ライフサイエンスコーパス: meristem

1 enes control auxin levels in the entire root meristem.

2 cell divisions in all cell types of the root meristem.

3 -days which was mediated via damaging apical meristem.

4 sport through the phloem to the shoot apical meristem.

5 is maintained cell autonomously in the shoot meristem.

6 (miRNAs), impairs cell division in the root meristem.

7 the mechanics and growth of the shoot apical meristem.

8 uniquely, to patterning of the inflorescence meristem.

9 to reduced cell division in the root apical meristem.

10 ruits due to an increased size of the floral meristem.

11 on of CUC genes requires STM mobility in the meristem.

12 scular bundles in the nodes and the axillary meristem.

13 ation/differentiation transition in the root meristem.

14 the stem cell population of the root apical meristem.

15 enlargement and multiplication of the shoot meristem.

16 on originally detached from the shoot apical meristem.

17 nce in stem cell maintenance in the vascular meristem.

18 vision and elongation activities in the root meristem.

19 the Arabidopsis (Arabidopsis thaliana) root meristem.

20 cally observed in cells that have exited the meristem.

21 transition to flowering in leaves and apical meristem.

22 ession via the CLV2 receptor in the proximal meristem.

23 -and thereby regulates the activity of shoot meristems.

24 n levels and disrupt primordia initiation in meristems.

25 atures with the organizers of root and shoot meristems.

26 wth-attenuating hormone across leaf and stem meristems.

27 f a self-organising system, similar to plant meristems.

28 is essential for a steady fuelling of plant meristems.

29 anization and developmental fate of axillary meristems.

30 ases the percentage of nodules with multiple meristems.

31 oot cell types, in embryos, and shoot apical meristems.

32 d between the oldest extinct and extant root meristems.

33 boundaries to establish identity of adjacent meristems.

34 ifferentiation in specialized regions called meristems.

35 on the establishment and activity of branch meristems.

36 ap (LRC) is the outermost tissue of the root meristem [1], and it is known to play an important role

37 ion is a conserved feature of vascular plant meristems [4].

38 ody arises through the activity of an apical meristem (a niche of cells or a single cell).

39 Within the root apical meristem, a group of slowly dividing quiescent center ce

40 rmined by a coordinated arrest of all active meristems, a process known as global proliferative arres

41 ses, especially those involved in repressing meristem activity and ABA-mediated dehydration pathways.

42 GSNOR maintains root meristem activity and prevents cell death via inhibiting

43 gy is shaped by factors that modulate floral meristem activity and size, and the identity, number and

44 ark-grown seedlings have reduced root apical meristem activity, as observed in the clasp-1 null mutan

45 transcription factor which regulates floral meristem activity.

46 the Arabidopsis (Arabidopsis thaliana) root meristem acts as an organizer that promotes stem cell fa

47 efects and an increase in dead cells in root meristems after CPT treatment demonstrates that there ar

48 In plants, apical meristems allow continuous growth along the body axis.

49 Axillary meristems (AMs) give rise to lateral shoots and are crit

50 plant architecture is determined by axillary meristems (AMs).

51 r, the position and the fate of the Axillary Meristems (AMs).

52 of leaf initiation, an enlarged shoot apical meristem and an increase in the number of juvenile leave

53 s, which promote cell production in the root meristem and cell expansion in the elongation zone.

54 constructs on the lengths of the root apical meristem and cortical cells in the elongation zone confi

55 is, GA 20-oxidation, is required in both the meristem and elongation zone.

56 d in the transition zone located between the meristem and elongation zones.

57 correlated with rounding of the shoot apical meristem and induction of TGSQA expression, a tulip gene

58 -of-function mutant Sln1d (4) also uncoupled meristem and inflorescence size from plant height.

59 eriphery and is strong throughout the floral meristem and intersepal regions.

60 e in phyllotaxis in vip3 was observed at the meristem and related to defects in spatial patterns of a

61 rs in roots after laser ablation removed the meristem and root cap.

62 eased CYCA3;4 levels result in aberrant root meristem and stomatal divisions, mimicking phenotypes of

63 ct bZIPs orchestrate floral induction at the meristem and that FAC formation is largely combinatorial

64 es at stage II and rescues mutant defects in meristem and tissue establishment.

65 egions of the shoot apical meristem, lateral meristem and young stems.

66 ranscription factor is expressed in axillary meristems and binds to the promoter of WUSCHEL, repressi

67 r layer and glume primordia of spikelet pair meristems and floral meristems, respectively.

68 florescence stem, and early arrest of floral meristems and floral organ primordia.

69 the vascular plants lack such indeterminate meristems and have an overall sporophyte form comprising

70 oles in the specification of axillary floral meristems and lemma identity.

71 VRN2 has a dual function, confining VRN2 to meristems and primordia, where it has specific developme

72 ranslocated out of the treated leaf to shoot meristems and roots than in plants grown under control c

73 s, with higher transcript levels in the root meristems and senesced nodules.

74 was found to promote cell divisions in root meristems and stimulate lateral root branching.

75 R5:YFP Upon auxin microapplication, both lfs meristems and TIBA-pin apices activated DR5:YFP expressi

76 oot and shoot growth, a smaller shoot apical meristem, and an enlarged root cap.

77 ed and deformed plastids in the shoot apical meristem, and develop a mass of callus tissue at the sho

78 part of complexes promoting flowering at the meristem, and little is known about the role of other bZ

79 ic activity, for example, of the root apical meristem, and position new sites of outgrowth, such as d

80 phase the arrested SAM behaves as a dormant meristem, and they strongly support AP2 as a master regu

81 New cells in the meristem are generated by stem cells and transit-amplify

82 Meristems are highly regulated structures ultimately res

83 During lateral root (LR) formation, new LR meristems are specified to support the outgrowth of LRs

84 Shoot apical meristems are stem cell niches that balance proliferatio

85 Our results suggest that AP2 controls meristem arrest by repressing genes related to axillary

86 Meristem arrest is also controlled genetically.

87 stained proliferative activity of sporophyte meristems at plants' shoot and root tips, a trait known

88 erentiate in the final iteration of axillary meristem branching.

89 plants to grow organs efficiently out of the meristem by reorganizing the cellular growth rather than

90 g, AGO10, maintains stem cell homeostasis in meristems by sequestration of miR165/6, a conserved miRN

91 Plant meristems carry pools of continuously active stem cells,

92 t PRR-TZF1-TOR molecular axis modulates root meristem cell proliferation by integrating both transcri

93 s of circadian core oscillators, affect root meristem cell proliferation mediated by Target Of Rapamy

94 s over-expressed in Arabidopsis, root apical meristem cell size increases, and morphogenetic capacity

95 n induced 'hypersensitive' response in which meristem cells become necrotic and kill E. solidaginis h

96 It also plays a role in transporting Zn to meristem cells in the TBs.

97 and ZmSCR1h transcripts accumulate in ground meristem cells of developing leaf primordia and in Zmscr

98 Because the cellular organization of meristems changes when root growth stops, it has been im

99 criptomic analysis, inducing AP2 activity in meristems close to arrest.

100 Like other meristems, thorn meristems contain stem cells but, in the case of thorns,

101 f BP, implying a non-cell autonomous mode of meristem control via one or more GSL metabolites.

102 We found that the precocious phenotypes of meristem-deficient mutants are a consequence of reduced

103 ted this question by examining the effect of meristem-deficient mutations on vegetative phase change

104 y, exert different roles in mediating floral meristem determinacy and ovule development, respectively

105 es for putative regulators of cell shape and meristem determinacy as well as a general signature of c

106 oral organ identity determination and floral meristem determinacy in the rosid species Arabidopsis (A

107 In addition, spikelet meristem determinacy is altered in the mutants, which pr

108 he drl genes regulate FM activity and impose meristem determinacy non-cell-autonomously from differen

109 ng bristle identity and maintaining spikelet meristem determinacy.

110 n vivo oxygen measurements, that plant shoot meristems develop embedded in a low-oxygen niche, and th

111 ted with primary root development, including meristem development and auxin homeostasis.

112 ity specification, floral organ development, meristem development and auxin signaling.

113 d formation, such as cell-wall biosynthesis, meristem development and epigenetic pathways.

114 lation of hormones, protein phosphorylation, meristem development and epigenetic processes.

115 the down-regulation of key genes involved in meristem development as the autumn progressed.

116 Another catabolite, acrylic acid, affects meristem development by influencing the progression of t

117 y wall CESA complex acts during shoot apical meristem development.

118 efined or their role, if any, in influencing meristem developmental dynamics.

119 which is expressed primarily in the axillary meristem dome and primordia and in developing stolons.

120 FAC promoting flowering at the shoot apical meristem, downstream of OsFD1.

121 force for activation of the growth-inhibited meristem during bud-break.

122 dule was recruited into vascular plant shoot meristems during evolution to promote indeterminacy, the

123 porary models of plant growth, mechanics and meristem dynamics(4-12).

124 ng in tulip, RNA sequencing was performed on meristem-enriched tissue collected under two contrasting

125 xpression in root tissues including the root meristem (ERF103), the quiescent center (ERF104) and the

126 ormative cell divisions that lead to de novo meristem establishment and tissue patterning associated

127 n fbl17 and increased cell death in the root meristem, even in the absence of genotoxic stress.

128 biosynthesis at boundary domains influences meristem fate decisions during inflorescence development

129 ributing to organ differentiation and flower meristem fate, and uniquely, to patterning of the inflor

130 ce drl1 mutant phenotypes by reducing floral meristem (FM) determinacy.

131 ant shoot development, which are involved in meristem formation and maintenance.

132 TM mobility is required to suppress axillary meristem formation during embryogenesis, to maintain mer

133 This extended phase of floral meristem formation, coupled with slower growth of sepals

134 l2ful3-null triple mutant, the inflorescence meristem formed a normal double-ridge structure, but the

135 evelop a rhizoid from one pole and a thallus meristem from the other, addition of exogenous auxins to

136 ed role for DELLA genes in controlling shoot meristem function and suggests how dissection of pleiotr

137 Meristem function is underpinned by numerous genes that

138 double-ridge structure, but then the lateral meristems generated vegetative tillers subtended by leav

139 (ARR1) control auxin distribution within the meristem, generating an instructive auxin minimum that p

140 In meristems, genetic networks, hormones, and signaling mol

141 e propose that cells in the Arabidopsis root meristem gradually transition from stem cell activity to

142 g networks involving a peptide hormone, root meristem growth factor 1 (RGF1)(1).

143 , CYCB1;1 TCP20 and NLP6&7 also support root meristem growth under N starvation.

144 The two meristems had a distinct contribution to the stem transc

145 ly found that in Arabidopsis SAMs, the HAIRY MERISTEM (HAM) family transcription factors form a conce

146 ulates stem cell numbers of the shoot apical meristem has exclusively been studied in Arabidopsis; as

147 profiles of different hormones within plant meristems has thus far remained scarce.

148 e findings uncover a mechanism that sustains meristem homeostasis through CLASP, and they advance our

149 invokes homeotic shifts in multiple distinct meristem identities, obscures a recurring theme emerging

150 y redundantly are required for inflorescence meristem identity and act as B-function repressors in th

151 t necessitates the coordinated regulation of meristem identity and maturation and lateral organ initi

152 al induction and the transcription of floral meristem identity genes during vernalization.

153 Furthermore, auxin regulates floral meristem identity genes, such as Matricaria inodora RAY2

154 ed by advanced seasonal expression of floral meristem identity genes.

155 of the boundary gene Liguleless1 and confers meristem identity partially independent of the COM2 path

156 This meristem identity pathway has conceptual implications fo

157 sis, play a major role in determining floral meristem identity together with FBP4, while contributing

158 The meristem identity was altered in dormant buds of transge

159 SPIKELET1 orthologue pleiotropically affects meristem identity, floral phyllotaxy and organ initiatio

160 floral number and similar to MFS1, promotes meristem identity.

161 transcription factor homologous to the LATE MERISTEM IDENTITY1 (LMI1) gene of Arabidopsis is the cau

162 n leaves, root vasculature, and shoot apical meristem, implicating it in both local and systemic sign

163 n in tissues including leaf and shoot apical meristem, implying their function in seed germination.

164 The stem cell niche and the size of the root meristem in plants are maintained by intercellular inter

165 ential growth across the epidermis below the meristem in the hypocotyl.

166 lso permitting broad accumulation outside of meristems in response to environmental cues, leading to

167 ime, alternating vegetative and reproductive meristems in the same individual.

168 ipts were predominantly present in the plant meristems, indicating that SPL13 is involved in regulati

169 edited dicotyledonous plants through de novo meristem induction.

170 We show that hypoxia localized to the shoot meristem inhibits the proteolysis of an N-degron-pathway

171 cell proliferation in internode intercalary meristems, inhibits endocytosis, and alters the distribu

172 ctive phenotypes, many related to defects in meristem initiation and maintenance.

173 tanding of the genetic networks that control meristem initiation and stem cell maintenance, including

174 transcription factors have roles in axillary meristem initiation.

175 icating that GA biosynthesis in the axillary meristem is essential for inducing stolon differentiatio

176 w RGF1 regulates the development of the root meristem is essential for understanding stem cell functi

177 n accumulation per unit time in shoot apical meristem is lower than that in root apical tissues in pe

178 early seedling development, the shoot apical meristem is protected from damage as the seedling emerge

179 s, Qian Shou kinase (QSK1) and inflorescence meristem kinase2, which under optimal growth conditions

180 in the subapical regions of the shoot apical meristem, lateral meristem and young stems.

181 s strongly expressed in shoot apices, floral meristems, lateral root primordia and all lateral organ

182 ion and is enriched in shoot and root apical meristems, lateral root primordia, the vascular system,

183 transcription factors, ARABIDOPSIS THALIANA MERISTEM LAYER 1 (ATML1) and its close homolog, define t

184 expressed in the cotyledon and shoot apical meristem, mainly in the cytosol, and that the epidermis

185 genes play an essential role in shoot apical meristem maintenance and floral organ development, and u

186 ATERAL ORGAN SUPRESSOR 1 (MpLOS1), regulates meristem maintenance and lateral organ development in Ma

187 in-containing protein, with no link to known meristem maintenance or flowering time pathways.

188 targeting genes potentially associated with meristem maintenance, flowering time, stomatal density,

189 r several GRCD genes in regulation of flower meristem maintenance, while functional diversification f

190 ntial functions in seed development and root meristem maintenance.

191 which impair cell-to-cell communication and meristem maintenance.

192 r and, furthermore, causes defects in apical meristem maintenance.

193 nal repressor WOX genes in embryogenesis and meristems maintenance, but the mechanism of this interac

194 ISTHALIANAHOMEOBOXGENE1 (ATH1) maintains the meristem marker gene SHOOT MERISTEMLESS (STM) expression

195 fusions and expression of hormone related or meristem marker genes.

196 loss of SPL/SPB function impaired canonical meristem maturation and flower initiation in tomato.

197 flowers is labile, demonstrating that floral meristem maturation involves the stabilisation of positi

198 nces depends on a precisely timed process of meristem maturation mediated by the transcription factor

199 ngthened the time interval over which floral meristems matured.

200 The rapidly proliferating cells in plant meristems must be protected from genome damage.

201 ized organ boundary gene candidate NO APICAL MERISTEM (NAM) supports the hypothesis that it establish

202 n lateral root primordia and the root apical meristem negatively regulates root system architecture.

203 ision activity in both shoot and root apical meristems observed in fbl17 loss-of-function mutants.

204 ding fields of cells within the shoot apical meristem of Arabidopsis show dynamic regulation of mean

205 uggest that WOX function in shoot and floral meristems of Arabidopsis is restricted to the modern WUS

206 ines the shoot stem cell niche in the apical meristems of many angiosperm species; we show that TI1 b

207 ing land plants) develop lateral organs from meristems of sporophytes and gametophytes, respectively.

208 h transcriptional competence in shoot apical meristems of tomato.

209 Inflorescence meristems of vrn1ful2ful3-null and vrn1ful2-null remaine

210 ptional profiling in developing shoot apical meristems of vrs3 suggested that VRS3 acts as a transcri

211 at the shoot apex, but not in the vegetative meristem or stem.

212 idopsis hypocotyl pushes the shoot-producing meristem out of the soil by rapid expansion of cells alr

213 high accumulation of Zn was observed in the meristem part.

214 4 clade genes in regulation of inflorescence meristem patterning was observed.

215 ipient floral primordia in the inflorescence meristem periphery and is strong throughout the floral m

216 the cytosol into the nucleus in cells at the meristem periphery, possibly triggering their differenti

217 Here, we used the growth and meristem phenotypes of Arabidopsis (Arabidopsis thaliana

218 The meristem produces lateral organs in specific patterns, r

219 dopsis, loss of the carboxypeptidase ALTERED MERISTEM PROGRAM1 (AMP1) produces an increase in the rat

220 The mitotic activity of root apical meristem (RAM) is critical to primary root growth and de

221 also ethylene is able to control root apical meristem (RAM) size through activation of the multistep

222 These findings connect meristem redox status and auxin in the control of maize

223 t is characterized by repeated initiation of meristems, regions of dividing cells that give rise to n

224 ental plasticity relies on the activities of meristems, regions where stem cells continuously produce

225 TM to activate STM as well as other axillary meristem regulatory genes.

226 When FveGA20ox4 is mutated, axillary meristems remain dormant or produce secondary shoots ter

227 auxin signaling domains in the early floral meristem remnants allowing for lateral domain identity a

228 Plant stem cell niches, the meristems, require long-distance transport of energy met

229 spatial, auxin-dependent, patterning at the meristem requires Paf1c.

230 mordia of spikelet pair meristems and floral meristems, respectively.

231 rrest of mitotic activity in the root apical meristem, resulting in a short-root phenotype.

232 tivity in meristems upon GPA, but found that meristems retain their identity and proliferative potent

233 s and depends on cell production in the root meristem (RM).

234 The AP2 genes maintain shoot apical meristem (SAM) activity in part by keeping WUSCHEL expre

235 pression of proHLP1::GUS in the shoot apical meristem (SAM) after HS coincides with TOR-E2Fa expressi

236 Lateral organs formed by the shoot apical meristem (SAM) are separated from surrounding stem cells

237 In flowering plants, the shoot apical meristem (SAM) contains a pluripotent stem cell populati

238 The extent to which the shoot apical meristem (SAM) controls developmental decisions, rather

239 The shoot apical meristem (SAM) enables the formation of new organs throu

240 , initiate at the flanks of the shoot apical meristem (SAM) following auxin maxima signals; however,

241 The shoot apical meristem (SAM) gives rise to all aerial plant organs.

242 Enlargement and doming of the shoot apical meristem (SAM) is a hallmark of the transition from vege

243 ell niche, contained within the shoot apical meristem (SAM) is maintained in Arabidopsis by the homeo

244 ifferent with model plants, the shoot apical meristem (SAM) of Moso is composed of six layers of cell

245 The shoot apical meristem (SAM) orchestrates the balance between stem cel

246 ewing stem cells located in the shoot apical meristem (SAM) produce leaves from the SAM peripheral zo

247 the distinct cell types of the shoot apical meristem (SAM) withstand ultraviolet radiation (UVR) str

248 through the phloem to reach the shoot apical meristem (SAM).

249 bps1 mutants to maintain their shoot apical meristem (SAM).

250 D-ZIPIII genes within the plant shoot apical meristem (SAM).

251 long the vertical axis of plant shoot apical meristems (SAMs), stem cells are located at the top whil

252 ns of pluripotent stem cells in shoot apical meristems (SAMs), which continuously produce new abovegr

253 aging, we showed that XyGs are important for meristem shape and phyllotaxis.

254 Meristems show a striking diversity in shape and size.

255 The de novo induction of gene-edited meristems sidesteps the need for tissue culture and prom

256 ical or genetic ablation of LRC cells affect meristem size [7, 8]; however, the molecular mechanisms

257 CLAVATA3 promoter is sufficient to restrict meristem size and promote leaf initiation.

258 t, under the control of cytokinin, regulates meristem size and root growth.

259 nd is required for proper regulation of root meristem size and root hair development.

260 Root meristem size and, consequently, root growth depend on t

261 ssociated protein CLASP sustains root apical meristem size by influencing microtubule organization an

262 We also found that SPL genes control meristem size by repressing WUSCHEL expression via a nov

263 nflorescence tip, revealed that DELLAs limit meristem size in Arabidopsis by directly upregulating th

264 hout affecting the canonical WUSCHEL-CLAVATA meristem size regulators (3) .

265 A), reduces primary root growth, root apical meristem size, and meristematic activity in Arabidopsis.

266 formation during embryogenesis, to maintain meristem size, and to precisely specify organ boundaries

267 P2 in a DELLA semi-dwarf background restored meristem size, but not stem growth, and accelerated flow

268 he LRC to control the position of the TZ and meristem size.

269 s developmental transition, thus controlling meristem size.

270 ematic tissues likely due to the presence of meristem-specific activation regulatory element identifi

271 the StHAP3 transcription factor that directs meristem-specific expression; and the StCASP1B2-like and

272 t increase in fruit size generating enlarged meristems that lead to flowers with extra organs and big

273 This induces meristems that produce shoots with targeted DNA modifica

274 Thorns arise from axillary shoot apical meristems that proliferate for a time and then terminall

275 It has been proposed that the meristems that undergo arrest at the end of the reproduc

276 phological changes occur in the shoot apical meristem, the expression of floral repressors in tulip i

277 ar cambium has a unique function among plant meristems, the stem-cell organizer of this tissue shares

278 Bark tissues develop from two lateral meristems; the phellogen (cork cambium) produces the out

279 zes cell fate in distinct zones of the shoot meristem thereby controlling the spatio-temporal dynamic

280 Like other meristems, thorn meristems contain stem cells but, in th

281 the activity of the secondary inflorescence meristem, thus controlling the number of flowers produce

282 xin response gradient, and the expression of meristem/tissue identity markers are impaired from the "

283 equire vernalization and/or dormancy for the meristem to change from a vegetative to floral state.

284 travels from the leaves to the shoot apical meristem to promote flowering.

285 equire a minimum chilling duration for their meristems to break dormancy and grow fruitfully.

286 ed and measured from organ initiation in the meristems to subsequent morphogenesis and differentiatio

287 lators are up-regulated in leaves during the meristem transition.

288 bitory hormones, and low mitotic activity in meristems upon GPA, but found that meristems retain thei

289 to infer molecular organization of the root meristem, we used a whole-genome approach to determine d

290 In contrast to complex plant meristems, we were unable to correlate the plant morphog

291 endent ecotypes, VRN2 is only active outside meristems when its proteolysis is inhibited in response

292 tem growth and the size of the inflorescence meristem, where flowers initiate.

293 ized groups of pluripotent stem cells termed meristems, which allow for the elaboration of the shoot,

294 is achieved by stem-cell-containing axillary meristems, which are initiated from a leaf axil meristem

295 display a squa phenotype developing axillary meristems, which can eventually turn into inflorescences

296 rly November and overwinter as inflorescence meristems, which complete floral development in spring.

297 Leaves are derived from the shoot apical meristem with three distinct axes: dorsoventral, proximo

298 ehave at the transcriptomic level as dormant meristems, with low mitotic activity and high expression

299 n adaptive mechanism involving protection of meristems within specialized structures named buds in or

300 l-cycle inhibitor KRP2 in the underlying rib meristem, without affecting the canonical WUSCHEL-CLAVAT