ライフサイエンスコーパス: mesenteric lymph node

1 ression and bacterial translocation into the mesenteric lymph node.

2 lular leak initiates immune responses in the mesenteric lymph node.

3 e system interactions with LjN6.2 within the mesenteric lymph node.

4 hese sequences were compartmentalized in the mesenteric lymph node.

5 es, but only the wild-type virus reached the mesenteric lymph node.

6 thereby preventing their accumulation in the mesenteric lymph node.

7 ion of DCs in rhesus macaque (Macaca mulata) mesenteric lymph node.

8 CII(+) macrophages in the lamina propria and mesenteric lymph nodes.

9 f Breg cells in the spleen as well as in the mesenteric lymph nodes.

10 d levels of enteric bacterial genomic DNA in mesenteric lymph nodes.

11 on and exhibited limited colonization of the mesenteric lymph nodes.

12 ed by standard microbiological techniques on mesenteric lymph nodes.

13 y induced both IFN-beta and IFN-gamma in the mesenteric lymph nodes.

14 inflammatory cytokine production within the mesenteric lymph nodes.

15 as correlated to a Th17 cell bias within the mesenteric lymph nodes.

16 egs within the intestinal lamina propria and mesenteric lymph nodes.

17 y measuring numbers of regulatory T cells in mesenteric lymph nodes.

18 ld-type (WT) mice in the Peyer's patches and mesenteric lymph nodes.

19 Salmonella from the intestinal tract to the mesenteric lymph nodes.

20 intestines and their draining lymphatics and mesenteric lymph nodes.

21 splenomegaly and massive enlargement of the mesenteric lymph nodes.

22 ology, and production of interferon-gamma in mesenteric lymph nodes.

23 the colon and subsequent migration into the mesenteric lymph nodes.

24 ential adoptive transfers of CD4+ cells from mesenteric lymph nodes.

25 ugh viable bacteria could be observed in the mesenteric lymph nodes.

26 re determined in duodenum, ileum, colon, and mesenteric lymph nodes.

27 -specific regulatory T cell induction in the mesenteric lymph nodes.

28 dritic cells became prominently activated in mesenteric lymph nodes.

29 tinued reduction in CD4(+) T cell numbers in mesenteric lymph nodes.

30 l accumulation in the inflamed intestine and mesenteric lymph nodes.

31 of the small intestine, Peyer's patches, and mesenteric lymph nodes.

32 f the surrounding mesenteric fat tissue, and mesenteric lymph nodes.

33 and immature in the fetal thymus, spleen and mesenteric lymph nodes.

34 nlarged germinal centers (GCs) in spleen and mesenteric lymph nodes; 3) enrichment in IRF4(+)CD138(-)

35 were detected in the Peyer's patches (4/5), mesenteric lymph nodes (4/5), spleens (4/10), livers (6/

36 In the mesenteric lymph node, a fourth subset expressed CCR9 an

37 These animals frequently had sterile mesenteric lymph nodes, a finding consistent with transl

38 tinal immune cells that traffic to liver and mesenteric lymph nodes, a process that occurs even durin

39 and CD30 x OX40-deficient OTII cells in the mesenteric lymph node after oral immunization.

40 They were exclusively found in the mesenteric lymph node after T cell-mediated colitis indu

41 lex host response in the Peyer's patches and mesenteric lymph nodes after oral infection with Y. ente

42 Compared with those in mesenteric lymph nodes, AI4 T cells entering PLNs underw

43 urther analyses showed that 55 +/- 7% of the mesenteric lymph node alpha4beta7+CD4 expressed L-select

44 ects were mediated through the limitation of mesenteric lymph node and intestinal DC accumulation and

45 Symptoms, intestinal inflammation, and mesenteric lymph node and intestine mucosal DCs were ass

46 ed germinal center B cells were found in the mesenteric lymph node and Peyer patches.

47 memory CD4+ T cells persist in the draining mesenteric lymph node and protect mice against reinfecti

48 These cells colonize the spleen and mesenteric lymph node and secrete IL-17A in vitro follow

49 PSGL-1 was detected in venules of mesenteric lymph node and small intestine by immunohisto

50 on remained on endothelium, localized within mesenteric lymph node and small intestine.

51 s, and restored the Th17 and Treg content in mesenteric lymph nodes and aorta.

52 seca et al. (2015) demonstrate disruption of mesenteric lymph nodes and associated lymphatics after Y

53 node homing receptors vigorously expanded in mesenteric lymph nodes and colon by day 6 after transpla

54 /acquired Treg (iTreg) cells was observed in mesenteric lymph nodes and colon from both groups, the s

55 -cadherin(+) DCs accumulated in the inflamed mesenteric lymph nodes and colon, had high expression of

56 ith reduced CD4(+) T cell counts in both the mesenteric lymph nodes and colon.

57 ion revealed commensal gut microflora in the mesenteric lymph nodes and elevated LPS levels in the se

58 ited increased Th2 cytokine responses in the mesenteric lymph nodes and elevated serum IgE and IgG1 l

59 bacterial translocation from the gut to the mesenteric lymph nodes and exhibited reduced liver regen

60 es and their distinct modes of action in the mesenteric lymph nodes and intestinal tissues.

61 -specific IL-17A and IL-22 production in the mesenteric lymph nodes and intestine.

62 tion and decreased the numbers of DCs in the mesenteric lymph nodes and lamina propria.

63 and enhances bacterial translocation to the mesenteric lymph nodes and liver, promoting the progress

64 germinal center B cells and plasma cells in mesenteric lymph nodes and more IgA-coated commensal bac

65 iated organized lymphoid tissues such as the mesenteric lymph nodes and Peyer patches.

66 ressing of FoxP3(+) T cells were measured in mesenteric lymph nodes and Peyer's patches cells.

67 ation of gut-homing effector T-cells in both mesenteric lymph nodes and Peyer's patches without obvio

68 d in organized lymphoid tissues, such as the mesenteric lymph nodes and Peyer's patches, as well as i

69 anges in adaptive and innate immune cells in mesenteric lymph nodes and spleen demonstrated that the

70 Mesenteric lymph nodes and spleen were the most heavily

71 lied in all organs except for lungs and that mesenteric lymph nodes and spleen were the most heavily

72 n ligand in cutaneous lymph nodes but not in mesenteric lymph nodes and spleen, demonstrating that ex

73 C in the Peyer's Patches (PP) but not in the mesenteric lymph nodes and spleen.

74 DC subsets were depleted from peripheral and mesenteric lymph nodes and spleens in monkeys with AIDS,

75 itic cells, and less Il10 gene expression in mesenteric lymph nodes and spleens.

76 e early accumulation of donor T cells in the mesenteric lymph nodes and subsequently in the colon.

77 T lymphocytes is significantly increased to mesenteric lymph nodes and to the inflamed paw in a mode

78 rred within the spleen, intestinal tract, or mesenteric lymph nodes and were present at higher propor

79 on of murine CD4(+) T cells from the spleen, mesenteric lymph node, and Peyer's patch.

80 decrease in IL-12p35 expression in colon and mesenteric lymph nodes, and a substantial increase in th

81 s, CCR9 and alpha4beta7, on donor T cells in mesenteric lymph nodes, and augmented the accumulation o

82 lls under inflammatory conditions in spleen, mesenteric lymph nodes, and colon tissue.

83 apoptosis in the esophagus, small intestine, mesenteric lymph nodes, and kidney in minks experimental

84 age populations in the spleen, kidney, skin, mesenteric lymph nodes, and liver are normal.

85 HSPCs in intestinal mucosa, Peyer's patches, mesenteric lymph nodes, and liver.

86 ess, CD25(+) CD4(+) T cells from the spleen, mesenteric lymph nodes, and Peyer's patches of orally to

87 reduced recruitment of MDSCs to the spleen, mesenteric lymph nodes, and primary tumor site.

88 nce of the bacterium in the Peyer's patches, mesenteric lymph nodes, and spleen, suggesting a role fo

89 F-beta transcription in the Peyer's patches, mesenteric lymph nodes, and spleen.

90 cruitment, and cytokine expression in ileum, mesenteric lymph nodes, and spleen.

91 T cells were assessed in the Peyer patches, mesenteric lymph nodes, and spleens by using flow cytome

92 an inflammatory signal that is propagated to mesenteric lymph nodes, and that can facilitate extraint

93 7 in the granulomas, but not in the draining mesenteric lymph nodes, and with a markedly suppressed S

94 However, whether GALT and/or mesenteric lymph nodes are required for intestinal Th17

95 from proximal and distal graft sections and mesenteric lymph nodes at 20 min, 12 hr, 7 day, and 6 mo

96 ositive PCR detection of M. canettii for 5/8 mesenteric lymph nodes at days 1 and 3 p.i. and 5/6 pool

97 ignant cells were identified on laparoscopic mesenteric lymph nodes biopsies.

98 n of CD11b(+)CD103(-) dendritic cells in the mesenteric lymph nodes, both of which could be reversed

99 ipts in the ileal Peyer's patches (IPPs) and mesenteric lymph nodes but on average only approximately

100 etected in nasal swab, nasal turbinates, and mesenteric lymph node, but no evidence of histopathologi

101 IL-17F was mainly produced in the spleen and mesenteric lymph nodes, but IL-23 was unaltered in Il17a

102 eferentially infects the Peyer's patches and mesenteric lymph nodes, causing an acute inflammatory re

103 In this report, we demonstrated that mesenteric lymph node CD103(-) DCs express, among other

104 3SL directly stimulated mesenteric lymph node CD11c(+) dendritic cells and induc

105 d heightened IL-4 and IL-17A production from mesenteric lymph node CD4(+) cells.

106 Mesenteric lymph node CD4(+) FoxP3(+) regulatory T cells

107 Strikingly, neonatal mesenteric lymph node CD4(+) T cells produced Yersinia-s

108 IL-33 augmented mesenteric lymph node cell secretion of IL-5, IL-13, IL-

109 t notably, suppression was transferable with mesenteric lymph node cells (MLNC) from infected animals

110 erpes virus entry mediator, because LIGHT Tg mesenteric lymph node cells do not cause intestinal infl

111 When enteroids are cocultured with CD90(+) mesenteric lymph node cells from IL-33-treated mice, IL-

112 Mesenteric lymph node cells from STAT6(-/-) mice with co

113 m-specific proliferative immune responses of mesenteric lymph node cells in C57BL/6J mice infected wi

114 ntal model for CD by adoptive transfer of Tg mesenteric lymph node cells into RAG(-/-) mice.

115 SEA-stimulated bulk mesenteric lymph node cells or CD4 T cells from 7-week-i

116 Mesenteric lymph node cells produced lower levels of cyt

117 Analyses of spleen and mesenteric lymph node cells showed no differences in tot

118 nalysis of cytokine production by spleen and mesenteric lymph node cells showed production of IL-4, I

119 Adhesion of mesenteric lymph node cells to mucosal addressin cell ad

120 L-10, and transforming growth factor beta in mesenteric lymph node cells, purified CD4 T cells, and i

121 secretion of interferon-gamma by stimulated mesenteric lymph node cells.

122 d TNF-alpha by schistosome egg Ag-stimulated mesenteric lymph node cells.

123 terial translocation, measured as numbers of mesenteric lymph node CFU of noninvasive Escherichia col

124 significant protection from both spleen and mesenteric lymph node colonization by M. avium subsp. pa

125 aled that a DeltasfpA strain is defective in mesenteric lymph node colonization.

126 ry-effector CD4(+) T cells in the spleen and mesenteric lymph nodes compared with wild-type mice; sCY

127 ubsets present within the lamina propria and mesenteric lymph node compartments based on expression o

128 thiocyanate dextran, bacterial counts in the mesenteric lymph nodes complex, and gut water content we

129 Likewise, plasmablast frequency in the mesenteric lymph node correlated with viremia.

130 Mesenteric lymph node cultures from VDR KO and B-VDR KO

131 when exposed to oral Ag, and 4-1BB-deficient mesenteric lymph node DC displayed weak RALDH activity a

132 the TNFR family, together with CD103, marked mesenteric lymph node DC with the highest level of RALDH

133 describe that p38alpha signaling in CD103(+) mesenteric lymph node DCs reciprocally regulates the dif

134 The phenotypes of these DCs and of mesenteric lymph node DCs were assessed by flow cytometr

135 reover, we identify a population of CD103(+) mesenteric lymph node dendritic cells (DCs) that induce

136 ritic cells, lamina propria macrophages, and mesenteric lymph node dendritic cells were examined.

137 activity of vitamin A-converting enzymes in mesenteric lymph node dendritic cells, along with increa

138 s, the production of IFN-gamma by spleen and mesenteric lymph node-derived CD4+ T cells was down-regu

139 TGF-beta on the differentiation of colon and mesenteric lymph node-derived murine Foxp3(-) IL-10(-) C

140 response and IL-4 production in the draining mesenteric lymph nodes during infection with the enteric

141 RORgammat(+) T cells were induced in mesenteric lymph nodes early after SFB colonization and

142 Apoptotic IECs were trafficked to mesenteric lymph nodes exclusively by the dendritic cell

143 CD8(+) T cells in the intestinal mucosa and mesenteric lymph nodes, express the cell adhesion molecu

144 In peripheral and mesenteric lymph nodes from animals with AIDS there was

145 Colonic mucosa and mesenteric lymph nodes from Ccl17-deficient mice produce

146 esponses against a soluble protein Ag and in mesenteric lymph node GC responses against gut-derived A

147 Mesenteric lymph node GCs were more resistant but became

148 om the draining lymph nodes/CNS route to the mesenteric lymph nodes/gut route, which ameliorated EAE

149 YopJ-dependent apoptosis in mesenteric lymph nodes has also been demonstrated in a m

150 ntestinal tissue, active germinal centers in mesenteric lymph nodes, IgG(+) and IgA(+) plasmablasts i

151 hanced C. jejuni invasion into the colon and mesenteric lymph nodes in Il10(-/-); Nod2(-/-) mice, com

152 of Foxp3(+) regulatory T cells increased in mesenteric lymph nodes in mice given MSCs.

153 enzymes were increased in the intestine and mesenteric lymph nodes in mice with active GVHD.

154 ry cells (CD25(+)CD127(-)) in the spleen and mesenteric lymph nodes in the mice treated with both Abs

155 Bacterial culture of mesenteric lymph nodes in these mice isolated K. pneumon

156 Cellularity of local mesenteric lymph nodes, including T- and B-lymphocytes,

157 T-bet(+) T cells and IFN-gamma production in mesenteric lymph nodes, increased expression of Ido1 in

158 DENV enhanced ZIKV infection, mainly in the mesenteric lymph node, indicating the potential for DENV

159 ed by a second wave of dissemination via the mesenteric lymph nodes (indirect pathway).

160 stitutively presented in both peripheral and mesenteric lymph nodes, leading to abortive activation a

161 NKT2 cells were abundant in the mesenteric lymph node (LN) of BALB/c mice and produced I

162 Mesenteric lymph nodes (LN) and spleen were differential

163 asma cells appeared first in the hepatic and mesenteric lymph nodes (LNs) and then at later times in

164 mphatics convey Ags and microbial signals to mesenteric lymph nodes (LNs) to induce adaptive immune r

165 from a reduction of CD103(+) DCs in draining mesenteric lymph nodes (LNs), which is associated with d

166 hat impair antigen and immune cell access to mesenteric lymph nodes (LNs), which normally sustain app

167 Lin(-)c-Kit(+) innate cell population in the mesenteric lymph node, lung, and liver.

168 alities include intestinal lymphangiectasia, mesenteric lymph node lymphadenopathy, and lymphangiogen

169 h evidence of bacterial translocation to the mesenteric lymph nodes, macrophage, and T-lymphocyte inf

170 ic lymphatic vessels and lymph drainage into mesenteric lymph nodes may be compromised.

171 During in vivo mesenteric lymph node migration assays, the absence of L

172 trate significant bacterial translocation to mesenteric lymph node (MLN) and liver following morphine

173 In ex vivo studies using whole mesenteric lymph node (MLN) as well as CD3(+) T-cells, w

174 We show that mudeltaCSR occurs in mouse mesenteric lymph node (MLN) B cells and is AID-dependent

175 Here we report that mesenteric lymph node (MLN) B cells protect mice from co

176 Cytokine production of purified mesenteric lymph node (MLN) B cells was examined by flow

177 and TH17 cytokine, and Tgfbeta expression in mesenteric lymph node (MLN) CD4(+) T cells and jejunum w

178 nfected mice by intraperitoneal injection of mesenteric lymph node (MLN) cells alone from H. hepaticu

179 Splenocytes and mesenteric lymph node (MLN) cells from hamsters infected

180 IgG1, and IgG2a and cytokine secretion from mesenteric lymph node (MLN) cells were analyzed.

181 lamina propria mononuclear cells (LPMC) and mesenteric lymph node (MLN) cells.

182 of alpha4beta7 and CCR9 by Peyer's patch and mesenteric lymph node (MLN) dendritic cells (DCs) in a r

183 cytes in the intestinal tissues, we isolated mesenteric lymph node (MLN) from naive wild type mice.

184 ype in the bone marrow (BM), spleen, and the mesenteric lymph node (MLN) of allergic and control mice

185 The impact of CT on DC subsets in the mesenteric lymph node (MLN) was assessed by flow cytomet

186 cellular apoptosis in Peyer's patches (PPs), mesenteric lymph node (MLN), and spleen.

187 rain 16M was consistently recovered from the mesenteric lymph node (MLN), spleen, and liver beginning

188 DCs of mesenteric lymph nodes (MLN) and joint regional lymph no

189 istinct immune responses concurrently in the mesenteric lymph nodes (MLN) and the spleen after i.p. a

190 The Peyer's patches (PP) and mesenteric lymph nodes (MLN) are structural components o

191 cell differentiation was investigated in the mesenteric lymph nodes (MLN) as well as in galectin-9-ex

192 We show that CCR9(+) T cells isolated from mesenteric lymph nodes (MLN) draining CD SB express a mo

193 was readily detectable in the intestine and mesenteric lymph nodes (MLN) for at least 1 week after i

194 e collected intestinal and other tissues and mesenteric lymph nodes (MLN) from SAMP mice.

195 ng those in adults arise very rapidly in the mesenteric lymph nodes (MLN) of neonates.

196 bone marrow and spleen of naive mice and in mesenteric lymph nodes (mLN) of Trichinella spiralis-inf

197 FN-gamma and TNF-alpha CSCs were detected in mesenteric lymph nodes (MLN) or spleen and Th2 (IL-4) CS

198 en for differentially expressed genes in pig mesenteric lymph nodes (MLN) responding to infection wit

199 ressing) L. monocytogenes recovered from the mesenteric lymph nodes (MLN) was extracellular within th

200 ria travel from the intestinal mucosa to the mesenteric lymph nodes (MLN), a key site for Ag presenta

201 d RRV were detected extraintestinally in the mesenteric lymph nodes (MLN), livers, lungs, blood, and

202 Lymphocytes isolated from ileum, mesenteric lymph nodes (MLN), spleen and thymus were lab

203 uction was impaired during T cell priming in mesenteric lymph nodes (MLN), which correlated with a re

204 te antigens presented by cells isolated from mesenteric lymph nodes (MLN).

205 sed effector T cell induction in the CLN and mesenteric lymph nodes (MLN).

206 propria dendritic cells (DCs) into draining mesenteric lymph nodes (MLN).

207 103, which is essential for migration to the mesenteric lymph nodes (MLN).

208 n of large numbers of myeloid cells into the mesenteric lymph nodes (MLNs) and colon.

209 at CD172a(+)CD11c(+) cells accumulate in the mesenteric lymph nodes (mLNs) and inflamed intestinal mu

210 g lymphocytes were purified from SAMP1/YitFc mesenteric lymph nodes (MLNs) and their ability to induc

211 ing of luminescent Salmonella identified the mesenteric lymph nodes (MLNs) as a major reservoir of re

212 ient T reg cells failed to accumulate in the mesenteric lymph nodes (MLNs) at early time points (2-5

213 e distribution of T and B lymphocytes in the mesenteric lymph nodes (MLNs) by flow cytometry.

214 The pathogen was detected in the mesenteric lymph nodes (MLNs) of irradiated mice 1-4 d p

215 T cells purified from mesenteric lymph nodes (MLNs) of orally immunized WT mic

216 ne receptor (Cxcr)5(+) CD4(+) T cells in the mesenteric lymph nodes (MLNs) of transiently n-6-fed mic

217 T cells were cultured with purified DCs from mesenteric lymph nodes (MLNs) or intestines of wild-type

218 Migration of Foxp3+ T cells from the mesenteric lymph nodes (MLNs) to the lamina propria occu

219 elium from where they are transported to the mesenteric lymph nodes (MLNs) within migrating immune ce

220 nting translocation of commensal bacteria to mesenteric lymph nodes (mLNs), and limiting colitogenic

221 lated from the intestinal lamina propria and mesenteric lymph nodes (MLNs), and the following paramet

222 ack CD103+CD11b- DCs in the lung, intestine, mesenteric lymph nodes (MLNs), dermis, and skin-draining

223 The mesenteric lymphatic system, including mesenteric lymph nodes (MLNs), is an important viral res

224 populations in the peripheral blood, liver, mesenteric lymph nodes (MLNs), jejunum, and bronchoalveo

225 enhances alloantigen presentation within the mesenteric lymph nodes (mLNs), mediated by donor CD103(+

226 It disseminates from gut to mesenteric lymph nodes (MLNs), spleen, and liver of infe

227 from the gut to systemic sites, such as the mesenteric lymph nodes (MLNs), via CD11b(+) migratory de

228 n the lamina propria (LP) and migrate to the mesenteric lymph nodes (MLNs), where they drive the diff

229 he lumen to a key immune inductive site, the mesenteric lymph nodes (MLNs).

230 atory responses in Peyer's patches (PPs) and mesenteric lymph nodes (MLNs).

231 nt a resident memory (Trm) population in the mesenteric lymph nodes (MLNs).

232 patches, limited dendritic cell migration to mesenteric lymph nodes [mLNs] causing reduced T cell-med

233 located in the bacterial translocation site (mesenteric lymph nodes [MLNs]) of unirradiated mice were

234 these cells in the liver, lung, spleen, and mesenteric lymph node, more than one week after transpla

235 ntestinal inflammation by activating gut and mesenteric lymph node myeloid cells.

236 Except for IgG3 in the IPPs and mesenteric lymph nodes, no stochastic pattern of Cgamma

237 it at high levels in the small intestine and mesenteric lymph node of young adult mice, suggesting a

238 SS2 was detected in mesenteric lymph nodes of 40% of challenged piglets.

239 of gammadelta-17 cells in the peripheral and mesenteric lymph nodes of adult IL-15Ralpha KO mice, but

240 population of DCs was also depleted from the mesenteric lymph nodes of B27-transgenic rats.

241 MAbs) produced from the Peyer's patches and mesenteric lymph nodes of BALB/c mice immunized intragas

242 Mtor gene expression to a greater extent in mesenteric lymph nodes of BALB/cAnPt mice than of DBA/2N

243 ing with the population of CD4+ cells in the mesenteric lymph nodes of BM-->tg epsilon26 mice.

244 Colon tissues and mesenteric lymph nodes of Card9-null mice had reduced le

245 persistence of Salmonella in the spleen and mesenteric lymph nodes of chronically infected mice.

246 CD4+ cells purified from the mesenteric lymph nodes of colitic BM-->tg epsilon26 mice

247 rase chain reaction of intestinal mucosa and mesenteric lymph nodes of Duoxa(-/-) mice that lack func

248 ency of CD4(+)CD25(+)Foxp3(+) T cells in the mesenteric lymph nodes of GF mice compared with SPF mice

249 regulate T cell homing, were also reduced in mesenteric lymph nodes of infected AMCase-deficient mice

250 ions is essential for their expansion in the mesenteric lymph nodes of infected mice.

251 required to maintain the Th2 response in the mesenteric lymph nodes of infected mice.

252 TLR4 conditioned the in vivo mobilization to mesenteric lymph nodes of intestinal migratory CD103(+)

253 ica infection within the Peyer's patches and mesenteric lymph nodes of mice.

254 ed mice readily accumulated in the colon and mesenteric lymph nodes of recipient mice, and they recon

255 Cells isolated from mesenteric lymph nodes of the transgenic mice had signif

256 om rectal biopsy specimens, bone marrow, and mesenteric lymph nodes of vaccinated infected, unvaccina

257 CCR9(high) memory-phenotype cells within the mesenteric lymph nodes of wild-type hosts.

258 as comparable to those found in the draining mesenteric lymph node or the spleen.

259 +) T cells preferentially home to spleen and mesenteric lymph nodes owing to increased expression of

260 e Gag-specific CD8+ T cell clonotypes in the mesenteric lymph nodes relative to rhesus macaques with

261 ted immune cell subsets from the spleens and mesenteric lymph nodes revealed an increase in PD-1(+)CD

262 Culture of mesenteric lymph nodes showed higher density of infectio

263 D69 by CD4 T cells isolated from the spleen, mesenteric lymph nodes, small intestinal lamina propria,

264 phimurium recovered from the lamina propria, mesenteric lymph nodes, spleen, and liver.

265 the mouse and are particularly prevalent in mesenteric lymph nodes, spleen, and liver.

266 d in cervical lymph nodes at 8 weeks, in the mesenteric lymph nodes, spleen, and Peyer's patches at 1

267 enous increase of SIV RNA in superficial and mesenteric lymph nodes, spleen, and the gastrointestinal

268 develop organized lymphoid tissues including mesenteric lymph nodes, splenic follicles and gut-associ

269 The appearance of i.p. injected cells in mesenteric lymph nodes suggests that the mesentery-assoc

270 The cavitating mesenteric lymph node syndrome (CMLNS) is a rare manifes

271 s were sacrificed on day 1 after injury, and mesenteric lymph node T cells were isolated.

272 f antigen-specific regulatory T cells in the mesenteric lymph nodes than mast cell-containing control

273 greater dissemination of the bacteria to the mesenteric lymph nodes than mice infected with wild-type

274 omal RNA gene sequencing; lamina propria and mesenteric lymph node tissues were analyzed by RNA seque

275 ired characterization of immune cells in the mesenteric lymph nodes, to delineate colonic immune nich

276 stantially in their ability to expand in the mesenteric lymph nodes, trigger proinflammatory cytokine

277 ed lymphatic transport of dendritic cells to mesenteric lymph nodes, two features likely to actively

278 hese CD8(+) T(reg) undergo conversion in the mesenteric lymph nodes under the influence of recipient

279 o initially differentiate in the GALT and/or mesenteric lymph nodes upon Ag encounter and subsequentl

280 nces in survival observed between resectable mesenteric lymph nodes versus unresectable masses in the

281 pattern and subsequently migrate toward the mesenteric lymph nodes via the mesenteric lymphatic capi

282 Affymetrix porcine GeneChip analysis of pig mesenteric lymph nodes was used to identify 848 genes sh

283 Spleen and mesenteric lymph node were collected, processed, and ana

284 endogenous TGF-beta, DCs from gut-associated mesenteric lymph nodes were capable of differentiating F

285 ed that 1% to 2% of lymphocytes in the graft mesenteric lymph nodes were CD4/CD25(HIGH+)/Foxp3(+) cel

286 Spleens and mesenteric lymph nodes were collected and analyzed for T

287 s (cDC) in the intestinal lamina propria and mesenteric lymph nodes were GFP(+) However, in vitro inf

288 The Peyer's patches and mesenteric lymph nodes were markedly enlarged with expan

289 pha(-/-)) mice that lack Peyer's patches and mesenteric lymph nodes were orally infected with murine

290 Both the ileum and the mesenteric lymph nodes were persistently infected for mo

291 PA-TG, the concentrations of free MPA in the mesenteric lymph nodes were significantly enhanced (up t

292 bacteria across the intestinal epithelium to mesenteric lymph nodes were significantly greater in wil

293 aive lymphocytes traffic to the gut-draining mesenteric lymph nodes where they undergo antigen-induce

294 uces tolerogenic XCR1(+) DC migration to the mesenteric lymph node, where Treg cells are induced and

295 with lymphoid organs, such as the spleen and mesenteric lymph nodes, where these same cells appear to

296 of the target genes in colonic biopsies and mesenteric lymph nodes which was accompanied with a dist

297 g of ILCs from the intestine to the draining mesenteric lymph nodes, which specifically for the LTi-l

298 -specific CD4 T cells in Peyer's patches and mesenteric lymph nodes, which was accompanied by increas

299 cell populations in the Peyer's patches and mesenteric lymph nodes, while 6'-sialyllactose also indu

300 arterial rim enhancement around the necrotic mesenteric lymph nodes, without venous wash-out.