ライフサイエンスコーパス: mesenteric artery

1 and Doppler ultrasonography of the superior mesenteric artery.

2 hepatic vein, common bile duct, and superior mesenteric artery.

3 in but only anti-TRPC6 inhibited activity in mesenteric artery.

4 have compared the data to that from a small mesenteric artery.

5 -2d also elicited vasodilation effect in rat mesenteric artery.

6 niform patterns of branching at the superior mesenteric artery.

7 as significantly greater in coronary than in mesenteric artery.

8 rived from the adult rat (or mouse) superior mesenteric artery.

9 increased in the Day28 low flow first order mesenteric artery.

10 tery arising independently from the superior mesenteric artery.

11 rminant of peripheral resistance - the small mesenteric artery.

12 from 1st to 7th order branches of guinea-pig mesenteric artery.

13 carba-UDP were studied in a model of the rat mesenteric artery.

14 rn endotoxemia on blood flow in the superior mesenteric artery.

15 e patient died of thrombosis in the superior mesenteric artery.

16 V) lodged between the aorta and the superior mesenteric artery.

17 limited to the distribution of the inferior mesenteric artery.

18 MP (Epac) in mediating vasorelaxation in rat mesenteric arteries.

19 ulates the myogenic response in cerebral and mesenteric arteries.

20 ecrease in contractile response was found in mesenteric arteries.

21 only in native rat renal arteries but not in mesenteric arteries.

22 alpha, limits TRPV4(EC) -eNOS signalling in mesenteric arteries.

23 roduction to increase myogenic tone in small mesenteric arteries.

24 s less in the endothelium of IH than in sham mesenteric arteries.

25 nd dilated intact or endothelium-denuded rat mesenteric arteries.

26 (SUR) 2-deficient [SUR2(-/-)] mouse myogenic mesenteric arteries.

27 of the structure and function of resistance mesenteric arteries.

28 of a change in endothelial cell Ca2+ in rat mesenteric arteries.

29 thetic neurotransmission in rat second-order mesenteric arteries.

30 f caveolae-like structures in SHR aortas and mesenteric arteries.

31 erwent stent placement in 79 stenotic (>70%) mesenteric arteries.

32 le for Cx40 in EDHF-mediated dilation of rat mesenteric arteries.

33 d to assess vascular function in third-order mesenteric arteries.

34 revealed expression of Kir6.1/SUR2B mRNAs in mesenteric arteries.

35 ed vasodilation of the isolated and perfused mesenteric arteries.

36 scle hyperpolarization and relaxation in rat mesenteric arteries.

37 uced surface and total KV 1.5 protein in rat mesenteric arteries.

38 of S1P synthesis reduced vasoconstriction of mesenteric arteries.

39 as well as flow-induced dilatation in murine mesenteric arteries.

40 found in thoracic aortas but not in superior mesenteric arteries.

41 rlipressin relaxed pulmonary and constricted mesenteric arteries.

42 teries from TgNotch3(R169C) mice, but not in mesenteric arteries.

43 ac trunk (50), hepatic artery (29), superior mesenteric artery (35), and other segments (4).

44 ction of the distal branches of the superior mesenteric artery (60 minutes) and reperfusion for 90 mi

45 nsport activity was investigated in VSMCs of mesenteric arteries after an NH4(+) prepulse.

46 relaxation of microvessels from the superior mesenteric artery after I/R was significantly attenuated

47 In small mesenteric arteries, alpha(1A)-subtype-specific antagoni

48 olic blood flow in the superior and inferior mesenteric arteries and celiac trunk (CT) compared with

49 ere studied on VSMs acutely dissociated from mesenteric arteries and HEK293 cells expressing Kir6.1/S

50 4) CaSR mRNA and protein were present in rat mesenteric arteries and in porcine coronary artery endot

51 ified a mechanosensing mechanism in isolated mesenteric arteries and in the renal circulation that re

52 GS5 caused augmented myogenic tone in intact mesenteric arteries and increased activation of protein

53 explain TRPV4(EC) -IK/SK channel coupling in mesenteric arteries and its absence in pulmonary arterie

54 ent in VSMCs of medium-sized vessels such as mesenteric arteries and larger retinal arterioles.

55 tural and functional integrity of resistance mesenteric arteries and lowered blood pressure in low-re

56 Meanwhile, mesenteric arteries and lung vascular endothelial cells

57 C5 antibodies inhibited SOCs in coronary and mesenteric arteries and portal vein but anti-TRPC6 block

58 dispersed myocytes from rabbit coronary and mesenteric arteries and portal vein.

59 olae integrity and density in SHR aortas and mesenteric arteries and the role played by caveolae in e

60 ion and function of the KCNE4 subunit in rat mesenteric arteries and to determine whether it has a fu

61 Mesenteric arteries and veins from female mice flown on

62 to induce rapid constriction in femoral and mesenteric arteries and veins in rats.

63 f this study was to test the hypothesis that mesenteric arteries and veins will exhibit diminished va

64 4) by means of surgical occlusion of distal mesenteric arteries and veins.

65 preferentially couple with IK/SK channels in mesenteric arteries and with eNOS in pulmonary arteries.

66 ith an ultrasonic flow probe on the superior mesenteric artery and a catheter into the superior mesen

67 ic flow probe was inserted into the superior mesenteric artery and a catheter into the superior mesen

68 Mesenteric artery and aortic transcriptome was profiled

69 half of the nerve plexus around the superior mesenteric artery and celiac axis.

70 Lower blood flow in the superior mesenteric artery and CT was correlated with HF severity

71 ce of [14C]lactate infused into the superior mesenteric artery and direct measurements of blood lacta

72 ng the aorta in continuity with the inferior mesenteric artery and portal vein in continuity with the

73 ity in coronary artery but inhibited SOCs in mesenteric artery and portal vein myocytes.

74 vation of SOCs in coronary artery similar to mesenteric artery and portal vein.

75 roperitoneal D3 located between the superior mesenteric artery and the aorta was seen on US in all pa

76 were placed around a branch of the superior mesenteric artery and the right femoral artery.

77 tructures, relative position of the superior mesenteric artery and vein.

78 given by the terminal branch of the superior mesenteric artery and venous outflow by a proximal segme

79 r (celiac, superior mesenteric, and inferior mesenteric arteries) and mediolateral (renal arteries) b

80 served in an artery with white fat (superior mesenteric artery) and in aorta from both male and femal

81 m colic branches of the superior or inferior mesenteric arteries, and selective transcatheter emboliz

82 ncreatic adenocarcinoma, celiac and superior mesenteric arteries, and superior mesenteric and portal

83 Rabbit aorta, mesenteric arteries, and the combination of 15-LO and cy

84 d with pulsed Doppler flow probes (renal and mesenteric arteries, and the descending abdominal aorta)

85 ume were measured in celiac artery, superior mesenteric artery, and main portal vein (MPV).

86 rricane eye, small bowel behind the superior mesenteric artery, and right-sided anastomosis.

87 Superior mesenteric artery aneurysm (SMAA) is an uncommon vascula

88 less invasive option for rupture of superior mesenteric artery aneurysm.

89 Significant remodeling of resistance mesenteric arteries (approximately 100-microm outside di

90 Mesenteric arteries are densely innervated and the nerve

91 olving both roots of the celiac and superior mesenteric artery are deemed unresectable by conventiona

92 of dissection of the celiac and/or superior mesenteric artery are rare; as far as we know, only 24 c

93 asoconstriction to NE was also determined in mesenteric arteries at 1, 5, and 7 d postlanding.

94 m efflux was increased in both the aorta and mesenteric artery at 24 hrs.

95 -sec reperfusion and reocclusion of superior mesenteric artery at the initiation of reperfusion.

96 e result of a threefold increase in superior mesenteric artery BFV (P < .0001).

97 ons in gastric volume (P < 0.0001), superior mesenteric artery blood flow (P < 0.0001), and velocity

98 ume, small bowel water content, and superior mesenteric artery blood flow and velocity were measured

99 In mesenteric arteries, BMAL1 bound to the promoter of and

100 channels co-localize with IK/SK channels in mesenteric arteries but not in pulmonary arteries, which

101 ) proteins modulate the myogenic response in mesenteric arteries, but involvement in other vascular b

102 thelium-denuded IPAs by 235 +/- 32 %, and in mesenteric arteries by 218 +/- 38 %.

103 ort hairpin RNA (shRNA) were transduced into mesenteric arteries by chemical loading plus liposomes.

104 r-vessel interface was noted at the superior mesenteric artery, celiac artery, or common hepatic arte

105 postischemia reperfusion (IR) injury of the mesenteric artery, characterized by marked neutrophil ad

106 itol (osmotic control), followed by superior mesenteric artery clamping for 60 minutes and 30 minutes

107 ed in tissue superfusates upon EFS of canine mesenteric artery (CMA), canine urinary bladder, and mur

108 s expressed in a variety of arteries and, in mesenteric arteries, co-localizes with Kv7.4, which is i

109 ed to EDHF, was significantly reduced in OHF mesenteric arteries compared with controls.

110 downregulated by lipopolysaccharide (LPS) in mesenteric arteries concordant with vascular hypocontrac

111 ) contact (r = -0.38), and post-CRT superior mesenteric artery contact (r = 0.34).

112 ood pressure, heart rate, aortic function or mesenteric artery contractile function, at either 3 or 6

113 endothelial cells isolated from plaque-free mesenteric arteries (CSE activity high) and plaque-conta

114 n analysis, lower blood flow in the superior mesenteric artery, CT (p < 0.04), and inferior mesenteri

115 rdial space until blood flow in the superior mesenteric artery decreased to half of baseline.

116 Pressure myography using mesenteric arteries demonstrated that relaxation respons

117 nd 71 cases of spontaneous isolated superior mesenteric artery dissection have been reported.

118 ntaneous isolated celiac artery and superior mesenteric artery dissections must be kept in mind in th

119 gh/low flow, the portal vein and first order mesenteric artery dynamically downregulate Tra2beta conc

120 TRPV4-C1-P2 complex in primary cultured rat mesenteric artery endothelial cells (MAECs) and HEK293 c

121 Finally, in pure cell populations of mouse mesenteric artery endothelial cells, we show that P2X(1)

122 sulin resistance, dyslipidaemia, obesity and mesenteric artery endothelial dysfunction in adult offsp

123 nfusion increased SBP (P<0.01) and decreased mesenteric artery endothelial function (P<0.01) in wild-

124 Small mesenteric artery endothelium-dependent dilation to acet

125 hich is similar to the pressure second-order mesenteric arteries experience in vivo, and that Ca(2+)

126 imary endothelial cells isolated from murine mesenteric arteries express functional Kir2.1 channels s

127 0.62] L.min(-1); P=3.8x10(-8)) and superior mesenteric artery flow (Deltamean, 0.76 [SD, 0.35] L.min

128 nocclusive intestinal ischemia, the superior mesenteric artery flow and RBC velocity correlated signi

129 tamponade (n = 12), which decreased superior mesenteric artery flow from 351 +/- 55 to 182 +/- 67 mL/

130 ance, reduced portal pressure (PP), superior mesenteric artery flow, mesenteric vascular density, por

131 e is used to provide inflow to the renal and mesenteric arteries followed by aortic relining with ste

132 I/R injury induced by clamping the superior mesenteric artery for 100 min with tissue analysis at 4

133 al ischemia (GI) was induced by clamping the mesenteric artery for 20 minutes and then reperfused for

134 al I/R was induced by occluding the superior mesenteric artery for 30 min followed by reperfusion for

135 sion by occlusion (clamping) of the superior mesenteric artery for 30 min, followed by unclamping and

136 njury by transient occlusion of the superior mesenteric artery for 30 min.

137 duced by temporary occlusion of the superior mesenteric artery for 30 mins, followed by 2 hrs of repe

138 inuously infused into the cranial (superior) mesenteric artery for 48 hours.

139 animals/group) by occlusion of the superior mesenteric artery for 90 min and subsequent reperfusion

140 Resistance mesenteric arteries from 6-month-old female rats fed the

141 We tested this hypothesis in resistant mesenteric arteries from Adipo-MROE mice using myography

142 , Rho-kinase II, and MYPT1 were increased in mesenteric arteries from endotoxemic rats, but the phosp

143 Myogenic tone was greater in mesenteric arteries from IH than sham control rat arteri

144 Isolated renal and mesenteric arteries from Lewis rats were incubated with

145 mpared with those fed the depleted diet, and mesenteric arteries from male and female rats fed the is

146 xpressed in thoracic aortas, small renal and mesenteric arteries from mice and rats of both sexes, as

147 mented myosin light chain phosphorylation in mesenteric arteries from mice with smooth muscle-specifi

148 An increased response to acetylcholine of mesenteric arteries from rats with cirrhosis (50% effect

149 SK3 protein was elevated in small mesenteric arteries from SK3T/T mice compared with wild-

150 Mesenteric arteries from SMC-specific Gprc5b-KOs showed

151 ly with the evoked contractile response of a mesenteric artery from a healthy Sprague Dawley rat.

152 constrictors, and their aortic, femoral, and mesenteric arteries had reduced contractile responses to

153 are noteworthy: FMD limited to the inferior mesenteric artery has not been previously reported, FMD

154 t ECs, but not smooth muscle cells, of small mesenteric arteries have Kir currents, which are substan

155 neurovascular transmission in isolated small mesenteric arteries have used either isometric recording

156 sease models such as portal hypertension and mesenteric artery high/low flow, the portal vein and fir

157 In preconstricted rat mesenteric arteries, highly diluted sangre de grado (1:1

158 difference in contractility of medium-sized mesenteric arteries; however, responsiveness of the aort

159 ording to their sources into simple inferior mesenteric artery (IMA), simple lumbar artery (LA), comp

160 th high or low ligation (LL) of the inferior mesenteric artery (IMA).

161 (i) vasorelaxation of rat mesenteric arteries in response to calcitonin gene-relat

162 with enhanced HNO-mediated vasorelaxation in mesenteric arteries in vitro and arteriolar dilation in

163 issues such as the rat portal vein and small mesenteric artery, in which E23 is spliced, as compared

164 the portal vein, small intestine, and small mesenteric artery, in which Mypt1 E23 is predominately i

165 In vitro, CRF injected into the inferior mesenteric artery increased distal colonic myoelectric a

166 e [14C]lactate was infused into the superior mesenteric artery, indicating increased first-pass clear

167 Using an in vivo mesenteric artery injury model and real-time continuous

168 tumor grade, lymph node positivity, superior mesenteric artery involvement), or treatment factors (eg

169 uced endothelium-dependent relaxation in OHF mesenteric arteries is due to impaired EDHF-mediated rel

170 ital and subjected to 30 minutes of superior mesenteric artery ischemia, followed by 4 hours of equia

171 on of P2Y receptor on the endothelium of rat mesenteric arteries leads to marked spreading dilatation

172 anal verge, CCI of 3 or more, high inferior mesenteric artery ligation (above left colic artery), in

173 h-old) were tested for vascular functions in mesenteric arteries (MA) and ion channel activities in s

174 The function of sympathetic nerves supplying mesenteric arteries (MA) and veins (MV) in rats was inve

175 nduced (P < 0.05) vasodilatation in isolated mesenteric arteries (MA) from protein-restricted pregnan

176 We studied mesenteric artery (MA) from transgenic mice expressing d

177 ere ligated so that the upstream first order mesenteric artery (MA1) is under chronic low flow and th

178 Resistance-sized mesenteric arteries (MAs) and pulmonary arteries (PAs) w

179 tion (KCl)-induced constriction of rat small mesenteric arteries (MAs) and veins (MVs) to the dilator

180 Abstract Mesenteric arteries (MAs) are studied widely in vitro bu

181 Isolated mesenteric arteries (MAs) from RGS2(-/-) mice showed an

182 hesis that functional sensory innervation of mesenteric arteries (MAs) is impaired for Old (24 months

183 The dilatory role for sensory innervation of mesenteric arteries (MAs) is impaired in Old ( approxima

184 usion (35 min) and reopening of the superior mesenteric artery, MC3R-null mice displayed a higher deg

185 ylation of key proteins in denuded rat small mesenteric artery, midsized caudal artery and thoracic a

186 In the mesenteric artery, more than 20 myocytes are required fo

187 ion conductances in freshly dispersed rabbit mesenteric artery myocytes at the single-channel level u

188 ctivates two distinct cation conductances in mesenteric artery myocytes by stimulation of AT1 recepto

189 ominant-negative Kv7.4 and Kv7.5 subunits in mesenteric artery myocytes reduced endogenous Kv7 curren

190 channel activity in freshly dispersed rabbit mesenteric artery myocytes using patch clamp recording a

191 In contrast, mesenteric artery myocytes, which express both KCNQ4 and

192 P(2)-induced inhibition of TRPC6 activity in mesenteric artery myocytes.

193 e OAG activates a homomeric TRPC6 channel in mesenteric artery myocytes.

194 d no effect on OAG-induced TRPC6 activity in mesenteric artery myocytes.

195 man KCNQ5 currents) and freshly isolated rat mesenteric artery myocytes.

196 reversal potentials in coronary compared to mesenteric artery myocytes.

197 hanism, reducing abundance at the surface of mesenteric artery myocytes.

198 showed that KCNE4 co-localized with Kv7.4 in mesenteric artery myocytes.

199 expression was found in the membrane of the mesenteric artery myocytes.

200 type IA, n=1; type IB, n=1; type II inferior mesenteric artery, n=2; type II lumbar artery, n=28; typ

201 l ventilation (CMV) over 60 mins of superior mesenteric artery occlusion and 60 mins of reperfusion.

202 ischemia-reperfusion groups, where superior mesenteric artery occlusion was maintained for 1 hr and

203 After superior mesenteric artery occlusion, intestinal permeability inc

204 gnal-regulated kinase (Erk) were assessed in mesenteric arteries of 3- (3M) and 9-month-old (9M) male

205 ed expression of HO-1 was found in aorta and mesenteric arteries of BDL rats in a close chronologic r

206 single myocytes freshly isolated from small mesenteric arteries of guinea-pig was used to investigat

207 and protein were significantly increased in mesenteric arteries of hypertensive animals, and pharmac

208 ompared IP3R expression and function between mesenteric arteries of normotensive and hypertensive ani

209 dothelial cells within isolated, pressurized mesenteric arteries of the rat.

210 onor spermine-NONOate (SPER-NO) in aorta and mesenteric arteries of WT mice.

211 In the mesenteric arteries, only ECs, but not smooth muscle cel

212 owel are diverse, ranging from occlusions of mesenteric arteries or veins to complicated bowel obstru

213 e coil embolization (hypogastric or inferior mesenteric artery) (OR = 2.1, P = 0.008).

214 Small mesenteric arteries (outside diameter, 50-150 microm) we

215 senteric artery, CT (p < 0.04), and inferior mesenteric artery (p = 0.056) was correlated with the pr

216 vasodilatation (FIV) assayed in pressurized mesenteric arteries pre-constricted with endothelin-1.

217 uced contractile differences between ex vivo mesenteric artery preparations.

218 al artery access to catheterize the inferior mesenteric artery, proceeding to the superior rectal art

219 inase into Triton X-100-permeabilized rabbit mesenteric artery provoked a Ca(2+)-free contraction.

220 ation in response to acetycholine in control mesenteric arteries remained after inhibition of nitric

221 tation and hyperpolarization of VSM in small mesenteric arteries requires BK(Ca) channels.

222 elialized Pkd2(+/-) resistance (fourth-order mesenteric) arteries responded to PE with a stronger con

223 Doppler ultrasonography of the superior mesenteric artery revealed a twofold increase in blood f

224 iac RI (0.78 versus 0.73, P = 0.04) superior mesenteric artery RI (0.89 versus 0.84, P = 0.005), and

225 intact, but not of endothelium-denuded, rat mesenteric artery segments, modulation of endothelial BK

226 s as a full agonist in relaxing rat isolated mesenteric artery segments.

227 min) of both canine and guinea-pig isolated mesenteric artery segments.

228 nts between renal arteries, and the inferior mesenteric arteries served as autografts.

229 Vascular function evaluated ex vivo in mesenteric arteries showed that in wild-type mice, CHF m

230 mathematical model of Ca(2+) dynamics in rat mesenteric arteries shows that a number of synchronizing

231 e placed in 692 renal arteries, 156 superior mesenteric arteries (SMA), and 50 celiac arteries.

232 obstruction by compression from the superior mesenteric artery (SMA) can be managed using minimally i

233 IIR was established by clamping the superior mesenteric artery (SMA) for 45 minutes followed by 120 m

234 Vasoconstriction of the superior mesenteric artery (SMA) is the earliest hemodynamic even

235 he final bile duct, pancreatic, and superior mesenteric artery (SMA) margins.

236 rtery (CHA) arising from either the superior mesenteric artery (SMA) or the aorta.

237 t vasodilatation of both rat aorta and small mesenteric artery (SMA) segments and reduced Phe-induced

238 Superior mesenteric artery (SMA) syndrome describes vascular comp

239 preoperative embolization of graft superior mesenteric artery (SMA) to facilitate intestinal graft r

240 sion of isolated neutrophils to rat superior mesenteric artery (SMA) vascular segments stimulated wit

241 octreotide on vascular tone in the superior mesenteric artery (SMA) was studied in portal-hypertensi

242 An aneurysm of the superior mesenteric artery (SMA) with a diameter of 2.2 cm was fo

243 low probe was positioned around the superior mesenteric artery (SMA), and cannulation of the pericard

244 epatic artery (HA) arising from the superior mesenteric artery (SMA), and increasing donor BMI were a

245 of celiac truncus (CT), orifice of superior mesenteric artery (SMA), vena cava inferior confluence (

246 ays was analyzed by western blot in superior mesenteric artery (SMA).

247 heter was placed selectively in the superior mesenteric artery (SMA).

248 , endothelium-dependent relaxations of small mesenteric arteries (SMAs).

249 olino-induced knockdown of KCNE4 depolarized mesenteric artery smooth muscle cells and resulted in th

250 the only SUR isoform expressed in SUR2(+/+) mesenteric artery smooth muscle cells, whereas SURs were

251 dogenous Kv7 currents in A7r5 rat aortic and mesenteric artery smooth muscle cells.

252 endent Cl- channel has been described in rat mesenteric artery smooth muscle cells.

253 ivated Ca(2+)-dependent Cl(-) channel in rat mesenteric artery smooth muscle cells.

254 teromeric channels natively expressed in rat mesenteric artery smooth muscle cells.

255 c Kv7.4/7.5 channels in A7r5 cells or native mesenteric artery smooth muscle Kv7.4/7.5 channels were

256 diet had catheters placed into the superior mesenteric artery so that the visceral adipose bed could

257 ric vein thrombosis, and 4 (3%) had superior mesenteric artery stricture or spasm.

258 Interestingly, HB-EGF had no effect on mesenteric arteries, suggesting a possible mechanistic b

259 fied the occurrence of an allograft superior mesenteric artery-superior mesenteric vein (SMA-SMV) AVF

260 BH(4) decreased ROS production in aorta and mesenteric arteries supernatant's of both SHR and normot

261 annular pancreas, duplication cyst, superior mesenteric artery syndrome, midgut volvulus, and diverti

262 mone; pancreatitis; cholelithiasis; superior mesenteric artery syndrome; ileus; pnemothorax; hemothor

263 le responses to these drugs were assessed in mesenteric arteries taken from animals at 24 hrs using w

264 In rat middle cerebral and mesenteric arteries the K(Ca)2.3 component of endotheliu

265 Using porcine swine mesenteric arteries, the effects of up to 6-day incubati

266 In isolated mesenteric arteries, the vasodilatation in response to t

267 Seventy-four (65%) had a superior mesenteric artery thromboembolism, 25 (22%) had a superi

268 ion, as demonstrated with the FeCl3 model of mesenteric artery thrombosis.

269 elium-dependent vasodilation in rat isolated mesenteric arteries through a G protein-coupled receptor

270 in portal vein but only weakly associated in mesenteric artery tissue lysates.

271 of EET production normalizes the response of mesenteric arteries to vasodilators, with beneficial eff

272 antly reduced the blood flow in the superior mesenteric artery to 53% of baseline.

273 hronic low flow and the adjacent first order mesenteric artery under chronic high flow.

274 by angiotensin II (Ang II) in native rabbit mesenteric artery vascular smooth muscle cells (VSMCs).

275 lial Kir channels contribute to FIV of mouse mesenteric arteries via an NO-dependent mechanism, where

276 SRF controls vasoconstriction in mesenteric arteries via vascular SM cell phenotypic modu

277 ive single TRPC channels in acutely isolated mesenteric artery VSMCs from wild-type (WT) and TRPC1-de

278 volved in activating TRPC1-based SOCs in rat mesenteric artery VSMCs.

279 tivating TRPC1-based SOCs in contractile rat mesenteric artery VSMCs.

280 sentery, and skin; plus aortic, carotid, and mesenteric artery walls.

281 and electrical field stimulation (P<0.05) in mesenteric arteries was also significantly increased in

282 otensin II-mediated constriction of isolated mesenteric arteries was blunted in OVE26EP1(-/-) mice, d

283 Vascular reactivity of the mesenteric arteries was not different.

284 Furthermore, NO-mediated relaxation in mesenteric arteries was significantly improved in DOCA-s

285 In the porcine experiments, the superior mesenteric artery was gradually obstructed during consec

286 unaltered whereas the conductance of SOCs in mesenteric artery was increased fourfold.

287 administered 60 minutes before the superior mesenteric artery was occluded for 90 minutes and reperf

288 fter a laparotomy, a section of second-order mesenteric artery was visualized in an organ bath after

289 Small resistance mesenteric arteries were connected to a pressure servo c

290 thesis alternating pairs of rat second order mesenteric arteries were ligated so that the upstream fi

291 nt responses to acetylcholine in pressurized mesenteric arteries were reduced in KW versus HW (P<0.01

292 The aorta and mesenteric artery were examined ex vivo for rubidium eff

293 This contrasts with the mesenteric artery, where Ca entry and ryanodine receptor

294 ocated at the branch points of the renal and mesenteric arteries, whereas lesions in this area were n

295 ium-dependent relaxation is impaired in T1DM mesenteric arteries, which is rescued by SOD mimetic tem

296 subunit mRNA increased significantly in the mesenteric artery while rubidium efflux was increased in

297 The response of rat mesenteric arteries with intact (+E) and denuded (-E) en

298 ETA relaxed endothelium-denuded rabbit small mesenteric arteries with maximum relaxations of 22.6 +/-

299 In the present study, treatment of rat mesenteric arteries with phenylephrine (PE) led to the i

300 the bleeding rate in the injured femoral and mesenteric arteries, with a complete hemorrhage arrest a