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1 surround a single HSPC attached to a single mesenchymal stromal cell.
2 further improve the therapeutic potential of mesenchymal stromal cells.
3 sing the patient's own immune or bone marrow mesenchymal stromal cells.
4 A, which expressed surface markers common to mesenchymal stromal cells.
5 by Mcl-1 or by interactions with bone marrow mesenchymal stromal cells.
6 h the recipient's respiratory epithelium and mesenchymal stromal cells.
7 y in promoting osteogenic differentiation of mesenchymal stromal cells.
8 ose-derived stromal cells and marrow-derived mesenchymal stromal cells.
9 patients as well as in vitro-cultured human mesenchymal stromal cells.
10 d defects in skull, articular cartilage, and mesenchymal stromal cells.
11 expressed in cells and tissues arising from mesenchymal stromal cells.
12 ng cord blood that was expanded ex vivo with mesenchymal stromal cells.
13 panded ex vivo in cocultures with allogeneic mesenchymal stromal cells.
14 l progenitor colony cells were surrounded by mesenchymal stromal cells.
15 through donor-derived mononuclear cells and mesenchymal stromal cells.
16 ates persistent expression into adulthood in mesenchymal stromal cells.
17 es, and an immunophenotype characteristic of mesenchymal stromal cells.
18 mors contained a mixture of cancer cells and mesenchymal stromal cells.
19 cts of gene expression by tumor-infiltrating mesenchymal stromal cells.
20 ile reducing passive stiffness compared with mesenchymal stromal cells.
21 ling detrimental cross-talk with bone marrow mesenchymal stromal cells.
22 duces collagen deposition in the bone marrow mesenchymal stromal cells.
24 Endomyocardial injections of iron-labeled mesenchymal stromal cells admixed with tissue dye were p
25 sibility of using autologous adipose-derived mesenchymal stromal cells (AdMSCs) for the treatment of
27 agen VII expressed by intradermally injected mesenchymal stromal cells also exhibited a similar half-
29 erse relationship between a subpopulation of mesenchymal stromal cells and cancer cells in the bone m
30 ation inhibits the regenerative potential of mesenchymal stromal cells and derived extracellular vesi
32 after injury were restored by treatment with mesenchymal stromal cells and derived vesicles but not w
33 gineered by inducing chondrogenesis of human mesenchymal stromal cells and devitalized by the impleme
34 The niche is perivascular, created partly by mesenchymal stromal cells and endothelial cells and ofte
35 y causes an immediate, transient response of mesenchymal stromal cells and endothelial cells followed
36 ramedullary bone and bone marrow using human mesenchymal stromal cells and endothelial colony-forming
37 rm repopulating of hematopoietic stem cells, mesenchymal stromal cells and endothelial progenitors.
38 ions reverted after treatment with wild-type mesenchymal stromal cells and extracellular vesicles but
41 The CD40/CD40L-assisted crosstalk between mesenchymal stromal cells and mast cells populating the
42 ncapsulation in microporous hydrogels, human mesenchymal stromal cells and osteoblasts spread rapidly
43 ne marrow myofibroblasts derive from Gli1(+) mesenchymal stromal cells and that a Gli inhibitor targe
45 on of adult human hMSCs (bone marrow-derived mesenchymal stromal cells) and cKit(+) cardiac cells.
46 t nonmalignant peripheral blood lymphocytes, mesenchymal stromal cells, and CD34-positive hematopoiet
47 signalling, adhesion to primary bone-marrow mesenchymal stromal cells, and proliferation of primary
48 plantation of cord-blood cells expanded with mesenchymal stromal cells appeared to be safe and effect
50 nvestigate the paracrine mechanisms by which mesenchymal stromal cells are protective in hypoxic pulm
51 Adventitial fibroblasts (AFs), a subtype of mesenchymal stromal cell, are the dominant constituents
52 re the safety, tolerability, and efficacy of mesenchymal stromal cell-based therapy in pilot clinical
53 In vitro studies have shown that bone marrow mesenchymal stromal cells (BM-MSC) protect AML blasts fr
55 ct of local injection of bone marrow-derived mesenchymal stromal cells (BM-MSCs) or adipose tissue-de
57 ve this challenge, human bone marrow-derived mesenchymal stromal cells (BM-MSCs) were used to efficie
60 stem cells (iHepSCs) and bone marrow derived mesenchymal stromal cells (BMSCs) were used to CBTs for
63 -inducible factor 1alpha was knocked down in mesenchymal stromal cells by lentiviral transfer of shor
64 ave shown convincingly in rodent models that mesenchymal stromal cells can prolong solid organ graft
65 Ms) as the target of mouse and human cardiac mesenchymal stromal cells (cMSC) with progenitor-like fe
66 te+/-SE, 0.01+/-0.002; P<0.001), multipotent mesenchymal stromal cell colony maximum (estimate+/-SE,
67 mate+/-SE, 0.01+/-0.002; P=0.002) in BM, and mesenchymal stromal cell colony maximum in PB (estimate+
71 n in BM stromal elements, including CD146(+) mesenchymal stromal cells, correlates with the degree of
80 lving lipid mediators contribute to improved mesenchymal stromal cell efficacy when exposed to carbon
83 in situ differentiation of human BM-derived mesenchymal stromal cells, enables the robust engraftmen
85 in vivo selection of cytokines that improve mesenchymal stromal cell engraftment into the heart both
86 he batch transduction of bone marrow-derived mesenchymal stromal cells ex vivo, followed by intramyoc
88 separation efficacy of >98% in pre-purified mesenchymal stromal cells, extracted from human dental p
91 ng lipid mediators, and their importance for mesenchymal stromal cells function using gene silencing.
93 modulatory efficacy of regulatory T cells or mesenchymal stromal cells has been demonstrated in vitro
97 In this study, human bone marrow-derived mesenchymal stromal cell (hMSCs) was used as a viral rep
101 Adipocytes differentiate from multipotent mesenchymal stromal cells (hMSCs) through adipogenesis,
104 acts directly on Leptin-Receptor-expressing mesenchymal stromal cells in adult bone marrow to influe
105 s significantly augmented in macrophages and mesenchymal stromal cells in inflamed human pulp tissues
106 and osteogenic activity, a critical role for mesenchymal stromal cells in osteogenesis, and temporal
108 g regulates adipogenesis and osteogenesis by mesenchymal stromal cells in the bone marrow in response
109 ation toward CXCL12 and reducing adhesion to mesenchymal stromal cells in vitro We also found that HI
110 of mesenchymal stem cells (MSCs, also called mesenchymal stromal cells) in endogenous repair and cell
111 logeneic human umbilical cord tissue-derived mesenchymal stromal cells, in comparison to corticostero
112 how that asporin regulates key properties of mesenchymal stromal cells, including self-renewal and mu
113 ing Dab2-deficient embryonic fibroblasts and mesenchymal stromal cells indicated that Dab2 promoted a
114 e nuclear and cellular morphologies of human mesenchymal stromal cells induced by micropillar pattern
116 planting ceramic scaffolds coated with human mesenchymal stromal cells into immune-deficient mice, we
117 brain injured mice receiving human amniotic mesenchymal stromal cells intravenously or intracerebrov
118 hours after surgery, mice were treated with mesenchymal stromal cells IV (1 x 10 cells in 0.05 mL of
119 ate alpha7 nicotinic receptor in bone marrow mesenchymal stromal cells leading to increased CXCL12 ex
122 ologic function in human adult lung-resident mesenchymal stromal cells (LR-MSCs) remain to be elucida
123 upregulated upon damage and is expressed in mesenchymal stromal cells, macrophages, and Paneth cells
128 about the potentially unfavorable effects of mesenchymal stromal cell (MSC) activation on the heart.
130 stretch have been investigated for inducing mesenchymal stromal cell (MSC) differentiation towards t
131 identify asporin (ASPN) as a novel, secreted mesenchymal stromal cell (MSC) factor in the tumor micro
134 But the mechanisms underlying OA-mediated mesenchymal stromal cell (MSC) osteogenic differentiatio
135 to isolate a quiescent and undifferentiated mesenchymal stromal cell (MSC) population from the bone
142 ll apoptosis, and could overcome bone marrow mesenchymal stromal cell (MSC)-induced chemoresistance.
143 ion (DR) in both ex vivo bone marrow-derived mesenchymal stromal cells (MSC) and in vitro 3T3-L1 prea
148 contributions to primary myelofibrosis from mesenchymal stromal cells (MSC) have been suggested by m
149 premetastatic niche by examining the role of mesenchymal stromal cells (MSC) in cancer cell homing.
150 umbilical vein endothelial cell (HUVEC) and mesenchymal stromal cells (MSC) onto the decellularized
151 ure of leukemia cells to bone marrow-derived mesenchymal stromal cells (MSC) promotes accumulation of
153 posure of cultured mouse bone marrow-derived mesenchymal stromal cells (MSC) to hypoxia or an adenovi
156 endothelial precursors, abundantly available mesenchymal stromal cells (MSC) were reprogrammed into i
157 (CAF) have been suggested to originate from mesenchymal stromal cells (MSC), but their relationship
158 ase IL1beta, which induces PGE2 synthesis in mesenchymal stromal cells (MSC), in turn activating beta
159 ytic leukemic (CLL) cells and marrow-derived mesenchymal stromal cells (MSCs) activates both cell typ
161 es osteogenic differentiation of multipotent mesenchymal stromal cells (MSCs) and lineage progression
166 vironments that modulate fate commitments of mesenchymal stromal cells (MSCs) are composed of chemica
178 ing in two murine models of BMF that Gli1(+) mesenchymal stromal cells (MSCs) are recruited from the
183 ed with long-term passaged (P10) aging human mesenchymal stromal cells (MSCs) could be used for bone
184 n iTreg co-cultured with bone marrow-derived mesenchymal stromal cells (MSCs) during short-term ex vi
185 y demonstrated that coculture of islets with mesenchymal stromal cells (MSCs) enhanced islet insulin
186 ) cells in the presence of bone marrow-human mesenchymal stromal cells (MSCs) enhanced the production
188 in preparation for a first-in-human trial of mesenchymal stromal cells (MSCs) for septic shock, we ap
189 n a rapid expansion in clinical trials using mesenchymal stromal cells (MSCs) from a variety of tissu
190 d the adipogenic potential of marrow-derived mesenchymal stromal cells (MSCs) from mice with decrease
191 the BM extracellular fluid were elevated and mesenchymal stromal cells (MSCs) had a reduced capacity
193 The therapeutic potential of donor-derived mesenchymal stromal cells (MSCs) has been investigated i
194 the past decade, the clinical application of mesenchymal stromal cells (MSCs) has generated growing e
196 f the identity and physiological function of mesenchymal stromal cells (MSCs) have been hampered by a
204 n that drives lung failure, while unmodified mesenchymal stromal cells (MSCs) have shown only modest
206 nt strategies have pursued the idea of using mesenchymal stromal cells (MSCs) in combination with ost
207 and safety of osteoarthritis treatment with mesenchymal stromal cells (MSCs) in humans and to obtain
208 strate expression exclusively in multipotent mesenchymal stromal cells (MSCs) in the bone marrow of t
209 eviously identified a resident population of mesenchymal stromal cells (MSCs) in the terminal airways
212 steering the chondrogenic differentiation of mesenchymal stromal cells (MSCs) into either permanent c
213 val, growth, and myogenic differentiation of mesenchymal stromal cells (MSCs) isolated from adipose o
215 one marrow (BMT) as well as ex vivo-expanded mesenchymal stromal cells (MSCs) leads to striking clini
216 ell viability and homogenous distribution of mesenchymal stromal cells (MSCs) observed throughout pri
217 the hypothesis that the favorable effects of mesenchymal stromal cells (MSCs) on infarct repair are m
218 ated when the CLL cells were cocultured with mesenchymal stromal cells (MSCs) or hyaluronic acid or w
222 e show that targeted deletion of Cxcl12 from mesenchymal stromal cells (MSCs) reduces normal HSC numb
223 ates to bone marrow, where interactions with mesenchymal stromal cells (MSCs) shape disease trajector
227 e marrow (BM) niche contains a population of mesenchymal stromal cells (MSCs) that provide physical s
230 ernative approaches aim to infuse allogeneic mesenchymal stromal cells (MSCs) to provide a more gener
231 local administration of bone marrow-derived mesenchymal stromal cells (MSCs) to these patients from
232 eutically relevant quantities of multipotent mesenchymal stromal cells (MSCs) via in vitro culture is
233 and insulin-like growth factor 1 (IGF1) from mesenchymal stromal cells (MSCs) was predicted to have t
234 luid (SF) in the knee serve as reservoirs of mesenchymal stromal cells (MSCs) with potential therapeu
236 CSF) secreted from human bone marrow-derived mesenchymal stromal cells (MSCs), all of which also cont
237 one marrow (BM) affects local cells, such as mesenchymal stromal cells (MSCs), leading to osteolysis
239 emia (CLL) cells interact in the marrow with mesenchymal stromal cells (MSCs), which can enhance CLL-
240 osteogenic properties of bone marrow-derived mesenchymal stromal cells (MSCs)-seeded bone micropartic
254 ant changes in the abundance of perivascular mesenchymal stromal cells (MSCs)/osteoprogenitors and os
256 ly administering vessel-forming cells (human mesenchymal stromal cells [MSCs] and endothelial colony-
257 = 40); arm 2: umbilical cord tissue-derived mesenchymal stromal cells (n = 120), CSI (n = 40); arm 3
259 deprivation and treated with human amniotic mesenchymal stromal cells or conditioned medium showed c
260 lled cortical impact received human amniotic mesenchymal stromal cells or phosphate-buffered saline i
261 deprivation were treated with human amniotic mesenchymal stromal cells or with their secretome (condi
262 ound that by inducing TGF-beta activation on mesenchymal stromal cells (pericytes), Amphiregulin indu
263 ndent phenotypic changes of nonhematopoietic/mesenchymal stromal cells play a key role in TD humoral
264 -gestation chorionic villus-derived placenta mesenchymal stromal cells (PMSCs) as a potential treatme
267 originate from BM, or of healthy BM-derived mesenchymal stromal cells, protected hemophilia A mice f
268 h lipopolysaccharide, conditioned media from mesenchymal stromal cells reduced astrogliosis, interleu
272 arrow (BM) fibrosis thought to be induced by mesenchymal stromal cells stimulated by overproduced gro
273 rved between the amount of FSTL1 produced by mesenchymal stromal cells, stromal ST2 cells, and monocy
274 A growing body of evidence demonstrates that mesenchymal stromal cells, such as stromal fibroblasts,
275 eview the available data on culture-expanded mesenchymal stromal cells tested in renal transplantatio
277 ous assessment of the safety and efficacy of mesenchymal stromal cell therapies to allow the translat
278 have produced promising results for HSCT and mesenchymal stromal cell therapy as alternatives to syst
280 poietic stem cell transplantation (HSCT) and mesenchymal stromal cell therapy have been proposed for
282 skin, and suggest an approach for improving mesenchymal stromal cell therapy in scleroderma and othe
283 te, no study has investigated the effects of mesenchymal stromal cell therapy on the blood-brain barr
284 In mice who survived experimental sepsis, mesenchymal stromal cell therapy protected blood-brain b
285 Ps) are metabolically coupled to bone marrow mesenchymal stromal cells through mitochondrial transfer
286 preconditioning with carbon monoxide allowed mesenchymal stromal cells to be administered later after
288 dicating that the exposure of human amniotic mesenchymal stromal cells to the injured tissue is not n
291 mmunocytes and, importantly, subtypes of VAT mesenchymal stromal cells (VmSCs) that are either immuno
295 y contrast, donor BM-derived mononuclear and mesenchymal stromal cells were more abundant and express
296 roinflammatory cytokines in BM-derived human mesenchymal stromal cells, which are part of the hematop
297 trated that IL11 acts on bone-marrow-derived mesenchymal stromal cells, which induce pro-tumorigenic
298 We hypothesized that preconditioning of mesenchymal stromal cells with carbon monoxide ex vivo w
300 , and activation of EphA3(+)/CD90(+)/Sca1(+) mesenchymal/stromal cells with an EphA3 agonist leads to