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1 istically with thyroid hormone to accelerate metamorphosis).
2 ation of larval insects into the adult form (metamorphosis).
3 ammals (puberty) and holometabolous insects (metamorphosis).
4 for 7 days and depurated until completion of metamorphosis.
5  leads to tadpole lethality at the climax of metamorphosis.
6 omorphic species, which undergo differential metamorphosis.
7 mations via a process we term macromolecular metamorphosis.
8 become integrated into the mature SEZ during metamorphosis.
9  from microbial infection during molting and metamorphosis.
10 or to the induction of larval settlement and metamorphosis.
11 nes adult morphogenesis in the hemimetabolan metamorphosis.
12 y system undergoes massive remodeling during metamorphosis.
13  to morphological changes upon initiation of metamorphosis.
14 -embryonic developmental transitions such as metamorphosis.
15 nitors at four stages, from embryogenesis to metamorphosis.
16 hanges of the Xenopus olfactory organ during metamorphosis.
17 d that they are eventually eliminated during metamorphosis.
18 based one (adult mode) as it transits beyond metamorphosis.
19 or on a novel one invented by the newt after metamorphosis.
20 hanges of the Xenopus olfactory organ during metamorphosis.
21  its regenerative ability of the limbs after metamorphosis.
22 lose their ability to regenerate limbs after metamorphosis.
23 ctivate circadian oscillator function during metamorphosis.
24 dpoles and led to tadpole lethality prior to metamorphosis.
25  histoblast nests to the hormonal control of metamorphosis.
26 and the organisation of pigment cells during metamorphosis.
27 y contributing to the correct culmination of metamorphosis.
28 nd the factors they produce stimulate animal metamorphosis.
29 ion in Dicer-1 knockdown individuals rescues metamorphosis.
30 terocyte identity during a defined window of metamorphosis.
31 nderstanding of the molecular basis of adult metamorphosis.
32 be specified twice, during embryogenesis and metamorphosis.
33 e same regulatory mechanism to promote adult metamorphosis.
34 nregulation of all three miRNAs seen late in metamorphosis.
35 e a reduced ability to successfully complete metamorphosis.
36  and begin to reform in the months following metamorphosis.
37 sults in eyeless animals, and is lethal peri-metamorphosis.
38 t a role for awd in ACP wing development and metamorphosis.
39 ntestinal progenitors that take place during metamorphosis.
40 ded to have lower infection prevalence after metamorphosis.
41 emoval of obsolete larval neurons during CNS metamorphosis.
42 erant (Met), plays a critical role in insect metamorphosis.
43 opmental stages but loses the ability during metamorphosis.
44  (DEOM) which occurs during the first 24h of metamorphosis.
45 olt timer that establishes a minimal time to metamorphosis.
46 horacicotropic hormone (PTTH) that initiates metamorphosis.
47 ryogenesis, larval development, and juvenile metamorphosis.
48 al stage and form the adult intestine during metamorphosis.
49 es acting downstream of JH and Met in insect metamorphosis.
50 idal to the eponymous stellate shape, during metamorphosis.
51  and 81.5% of P. regilla were infected after metamorphosis.
52 to recover from Bd infection as they undergo metamorphosis.
53  loses its bile ducts and gallbladder during metamorphosis.
54 ster molecular genetic tools to study insect metamorphosis.
55 the rod loss phenotype was not obvious after metamorphosis.
56 e ecdysteroid molting hormones that regulate metamorphosis.
57 al changes in X. tropicalis intestine during metamorphosis.
58 it is required broadly for patterning during metamorphosis.
59 e to produce MACs are capable of stimulating metamorphosis.
60 xillary and labial appendages, are formed at metamorphosis.
61 uired for adult intestinal stem cells during metamorphosis.
62 h of the pentaradial adult rudiment prior to metamorphosis.
63  increases in osmoregulatory capacity during metamorphosis.
64 es without affecting axons during Drosophila metamorphosis.
65 gateway for the tissue development preceding metamorphosis.
66 cle exit and terminal differentiation during metamorphosis.
67  that the mif1 gene is required for inducing metamorphosis.
68 formation of the larva into the adult during metamorphosis.
69 to new morphologies driven by macromolecular metamorphosis.
70 ms can lead to fates as diverse as death and metamorphosis.
71 the remodeling of cartilage tissue, prior to metamorphosis.
72 een in the shift from incomplete to complete metamorphosis.
73 P reconstitution, ensuring the completion of metamorphosis.
74 aevis before, during, and after TH-dependent metamorphosis.
75 into the different processes involved during metamorphosis.
76 o control Drosophila wing development during metamorphosis.
77  through metamorphosis) or during T3-induced metamorphosis.
78 mation of adult intestinal stem cells during metamorphosis.
79 developing offspring until tadpoles complete metamorphosis [1-3].
80 h and maturation, represented by molting and metamorphosis [2].
81 meostatic adjustment to starvation but start metamorphosis 4 d after feeding onset, regardless of lar
82  small amounts of cardiac glycosides through metamorphosis, a trait that has been optimized in monarc
83 ent feeding period to ensure that they begin metamorphosis above critical weight.
84 d mitigation strategies need to consider how metamorphosis affects the movement of materials between
85                                              Metamorphosis also increased delta(15)N by approximately
86 rus infection decreased survival and delayed metamorphosis, although chronic corticosterone exposure
87 tion underlying differences in the timing of metamorphosis among three spadefoot toads with different
88 o interpret, as larval neurons degenerate at metamorphosis and a tripartite nervous system differenti
89  that account for effects of contaminants on metamorphosis and adult insect emergence for the develop
90 t predominantly loses these abilities during metamorphosis and adulthood.
91  expressed in the intestine at the climax of metamorphosis and are induced by T3.
92 eroid-dependent program at the initiation of metamorphosis and are the primary phagocytic cell type i
93 larval secondary lineage projections through metamorphosis and bfy identifying each neuroglian-positi
94 ll type specification in embryos and adults, metamorphosis and body plan patterning.
95 are not manifest until later in life (during metamorphosis and emergence).
96 , could be linked with biological effects on metamorphosis and gonadal phenotypes, respectively, that
97 In Rhodnius prolixus, both the physiology of metamorphosis and its hormonal control are known in deta
98 rowth and development of the tadpoles during metamorphosis and leads to tadpole lethality at the clim
99 nsidered to be involved in their settlement, metamorphosis and locomotion.
100                                       Insect metamorphosis and mammalian puberty exhibit similar desi
101                                       Larval metamorphosis and recruitment represent critical life-hi
102 arval bacteriome dissociates at the onset of metamorphosis and releases bacteriocytes that undergo en
103 ls were significantly elevated by stage 5 of metamorphosis and remained elevated through stage 7, whe
104                                      Time to metamorphosis and survival depended on both corticostero
105 dentify bacterial cues that stimulate animal metamorphosis and test hypotheses addressing their mecha
106 3 target genes during natural and T3-induced metamorphosis and that Dot1L is itself a T3 target gene.
107 te that GHR and PRLR may both participate in metamorphosis and that GHR may mediate SW acclimation.
108 posed to 17alpha-ethynylestradiol throughout metamorphosis and the early postmetamorphic period.
109  conservation between MB axon pruning during metamorphosis and the refinement of ectopic larval neuro
110 nge) (LT) or 21-22 degrees C (range) (HT) to metamorphosis and then transferred to 21-22 degrees C.
111 P. luteoviolacea from inducing settlement or metamorphosis and three MAPK inhibitors, we established
112 d hormone (the primary morphogen controlling metamorphosis) and corticosterone (a stress hormone acti
113              Editing levels rise strongly at metamorphosis, and Adar(5G1) null mutant flies lack edit
114 nica with an miR-2 inhibitor, which impaired metamorphosis, and by treating Dicer-1-depleted individu
115 re persisted from the tadpole stage, through metamorphosis, and following probiotic treatment.
116  in tadpoles from the pond of origin, across metamorphosis, and in toadlets via microbial fingerprint
117              Tadpoles retained Se throughout metamorphosis, and partitioned the element predominantly
118 ns during embryogenesis, remodel them during metamorphosis, and repair them following injury.
119 ages, stressful and stochastic events during metamorphosis, and stressful environmental conditions at
120 lopment rate, mass at metamorphosis, date of metamorphosis, and survival.
121 ependent transcription factor that represses metamorphosis, and that depletion of Kr-h1 expression in
122 xposure mortality in the larval stage and at metamorphosis, and very strongly reduced adult lifespan.
123 r key innovations, such as wings or complete metamorphosis are usually invoked as potential evolution
124 rent larval activities (e.g., attachment and metamorphosis) are under the control of different neural
125 ue degeneration and remodeling during anuran metamorphosis as a mechanism for altering tissue-specifi
126        To establish X. tropicalis intestinal metamorphosis as a model for adult organogenesis, we ana
127 tinal remodeling during T3-dependent Xenopus metamorphosis as a model for organ maturation and format
128 s of hemimetabolous insects (with incomplete metamorphosis) as a research model.
129 can induce tubeworm and coral metamorphosis; Metamorphosis-Associated Contractile structures (MACs) a
130  producing ordered arrays of phage tail-like metamorphosis-associated contractile structures (MACs).
131 teoviolacea initiate cilia loss and activate metamorphosis-associated transcription; finally, signali
132                                  Since early metamorphosis at a larger size has potential fitness adv
133                       Salamanders that delay metamorphosis attain significantly larger body sizes as
134 ntify a location on the sea floor to undergo metamorphosis based on the presence of specific bacteria
135 cation on the seafloor to settle and undergo metamorphosis based on the presence of specific surface-
136               After 6 days of exposure, when metamorphosis began, larval survival was unaffected by z
137                  Species undergoing complete metamorphosis (biphasic and direct-developing) exhibit g
138      Lipid stores are consumed shortly after metamorphosis but contribute little to energy metabolism
139 lier breeding and larval survival or mass at metamorphosis, but earlier breeding was associated with
140 abolous insects suggests that holometabolous metamorphosis combines patterning processes of both late
141                                           At metamorphosis, comparisons of RNAi phenotypes indicate t
142                                 We show that metamorphosis could be induced by precocene treatment in
143  subsequent larval development rate, mass at metamorphosis, date of metamorphosis, and survival.
144              At both sites, survival through metamorphosis declined with increasing variability of st
145 nction of Hox genes and Tc-hth/Tc-exd during metamorphosis did not match predictions based on embryon
146 ticularly sensitive due to the potential for metamorphosis-driven mobilization, which could transfer
147                              At the onset of metamorphosis, Drosophila salivary gland cells undergo a
148 tants have severe defects in pupariation and metamorphosis due to a lack of activation of ecdysone-re
149  gene expression initiate periodic molts and metamorphosis during insect development.
150 tional checkpoints, which inhibit precocious metamorphosis during nutrient restriction in undersized
151                        With the evolution of metamorphosis, ecdysteroids acquired a metamorphic funct
152 e isotopes and contaminants), and found that metamorphosis effects varied greatly.
153 ion of a number of processes associated with metamorphosis, either in the less modified hemimetabolan
154 us, adaptive developmental plasticity during metamorphosis enables spinal CPG-driven extraocular moto
155         In holometabolous symbiotic insects, metamorphosis entails a complete and abrupt internal reo
156 tments, tadpoles caused salamanders to reach metamorphosis faster and larger.
157 n of flight initiated the trajectory towards metamorphosis, favoring enhanced differences between juv
158 aquatic larval stage, a brief and pronounced metamorphosis, followed by a terrestrial adult stage.
159 iator complex, causing them to shrink during metamorphosis, followed by nuclear accumulation of Prosp
160 helial stem cell proliferation at the end of metamorphosis (for the few that survive through metamorp
161  a complex life history marked by a dramatic metamorphosis from a benthic filter-feeding ammocoete la
162                                              Metamorphosis from larvae to adult can cause large chemi
163 y variable affecting the timing of amphibian metamorphosis from tadpoles to tetrapods, through the pr
164 f biochemical cues from bacteria that induce metamorphosis has been a mystery.
165 ng hypochord-induced loss of the tail during metamorphosis has enabled the evolution of the unique an
166                                              Metamorphosis has profoundly influenced salamander crani
167 e presynaptic and become apparent only after metamorphosis, highlighting a delayed response to a sign
168        Hydroides' dependency on bacteria for metamorphosis highlights the importance of external stim
169              The second step was to complete metamorphosis, holometaboly, and occurred by profoundly
170 ntact with surface-bound bacteria to undergo metamorphosis; however, the mechanisms that underpin thi
171                      These data suggest that metamorphosis impairment caused by Dicer-1 and miRNA dep
172 sponse variables, including size and mass at metamorphosis in A. maculatum, but at a reduced strength
173 ritic arborization neurons degenerate during metamorphosis in an ecdysone-dependent manner.
174 ly similar to thyroid hormone (TH)-regulated metamorphosis in anuran amphibians.
175     We studied the regulation of molting and metamorphosis in bed bugs with a goal to identify key pl
176 e final step of miRNA biosynthesis, prevents metamorphosis in Blattella germanica.
177 3 is the key determinant that promotes adult metamorphosis in both hemimetabolous and holometabolous
178 of larval sensory neurons is replaced during metamorphosis in both sensory epithelia.
179 d gating mechanism to help ensure productive metamorphosis in Drosophila.
180 suggests that mechanisms of bacteria-induced metamorphosis in Hydroides may have conserved features i
181 o zinc and warming before, during, and after metamorphosis in Ischnura elegans damselflies from high-
182 lignaria We demonstrate that starvation cues metamorphosis in O. lignaria and that a critical weight
183 unveil a remarkable potential for structural metamorphosis in proteins and demonstrate key principles
184 re we show that hdc is expressed just before metamorphosis in sensory neurons that undergo remodeling
185 oroaniline (3,4-DCA) on thyroid function and metamorphosis in tadpoles of Lithobates catesbeianus.
186 tween Kr-h1, E93 and JH in the regulation of metamorphosis in the bed bugs.
187  T3 directly activated the c-Myc gene during metamorphosis in the intestine via binding of the T3 rec
188                                  Survival to metamorphosis in the laboratory was strongly affected by
189 bility in streams decreases survival through metamorphosis in the salamander Gyrinophilus porphyritic
190 ing thyroid hormone (T3)-dependent amphibian metamorphosis in two highly related species, the pseudo-
191  corticosterone exposure accelerated rate of metamorphosis in uninfected larvae.
192 ave characterized a period during behavioral metamorphosis in which zebrafish are highly reactive to
193                       Using the T3-dependent metamorphosis in Xenopus tropicalis as a model, we show
194 of tadpoles and the inverse of their size at metamorphosis) in our tadpole-parasitic cercarial (trema
195  Hox is initiated in the late larva prior to metamorphosis, in preparation for the transition to the
196 s cultripes and Spea multiplicata accelerate metamorphosis, increase standard metabolic rate (SMR), a
197                       Abalone settlement and metamorphosis increased from 11% in the absence of CCRA
198    Acute corticosterone exposure accelerated metamorphosis increased survival in infected larvae.
199     The first JH was described in 1934 as a "metamorphosis inhibitory hormone" in Rhodnius prolixus b
200  for tadpole growth and development prior to metamorphosis into a frog.
201  its brief journey as a larva to its radical metamorphosis into adult form-and relate these features
202 ts for all three thoracic neuromeres through metamorphosis into the adult.
203 opolymer or hyperbranched polymer undergoes 'metamorphosis' into comb, star and hydrophobic block cop
204        Other studies have found that earlier metamorphosis is associated with increased postmetamorph
205 polysulfide dissolution by understanding the metamorphosis is essential for realizing stable and high
206                                              Metamorphosis is often characterized by profound changes
207                      The evolution of insect metamorphosis is one of the most important sagas in anim
208                                    Amphibian metamorphosis is strikingly similar to postembryonic dev
209                                              Metamorphosis is widespread across the animal kingdom an
210                    Although bacteria-induced metamorphosis is widespread among metazoans, little is k
211 d zinc concentrations at different stages of metamorphosis: larval, subimago, and imago.
212 arbons (PAHs) were predominantly lost during metamorphosis leading to approximately 2 to 125-fold hig
213 ingomyelin-rich cells undergoes a structural metamorphosis, leading to the assembly of nanopores at t
214    Toxicity included stunted growth, delayed metamorphosis, malformations, organ pathology, and DNA d
215                           Thus, death during metamorphosis may be a key mechanism explaining how stre
216    These results support the hypothesis that metamorphosis may be a survival bottleneck, particularly
217 he nerve-associated progenitors lasting into metamorphosis may have facilitated the evolution of adul
218 y factors that can induce tubeworm and coral metamorphosis; Metamorphosis-Associated Contractile stru
219 gent, insight into the triggers of Hydroides metamorphosis might lead to practical strategies for fou
220                                During insect metamorphosis, neuronal networks undergo extensive remod
221 either the development of flight or complete metamorphosis nor the Cretaceous Terrestrial Revolution
222 Pt-Sb platform supports the fluoride-induced metamorphosis of a stiboranyl X ligand into a stiborane
223 ellular vesicles in eliciting settlement and metamorphosis of benthic marine larvae.
224                     During the corresponding metamorphosis of CPPase to FPPase, cyclopropanation and
225            Our numerical simulations predict metamorphosis of fast Abrikosov vortices into mixed Abri
226 activity during the N-terminal to C-terminal metamorphosis of FPPase to CPPase, with product selectiv
227                              Average time to metamorphosis of frogs was delayed by 30% in the presenc
228 s of phage tail-like structures that trigger metamorphosis of H. elegans.
229                                              Metamorphosis of larvae of the tubeworm Hydroides elegan
230 ributed marine bacterium that stimulates the metamorphosis of marine animal larvae, an important bact
231 ich play an important role in triggering the metamorphosis of swimming tadpoles.
232                                   An unusual metamorphosis of the CCN atom triad, from a near sp 1-az
233 ing processes of both late embryogenesis and metamorphosis of the hemimetabolous life cycle.
234 aralogs, but not dachshund, are required for metamorphosis of the maxillary endites.
235 olacea biofilms producing MACs stimulate the metamorphosis of the tubeworm Hydroides elegans, TBP bio
236                                       Insect metamorphosis often results in substantial chemical chan
237 t the larva must surpass before it can enter metamorphosis on a normal schedule, and the inhibitory a
238 for only 12-24 h was sufficient to result in metamorphosis on day 4, regardless of further feeding or
239                    Thus, the main effects of metamorphosis on insect chemistry were large declines in
240 sects are generally hypothesized to initiate metamorphosis once they attain a critical weight.
241                                       During metamorphosis, one eye migrates to the contralateral sid
242 hing patterns in a stepwise fashion from mid-metamorphosis onwards.
243  decisions of when and where animals undergo metamorphosis, optimizing conditions for adult developme
244 amorphosis (for the few that survive through metamorphosis) or during T3-induced metamorphosis.
245 ly, is not essential for larval development, metamorphosis, or maintenance of adulthood.
246  flatfish and transcriptomic analyses during metamorphosis point to a role for thyroid hormone and re
247 gument were compared during the larval-pupal metamorphosis process of the S. exigua wild type (SEW) a
248 d explore why, the developmental decision of metamorphosis relies on cues from environmental bacteria
249                      This limited structural metamorphosis represents a previously unidentified envir
250 hese transcription factors in embryogenesis, metamorphosis, reproduction, and homeostasis.
251 uring thyroid hormone (T3)-dependent Xenopus metamorphosis resembles postembryonic intestinal maturat
252 the first instar (L1) larval stage or during metamorphosis, respectively.
253 nique molecular switch occurs during lamprey metamorphosis resulting in distinct gill carbonic anhydr
254 ounding functional constraints on variation, metamorphosis seems to have promoted the morphological e
255 ably expressed markers, differentiate during metamorphosis, sending terminal axonal and dendritic bra
256 ent therefore corresponds to a TH-controlled metamorphosis, sensitive to endocrine disruption.
257                      The T3-dependent anuran metamorphosis serves as a model to study postembryonic d
258 d ca18 and protein expression in gill across metamorphosis show that the ca19 levels are highest in a
259 of genes known to be involved in molting and metamorphosis showed high levels of Kruppel homolog 1 [K
260 ommodation of endocrine pathways controlling metamorphosis, showing how phenotypic plasticity within
261 roendocrine signaling cascade that initiates metamorphosis, similar to the way in which their mammali
262 larval newt, but this changes abruptly after metamorphosis so that the formation of anterior and post
263 nd physiological adaptations such as flight, metamorphosis, sociality, and chemoperception.
264 s and staining intensity further change over metamorphosis, suggesting compositional rearrangement.
265 n larvae fed ad libitum eventually underwent metamorphosis, suggesting that some secondary mechanism
266 etween the start of feeding and the onset of metamorphosis suggests that larvae possess a molt timer
267 were 4.1-4.3 times more likely to survive to metamorphosis than tadpoles in 45-day mesocosms.
268 mammalian immune system may be linked to the metamorphosis that allows them to transfer from mammals
269                                       During metamorphosis, the epithelium of the PC is rearranged in
270                                       During metamorphosis, the larval epithelium degenerates and adu
271                                     Early in metamorphosis, the LNs increase from 8 to 11 in number,
272 itially concentrated in the gut; however, by metamorphosis, the majority were found in other tissues.
273 ure with regard to carry-over effects across metamorphosis: their dependence on hatching period, and
274 s, quantified settlement success and size at metamorphosis, then outplanted juveniles to Tomales Bay,
275              For individuals who survived to metamorphosis, there was only a weak negative effect of
276              Although the tadpoles completed metamorphosis, they accumulated among the highest concen
277 ling of lineage tracts disappearing early in metamorphosis, they were unable extend the identificatio
278 ptor tyrosine kinase (RTK) Torso to initiate metamorphosis through the release of ecdysone.
279 ith an miR-2 mimic to allow nymphal-to-adult metamorphosis to proceed.
280                 We show that dac acts during metamorphosis to restrict sex comb development to the ap
281  transformations that occur during amphibian metamorphosis to show that PNNs can be highly dynamic in
282                    While bacteria-stimulated metamorphosis underpins processes such as the fouling of
283 y during summers influences survival through metamorphosis, using capture-mark-recapture data from Me
284 hat breeding took place, the average date of metamorphosis was 1 day earlier.
285            This severe delayed effect across metamorphosis was especially remarkable in high-latitude
286 iuron at 34 degrees C and an acceleration of metamorphosis was observed for the same group.
287 , critically required for insect molting and metamorphosis was selected as a potential target.
288  mechanisms by which bacteria promote animal metamorphosis, we begin to illustrate how, and explore w
289  larvae taken prior to observed mortality at metamorphosis, we found that exposure to OSS and exogeno
290 amining Drosophila tracheal outgrowth during metamorphosis, we show that progenitors follow a stereot
291 ding delayed development and reduced size at metamorphosis, were associated with elevated metalloid e
292   Purified Mif1 is sufficient for triggering metamorphosis when electroporated into tubeworm larvae.
293 ting that some secondary mechanism regulates metamorphosis when provisions are not completely consume
294        It forms as the tail regresses during metamorphosis, when locomotion changes from an axial-dri
295 ogrammed cell death occurs during Drosophila metamorphosis, when most of the larval tissues are destr
296 re highest in ammocoetes and decrease during metamorphosis while ca18 shows the opposite pattern with
297 iched by approximately 1 per thousand during metamorphosis, while delta(13)C used to estimate diet, w
298                                       During metamorphosis, Xenopus switches its locomotory mechanism
299  only during the larval stage but also after metamorphosis, yet notably only in low-latitude damselfl
300 ons subsequently decreased with the onset of metamorphosis, yet remained quite elevated.

 
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