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1  clonal sweep of stable subclones capable of metastasizing.
2  the epidermis and invades the dermis before metastasizing.
3 l cancer cells within a tumor are capable of metastasizing.
4 d to assess patient prognosis and to prevent metastasizing.
5 luence could reduce the chance of a melanoma metastasizing.
6 an dramatically accelerate tumorigenesis and metastasizing ability of prostate cancer cells.
7 expression was virtually diminished, and the metastasizing ability was lost after cells were stably t
8 d cells to acquire estrogen independence and metastasizing ability.
9 elial cell line, Rama 37, yields benign, non-metastasizing adenomatous tumours in syngeneic Furth-Wis
10 exus required for the survival and growth of metastasizing and circulating tumor cells in vivo.
11                                Boundaries of metastasizing and non-metastasizing primary tumors were
12 our screening study comparing mRNA levels in metastasizing and nonmetastasizing endometrial carcinoma
13 hether lymph node metastases were capable of metastasizing and therefore, if lymphadenectomy was stil
14 fferentiated disease into a more aggressive, metastasizing, and fatal malignancy.
15 (SCLC) is an aggressive, rapidly growing and metastasizing, and highly fatal neoplasm.
16 sed in invasive breast carcinomas capable of metastasizing, and it may be clinically useful in patien
17 arose several millennia ago and survives by "metastasizing" between hosts through cell transfer.
18            In a mouse model of spontaneously metastasizing breast cancer (MMTV-PyV mT), we found that
19 line with genomic DNA fragments from a human metastasizing breast cancer cell line yields cells which
20                                              Metastasizing cancer cells are able to withstand high le
21                                              Metastasizing cancer cells are uniquely vulnerable to im
22 uld not only increase genomic instability in metastasizing cancer cells but could also cause DNA dama
23 onmental cues in the regulation of miRNAs in metastasizing cancer cells has not been studied.
24                                      Whether metastasizing cancer cells reach lymph nodes via intratu
25 y, attention has been drawn to the fact that metastasizing cancer cells selectively and dynamically a
26 ese results suggest that host B7x may enable metastasizing cancer cells to escape local antitumor imm
27        The reduction in miR-193b enabled the metastasizing cancer cells to invade and proliferate int
28 ntoic membrane (CAM), and mRNA levels in the metastasizing cancer cells were evaluated by species-spe
29  suppresses tumor metastasis, likely because metastasizing cancer cells with high expression of SLC7A
30 ay lead to changes in gene expression of the metastasizing cancer cells, although such changes have n
31 nologies that capture metabolic processes in metastasizing cancer cells.
32 ick, adaptable switch to promote survival of metastasizing cancer cells.
33 and even less is known about the identity of metastasizing cancer cells.
34 a mimicry of skeletal cellular phenotypes by metastasizing cancer cells.
35  of lymphovascular invasion exhibited by all metastasizing cancers.
36 potential therapeutic target for lung-tropic metastasizing cancers.
37 onfer differences in metastatic potential as metastasizing cells depend on MCT1 to manage oxidative s
38       After separating from a primary tumor, metastasizing cells enter the circulatory system and int
39 votal step in cancer disease progression, as metastasizing cells often lose firm attachment to surrou
40 esent within tumors, how AXL is activated in metastasizing cells remains unclear.
41 but to later become a metabolic liability in metastasizing cells that is counteracted by decreased TI
42 ove cancer therapy, it is critical to target metastasizing cells.
43 ty acid synthesis (FAS), essential for brain-metastasizing cells; SCD1 knockout abolished the p53-dep
44  known to be elevated in patients with liver-metastasizing colon cancer; thus, cholecystokinin (CCK)
45 on and LOH of genes on chromosome 3 occur in metastasizing disomy 3 UM and are detectable by aSNP ana
46 f chromosome 3 were seen in the remaining 17 metastasizing disomy 3 UMs by MLPA.
47 etastasizing monosomy 3 uveal melanoma; iii) metastasizing disomy 3 uveal melanoma; and iv) monosomy
48 play a dominant role by invading further and metastasizing earlier.
49                                              Metastasizing EOCs engage in integrin-mediated adhesion
50 ring epithelial-to-mesenchymal transition of metastasizing epithelial tumors, we generated citrullina
51 f g-E and g-EAR delayed bioluminescence from metastasizing ES-2-luc cells for 2 and 3weeks, respectiv
52 tly, an in vivo model of human breast cancer metastasizing from the orthotopic site to bone does not
53 ancers, it inhibits primary lung tumors from metastasizing in a mouse model system.
54 in real time, fluorescent tumors growing and metastasizing in live mice.
55 tatic lesions to the oral mucosa; only cases metastasizing in the oral mucosa, gingiva, and periodont
56                                       Benign metastasizing leiomyoma (BML) is a rare condition descri
57 ction with the diagnosis of pulmonary benign metastasizing leiomyoma being stated.
58        We present a case of pulmonary benign metastasizing leiomyoma in a female patient admitted to
59                             Pulmonary benign metastasizing leiomyoma is a rare entity.
60 es confirmed the initial diagnosis of benign metastasizing leiomyoma with no evidence of neoplastic c
61 age peptides home may serve as receptors for metastasizing malignant cells.
62 T1 tumor, a highly aggressive, spontaneously metastasizing mammary carcinoma, followed by surgical re
63 l targets for the diagnosis and treatment of metastasizing MB in humans.
64  experienced in the blood limits survival of metastasizing melanoma cells, SELENOO might be able to a
65        Among the 71 patients who had died of metastasizing melanoma, there was no difference in time
66          Monosomy 3 was detected in 47 of 71 metastasizing melanomas (66.1%) and was significantly as
67                                 Successfully metastasizing melanomas underwent reversible metabolic c
68 ndling between efficiently and inefficiently metastasizing melanomas, with circulating lactate being
69 plantation, particularly among inefficiently metastasizing melanomas.
70  uveal melanoma with long-term survival; ii) metastasizing monosomy 3 uveal melanoma; iii) metastasiz
71 gene expression signature for distinguishing metastasizing (N+) from nonmetastasizing (N0) squamous c
72 ioleiomyomatosis (LAM) is a rare, low-grade, metastasizing neoplasm that arises from an unknown sourc
73 young women and is classified as a low-grade metastasizing neoplasm.
74 the view of LAM as a low-grade, destructive, metastasizing neoplasm.
75 y as those caused by low-grade or high-grade metastasizing neoplasms, polyclonal or monoclonal lympho
76 Because of the intimate relationship between metastasizing neoplastic cells and vascular as well as e
77 on of a tumor suppressor miRNA, miR-193b, in metastasizing OvCa cells.
78          Boundaries of metastasizing and non-metastasizing primary tumors were readily defined, leadi
79 ically promotes the invasive ability of bone-metastasizing prostate (and breast) cancer cells but not
80 type are indistinguishable from those of the metastasizing prostate tumors but distinctly different f
81 nesis in vivo remains poor, in particular in metastasizing solid cancers.
82 eatment of hematologic malignancies and bone-metastasizing solid tumors.
83                                       In the metastasizing state, the course of the disease is often
84                    Higher levels of LRV-1 in metastasizing strains of L.V. guyanensis have been docum
85                      Moreover, we found that metastasizing subclones preferentially originate from th
86  array of cell membrane molecules on a liver-metastasizing subline of B16 melanoma versus the parenta
87 gionally through the lymphatic system before metastasizing systemically through the blood(1-4); howev
88                     Prostate carcinoma cells metastasizing to bone must initially adhere to endotheli
89 or osteolytic lesions caused by cancer cells metastasizing to bone.
90                  The lowest T category tumor metastasizing to both regional nodes and distant locatio
91 m the ECM are capable of evading anoikis and metastasizing to different distant organs.
92 fest distinct gene expression patterns after metastasizing to different organs.
93 t melanoma is highly invasive and capable of metastasizing to distant sites where it is typically res
94 in syngeneic C57Bl/6 mice, which form tumors metastasizing to draining lymph nodes and subsequently t
95 was inhibited, and the number of tumor cells metastasizing to lung was reduced significantly by combi
96 ly fluorescent pancreatic tumors growing and metastasizing to multiple sites in live mice.
97 c lesions to the gingiva compared with cases metastasizing to other oral mucosal sites.
98                    Breast cancers capable of metastasizing to the brain showed evidence of altered li
99 molecular basis of breast cancers capable of metastasizing to the brain-a principal cause of death in
100                                       Tumors metastasizing to the CNS and leptomeninges (LM) are asso
101 inal tract origin, have a predisposition for metastasizing to the liver.
102 nearly 30% of the PDX models were capable of metastasizing to the lungs.
103  the Fallopian tube's epithelium (TE) before metastasizing to the ovary.
104  This mechanotransduced pathway may regulate metastasizing tumor cell adhesion.
105         A mutual relationship exists between metastasizing tumor cells and components of the coagulat
106  contribute to the close association between metastasizing tumor cells and peri-tumor lymphatic densi
107            Importantly, the omentum collects metastasizing tumor cells and supports tumor growth by i
108          Reduced recruitment of monocytes to metastasizing tumor cells in Fuc-TVII(-/-) mice correlat
109        Thus, the recruitment of monocytes to metastasizing tumor cells is facilitated by endogenous s
110                                              Metastasizing tumor cells migrate through the surroundin
111                                              Metastasizing tumor cells need to reprogram their metabo
112                                              Metastasizing tumor cells predominantly utilize mitochon
113 rise from a multi-step process that requires metastasizing tumor cells to adapt to signaling input fr
114 , may enable a molecular mimicry that allows metastasizing tumor cells to downregulate MHC class I, t
115                                           As metastasizing tumor cells undergo epithelial to mesenchy
116                                              Metastasizing tumor cells use matrix metalloproteases, s
117 The recruitment of inflammatory monocytes to metastasizing tumor cells was dependent on the local end
118 s critically dependent on the interaction of metastasizing tumor cells with cells in the local microe
119      PMo established early interactions with metastasizing tumor cells, scavenged tumor material from
120 reast) cancer cells but not that of non-bone-metastasizing tumor cells.
121 ns regulates migratory and invasive nexus of metastasizing tumor cells.
122 veloping insect embryos, skeletal muscle and metastasizing tumor cells.
123 n vivo cancer cell tracking in spontaneously metastasizing tumor models still poses a challenge requi
124 arcinoma differs from that of hematogenously metastasizing tumors because ovarian cancer cells primar
125                           A vast majority of metastasizing tumors have mutations in the BAP1 gene.
126 ne confers the ability to form spontaneously metastasizing tumors in ovariectomized nude mice without
127                 Median pO2 was 7.5 mm Hg for metastasizing tumors versus 20 mm Hg for nonmetastasizin
128 BAP1) on chromosome 3p21.1 in 26 of 31 (84%) metastasizing tumors, including 15 mutations causing pre
129 emalignant lesions and lower TIGAR levels in metastasizing tumors.
130 tasis, which is hardly found in aggressively metastasizing tumors.
131 ogically normal prostate tissues surrounding metastasizing tumors.
132 iated macrophages and their interaction with metastasizing tumour cells, is dependent on CCL2 synthes
133  and metabolic adaptation of established and metastasizing tumours.
134  16 genes showed LOH exclusively in disomy 3 metastasizing UM, suggesting a potential role for these
135 ozen tissue samples from four fatal disomy 3 metastasizing UMs and three disomy 3 tumors with >5 year
136                                          Two metastasizing UMs that had been classified as disomy 3 b
137 gosity (LOH) on chromosome 3 in the disomy 3 metastasizing UMs that were not deleted or showing LOH i
138 (FFPE) whole tumor sections from 19 disomy 3 metastasizing UMs.
139 ng finding of our study was that even before metastasizing, VEGF-A-overexpressing primary tumors indu
140 atment of ovarian cancer, a tumour known for metastasizing within the peritoneal cavity.

 
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