ライフサイエンスコーパス: metastasis to bone

1 with a known role in promoting breast cancer metastasis to bone.

2 utant p53R280K to prevent breast cancer cell metastasis to bone.

3 urine models in the study of prostate cancer metastasis to bone.

4 (4T1/Red) to visualize tumor development and metastasis to bone.

5 the role of the IGF system in neuroblastoma metastasis to bone.

6 dramatically up-regulates their spontaneous metastasis to bone.

7 ism for both primary tumor proliferation and metastasis to bone.

8 hesion in the predilection of prostate tumor metastasis to bone.

9 ction but were not protected from tumor cell metastasis to bone.

10 wth factor (TGF)-beta promotes breast cancer metastasis to bone.

11 signaling mechanisms could contribute to PCa metastasis to bone.

12 As, reduced the tumor growth and spontaneous metastasis to bone.

13 the molecule is also required for effective metastasis to bone.

14 elevated PTHLH expression and organ-specific metastasis to bone.

15 Prostate cancer mortality results from metastasis to bone and hormone-independent tumor growth.

16 subpopulations with a higher propensity for metastasis to bone and the central nervous system.

17 o and colleagues show tumor exosomes enhance metastasis to bones and lungs, which is diminished by re

18 SA], prostatic acid phosphate) for suspected metastasis to bones and lymph nodes.

19 In a model of tumor metastasis to bone, bone tumor burden was decreased in t

20 and mortality associated with breast cancer metastasis to bone has motivated efforts to elucidate tu

21 ell migration and proliferation in vitro and metastasis to bone in vivo using a novel "human breast c

22 Metastasis to bone is a major cause of morbidity in brea

23 he molecular mechanism leading to the cancer metastasis to bone is poorly understood but yet determin

24 Metastasis to bone is the major clinical concern during

25 Metastasis to bones is determined by both intrinsic trai

26 Cancer pain, especially pain caused by metastasis to bone, is a severe type of pain, and unless

27 therapeutic strategies against breast cancer metastasis to bone it would worthwhile to keep in mind t

28 Metastasis to bone, liver and lungs is the primary cause

29 Metastasis to bone, lungs, liver, and brain involves dis

30 Early detection of tumor metastasis to bone marrow (BM) using conventional histoc

31 tumors, CXCL12-gamma significantly increased metastasis to bone marrow and other sites.

32 Prostate cancer metastasis to bone marrow involves initial adhesion of t

33 blockade immediately upon detection of tumor metastasis to bone may help preserve the integrity and u

34 ave association with patient outcomes, first metastasis to bone, nor with bone recurrence at any time

35 ME(2) inhibited tumor growth in soft tissue, metastasis to bone, osteolysis, and tumor growth in bone

36 ined whether 2ME(2) could effectively target metastasis to bone, osteolytic tumors, and soft tissue t

37 1.66, 95% CI = 0.982-2.8) and time to first metastasis to bone (p-value = 0.04, HR = 2.23, 95% CI =

38 or stefin A is a suppressor of breast cancer metastasis to bone that is coexpressed with cathepsin B

39 2 in a suitable mouse model of breast cancer metastasis to bone using the whole-body luciferase imagi

40 icate that RANKL promotes breast cancer cell metastasis to bone via its pro-migratory effect on breas

41 TGF-beta in the development of breast cancer metastasis to bone, via the TGF-beta receptor-mediated s

42 ociated with breast cancer cell invasion and metastasis to bone, we propose that the interaction with

43 e involvement of E-cadherin in breast cancer metastasis to bone, we used an in vivo model in which os

44 Tumor cell growth and metastasis to bone were monitored and bone volume was de

45 thotopically, PC3-mm2-shTNC clones decreased metastasis to bone, while C4-2b-TNC-overexpressing cells