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1 eveal that GABRP responds more like a type-B metabotropic receptor.
2 entiation, desensitization and activation by metabotropic receptors.
3 hyperpolarization with activation of group I metabotropic receptors.
4  NMDA as well as non-NMDA receptors, but not metabotropic receptors.
5 rly changes of the density of ionotropic and metabotropic receptors.
6 e, serotonin, and norepinephrine) signal via metabotropic receptors.
7 ing of striatal neurons relies critically on metabotropic receptors.
8 tal neurons is tightly controlled by various metabotropic receptors.
9 trocytes contain BDNF and increase levels of metabotropic receptors.
10  can an abnormal activation of glutamatergic metabotropic receptors.
11 ity that also depends on activation of these metabotropic receptors.
12 e downstream actions for both ionotropic and metabotropic receptors.
13   Presynaptic Ca2+ channels are inhibited by metabotropic receptors.
14 mine diffuses to more distant, slower-acting metabotropic receptors.
15 ntly in response to activation of excitatory metabotropic receptors.
16 nervous system and is typically mediated via metabotropic receptors.
17 s mediate diffuse signaling at extrasynaptic metabotropic receptors.
18  and GABA signal through both ionotropic and metabotropic receptors.
19 ase was potentiated by activation of group I metabotropic receptors.
20 litate release of norepinephrine to activate metabotropic receptors.
21                 Selective antagonists of the metabotropic receptors 1 (mGluR1), +/-2-methyl-4-carboxy
22 utation analysis revealed that the glutamate metabotropic receptor 3 (GRM3) gene gained a premature s
23 tamate NMDA receptor subunit 2D (Grin2d) and metabotropic receptor 4 (Grm4) and downregulation of the
24 ]: rs9641220, P = 6.0x10(-10)) and glutamate metabotropic receptor 5 (GRM5)/TYR (lead SNP: rs661177,
25  Ca(2+) signals and the absence of glutamate metabotropic receptor 5 in adults.
26      This plasticity was not specific to the metabotropic receptor activating the GIRK channels, as d
27                    The excitatory effects of metabotropic receptor activation in the LGN appear to be
28 hanism by which ionotropic signals can shape metabotropic receptor activity in neurons and influence
29                       Lastly, we applied the metabotropic receptor agonist DHPG to dendrites and obse
30                         A selective group II metabotropic receptor agonist inhibited NMDA-stimulated
31 , 3-dicarboxylic acid (ACPD, a glutamatergic metabotropic receptor agonist), serotonin, or 2-methylth
32 , the effects of an intrastriatally injected metabotropic receptor agonist, trans-(1S,3R)-1-amino-1,3
33 how little sequence homology with most other metabotropic receptors and are important modulators of s
34 ient increase in phosphorylation mediated by metabotropic receptors and attenuated by inhibitors of c
35 e, bipolar neurones, have glutamate APB-type metabotropic receptors and can be identified by the pres
36            Rather, the potentiation involves metabotropic receptors and intracellular Ca2+ release fr
37 t NMDA receptor glutamate recognition sites, metabotropic receptors and opioid binding sites.
38                  They express ionotropic and metabotropic receptors, and can release gliotransmitters
39 neurotransmitters, through the activation of metabotropic receptors, and can release the gliotransmit
40 netic silencers include ion pumps, channels, metabotropic receptors, and tools that damage the neurot
41                                          The metabotropic receptor antagonist AP-3 was without effect
42         To evoke a transduction current, the metabotropic receptor antagonist LY341495 was applied to
43 xylic acid (AIDA) but not by the presynaptic metabotropic receptor antagonists alpha-methyl-4-phospho
44 s well as by the non-selective and selective metabotropic receptor antagonists L-(+)-2-amino-3-phosph
45 the protein-synthesis-dependent functions of metabotropic receptors are exaggerated in fragile X synd
46                                              Metabotropic receptors are responsible for so-called 'sl
47                                    NMDAR and metabotropic receptors bidirectionally regulate TNiK pho
48  ionic environment, or (b) by stimulation of metabotropic receptors; brief oscillations can even occu
49  postulate that activation of ionotropic and metabotropic receptors by C-fiber nociceptor afferents a
50 mate to the IP(3) signaling pathway, group I metabotropic receptors can increase intracellular Ca(2+)
51 s, an all-or-none activation profile, and no metabotropic receptor component, thought to be a main in
52  (EPSPs), a graded activation profile, and a metabotropic receptor component; thought to be modulator
53 alent hypothesis proposes that activation of metabotropic receptors coupled to the phosphatidylinosit
54  cannot be phosphorylated by CaMKII enhances metabotropic receptor-dependent depression in the hippoc
55 ated' channel whose activation downstream of metabotropic receptors elicits inflammatory pain or itch
56 injected with mRNA for GirK2 and the mGluR1a metabotropic receptor, exposure to glutamate potentiated
57 ta suggest that glutamate, acting at several metabotropic receptors expressed by astrocytes, could mo
58 es of membrane receptors, the ionotropic and metabotropic receptor families, which differ in their st
59 ine the brainwide expression patterns of all metabotropic receptors for acetylcholine, GABA, and glut
60  remain stable, expression of ionotropic and metabotropic receptors for all these neurotransmitter sy
61 In addition to their physiological function, metabotropic receptors for neurotransmitter gamma-aminob
62                  Although the ionotropic and metabotropic receptors for synaptically released glutama
63 ge number of genes coding for ionotropic and metabotropic receptors for various neurotransmitters-glu
64     Stimulation of gamma-aminobutyric acid B metabotropic receptors (GBRs) by baclofen reduces the in
65 ed by presynaptically expressed Gi/o-coupled metabotropic receptor (Gi/o-GPCR) activation.
66 K-801) glutamate receptor antagonists, and a metabotropic receptor glutamate agonist (ACPD), decrease
67 ling cascades consisting of ion channels and metabotropic receptors govern the electrical properties
68 ereas modulators also are likely to activate metabotropic receptors having a slow and prolonged posts
69 were used to investigate the role of group I metabotropic receptors in late-phase long-term potentiat
70  (GCP II) in order to explore a role for the metabotropic receptors in schizophrenia therapeutics.
71                                         Many metabotropic receptors in the nervous system act through
72  the presence of ionotropic and cell surface metabotropic receptor inhibitors.
73               GPR39 encodes a Zn(2+)-binding metabotropic receptor known to modulate excitatory and i
74      We also achieve 2P photoactivation of a metabotropic receptor, LimGluR3, with a new mGluR-specif
75 receptor on the neurones was identified as a metabotropic receptor linked to activation of phospholip
76 te and generated by stimulation of glutamate metabotropic receptors linked to G proteins and activati
77 on of mGluR5, demonstrating that H1b affects metabotropic receptor localization via a direct protein-
78 uctural sites that have been associated with metabotropic receptor localization.
79 rning mechanisms, and suggest that targeting metabotropic receptors may be useful in treating psychia
80 inputs activating very slow EPSPs (i.e., via metabotropic receptors) may be able to inactivate I(T) w
81                                    Glutamate metabotropic receptor mediated mechanisms have been impl
82     Glia demonstrated no evidence of NMDA or metabotropic receptor-mediated currents or membrane cond
83                          The identity of the metabotropic receptor mediating slow neuromodulatory eff
84  upstream glutamatergic transmission via the metabotropic receptor MGL-1 and mediated by the broadly
85                            Activation of the metabotropic receptor mGluR1 and inhibition of I(h) chan
86 ainate receptor (GluR5, GluR6, and GluR7) or metabotropic receptor (mGluR1-6) subtypes.
87 ut the AMPA receptor subunit, GluR4, and the metabotropic receptor, mGluR1 alpha, were found only at
88 icroscopic analysis reveals that the group I metabotropic receptor mGluR1alpha is significantly enric
89 t GluR1, the NMDA receptor subunit NR2A, the metabotropic receptor mGluR1alpha, the plasticity factor
90                         Presynaptic group II metabotropic receptors (mGluR2 and mGluR3) are among dif
91 spartylglutamate (NAAG) activates a group II metabotropic receptor, mGluR3.
92  signaling mediated by the group 1 glutamate metabotropic receptor, mGluR5, contributes to the longer
93 e cation channels, an effect mediated by the metabotropic receptor mGluR6.
94 ent of spinal glutamate receptors, including metabotropic receptors (mGluRs) and NMDA, in 5-HT(2A)R-i
95                        Blockade of group III metabotropic receptors (mGluRs) counteracted the effect
96                        Activation of group I metabotropic receptors (mGluRs) is required for anti-Heb
97 es indicate that glutamate may also activate metabotropic receptors (mGluRs) to modulate the excitabi
98 a protective role may be played by group III metabotropic receptors (mGluRs), which are uniquely loca
99 entually understand the molecular aspects of metabotropic receptor modulation of the N-type Ca2+ chan
100 ouple with this G-protein (group I glutamate metabotropic receptors, neurotensin type 1) could simila
101 nstrate that hypothalamic astrocytes contain metabotropic receptors of the metabotropic glutamate rec
102 cone stimuli suppress glutamate release onto metabotropic receptors of the S cone bipolar cell dendri
103 rom climbing fibers activates ionotropic and metabotropic receptors on Golgi cells through spillover-
104 m cortex and brainstem are known to activate metabotropic receptors on relay cell dendrites at which
105 that this is mediated by both ionotropic and metabotropic receptors on the same nerve terminal.
106 )) channels in retinal neurons by means of a metabotropic receptor pathway.
107                                   Inhibitory metabotropic receptors reduce Ca(2+) entry through activ
108 as recently demonstrated that a rat group II metabotropic receptor (rm-GluR2) is capable of coupling
109  extrasynaptic activation of the slow-acting metabotropic receptor, SER-2.
110 onsible for the use-dependent termination of metabotropic receptor signaling in embryonic sensory neu
111 in regulating affective states by modulating metabotropic receptor signaling pathways and neural acti
112                                    Moreover, metabotropic receptor stimulation evidently exerts oppos
113                   These results suggest that metabotropic receptor stimulation leads to the death of
114                          The consequences of metabotropic receptor stimulation, including activation
115                           Acting at a single metabotropic receptor subtype, ACh exerts two opposing a
116 layed >10000-fold selectivity over all other metabotropic receptor subtypes plus a wide range of othe
117 ls are sensitive to the action of glycine at metabotropic receptors, suggesting this signal regulates
118        In FXS, sAPPalpha signals through the metabotropic receptor that, activating the MAP kinase pa
119 ion of glutamate and acetylcholine acting on metabotropic receptors that are central to hippocampal f
120 cetylcholine receptors are G-protein-coupled metabotropic receptors that control the excitability of
121                       PFs activate glutamate metabotropic receptors that increase phosphoinositide tu
122 le in the kinetics of G-protein cascades for metabotropic receptors throughout the body.
123 d onto retinal ON bipolar neurons binds to a metabotropic receptor to activate a heterotrimeric G-pro
124 thelium, confirming the localization of this metabotropic receptor to gustatory cells.
125 ion, synaptically released glutamate acts on metabotropic receptors to excite neurons on a slower tim
126       Further more, the coupling of specific metabotropic receptors to the various neuronal-specific
127 gamma subunits) are critical for coupling of metabotropic receptors to their downstream effectors.
128 ising Galpha and Gbetagamma subunits, couple metabotropic receptors to various downstream effectors a
129 ells that are engineered to express a chosen metabotropic receptor, use the G(q) protein-coupled rece
130 tive agonists indicated that the predominant metabotropic receptor was the L-2-amino-4-phosphonobutyr
131        At this synapse, glutamate binds to a metabotropic receptor which couples to the closure of a
132 ntracellular calcium following activation of metabotropic receptors, which may trigger glutamate secr
133 dritic spines of neurons, local retention of metabotropic receptors within dendritic ER provides a po
134                               Thus, Group II metabotropic receptors within or very near the amygdala

 
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