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1 at PL cells were haploid relative to diploid metacyclics.
2 n was associated with a high mean percent of metacyclics (66%-82%) rather than total GMPL (2.0 x 10(4
3   To study this, we measured the adhesion of metacyclic and logarithmic-phase L. major promastigotes
4 Abs effectively bind to and agglutinate both metacyclic and procyclic L. major promastigotes.
5 e phosphorylation specifically in infectious metacyclics and amastigotes and promoting parasite survi
6 e RNA binding protein 6 to obtain infectious metacyclics and determined their protein and mRNA repert
7                               We showed that metacyclics are quiescent cells, and propose this influe
8 ed inoculum and, along with nondividing late metacyclics, are infectious to and exacerbate the pathol
9  could readily differentiate into infectious metacyclic cells but these were unable to establish infe
10 somes must develop into mammalian-infectious metacyclic cells in the fly's salivary glands (SGs) befo
11          During differentiation to infective metacyclics, d-arabinopyranose (d-Arap) caps the LPG sid
12 on (171 bp) is required for full activity in metacyclic-derived trypanosomes.
13 on very close to the initiation site used in metacyclic-derived trypanosomes.
14 Our results demonstrate that amastigotes and metacyclics efficiently enter and activate DCs of both g
15                Moreover, they expressed less metacyclic-enriched transcripts, displayed increased sen
16 express VSG from a separate set of telomeric metacyclic ESs (MESs).
17 and the genes that govern the development of metacyclic form parasites.
18 and failed to differentiate to the infective metacyclic form.
19 oodstream forms in mammals and procyclic and metacyclic forms (MFs) in tsetse fly hosts.
20 e parasite to transform from procylic to the metacyclic forms affecting the parasite's potential to c
21 setse, including the generation of infective metacyclic forms expressing the variant surface glycopro
22 tural transmission, we used sand fly-derived metacyclic forms of L. major and preexposed the injectio
23                               The infectious metacyclic forms of Trypanosoma brucei result from a com
24  developmental program leading to infectious metacyclic forms preadapted to survive in the mammalian
25 le concerning the gene expression profile of metacyclic forms.
26 eath occurred not by apoptosis or changes in metacyclic gene expression, but from catastrophic proble
27                                              Metacyclics have a largely bloodstream-form type transcr
28 sites develop from amastigotes to infectious metacyclics, highlighting recent findings concerning the
29 w that a mucin expressed and secreted by the metacyclic infective form of T. cruzi, AgC10, is able to
30 dermal forehead challenge infection with 107 metacyclic L. amazonensis promastigotes at 4 wk demonstr
31 the parasite inoculation site, we introduced metacyclic L. infantum chagasi promastigotes intradermal
32                      Now we demonstrate that metacyclic L. major promastigotes are poor inducers of I
33                                              Metacyclic Leishmania infantum chagasi promastigotes wer
34 somes did not result in cells becoming more 'metacyclic-like'.
35                  Of biological significance, metacyclic lipophosphoglycan lacks the glucose residues
36                                              Metacyclic lpg5A(-)/lpg5B(-) promastigotes showed strong
37 lopmental checkpoints in epimastigote (EMF), metacyclic (MCF), and BSF.
38 d the largest changes between procyclics and metacyclics, observed at both the transcript and protein
39 th bifurcation into either replicating early metacyclics or attached and detached haptomonads.
40 e spt2- failed to differentiate to infective metacyclic parasites and died instead.
41 to bind to sand fly midguts in vitro whereas metacyclic parasites and LPG lost this capacity.
42                             Blocking the few metacyclic parasites deposited in saliva from further de
43 te this differential effect of procyclic and metacyclic parasites in terms of IL-12 induction, both s
44 though the level of scAra caps is maximal in metacyclic parasites, scbetaAraT activity is maximal in
45 t of epimastigotes and infectious, quiescent metacyclic parasites.
46 ation of one member (named SGM1.7) on mature metacyclic parasites.
47 -mice were inoculated with 250, 500, and 750 metacyclic parasites.
48 vidual cell level data also confirm that the metacyclic pool is diverse, and that each parasite expre
49 ring this process that leads to an activated metacyclic primed for invasion.
50 , since blocking macrophage Mac-1 diminishes metacyclic promastigote adhesion to a greater extent tha
51 lysosome but largely failed to eliminate the metacyclic promastigote life cycle stage of the parasite
52  attaching to the gut wall, to a nondividing metacyclic promastigote stage that is unable to attach t
53  the sand fly vector to the highly infective metacyclic promastigote stage.
54 ein kinases involved in differentiation from metacyclic promastigote to amastigote, growth and surviv
55 iding further support for the role of PGs in metacyclic promastigote virulence.
56            We find that the human-infective (metacyclic promastigote) forms display 'run and tumble'
57 tion between myeloid-derived human DC and Lm metacyclic promastigotes (infectious-stage parasites) to
58 that: 1) they are expressed predominantly in metacyclic promastigotes (the form in the insect vector
59 n L. amazonensis enhanced differentiation of metacyclic promastigotes and amastigotes, but the parasi
60 tiation into the vertebrate infective forms, metacyclic promastigotes and amastigotes.
61  the parasite during differentiation both to metacyclic promastigotes and to amastigotes, autophagoso
62 f inoculation of infectious Leishmania major metacyclic promastigotes by sand flies.
63 e mice were challenged by inoculation of 100 metacyclic promastigotes in the ear dermis.
64 s leishmaniasis using 10(2) Leishmania major metacyclic promastigotes inoculated into the footpads of
65 wth of L. major following inoculation of 100 metacyclic promastigotes into the ear dermis.
66                               L. amazonensis metacyclic promastigotes lacking one SODA allele failed
67 infected intradermally in the ear with 10(5) metacyclic promastigotes of L. amazonensis together with
68                               The infectious metacyclic promastigotes of Leishmania protozoa establis
69 more abundantly expressed in amastigotes and metacyclic promastigotes than in procyclic promastigotes
70 e efficient adhesion of complement-opsonized metacyclic promastigotes to cells expressing both recept
71 the initial complement-dependent adhesion of metacyclic promastigotes to human monocyte-derived macro
72 igotes and, to a much lesser extent, that of metacyclic promastigotes to induce IL-12 were enhanced b
73 depends on the ability of insect-transmitted metacyclic promastigotes to invade mammalian hosts, diff
74  The stable adhesion of complement-opsonized metacyclic promastigotes to Mac-1 is a prerequisite for
75                                        1,000 metacyclic promastigotes were coinoculated with a saliva
76 tures of natural transmission: low dose (100 metacyclic promastigotes) and inoculation into a dermal
77      In contrast to the infective parasites (metacyclic promastigotes), the procyclic promastigotes c
78 )O(2), generate a greater yield of infective metacyclic promastigotes, and have increased virulence.
79 old was also required for the development of metacyclic promastigotes, as SODA/DeltasodA cultures wer
80 ventional experimental model employing 10(6) metacyclic promastigotes, in which the rapid development
81         Apart from isolated mammal-infective metacyclic promastigotes, little is known about the tran
82 racellular Leishmania to mammalian-infective metacyclic promastigotes, perhaps orchestrating the clea
83 us leishmaniasis (Lm), once loaded with live metacyclic promastigotes, were found to reactivate autol
84 for the phagocytosis of complement-opsonized metacyclic promastigotes.
85 fective procyclic promastigotes to infective metacyclic promastigotes.
86 SP and MSP-like protein (MLP), from virulent metacyclic promastigotes.
87 lenged in the ear with live Leishmania major metacyclic promastigotes.
88 3' UTRs uncovered: (1) previously identified metacyclic-specific expressed genes; (2) cloned genes wh
89 ogarithmic phase of growth to the infectious metacyclic stage does not affect this interaction.
90 t activated when VSG synthesis begins in the metacyclic stage in the tsetse fly salivary glands, are
91 of Leishmania infantum chagasi to a virulent metacyclic stage, as did the expression of PGF2alpha syn
92 nd differentiation into the animal-infective metacyclic stage.
93 sidues upon differentiation to the infective metacyclic stage.
94 um before differentiating into the infective metacyclic stages.
95 ms and subsequent purification of infectious metacyclics supports the existence of an RNA-recognition
96 hagosomes being particularly numerous during metacyclic to amastigote form transformation.
97 resent the epimastigote, and pre- and mature metacyclic trypanosome developmental stages.
98                                   Infectious metacyclic trypanosomes present within the tsetse fly ve
99                                              Metacyclic trypanosomes, which inhabit the tsetse saliva
100 ng to each gene in both the epimastigote and metacyclic trypomastigote developmental stages.
101 hosphorylase a by T. cruzi epimastigotes and metacyclic trypomastigote extracts.
102 tigotes undergo before they develop into the metacyclic trypomastigote stage.
103 rm resembles the transition to the infective metacyclic trypomastigote stage.
104 differentiation from the epimastigote to the metacyclic trypomastigote stage.
105                            Trypanosoma cruzi metacyclic trypomastigotes (MT), but not blood form tryp
106 deficient epimastigotes readily converted to metacyclic trypomastigotes and efficiently infected mamm
107                       We found that T. cruzi metacyclic trypomastigotes induced microvesicle release
108 i sp. are established after the injection of metacyclic trypomastigotes into the skin dermis by the t
109 d to extracellular amastigote-like cells and metacyclic trypomastigotes more rapidly than wild-type p
110 native conjunctival challenges with T. cruzi metacyclic trypomastigotes using a combination of immuno
111 fically reacted with a 55-kDa TcGP63 form in metacyclic trypomastigotes, suggesting stage-specific ex
112 ulture-derived trypomastigotes but 55 kDa in metacyclic trypomastigotes.
113 astigotes but distributed intracellularly in metacyclic trypomastigotes.
114 acellular and lacking in N-linked glycans in metacyclic trypomastigotes.
115    Determination of 23 kb of sequence at the metacyclic variant antigen type 4 (MVAT) vsg expression
116 e-associated gene I (ESAG-I) family occur in metacyclic variant antigen type 4 bloodstream trypanosom
117 e of these ESAG-I mRNAs are derived from the metacyclic variant antigen type 4 variant surface glycop
118                            Activation of the metacyclic variant antigen type 7 (MVAT7) variant surfac
119 ch parasite expresses only one of the unique metacyclic variant surface glycoprotein (mVSG) coat prot
120 ich protein, a marker for epimastigotes, and metacyclic variant surface glycoprotein.
121 Certain VSGs are preferentially expressed in metacyclic versus bloodstream stages as a result of diff
122                                 However, the metacyclic VSG (M-VSG) genes, a small subset activated w
123 f Trypanosoma brucei rhodesiense expresses a metacyclic vsg as a monocistronic RNA from a promoter lo
124 The telomere-linked donor VSG 10.1 resembles metacyclic VSG expression sites, and is preceded by a cl
125 sion Site (BES)-linked VSGs and silencing of metacyclic VSGs (mVSGs) in BF cells are essential for an
126 ng metabolic enzymes exhibited expression in metacyclics with features of both procyclic and bloodstr

 
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