to build integrative models than memory reconsolidation.
21 ecent advances in our understanding of human memory reconsolidation.
22 y reactivation significantly attenuated fear memory reconsolidation.
23 ing how these findings relate to theories of memory reconsolidation.
24 induced synaptic enhancements mediating fear memory reconsolidation.
25 egulated and required in the amygdala for IA memory reconsolidation.
26 n during retrieval and shortly after, during memory reconsolidation.
27 induction of hippocampus-dependent avoidance memory reconsolidation.
28 ption or modification through the process of memory reconsolidation.
29 diated translational control is required for memory reconsolidation.
30 anisms in hippocampus that are necessary for memory reconsolidation.
31                                       During memory reconsolidation, a memory that is recalled become
32 es can be weakened through interference with memory reconsolidation, a process by which memories are
33               Synaptic mechanisms underlying memory reconsolidation after retrieval are largely unkno
34                       As such, disruption of memory reconsolidation after retrieval has received atte
35 y analyzing the role of specific pathways in memory reconsolidation after retrieval.
36 akly learned memories.SIGNIFICANCE STATEMENT Memory reconsolidation allows existing memories to be up
37 B1R antagonism (AM251; 3 mg/kg, i.p.) during memory reconsolidation altered (1) subsequent drug conte
38 that CB1R signaling is necessary for cocaine-memory reconsolidation and associated glutamatergic plas
39                                  We question memory reconsolidation and emotional arousal as sufficie
40 ses, suggesting that CaN affects cocaine-cue memory reconsolidation and extinction by altering T-LA s
41  electrophysiological indices of cocaine cue memory reconsolidation and extinction.
42 ation of a conditioned fear stimulus induces memory reconsolidation and fear memory persistence, whil
43 am substrates are crucial in alcohol-related memory reconsolidation and highlight this pathway as a t
44 ression, we consider the effects of N(2)O on memory reconsolidation and propose a memory-based mechan
45 R) signaling is critical for context-cocaine memory reconsolidation and subsequent drug context-induc
46 s the destabilization required for avoidance memory reconsolidation and suggest that the generation o
47 the hypothesis that memory retrieval induces memory reconsolidation and suggest that the hypothesis n
48 in the BLA is an important modulator of fear memory reconsolidation and that its effects on memory ar
49  NMDAR subtypes in the mechanisms underlying memory reconsolidation and to further understand the con
50 f adrenergic signaling in alcohol-associated memory reconsolidation, and suggest a pharmacological ta
51 omplex activity leads to impairments in fear memory reconsolidation, and that elevating histone acety
52 xplicit memory, emotion-memory interactions, memory reconsolidation, and the relationship between aut
53              Lane et al. view the process of memory reconsolidation as a main ingredient of psychothe
54       Furthermore, CB1R antagonism increased memory reconsolidation-associated spontaneous EPSC (sEPS
55 ar requirement for memory consolidation), in memory reconsolidation at two time points after training
56 ch has shown that extinction training during memory reconsolidation attenuates the recovery of fear m
57 relapse prevention in drug addiction through memory reconsolidation blockade.
58 een widely implicated as being necessary for memory reconsolidation, but gene transcription mechanism
59 eptors (NMDARs) is known to be important for memory reconsolidation, but while most studies show that
60 rnu ammonis 3 DH subregion (dCA3) in cocaine-memory reconsolidation by utilizing the temporal and spa
61              While initial demonstrations of memory reconsolidation came mainly from animal studies,
62                                   Therefore, memory reconsolidation can be both disrupted and enhance
63                             Manipulations of memory reconsolidation can interfere with the ability of
64            Hence, targeted interference with memory reconsolidation can weaken maladaptive context-dr
65  exhibited increased c-Fos expression during memory reconsolidation co-expressed the glutamatergic ne
66                                Disruption of memory reconsolidation decreases the performance of lear
67 activation, and time-dependent, indicating a memory-reconsolidation deficit.
68                    The target article argues memory reconsolidation demonstrates how therapeutic chan
69 a selective enhancement and an impairment of memory reconsolidation dependent on amygdalar PKA.
70                           However, avoidance memory reconsolidation depends on a set of still loosely
71          These results indicate that spatial memory reconsolidation depends on the functionality of L
72 what is known about fear-motivated avoidance memory reconsolidation derives from studies based on fea
73 dministration (3 or 10 ng/hemisphere) during memory reconsolidation did not augment subsequent cocain
74 of fear conditioning, dysregulated circuits, memory reconsolidation, epigenetics, and genetic factors
75                         These data show that memory reconsolidation for a cocaine-paired stimulus is
76           Although disrupting the process of memory reconsolidation has a great potential for clinica
77 es in adolescents, building on principles of memory reconsolidation in adults.
78 , these results suggest that contextual drug-memory reconsolidation in Pavlovian and instrumental set
79 ound that one normal function of hippocampal memory reconsolidation in rats is to modify the strength
80 rgent forms of psychotherapy is a process of memory reconsolidation integrating new emotional experie
81                                              Memory reconsolidation is a fundamental plasticity proce
82                       Authors postulate that memory reconsolidation is a key common neurobiological p
83                  My commentary suggests that memory reconsolidation is an element within self-reorgan
84                         Furthermore, cocaine-memory reconsolidation is associated with CB1R-dependent
85 psychotherapeutic change comes about through memory reconsolidation is compelling; however, the model
86                                              Memory reconsolidation is considered to be the process w
87               Here, the adaptive function of memory reconsolidation is explored in more detail, with
88                                              Memory reconsolidation is hypothesized to be a mechanism
89 ve episodes, and the disruption of affective memory reconsolidation is one plausible mechanism throug
90                                         That memory reconsolidation is the process underlying decisiv
91 ontribution of BLA CB1R signaling to cocaine-memory reconsolidation is unknown.
92                            In the process of memory reconsolidation, it is now clear that every time
93                                              Memory reconsolidation may provide such a target for dis
94                 Thus, applying HDACis during memory reconsolidation might constitute a treatment opti
95 uss the implications of the target article's memory reconsolidation model in the development of more
96  knowledge about the precise rules governing memory reconsolidation of aversive memories that might b
97 ockdown of Arc/Arg3.1 in the LA impairs fear memory reconsolidation of both a recent (1-d-old) as wel
98 indicate that the hippocampus is involved in memory reconsolidation only when reactivation results in
99 we found that the hippocampus is involved in memory reconsolidation only when the animals acquired th
100                             Interfering with memory reconsolidation or inducing memory extinction are
101 heir reactivation through retrieval during a memory reconsolidation process that depends on protein s
102 ating memory trace or on the efficacy of the memory reconsolidation process.
103  results demonstrate how the consequences of memory reconsolidation processes are represented in the
104 look at the functional role of Arc/Arg3.1 in memory reconsolidation processes in the mammalian brain.
105         The functional role of Arc/Arg3.1 in memory reconsolidation processes, however, has not been
106          I suggest it is premature to assume memory reconsolidation provides a unifying model of psyc
107 y and its role in both implicit and explicit memory reconsolidation, rather than balances concerning
108 R antagonism, during, but not after, cocaine-memory reconsolidation reduced drug context-induced coca
109  the hippocampus in fear-motivated avoidance memory reconsolidation remains controversial.
110                 We hypothesized that cocaine-memory reconsolidation requires cannabinoid type 1 recep
111                                              Memory reconsolidation requires the gamma2alpha'1 mushro
112 fore memory retrieval, but not after (during memory reconsolidation), reverses the cocaine-evoked pre
113        Intra-BLA AM251 administration during memory reconsolidation selectively increased this cocain
114                                              Memory reconsolidation that incorporates new emotional e
115                     Lane et al. propose that memory reconsolidation through new emotional experiences
116           Here we show that blockade of drug memory reconsolidation, through infusion of Zif268 antis
117 ocedure to determine what was learned during memory reconsolidation under normal retrieval conditions
118    This reduction in relapse after disrupted memory reconsolidation was not only seen after several h
119 hesis in the DH is not necessary for cocaine-memory reconsolidation, we postulate that recurrent pyra
120 xtinction reflects a behavioural analogue of memory reconsolidation, we show that prevention of conte
121 consolidation, (c) brain plasticity, and (d) memory reconsolidation; we finish with a summary of the
122                               The process of memory reconsolidation - where stored memories become br
123 articipation of the hippocampus in avoidance memory reconsolidation, which is associated with an incr
124 on of the molecular underpinnings of cocaine-memory reconsolidation will likely facilitate the develo
125 at timing extinction to coincide with threat memory reconsolidation would prevent the return of defen

 
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