ライフサイエンスコーパス: mesenteric

1 refore, blocking IL-6 cytokine signaling in (mesenteric) adipocytes may be a novel approach to blunti

2 clusters, in mice, the serous surface of the mesenteric adipose streaks contains lymphocyte-rich orga

3 y unappreciated diversity of microbes in the mesenteric adipose tissue (MAT) surrounding the GI tract

4 lso documents decreased lipogenic pathway in mesenteric adipose tissue after HFD and/or OVX, independ

5 creeping fat" (CrF), defined as expansion of mesenteric adipose tissue around the inflamed and fibrot

6 ravasation of lymphocytes to the omental and mesenteric adipose tissues is partly mediated by L-selec

7 r-HuBDNF alone could cause mesenteric afferent mechanical hypersensitivity independ

8 augmented discharges in isolated intestinal mesenteric afferent nerves.

9 nstrated a temporally associated increase in mesenteric and colonic vascularity with an increase in m

10 nstrated a temporally associated increase in mesenteric and colonic vascularity with an increase in m

11 ch were generally accompanied by significant mesenteric and hindquarters, but not renal, vasoconstric

12 ase (eNOS) are present in the endothelium of mesenteric and pulmonary arteries.

13 effect on the resting diameter of resistance mesenteric and pulmonary arteries.

14 While 10% to 30% of mesenteric and submandibular lymph node CD4(+) cells bec

15 11,12-epoxyeicosatrienoic acid (EET) induces mesenteric arterial vasodilation, which contributes to t

16 endothelial cells isolated from plaque-free mesenteric arteries (CSE activity high) and plaque-conta

17 Resistance-sized mesenteric arteries (MAs) and pulmonary arteries (PAs) w

18 Isolated mesenteric arteries (MAs) from RGS2(-/-) mice showed an

19 The dilatory role for sensory innervation of mesenteric arteries (MAs) is impaired in Old ( approxima

20 explain TRPV4(EC) -IK/SK channel coupling in mesenteric arteries and its absence in pulmonary arterie

21 Meanwhile, mesenteric arteries and lung vascular endothelial cells

22 olae integrity and density in SHR aortas and mesenteric arteries and the role played by caveolae in e

23 preferentially couple with IK/SK channels in mesenteric arteries and with eNOS in pulmonary arteries.

24 Mesenteric arteries are densely innervated and the nerve

25 channels co-localize with IK/SK channels in mesenteric arteries but not in pulmonary arteries, which

26 downregulated by lipopolysaccharide (LPS) in mesenteric arteries concordant with vascular hypocontrac

27 imary endothelial cells isolated from murine mesenteric arteries express functional Kir2.1 channels s

28 We tested this hypothesis in resistant mesenteric arteries from Adipo-MROE mice using myography

29 An increased response to acetylcholine of mesenteric arteries from rats with cirrhosis (50% effect

30 Mesenteric arteries from SMC-specific Gprc5b-KOs showed

31 t ECs, but not smooth muscle cells, of small mesenteric arteries have Kir currents, which are substan

32 with enhanced HNO-mediated vasorelaxation in mesenteric arteries in vitro and arteriolar dilation in

33 vasodilatation (FIV) assayed in pressurized mesenteric arteries pre-constricted with endothelin-1.

34 Vascular function evaluated ex vivo in mesenteric arteries showed that in wild-type mice, CHF m

35 BH(4) decreased ROS production in aorta and mesenteric arteries supernatant's of both SHR and normot

36 of EET production normalizes the response of mesenteric arteries to vasodilators, with beneficial eff

37 lial Kir channels contribute to FIV of mouse mesenteric arteries via an NO-dependent mechanism, where

38 Small resistance mesenteric arteries were connected to a pressure servo c

39 nt responses to acetylcholine in pressurized mesenteric arteries were reduced in KW versus HW (P<0.01

40 m colic branches of the superior or inferior mesenteric arteries, and selective transcatheter emboliz

41 d with pulsed Doppler flow probes (renal and mesenteric arteries, and the descending abdominal aorta)

42 Using porcine swine mesenteric arteries, the effects of up to 6-day incubati

43 uced surface and total KV 1.5 protein in rat mesenteric arteries.

44 of S1P synthesis reduced vasoconstriction of mesenteric arteries.

45 as well as flow-induced dilatation in murine mesenteric arteries.

46 rlipressin relaxed pulmonary and constricted mesenteric arteries.

47 found in thoracic aortas but not in superior mesenteric arteries.

48 alpha, limits TRPV4(EC) -eNOS signalling in mesenteric arteries.

49 f caveolae-like structures in SHR aortas and mesenteric arteries.

50 d to assess vascular function in third-order mesenteric arteries.

51 Mesenteric arterioles from wild type (WT) and SMTNL1 glo

52 ac trunk (50), hepatic artery (29), superior mesenteric artery (35), and other segments (4).

53 th high or low ligation (LL) of the inferior mesenteric artery (IMA).

54 IIR was established by clamping the superior mesenteric artery (SMA) for 45 minutes followed by 120 m

55 An aneurysm of the superior mesenteric artery (SMA) with a diameter of 2.2 cm was fo

56 of celiac truncus (CT), orifice of superior mesenteric artery (SMA), vena cava inferior confluence (

57 Mesenteric artery and aortic transcriptome was profiled

58 Superior mesenteric artery aneurysm (SMAA) is an uncommon vascula

59 less invasive option for rupture of superior mesenteric artery aneurysm.

60 e result of a threefold increase in superior mesenteric artery BFV (P < .0001).

61 ons in gastric volume (P < 0.0001), superior mesenteric artery blood flow (P < 0.0001), and velocity

62 ume, small bowel water content, and superior mesenteric artery blood flow and velocity were measured

63 nfusion increased SBP (P<0.01) and decreased mesenteric artery endothelial function (P<0.01) in wild-

64 0.62] L.min(-1); P=3.8x10(-8)) and superior mesenteric artery flow (Deltamean, 0.76 [SD, 0.35] L.min

65 nocclusive intestinal ischemia, the superior mesenteric artery flow and RBC velocity correlated signi

66 tamponade (n = 12), which decreased superior mesenteric artery flow from 351 +/- 55 to 182 +/- 67 mL/

67 animals/group) by occlusion of the superior mesenteric artery for 90 min and subsequent reperfusion

68 tumor grade, lymph node positivity, superior mesenteric artery involvement), or treatment factors (eg

69 hanism, reducing abundance at the surface of mesenteric artery myocytes.

70 teromeric channels natively expressed in rat mesenteric artery smooth muscle cells.

71 c Kv7.4/7.5 channels in A7r5 cells or native mesenteric artery smooth muscle Kv7.4/7.5 channels were

72 annular pancreas, duplication cyst, superior mesenteric artery syndrome, midgut volvulus, and diverti

73 ion, as demonstrated with the FeCl3 model of mesenteric artery thrombosis.

74 tivating TRPC1-based SOCs in contractile rat mesenteric artery VSMCs.

75 volved in activating TRPC1-based SOCs in rat mesenteric artery VSMCs.

76 In the porcine experiments, the superior mesenteric artery was gradually obstructed during consec

77 fter a laparotomy, a section of second-order mesenteric artery was visualized in an organ bath after

78 served in an artery with white fat (superior mesenteric artery) and in aorta from both male and femal

79 rricane eye, small bowel behind the superior mesenteric artery, and right-sided anastomosis.

80 r-vessel interface was noted at the superior mesenteric artery, celiac artery, or common hepatic arte

81 al artery access to catheterize the inferior mesenteric artery, proceeding to the superior rectal art

82 e patient died of thrombosis in the superior mesenteric artery.

83 V) lodged between the aorta and the superior mesenteric artery.

84 hepatic vein, common bile duct, and superior mesenteric artery.

85 The mesenteric AT (MeAT) was collected on d 0, 4, 7 (early s

86 othesized that blunted postprandial superior mesenteric blood flow responses would be more common in

87 le, jejunal, and ileal atresia with aberrant mesenteric blood supply, and syndactyly.

88 sufficient to regulate vascular tone of the mesenteric blood vessels where the adult parasites resid

89 nally life-threatening consequences, such as mesenteric, bowel, ureteral, and/or bladder obstruction.

90 ted the migration of specific B cells to the mesenteric but not draining lymph nodes.

91 insulin's ability to suppress lipolysis from mesenteric but not epididymal adipocytes.

92 g endosymbionts to midgut sites where future mesenteric caeca will develop.

93 sis and ascites was markedly enhanced in the mesenteric circulation compared to the thoracic aorta.

94 r injury with thrombin microinjection in the mesenteric circulation of mice, we have demonstrated tha

95 In inflamed vessels of the mesenteric circulation, VWF recruited S. lugdunensis to

96 /1000 person-years) among women for whom the mesenteric defects had been closed during the primary pr

97 r of patients with preexisting peritoneal or mesenteric disease.

98 , while promoting tissue accumulation in the mesenteric fat and spleen.

99 A blunted mesenteric flow response was associated with increased l

100 s to examine the independent associations of mesenteric flow response with blood pressure status and

101 ement (odds ratio, 3.9; 95% CI: 1.3, 12) and mesenteric fluid (odds ratio, 3.6; 95% CI: 1.0, 12.8) we

102 in was injected into the celiac and superior mesenteric ganglia (CSMG) of rats.

103 ve fibers terminating in the celiac-superior mesenteric ganglia form varicose-like structures surroun

104 he DMN, which project to the celiac-superior mesenteric ganglia, significantly increase splenic nerve

105 onfidence interval [CI]: 2.6, 23.5), diffuse mesenteric haziness (odds ratio, 6.1; 95% CI: 2.5, 15.2)

106 cement, a closed-loop mechanism, and diffuse mesenteric haziness) can accurately predict strangulatio

107 llowing BAT is thought to occur secondary to mesenteric hematoma formation or mesenteric tear complic

108 mary ovarian mass(es), presence of definable mesenteric implants and infiltration, presence of other

109 Our patient did not have any mesenteric injury or hematoma on initial abdominal CT.

110 = 2.40) and left upper quadrant (OR = 1.19), mesenteric involvement (OR = 7.10), and lymphadenopathy

111 Mesenteric involvement at CT was an indicator of signifi

112 Mesenteric involvement at CT was associated with signifi

113 n, presence of PD in gastrohepatic ligament, mesenteric involvement, and supradiaphragmatic lymphaden

114 patients), cardiac ischemia (6.6% vs 5.6%), mesenteric ischemia (3.2% vs 2.6%), and peripheral ische

115 investigate the prognostic factors of acute mesenteric ischemia (AMI) in ICU patients.

116 ment of operative risk (standard deviations: mesenteric ischemia 20.2% vs 23.2%, P = 0.01; gastrointe

117 e initial workup in cases of suspected acute mesenteric ischemia because it can rule out other causes

118 clinician to identify patients in whom acute mesenteric ischemia develops.

119 Acute mesenteric ischemia is an abdominal emergency because re

120 Acute mesenteric ischemia is associated with high morbidity an

121 raluminally into the ileum before 45 minutes mesenteric ischemia or before reperfusion in non-CH4 pro

122 n after four cycles (n = 6), or nonocclusive mesenteric ischemia was induced by pericardial tamponade

123 The incidence of nonocclusive mesenteric ischemia was particularly high.

124 The diagnosis of acute mesenteric ischemia was proposed based on evidence of po

125 ons (transaminitis, ileus, Ogilvie syndrome, mesenteric ischemia) among critically ill patients with

126 laparotomy confirmed extensive nonocclusive mesenteric ischemia, and the patient rapidly died of mul

127 to detect and treat thromboses can result in mesenteric ischemia, chronic cavernous transformation, a

128 767) with four detailed clinical vignettes (mesenteric ischemia, gastrointestinal bleed, bowel obstr

129 , viral colitis, inflammatory enterocolitis, mesenteric ischemia, radiation-induced gastrointestinal

130 ue damage, we also studied its expression in mesenteric ischemia-reperfusion (I/R) injury.

131 Mesenteric ischemia-reperfusion injury was induced in ma

132 case vignette of a frail patient with acute mesenteric ischemia.

133 atient eventually died because of subsequent mesenteric ischemia.

134 s play an important role in the diagnosis of mesenteric ischemia.

135 nhibited repair of damaged mucosa induced by mesenteric ischemia/reperfusion in the small intestine a

136 f the gut barrier function after exposure to mesenteric ischemia/reperfusion.

137 compared with surgeons in the control group [mesenteric ischemia: 43.7% vs 64.6%, P < 0.001 (RCV = 25

138 Five mesenteric ischemic events occurred in the conservative-

139 icated by gastric, main portal vein, splenic mesenteric junction, and splenic vein occlusions; hence,

140 lso present in the main portal vein, splenic mesenteric junction, and splenic vein, causing an engorg

141 Mesenteric lesions involved intense arterial remodeling,

142 quences in axillary, brachial, inguinal, and mesenteric LNs were virtually identical, and a substanti

143 We identified three small mesenteric LNs, distinct from small intestinal LNs, whic

144 exclusively identified in skin-draining and mesenteric LNs, respectively.

145 t-derived inflammatory mediators carried via mesenteric lymph (ML).

146 icient targeting of LPV to HIV reservoirs in mesenteric lymph and MLNs.

147 o efficiently deliver lopinavir (LPV) to the mesenteric lymph and MLNs.

148 Exosomes in postshock mesenteric lymph are key mediators of acute lung injury

149 In ex vivo studies using whole mesenteric lymph node (MLN) as well as CD3(+) T-cells, w

150 and TH17 cytokine, and Tgfbeta expression in mesenteric lymph node (MLN) CD4(+) T cells and jejunum w

151 cytes in the intestinal tissues, we isolated mesenteric lymph node (MLN) from naive wild type mice.

152 They were exclusively found in the mesenteric lymph node after T cell-mediated colitis indu

153 ects were mediated through the limitation of mesenteric lymph node and intestinal DC accumulation and

154 ed germinal center B cells were found in the mesenteric lymph node and Peyer patches.

155 Mesenteric lymph node CD4(+) FoxP3(+) regulatory T cells

156 When enteroids are cocultured with CD90(+) mesenteric lymph node cells from IL-33-treated mice, IL-

157 Likewise, plasmablast frequency in the mesenteric lymph node correlated with viremia.

158 Mesenteric lymph node cultures from VDR KO and B-VDR KO

159 ritic cells, lamina propria macrophages, and mesenteric lymph node dendritic cells were examined.

160 activity of vitamin A-converting enzymes in mesenteric lymph node dendritic cells, along with increa

161 alities include intestinal lymphangiectasia, mesenteric lymph node lymphadenopathy, and lymphangiogen

162 ntestinal inflammation by activating gut and mesenteric lymph node myeloid cells.

163 omal RNA gene sequencing; lamina propria and mesenteric lymph node tissues were analyzed by RNA seque

164 Spleen and mesenteric lymph node were collected, processed, and ana

165 etected in nasal swab, nasal turbinates, and mesenteric lymph node, but no evidence of histopathologi

166 DENV enhanced ZIKV infection, mainly in the mesenteric lymph node, indicating the potential for DENV

167 uces tolerogenic XCR1(+) DC migration to the mesenteric lymph node, where Treg cells are induced and

168 TGF-beta on the differentiation of colon and mesenteric lymph node-derived murine Foxp3(-) IL-10(-) C

169 mphatics convey Ags and microbial signals to mesenteric lymph nodes (LNs) to induce adaptive immune r

170 DCs of mesenteric lymph nodes (MLN) and joint regional lymph no

171 ria travel from the intestinal mucosa to the mesenteric lymph nodes (MLN), a key site for Ag presenta

172 uction was impaired during T cell priming in mesenteric lymph nodes (MLN), which correlated with a re

173 sed effector T cell induction in the CLN and mesenteric lymph nodes (MLN).

174 elium from where they are transported to the mesenteric lymph nodes (MLNs) within migrating immune ce

175 The mesenteric lymphatic system, including mesenteric lymph nodes (MLNs), is an important viral res

176 populations in the peripheral blood, liver, mesenteric lymph nodes (MLNs), jejunum, and bronchoalveo

177 It disseminates from gut to mesenteric lymph nodes (MLNs), spleen, and liver of infe

178 from the gut to systemic sites, such as the mesenteric lymph nodes (MLNs), via CD11b(+) migratory de

179 patches, limited dendritic cell migration to mesenteric lymph nodes [mLNs] causing reduced T cell-med

180 located in the bacterial translocation site (mesenteric lymph nodes [MLNs]) of unirradiated mice were

181 s, and restored the Th17 and Treg content in mesenteric lymph nodes and aorta.

182 ith reduced CD4(+) T cell counts in both the mesenteric lymph nodes and colon.

183 tion and decreased the numbers of DCs in the mesenteric lymph nodes and lamina propria.

184 and enhances bacterial translocation to the mesenteric lymph nodes and liver, promoting the progress

185 germinal center B cells and plasma cells in mesenteric lymph nodes and more IgA-coated commensal bac

186 ressing of FoxP3(+) T cells were measured in mesenteric lymph nodes and Peyer's patches cells.

187 ation of gut-homing effector T-cells in both mesenteric lymph nodes and Peyer's patches without obvio

188 d in organized lymphoid tissues, such as the mesenteric lymph nodes and Peyer's patches, as well as i

189 anges in adaptive and innate immune cells in mesenteric lymph nodes and spleen demonstrated that the

190 ositive PCR detection of M. canettii for 5/8 mesenteric lymph nodes at days 1 and 3 p.i. and 5/6 pool

191 ry-effector CD4(+) T cells in the spleen and mesenteric lymph nodes compared with wild-type mice; sCY

192 Apoptotic IECs were trafficked to mesenteric lymph nodes exclusively by the dendritic cell

193 Bacterial culture of mesenteric lymph nodes in these mice isolated K. pneumon

194 ic lymphatic vessels and lymph drainage into mesenteric lymph nodes may be compromised.

195 Mtor gene expression to a greater extent in mesenteric lymph nodes of BALB/cAnPt mice than of DBA/2N

196 regulate T cell homing, were also reduced in mesenteric lymph nodes of infected AMCase-deficient mice

197 required to maintain the Th2 response in the mesenteric lymph nodes of infected mice.

198 TLR4 conditioned the in vivo mobilization to mesenteric lymph nodes of intestinal migratory CD103(+)

199 om rectal biopsy specimens, bone marrow, and mesenteric lymph nodes of vaccinated infected, unvaccina

200 e Gag-specific CD8+ T cell clonotypes in the mesenteric lymph nodes relative to rhesus macaques with

201 ted immune cell subsets from the spleens and mesenteric lymph nodes revealed an increase in PD-1(+)CD

202 The appearance of i.p. injected cells in mesenteric lymph nodes suggests that the mesentery-assoc

203 pattern and subsequently migrate toward the mesenteric lymph nodes via the mesenteric lymphatic capi

204 s (cDC) in the intestinal lamina propria and mesenteric lymph nodes were GFP(+) However, in vitro inf

205 aive lymphocytes traffic to the gut-draining mesenteric lymph nodes where they undergo antigen-induce

206 of the target genes in colonic biopsies and mesenteric lymph nodes which was accompanied with a dist

207 apoptosis in the esophagus, small intestine, mesenteric lymph nodes, and kidney in minks experimental

208 HSPCs in intestinal mucosa, Peyer's patches, mesenteric lymph nodes, and liver.

209 an inflammatory signal that is propagated to mesenteric lymph nodes, and that can facilitate extraint

210 ntestinal tissue, active germinal centers in mesenteric lymph nodes, IgG(+) and IgA(+) plasmablasts i

211 T-bet(+) T cells and IFN-gamma production in mesenteric lymph nodes, increased expression of Ido1 in

212 ired characterization of immune cells in the mesenteric lymph nodes, to delineate colonic immune nich

213 ed lymphatic transport of dendritic cells to mesenteric lymph nodes, two features likely to actively

214 with lymphoid organs, such as the spleen and mesenteric lymph nodes, where these same cells appear to

215 the colon and subsequent migration into the mesenteric lymph nodes.

216 re determined in duodenum, ileum, colon, and mesenteric lymph nodes.

217 -specific regulatory T cell induction in the mesenteric lymph nodes.

218 dritic cells became prominently activated in mesenteric lymph nodes.

219 tinued reduction in CD4(+) T cell numbers in mesenteric lymph nodes.

220 ld-type (WT) mice in the Peyer's patches and mesenteric lymph nodes.

221 The maximum levels of LPV in mesenteric lymph were 1.6- and 16.9-fold higher than pro

222 subset of patients, the disease is caused by mesenteric lymphadenopathy in response to (viral) infect

223 te toward the mesenteric lymph nodes via the mesenteric lymphatic capillaries.

224 litate the delivery of unmodified LPV to the mesenteric lymphatic system and resulted in undetectable

225 The mesenteric lymphatic system, including mesenteric lymph

226 layer to enter the blood circulation via the mesenteric lymphatic system.

227 ducing the size of HIV reservoirs within the mesenteric lymphatic system.

228 The potential for mesenteric lymphatic targeting and bioconversion to LPV

229 These observations suggest that downstream mesenteric lymphatic vessels and lymph drainage into mes

230 Using mesenteric lymphatic vessels from C57BL/6J, Ub-CreER(T2)

231 Mesenteric lymphatic vessels from high-fructose diet-ind

232 Mesenteric lymphatic vessels from MetSyn or LPS-injected

233 orphologic features and functional status of mesenteric lymphatics in CD.

234 ractile properties and valvular functions of mesenteric lymphatics, developed a surgical model for va

235 tion of an exhaled endogenous gas with acute mesenteric macro- and microvascular flow changes.

236 ce status 1, and R1-direct positive superior mesenteric/medial margin resection status were all signi

237 Single perfused rat mesenteric microvessels were perfused with fluorescent e

238 B4 did not overexpress VEGF or have signs of mesenteric neovascularization, and developed less-severe

239 al preparations with attached splanchnic and mesenteric nerves were used to study mechanosensory and

240 tal of 159 patients treated, 81 had baseline mesenteric or peritoneal disease, among whom 5 (6%) expe

241 n lead to bowel obstruction in patients with mesenteric or peritoneal disease, likely by inducing inf

242 r are pancreatic saponification, heterotopic mesenteric ossification, and pseudolipoma of the capsule

243 Mesenteric panniculitis (MP) is a radiological finding a

244 e epiploic appendagitis, omental infarction, mesenteric panniculitis, and encapsulated fat necrosis.

245 17beta-Estradiol was effective in improving mesenteric perfusion and reducing intestinal edema and h

246 wmetry and intravital microscopy to evaluate mesenteric perfusion; (b) histopathological analysis; (c

247 proximately 60% of Nppb-/- females developed mesenteric polyarteritis-nodosa (PAN)-like vasculitis in

248 Analyzing contraction of mesenteric resistance arteries supported the biological

249 asodilatation via nitric oxide (NO) in mouse mesenteric resistance arteries.

250 asodilatation via nitric oxide (NO) in mouse mesenteric resistance arteries.

251 he vasoconstrictor responses of cerebral and mesenteric resistance arteries.

252 ffect on endothelium-dependent relaxation in mesenteric resistance artery.

253 ncement of pressure-induced contractility of mesenteric resistance vessels by influencing the activit

254 trical perivascular nerve stimulation in rat mesenteric small arteries causes a large beta1-adrenocep

255 In mesenteric small arteries of anaesthetized rats, EFS fai

256 The best signs were mesenteric swirl (sensitivity and specificity, 86%-89% a

257 and sensitivity for diagnosis of IH included mesenteric swirl and SBO, the model with the highest spe

258 st overall accuracy and sensitivity included mesenteric swirl and SBO, with a diagnostic odds ratio o

259 owing previously established CT signs of IH: mesenteric swirl, small-bowel obstruction (SBO), mushroo

260 econdary to mesenteric hematoma formation or mesenteric tear complications.

261 Critically, TNFDeltaARE mice also present mesenteric tertiary lymphoid organs and have altered lym

262 al failure-associated liver disease or porto-mesenteric thrombosis.

263 to characterize the lymphatic vasculature in mesenteric tissue from controls or patients with CD.

264 n's disease, the characteristic expansion of mesenteric tissue on sites of intestinal inflammation is

265 stricted intact arteries isolated from mouse mesenteric tissue.

266 Liver and mesenteric tissues were collected and analyzed in angiog

267 Adult rat mesenteric tissues were harvested and cultured for three

268 l lymphadenopathy, right hydronephrosis, and mesenteric tumor deposits ( Fig 1A ).

269 e features (with inflammatory bowel disease, mesenteric vascular diseases, or other conditions).

270 Postprandial mesenteric vascular dysfunction is associated with LVH a

271 ographic interface between primary tumor and mesenteric vasculature.

272 eceptor agonists able to induce systemic and mesenteric vasoconstriction have shown their usefulness

273 ich was associated with decreased aortic and mesenteric vasoconstriction in hypertensive Sphk1(-/-) m

274 evaluated images for two new signs, superior mesenteric vein (SMV) "beaking" and "criss-cross" of the

275 rojejunostomy line in two patients, superior mesenteric vein (SMV) thrombosis in two patients, and in

276 y measurements were obtained in the superior mesenteric vein (SMV), splenic vein (SV), portal vein (P

277 portal vein graft to the recipient inferior mesenteric vein anastomosis.

278 ze with abutment of the portal vein-superior mesenteric vein confluence for less than 180 degrees .

279 xpectation of posttransplant cure, extensive mesenteric vein thrombosis and intestinal infarction, to

280 DVICE 11: Treatment of incidental portal and mesenteric vein thrombosis depends on estimated impact o

281 ptomatic deep vein thrombosis and portal and mesenteric vein thrombosis, but there are unresolved iss

282 l vein stump was performed from the inferior mesenteric vein to the umbilical portion of the left por

283 d splenic vein, causing an engorged inferior mesenteric vein.

284 colonic ischemia, the presence of gas in the mesenteric veins but not in the portal vein.

285 ceptor 7/8 (TLR7/8)-mediated inflammation of mesenteric veins, platelet activation drives the rapid m

286 position that leads to it receiving all the mesenteric venous blood, combined with its unique micro

287 l cells also increased leukocyte adhesion in mesenteric venules and increased the frequency of neutro

288 (von Willebrand factor)-platelet strings in mesenteric venules and that this is dependent on PAD4 en

289 Intravital microscopy of mouse mesenteric venules demonstrated that PD1n-3 DPA and RvD5

290 tion after ferric chloride-induced injury of mesenteric venules.

291 in F8-/-mice in the ferric chloride-induced mesenteric vessel injury model.

292 m that triggers endothelial communication to mesenteric vessel muscle cells, leading to vasoconstrict

293 In ex vivo rings, aortic and mesenteric vessels from SHR treated with DHI exhibited s

294 emman phosphorylation in vitro in aortic and mesenteric vessels using wire myography and membrane pot

295 ein (SMV) "beaking" and "criss-cross" of the mesenteric vessels.

296 odel of a tumor that commonly involves major mesenteric vessels.

297 A transcriptome analysis comparing mesenteric visceral AT (vAT) of HF and HF/DDE groups rev

298 okine-induced lipolysis may be restricted to mesenteric white adipose tissue and that it contributes

299 ivity of mast cells to fungi was tested with mesenteric windows (ex vivo) and the human mast cell lin

300 agonist) induced mast cell degranulation in mesenteric windows and HMC-1 cells responded to fungal a