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1 N-gamma into a mouse model of an established metastatic brain tumor.
2 iously irradiated patients with recurrent or metastatic brain tumors.
3 ntially enhance the management of primary or metastatic brain tumors.
4 hift in the use of natural product drugs for metastatic brain tumors.
5 uld be based on the histologic origin of the metastatic brain tumor and not on the lipophilicity of t
6 immune cellular landscape of ICB's effect on metastatic brain tumors and offers insights into potenti
7 s, which are rarely used in the treatment of metastatic brain tumors because they are thought to not
8 ly used, effective treatment for primary and metastatic brain tumors, but it also produces radiation-
9 eased tissue concentrations of paclitaxel in metastatic brain tumors compared with gliomas, support t
12 Xenograft mouse models of glioblastoma and metastatic brain tumors (from lung and breast cancer) we
15 guidelines for the treatment of adults with metastatic brain tumors, including systemic therapy and
16 enfluridol treatment inhibited the growth of metastatic brain tumors introduced by intracardiac or in
17 er, P-gp expression in the neovasculature of metastatic brain tumors is similar to the P-gp expressio
18 in developing new treatments for primary and metastatic brain tumors is the requirement for crossing
19 imary brain tumors (mouse glioblastoma), and metastatic brain tumors (mouse breast cancer metastasis)
20 fteen patients with progressive or recurrent metastatic brain tumors that failed to respond to surger
22 rd of care for the chemotherapy treatment of metastatic brain tumors, which has been generally limite