ライフサイエンスコーパス: mesenchymal

1 form switching during the transition between mesenchymal and epithelial cellular states, which is a k

2 are driven by distinctive crosstalk between mesenchymal and epithelial subsets of cells observed in

3 Deficiency of Cx43 delayed mesenchymal and osteogenic regeneration while in vivo AM

4 equencing (scRNA-seq) analysis reveals novel mesenchymal and transitional alveolar epithelial states

5 types differed in nature (lymphoid, myeloid, mesenchymal) and abundance of tumor-infiltrating cells.

6 undamental functions for various epithelial, mesenchymal, and immune cells.

7 n is found to affect both haematopoietic and mesenchymal BM cells and we specifically identify a dram

8 al Hh ligands not only regulate a variety of mesenchymal cell behaviors, but they also direct these m

9 allel to the core-Vangl2 PCP axis to control mesenchymal cell clustering.

10 Mesenchymal cell crawling is a critical process in norma

11 gical importance is undisputed, the level of mesenchymal cell heterogeneity within and between organs

12 tify the key players establishing epithelial-mesenchymal cell plasticity during reversible and irreve

13 Interestingly, we found increased pulpal mesenchymal cell proliferation in the presumptive root f

14 ng hepatocytes, endothelial cells (ECs), and mesenchymal cells (MCs) and recently reported the mass p

15 iments to determine the fates of peribiliary mesenchymal cells (PMCs) that surround the bile duct aft

16 xpression of growth factors (Nrg2, Rspo3) by mesenchymal cells acting in a paracrine fashion on lumin

17 pithelial cells into multipolar and invasive mesenchymal cells before differentiating into nonmyocyte

18 ling the organized proliferation of adjacent mesenchymal cells by regulating proper Wnt4 expression d

19 n the CompuCell3D simulation environment) of mesenchymal cells crawling on a two-dimensional substrat

20 ensity in the interstitial area of Nestin(+) mesenchymal cells expressing CXCL12 and myeloid cells ex

21 Chemotaxis of fibroblasts and other mesenchymal cells is critical for embryonic development

22 nd found that co-targeting of epithelial and mesenchymal cells is likely to be the most effective str

23 The gene expression pattern in the cryptal mesenchymal cells showed that receptors of the hormone/c

24 2 processes, providing a source for diverse mesenchymal cells that support formation of the highly f

25 Gli1 was a marker of mesenchymal cells that surround the biliary tree but not

26 l cell behaviors, but they also direct these mesenchymal cells to secrete additional soluble factors

27 crosstalk between epithelium and neighboring mesenchymal cells underpin the generation of different p

28 The mesenchymal cells were considerably more heterogeneous b

29 A-expressing cells were identified as FN1(+) mesenchymal cells which are responsible for the precocio

30 Prominent accumulation of mesenchymal cells within tumors has long been appreciate

31 WNT5A, which is secreted by surrounding mesenchymal cells, acts as a guidance cue to orchestrate

32 These changes result from an infiltration of mesenchymal cells, an important cell type in the pathoge

33 -term viral replication in hematopoietic and mesenchymal cells, but not epithelial cells (IECs), in t

34 e perform an unbiased interrogation of tumor mesenchymal cells, delineating the co-existence of disti

35 icroenvironmental characteristics of colonic mesenchymal cells, including the intrinsic involvement o

36 es on the interaction between epithelial and mesenchymal cells, providing a simple system to investig

37 When Notch or zfh1 are depleted in the mesenchymal cells, tumor growth is compromised.

38 riched with highly plastic hybrid epithelial/mesenchymal cells, which display invasive features and a

39 that restores a mucociliated epithelium from mesenchymal cells.

40 uired for digit identity in a subset of limb mesenchymal cells.

41 chondrogenic cells, and ITGA7+ smooth muscle-mesenchymal cells.

42 o the effect of co-culturing with immune and mesenchymal cells.

43 and increased the proliferation of handplate mesenchymal cells.

44 dinated signals from adjacent epithelial and mesenchymal cells.

45 afts) caused changes in the tumor epithelial-mesenchymal character.

46 p53DeltaIECAktE17K tumors resemble the human mesenchymal colorectal cancer subtype (CMS4), which is c

47 erentiated populations in the epithelial and mesenchymal compartments underlying the coordinated expa

48 thway, as well as conservation of epithelial/mesenchymal cross talk in the intestine, has meant that

49 at therapy-resistant cells associated with a mesenchymal dedifferentiated state displayed elevated re

50 These data suggest mesenchymal-derived NRG1 is a potent mediator of tissue

51 roles of ILF3 in immune response, epithelial-mesenchymal differential editing sites are enriched in g

52 ogy, sEOC maintains a high expression of the mesenchymal effector, TWIST-1.

53 erwent a gradually progressing epithelial-to-mesenchymal (EMT) phenotype following a 21-day exposure

54 unctionally capture the establishment of the mesenchymal-epithelial interface (Nes, Smo, Wnt5a, Nog)

55 epithelial-mesenchymal transition (EMT) and mesenchymal-epithelial transition (MET), in primary tumo

56 duced self-renewal and genes associated with mesenchymal-epithelial transition in renal tubule-formin

57 These findings demonstrate the efficacy of mesenchymal EVs as a therapeutic to reduce injury-relate

58 pancreatic mesenchyme by testing identified mesenchymal factors in an iPSC-based differentiation mod

59 Acquisition of mesenchymal features has been associated with drug resis

60 ber of cancer cell lines with characteristic mesenchymal features show simultaneous up-regulation of

61 in patients receiving statins show enhanced mesenchymal features.

62 can transcription, and enhanced migration of mesenchymal fibroblasts.

63 worst-prognosis subtype was characterized by mesenchymal gene signatures.

64 JD2B reduced TGF-beta2-induced expression of mesenchymal genes, prevented the alterations in endothel

65 cizumab resistance in GBM is associated with mesenchymal/glycolytic shifts involving YKL-40 and ZEB1.

66 produced FGF9 is principally responsible for mesenchymal growth, whereas epithelium-produced FGF9 and

67 the most common of these uncommon tumors are mesenchymal hamartoma and undifferentiated embryonal sar

68 al modeling approach to show that epithelial-mesenchymal heterogeneity can emerge from the noise in t

69 Epithelial-mesenchymal heterogeneity implies that cells within the

70 gram of SCC25, a representative cell type of mesenchymal HNSCC and its normal oral keratinocyte count

71 ng in mesoderm is critical to confer trachea mesenchymal identity in human and mouse.

72 rmine the effects of hyperoxia on epithelial-mesenchymal interactions and to define the mediators of

73 f the key ECM markers in the lung epithelial-mesenchymal interface both at the RNA and protein levels

74 highly expressed in patients with aggressive mesenchymal-like breast cancer.

75 Strikingly, GPX8 knockout in mesenchymal-like cells (MDA-MB-231) resulted in an epith

76 lose their endothelial identity and acquire mesenchymal-like features.

77 n depends on the coordinated movement of its mesenchymal-like leading cells and trailing cells, which

78 uronic acid is sufficient to inhibit several mesenchymal-like properties including cellular invasion

79 neural stem cells involves an epithelial-to-mesenchymal-like transition of a continuous stripe of ce

80 endothelin receptor antagonization inhibited mesenchymal lineage conversion in TGFbeta1-exposed human

81 cycle delay, and decreases in expression of mesenchymal marker genes SNAI1, SNAI2, and TWIST1.

82 ST6Gal-I expression displayed enrichment in mesenchymal markers (N-cadherin, slug, snail, fibronecti

83 hanced in vitro cell migration and invasion, mesenchymal markers (through the ERK-ZEB1-vimentin axis

84 RII simultaneously expressed endothelial and mesenchymal markers and transcription factors regulating

85 Mesenchymal markers snail and MMP14 were upregulated in

86 estigate expression levels of epithelial and mesenchymal markers.

87 EGFR correlates with increased epithelial to mesenchymal, migration and invasion, and metastasis.

88 ed selection of lung cancer cells displaying mesenchymal morphology with deficient SOX2 expression, w

89 del enables reconstruction of the epithelial-mesenchymal-myeloid unit of the distal lung, thereby ope

90 and maintains WNT signaling with underlying mesenchymal neighbors, while its overlying epithelial ce

91 Here we provide in vivo evidence that the mesenchymal niche controls tumour initiation in trans.

92 ly, single-cell RNA-sequencing analyses of a mesenchymal niche model showed that fibroblast-derived P

93 pairs normal hematopoiesis by remodeling the mesenchymal niche.

94 can exhibit different phenotypes-epithelial, mesenchymal, or one or more hybrid epithelial-mesenchyma

95 circulating CD45-CD31-PDPN+ preinflammatory mesenchymal, or PRIME, cells in the blood from patients

96 a receptor tyrosine kinase found in cells of mesenchymal origin such as fibroblasts and pericytes.

97 ng FAK_SRC to upregulate SNAIL and acquire a mesenchymal phenotype and (ii) FAK_SRC inhibition of cel

98 AFs showed that CAFs shape the epithelial-to-mesenchymal phenotype and confer gemcitabine resistance

99 factors known to promote a hybrid epithelial-mesenchymal phenotype can alter the phenotypic compositi

100 on of EGFR-mutant lung cancer cells with the mesenchymal phenotype diminished SOX2 expression and TKI

101 te that CBFbeta is essential to maintain the mesenchymal phenotype of triple-negative breast cancer c

102 inct subtypes; tumors that shift towards the mesenchymal phenotype upon recurrence are generally asso

103 pe in CRC cells while disordered ECM drove a mesenchymal phenotype, similar to well and poorly differ

104 stic link between ARID1A defects and a quasi-mesenchymal phenotype, suggesting that deleterious mutat

105 n promoted TKI sensitivity and inhibited the mesenchymal phenotype.

106 onditions, and supports the acquirement of a mesenchymal phenotype.

107 ncrease as the cells become resistant with a mesenchymal phenotype.

108 icity along EMT and suggests that hybrid and mesenchymal phenotypes arise through independent molecul

109 To what extent does the acquisition of mesenchymal phenotypes by tumor epithelial cells contrib

110 in cellular organization from epithelial to mesenchymal phenotypes, which leads to functional change

111 esenchymal, or one or more hybrid epithelial-mesenchymal phenotypes.

112 To investigate the role of mesenchymal plasticity in PDAC progression, we generated

113 The epithelial-mesenchymal plasticity, in tight association with stemne

114 the population-level dynamics of epithelial-mesenchymal plasticity.

115 Instead, an PDGFRA(HI)/F3(HI)/DLL1(HI) mesenchymal population lines the crypt-villus axis and i

116 ptome, and functional studies, we identified mesenchymal populations with different roles during panc

117 to examine how cell-extrinsic forces impact mesenchymal progenitor cell (MPC) fate.

118 tated the osteogenic differentiation of bone mesenchymal progenitor cells (BMSCs).

119 carring of the lung mediated by pathological mesenchymal progenitor cells (MPCs) that manifest autono

120 Dental mesenchymal progenitor cells in the dental follicle lie

121 so regulates the differentiation of resident mesenchymal progenitor cells.

122 airway smooth muscle mass via recruitment of mesenchymal progenitors to the airway smooth muscle bund

123 The cardiac stroma contains multipotent mesenchymal progenitors.

124 sphatases kappa led to acquisition of a full mesenchymal, rather than a hybrid, phenotype.

125 over, and additional phenotypes that suggest mesenchymal roles of CTNND1.

126 7b and regulates their expression to control mesenchymal specification to cartilage and smooth muscle

127 th muscle, coupling epithelial identity with mesenchymal specification.

128 tracellular matrix remodeling in the case of mesenchymal spheroids and different modes of cell migrat

129 with lower apparent surface tension than for mesenchymal spheroids with higher surface tension.

130 to the expression of markers associated with mesenchymal state and embryonic cellular phenotypes.

131 keeping cancer cells in a highly metastatic mesenchymal state.

132 Ns that underlie the extreme melanocytic and mesenchymal states and the intermediate state.

133 action signatures of different epithelial or mesenchymal states.

134 Bone marrow (BM) mesenchymal stem and progenitor cells (MSPCs) are a crit

135 ed regenerative and antibacterial effects of mesenchymal stem cell (MSCs), we evaluated the safety an

136 sterix is a critical transcription factor of mesenchymal stem cell fate, where its loss or loss of Wn

137 Bone marrow mesenchymal stem cell-derived extracellular vesicles (MS

138 morphological changes of bone marrow-derived mesenchymal stem cells (BM-MSC), and Michigan Cancer Fou

139 Bone mesenchymal stem cells (BMSCs) on the 3D nanofiber assem

140 modeling, the differentiation of bone marrow mesenchymal stem cells (BMSCs) to osteoblasts and chondr

141 eous iron overload clearance and bone marrow mesenchymal stem cells (BMSCs) transplantation following

142 esenchymal stem cells (PDLMSCs) and gingival mesenchymal stem cells (GMSCs).

143 o manipulate nuclear mechanosensing in human mesenchymal stem cells (hMSCs) in vitro.

144 during the in vitro differentiation of human mesenchymal stem cells (hMSCs) into chondrocytes.

145 methylation changes during chondrogenesis of mesenchymal stem cells (MSCs) by Infinium 450 K methylat

146 ring of plasma membrane viscosity changes in mesenchymal stem cells (MSCs) during osteogenic and chon

147 unomodulatory effects of bone marrow derived mesenchymal stem cells (MSCs) has been widely studied an

148 Inconsistent therapeutic efficacy of mesenchymal stem cells (MSCs) in regenerative medicine h

149 Therapeutic factors secreted by mesenchymal stem cells (MSCs) promote angiogenesis in vi

150 Mesenchymal stem cells (MSCs), are excellent candidates

151 ze thermal and mechanical signals applied to mesenchymal stem cells (MSCs).

152 ells, and comparison of periodontal ligament mesenchymal stem cells (PDLMSCs) and gingival mesenchyma

153 The mutation occurs in differentiating mesenchymal stem cells and associates with an impaired o

154 , VEGF produced by mixed retinal cells or by mesenchymal stem cells exerted a paracrine neuroprotecti

155 enefits than their counterparts derived from mesenchymal stem cells in some measures.

156 ting ER+ breast cancer cells and bone marrow mesenchymal stem cells to represent DTCs in a bone marro

157 l implant treatment starts with diffusion of mesenchymal stem cells to the wounded region and their s

158 of therapeutic agents include vasodilators, mesenchymal stem cells, antiinflammatory agents, antiinf

159 tumor origin cells, and potentially BECs and mesenchymal stem cells, which give rise to Kaposi sarcom

160 ated that pASCs exhibited characteristics of mesenchymal stem cells.

161 processes involving mural cells and derived mesenchymal stem cells.

162 erial-induced phenotypical response of human mesenchymal stem cells.

163 Transplantation of mesenchymal stem cells/multipotent stromal cells (MSCs)

164 Multipotent Mesenchymal Stem/Stromal Cells (MSCs) are widely used in

165 Furthermore, we introduce a mesenchymal stromal cell derived from human olfactory ti

166 Ms) as the target of mouse and human cardiac mesenchymal stromal cells (cMSC) with progenitor-like fe

167 Engraftment of engineered mesenchymal stromal cells (eMSCs) together with CD34(+)

168 Human mesenchymal stromal cells (hMSCs) are a promising source

169 We report that BM mesenchymal stromal cells (MSC) undergo massive damage t

170 endothelial precursors, abundantly available mesenchymal stromal cells (MSC) were reprogrammed into i

171 Bone marrow mesenchymal stromal cells (MSCs) have been studied for d

172 ernative approaches aim to infuse allogeneic mesenchymal stromal cells (MSCs) to provide a more gener

173 Prior studies that have transplanted mesenchymal stromal cells (MSCs) without co-administrati

174 lly on interleukin (IL)-33 produced by local mesenchymal stromal cells (mSCs).

175 iated by cell therapy with human bone marrow mesenchymal stromal cells (MSCs).

176 mmunocytes and, importantly, subtypes of VAT mesenchymal stromal cells (VmSCs) that are either immuno

177 ly administering vessel-forming cells (human mesenchymal stromal cells [MSCs] and endothelial colony-

178 Mesenchymal stromal cells did not alter clinical score o

179 We find that mesenchymal stromal cells engineered to express membrane

180 Mesenchymal stromal cells mitigated these cognitive and

181 h lipopolysaccharide, conditioned media from mesenchymal stromal cells reduced astrogliosis, interleu

182 upregulated upon damage and is expressed in mesenchymal stromal cells, macrophages, and Paneth cells

183 partly due to the dysfunction of bone marrow mesenchymal stromal/stem cells (MSCs) during aging.

184 This study reveals intestinal mesenchymal structure at fine anatomic, molecular, and f

185 veals that intact glycopeptide signatures of mesenchymal subtype are associated with a poor clinical

186 led that a highly invasive growth pattern of mesenchymal subtype tumor cells is responsible for its p

187 hich showed higher levels of efficacy in the mesenchymal subtype.

188 ification schemes and high robustness of the mesenchymal subtype.

189 The mesenchymal Tbx4 expression relies on endodermal Wnt act

190 s include adaptive immune system activation, mesenchymal tissue priming and so-called 'remote' (non-i

191 exposed cells on the surface of a disrupted mesenchymal tissue.

192 XN/LPXN binding promoted the transition from mesenchymal to amoeboid mode of movement as well as augm

193 ls and that CBFbeta-depleted cells undergo a mesenchymal to epithelial transition (MET) and re-organi

194 nical EphA2-S897E in melanoma cells led to a mesenchymal-to-amoeboid transition driven by Cdc42 activ

195 A role for mesenchymal-to-endothelial transition in the angiogenic

196 y development, nephron progenitors undergo a mesenchymal-to-epithelial transition and eventually diff

197 downregulated SOX2 and induced epithelial-to-mesenchymal transdifferentiation accompanied by increase

198 ral despite cells undergoing highly invasive mesenchymal transformation and cell migration.

199 e networks of immune-suppression, epithelial-mesenchymal transition (EMT) and angiogenesis as the key

200 but also induced an AKT-dependent epithelial-mesenchymal transition (EMT) and beta-catenin nuclear tr

201 red in transition states, such as epithelial-mesenchymal transition (EMT) and mesenchymal-epithelial

202 te conditioned media (CM) induced epithelial-mesenchymal transition (EMT) and migration in both prima

203 The epithelial-to-mesenchymal transition (EMT) and the unjamming transitio

204 own to causally contribute to the epithelial-mesenchymal transition (EMT) and tumor metastasis.

205 tion, cytotrophoblasts undergo epithelial-to-mesenchymal transition (EMT) as they differentiate into

206 Epithelial-mesenchymal transition (EMT) encompasses dynamic changes

207 Epithelial-to-mesenchymal transition (EMT) has been associated with ca

208 Modulators of epithelial-to-mesenchymal transition (EMT) have recently emerged as no

209 ways, both cytokines also induced epithelial-mesenchymal transition (EMT) in lung cancer cells and pr

210 ivation leads to the induction of epithelial-mesenchymal transition (EMT) in pancreatic ductal adenoc

211 The epithelial-mesenchymal transition (EMT) is a process by which cells

212 al polarity, and appearance of epithelial-to-mesenchymal transition (EMT) markers.

213 xpression in association with the epithelial mesenchymal transition (EMT) occurs frequently during tu

214 e that loss of ARID1A promotes an epithelial-mesenchymal transition (EMT) phenotype and sensitizes PD

215 es revealed that Sfrp1 induced an epithelial-mesenchymal transition (EMT) phenotype in tumor cells wi

216 The epithelial-mesenchymal transition (EMT) programs promote SC and CSC

217 An epithelial-mesenchymal transition (EMT) represents a basic morphoge

218 es, specifically in regard to the epithelial mesenchymal transition (EMT) structural component in mal

219 isplaced retinal cells undergo epithelial to mesenchymal transition (EMT) to form contractile membran

220 In human breast carcinomas, epithelial-to-mesenchymal transition (EMT) upregulates LARP6 expressio

221 The proteolytic profile and epithelial-mesenchymal transition (EMT) were investigated using zym

222 he ESR1 Y537S mutation induced an epithelial-mesenchymal transition (EMT) with cells exhibiting enhan

223 ming is known to occur during the epithelial-mesenchymal transition (EMT), a process that promotes me

224 d that TBL1XR1 promoted stemness, epithelial-mesenchymal transition (EMT), and lung and lymphatic met

225 creased ECM stiffness promotes epithelial-to-mesenchymal transition (EMT), cell invasion, and metasta

226 catenin/TCF4-dependent partial epithelial-to-mesenchymal transition (EMT), followed by the establishm

227 Schwann cell (SC) demyelination, epithelial-mesenchymal transition (EMT), loss of sensation and neur

228 s during labor were subject to epithelial-to-mesenchymal transition (EMT), mediated, in part, by the

229 lular matrix (ECM) remodeling and epithelial mesenchymal transition (EMT), provide mechanistic pathwa

230 HCC pathology involves epithelial to mesenchymal transition (EMT), the basis of cancer phenot

231 cells, although unable to induce epithelial-mesenchymal transition (EMT), VAL exerts potent pro-inva

232 data demonstrate the induction of epithelial-mesenchymal transition (EMT), which included enhanced ex

233 ulates differential expression of epithelial-mesenchymal transition (EMT)-associated genes, including

234 OE CMs present an epithelial-mesenchymal transition (EMT)-like regenerative response

235 vasion and metastasis by inducing epithelial-mesenchymal transition (EMT).

236 ss of cell invasion, adhesion and epithelial-mesenchymal transition (EMT).

237 epithelial cell migration and epithelial to mesenchymal transition (EMT).

238 acinar homeostasis, we induced epithelial-to-mesenchymal transition (EMT).

239 aling and subsequent induction of epithelial-mesenchymal transition (EMT).

240 lls, as a model for physiological epithelial-mesenchymal transition (EMT).

241 ogenic pathways and may affect epithelial-to-mesenchymal transition (EMT).

242 and endoderm germ layers through epithelial-mesenchymal transition (EMT).

243 y potentiates Pi triggered lethal epithelial-mesenchymal transition (EMT).

244 n, invasion, drug resistance, and epithelial-mesenchymal transition (EMT).

245 s emanating from epithelia via epithelial-to-mesenchymal transition (EMT).

246 Endothelial-to-mesenchymal transition (EndMT) is a cellular transdiffer

247 ed angiogenesis by downregulating epithelial-mesenchymal transition and angiogenic pathways.

248 imentin is a classic marker of epithelial-to-mesenchymal transition and is therefore an ideal target

249 h as E-cadherin, which suppresses epithelial-mesenchymal transition and metastasis.

250 They undergo epithelial-to-mesenchymal transition and migrate through the developin

251 stress and interferon responses, epithelial-mesenchymal transition and protein metabolism.

252 to be a mediator of Aldo-induced endothelial-mesenchymal transition and proteoglycan secretion in val

253 cellular matrix genes involved in epithelial-mesenchymal transition are expressed most differently.

254 dent AR signaling mediated HCC epithelial-to-mesenchymal transition by regulating the transcription f

255 progenitor cells that undergo epithelial-to-mesenchymal transition displaying characteristic convers

256 dentifying E-box motifs common to epithelial-mesenchymal transition driver transcription factors in s

257 ted cancer stemness and hybrid epithelial-to-mesenchymal transition features in advanced-stage patien

258 TBXT promotes mesenchymal transition in a subset of common carcinomas,

259 AGRN promoted epithelial-to-mesenchymal transition in primary tumors, whereas SERPIN

260 ts led to enhanced hypoxia and epithelial-to-mesenchymal transition in the primary tumors, elevated l

261 l activation markers and induced endothelial-mesenchymal transition in valvular endothelial cells, re

262 Positivity for markers of an endothelial-to-mesenchymal transition indicates possible endothelial or

263 ial and mesenchymal tumors, since epithelial-mesenchymal transition is a key paradigm for metastasis.

264 oliferation, and activation of epithelial-to-mesenchymal transition leading to increased migration of

265 ocytosis, suppressed invasion and epithelial-mesenchymal transition of ovarian cancer cells.

266 ells and platelets and induced epithelial-to-mesenchymal transition of tumor cells, promoting metasta

267 d in basal cells exhibiting an epithelial-to-mesenchymal transition signature, tumorigenesis proceeds

268 rylated STAT3, and markers of the epithelial-mesenchymal transition than nonneoplastic tissues.

269 ysiology: from the epithelium and epithelial-mesenchymal transition to innate and adaptive immunity p

270 YAP/TEAD engage the epithelial-to-mesenchymal transition transcription factor SLUG to dire

271 ve suggested that the zinc-finger epithelial-mesenchymal transition transcription factor Snai2 (also

272 used to evaluate the CSC, EMT (epithelial-to-mesenchymal transition), and metabolic profiles.

273 ociated with moderate hypoxia, epithelial-to-mesenchymal transition, and inflammatory responses with

274 ctivation of NF-kappaB signaling, epithelial-mesenchymal transition, and lung tumor metastasis.

275 antly induced tumor migration, epithelial-to-mesenchymal transition, and matrix remodeling.

276 ts of cancer progression, such as epithelial-mesenchymal transition, cell migration, and cell invasio

277 y oncogenic processes, such as epithelial-to-mesenchymal transition, cellular migration, invasion, an

278 -3p overexpression also inhibited epithelial mesenchymal transition, expression of Slug, TGF-beta3, p

279 her tumor cells have gone through epithelial-mesenchymal transition, is determinant for the extension

280 nscription factors regulating endothelial-to-mesenchymal transition, similar to TGFbeta1-stimulated e

281 ZEB1, a master epithelial-to-mesenchymal transition-inducing transcription factor, in

282 st1b-a well-known regulator of epithelial-to-mesenchymal transition.

283 capacity plus clear signs of endothelial to mesenchymal transition.

284 944 cells, accompanied by reduced epithelial-mesenchymal transition.

285 inoma, characterized by extensive epithelial-mesenchymal transition.

286 l gene programs that stimulate epithelial-to-mesenchymal transition; however, it is not clear how the

287 and epithelial subsets of cells observed in mesenchymal Tsc2-deficient lungs.

288 umor cells towards a motile, metastatic, and mesenchymal tumor phenotype, mediates resistance to conv

289 rences in RNA editing between epithelial and mesenchymal tumors and a novel mechanism of editing-depe

290 RNA editing profiles between epithelial and mesenchymal tumors, since epithelial-mesenchymal transit

291 essential extracellular matrix receptors in mesenchymal tumors, which mediate cancer progression and

292 sive guidelines on the management of thyroid mesenchymal tumours are currently lacking.

293 Benign thyroid mesenchymal tumours have excellent prognosis, whereas th

294 is, whereas the outcome of malignant thyroid mesenchymal tumours is variable.

295 sms of tumorigenesis have been implicated in mesenchymal tumours ranging from chondroblastoma and gia

296 Soft tissue sarcomas (STSs) are mesenchymal tumours where cytotoxic chemotherapy remains

297 y and aggressive nature of malignant thyroid mesenchymal tumours, a multidisciplinary team-based appr

298 chemoradiotherapy used for malignant thyroid mesenchymal tumours.

299 lysis showed that the dynamic epithelial and mesenchymal Wnt10a expression pattern occurred during ro

300 ion in the PCLS model.Conclusions: Increased mesenchymal Wnt5A during saccular-stage hyperoxia injury