ライフサイエンスコーパス: methamphetamine

1 of dopamine neurotransmission in response to methamphetamine.

2 administration of MDMA, its enantiomers, and methamphetamine.

3 after starting PrEP; 5 reported injection of methamphetamine.

4 a genetically-associated thermal response to methamphetamine.

5 male/female rhesus monkeys self-administered methamphetamine (0.03 mg/kg/injection, i.v.) under a fix

6 male Sprague-Dawley rats to self-administer methamphetamine (0.1 mg/kg/infusion) for 9 hours daily f

7 pellets for 6 days (6 h per day) for either methamphetamine (0.1 mg/kg/infusion) or heroin (0.1 mg/k

8 ster palatable food pellets (6 d, 6 h/d) and methamphetamine (12 d, 6 h/d).

9 lanted radiotelemetry, following exposure to methamphetamine (12.5 and 100 mg/ml), MDPV (25, 50, and

10 procedure between the palatable solution and methamphetamine (14 days).

11 drocodone) and five stimulants (amphetamine, methamphetamine, 3,4-methylenedioxymethamphetamine (MDMA

12 e 1% + maltodextrin 1%, 6 h/day, 6 days) and methamphetamine (6 h/day, 12 days).

13 ecently injected heroin (81%) and/or crystal methamphetamine (71%).

14 Methamphetamine, a potent psychostimulant, is a highly a

15 s highly dysregulated by the psychostimulant methamphetamine, a substrate for the dopamine transporte

16 cal profiles in animal models of cocaine and methamphetamine abuse.

17 neural responses to drug cues in humans with methamphetamine addiction and suggests that naltrexone m

18 Methamphetamine addiction is mimicked in rats that self-

19 c, a key neurochemical event contributing to methamphetamine addiction liability.

20 1R as a putative target for the treatment of methamphetamine addiction.

21 ibution of Taar1 and Oprm1 variants to human methamphetamine addiction.

22 ct has been shown for alcohol, nicotine, and methamphetamine addictions, but not for heroin or cocain

23 gy may be helpful for recovering cocaine and methamphetamine addicts.

24 djusted OR = 1.94; 95% CI = 1.21 to 3.11) or methamphetamine (adjusted OR = 5.40; 95% CI = 1.92 to 15

25 teins that rapidly decreased at 30 min after methamphetamine administration in H1(+/-) mice.

26 or been offered opioids, cocaine, cannabis, methamphetamine, alcohol, or tobacco.

27 Methamphetamine also interacts with the sigma-1 receptor

28 articipants, and rats chronically exposed to methamphetamine, an elicitor of paranoia in humans.

29 porter protein, dopamine uptake, or striatal methamphetamine and amphetamine metabolite levels.

30 creased mGlu1 function, in the incubation of methamphetamine and cocaine craving.

31 showed, for drugs with weekly cycles (MDMA, methamphetamine and cocaine in this sample), sampling st

32 in midbrain D2/D3 BPnd was detected between methamphetamine and control groups, midbrain D2/D3 BPnd

33 for neuromuscular blockers, drugs of abuse (methamphetamine and fentanyl), anesthetics, neurotoxins,

34 e (using a discrete choice procedure between methamphetamine and food; 20 trials/d) for 19 d.

35 UCP1 knockout (UCP1KO) mice exhibit blunted methamphetamine and fully inhibited noradrenaline thermo

36 transmitted infections, and higher rates of methamphetamine and gamma hydroxybutyrate use when compa

37 sing ToF-SIMS and DESI-MS, as the signals of methamphetamine and heroin were completely suppressed by

38 eatment of human primary neurons (HPNs) with methamphetamine and HIV gp120 and Tat increase dynamin-r

39 Methamphetamine and HIV proteins increased microtubule-a

40 Methamphetamine and methadone e-liquids were prepared at

41 little is known about the effect of comorbid methamphetamine and tobacco use on human fetal brain dev

42 ific genes, including Azi2 in sensitivity to methamphetamine and Zmynd11 in anxiety-like behavior.

43 f the pharmaceuticals morphine, codeine, and methamphetamine and, for context, compared it with nitra

44 rmalities reported in children with prenatal methamphetamine and/or tobacco exposure are present at b

45 Collectively, prenatal methamphetamine and/or tobacco exposure may lead to dela

46 Exposures: Prenatal methamphetamine and/or tobacco exposure.

47 Methamphetamine and/or tobacco use by pregnant women rem

48 UCP3 (UCP3KO) have impaired sympathomimetic (methamphetamine) and completely abrogated lipopolysaccha

49 choose between an addictive drug (heroin or methamphetamine) and social interaction.

50 rsor: stimulant drugs, such as ephedrine and methamphetamine, and certain antidepressants.

51 rated from inkjet-printed arrays of cocaine, methamphetamine, and heroin with a deposited-mass rangin

52 hdrawal from self-administration of cocaine, methamphetamine, and other drugs of abuse, a phenomenon

53 hold values for the detection of cocaine and methamphetamine as proof-of-principle with 0.25 and 0.75

54 proach to ameliorate the effects of HIV- and methamphetamine-associated cognitive deficits.

55 reactivity, and attentional bias toward the methamphetamine-associated cue, compared with the placeb

56 the clinical characteristics and outcomes of methamphetamine-associated ICH (Meth-ICH) versus Non-Met

57 Methamphetamine-associated psychosis (MAP) involves wide

58 sed methamphetamine choice, whereas removing methamphetamine availability decreased methamphetamine c

59 rescued, converting them to levels found in methamphetamine-avoiding animals.

60 T results for cocaine, fentanyl, heroin, and methamphetamine before vs after US declaration of corona

61 the reinforcing effects of both cocaine and methamphetamine but did not exhibit psychostimulant beha

62 urthermore, NAc core MeCP2 knockdown reduces methamphetamine, but not saccharin, SA on a progressive

63 d activity-based sleep measures disrupted by methamphetamine by decreasing sleep latency and increasi

64 ysregulated in HIV infection, and the use of methamphetamine can further negatively affect these prot

65 e, whereas bupropion treatment did not alter methamphetamine choice up to doses that decreased rates

66 Methamphetamine choice was not significantly altered whe

67 Risperidone treatment increased methamphetamine choice, whereas bupropion treatment did

68 Removing food availability increased methamphetamine choice, whereas removing methamphetamine

69 oving methamphetamine availability decreased methamphetamine choice.

70 analysis was performed for four drugs (MDMA, methamphetamine, cocaine, methadone) collected through a

71 oids, benzodiazepines, fentanyl derivatives, methamphetamines, cocaine, substituted methylenedioxyphe

72 Thus, we show that methamphetamine combined with HIV proteins inhibits mito

73 cur much more rapidly during withdrawal from methamphetamine compared with cocaine.

74 in mice selectively bred for high versus low methamphetamine consumption.

75 ently developed a rat model of incubation of methamphetamine craving after choice-based voluntary abs

76 introduced an animal model of incubation of methamphetamine craving after choice-based voluntary abs

77 osteric modulator, AZD8529, on incubation of methamphetamine craving after forced or voluntary abstin

78 ine D1 and D2 receptors in the incubation of methamphetamine craving after voluntary abstinence and t

79 Our results show that incubation of methamphetamine craving after voluntary abstinence gener

80 in NAc core is critical to the incubation of methamphetamine craving after voluntary abstinence.

81 rate a role for the NAc in the incubation of methamphetamine craving and describe adaptations in syna

82 voluntary abstinence prevents incubation of methamphetamine craving in rats.

83 Incubation of methamphetamine craving was associated with CP-AMPAR acc

84 relationship between midbrain D2/D3 BPnd and methamphetamine craving was not detected.

85 ly increases after withdrawal (incubation of methamphetamine craving), but the underlying mechanisms

86 d abstinence than after 1 day (incubation of methamphetamine craving).

87 ary abstinence than after 1 d (incubation of methamphetamine craving).

88 translation was elevated after incubation of methamphetamine craving, as recently found for cocaine.

89 wal interval when animals show incubation of methamphetamine craving.

90 ced seeking tests demonstrated incubation of methamphetamine craving.

91 similar plasticity accompanies incubation of methamphetamine craving.

92 voluntary abstinence prevents incubation of methamphetamine craving.

93 Reducing frequency of illicit opioid or methamphetamine/crystal use without abstinence was assoc

94 Reducing frequency of illicit opioid or methamphetamine/crystal use without abstinence was assoc

95 oad (VL) and reduced use of illicit opioids, methamphetamine/crystal, cocaine/crack, and marijuana, r

96 addition, reducing use of illicit opioids or methamphetamine/crystal, even without abstinence, was al

97 within-subject design and underwent a visual methamphetamine cue-reactivity task during two blood-oxy

98 tic resonance imaging (fMRI) measures during methamphetamine cue-reactivity to elucidate the role of

99 y HIV serostatus (HIV+ or HIV-) and lifetime methamphetamine dependence (METH+ or METH-).

100 lt men and women who met DSM-IV criteria for methamphetamine dependence and were enrolled in a reside

101 The extent to which these features of methamphetamine dependence are interrelated, however, is

102 itochondrial function.SIGNIFICANCE STATEMENT Methamphetamine dependence is a significant public healt

103 s from patients diagnosed with MAP (N = 12), methamphetamine dependence without psychosis (MA; N = 14

104 In Study 2, an independent sample of 20 methamphetamine-dependent and 18 control subjects comple

105 In Study 1, 19 methamphetamine-dependent and 26 healthy control subject

106 Here, we show that methamphetamine-dependent attenuation of GABABR-GIRK cur

107 The methamphetamine-dependent decrease in GABABR-GIRK curren

108 the GIRK3 subunit appeared critical for the methamphetamine-dependent decrease of GABABR-GIRK curren

109 nk that may help explain high impulsivity in methamphetamine-dependent individuals.

110 striatum, orbitofrontal cortex and insula in methamphetamine-dependent participants, but a positive r

111 In methamphetamine-dependent research participants, impulsi

112 In this study, 21 methamphetamine-dependent subjects and 23 healthy contro

113 Methamphetamine-dependent users exhibited a positive rel

114 trong preference for the palatable food over methamphetamine during the choice-based voluntary abstin

115 in imaging studies in children with prenatal methamphetamine exposure (PME) suggest structural and fu

116 Methamphetamine exposure in rats recapitulates this impa

117 For cocaine and methamphetamine, expression of incubated craving ultimat

118 We trained rats to self-administer methamphetamine for 20 days.

119 re, we show that stimulants like cocaine and methamphetamine greatly increase HS content and sulfatio

120 eceptor availability (BPnd) was lower in the methamphetamine group, as shown previously, the groups d

121 l mean cortical gray-matter thickness in the methamphetamine group, but no association was found betw

122 In the methamphetamine group, mean cortical gray-matter thickne

123 social behaviors and vocalizations, whereas methamphetamine had only modest effects on affiliative b

124 ith the nation's focus on the opioid crisis, methamphetamine has made a comeback, potentially increas

125 powdered mixtures of illicit drugs (cocaine, methamphetamine, heroin, fentanyl, and its analogues), a

126 the more methamphetamine users chose to view methamphetamine images, specifically vs pleasant images

127 xposure among people who reported ever using methamphetamine in a nationally representative survey.

128 Through interactions with DAT, methamphetamine increases extracellular dopamine levels

129 neurons, the combination of HIV proteins and methamphetamine increases oxidative stress, DRP1-mediate

130 mate receptor adaptations in NAc core during methamphetamine incubation.

131 us, both receptor subtypes may contribute to methamphetamine-induced alterations in cortical morpholo

132 at or near the plasma membrane and decreases methamphetamine-induced Ca(2+) signaling, providing pote

133 dynamic mitochondrial function that informs methamphetamine-induced cellular adaptations associated

134 ng increased synaptic hnRNP H with decreased methamphetamine-induced dopamine release and behaviors,

135 re, H1(+/-) mice showed a robust decrease in methamphetamine-induced dopamine release in the NAc with

136 Hnrnph1 mutation also blunted methamphetamine-induced dopamine release in the NAc, a k

137 We conclude that H1(+/-) decreases methamphetamine-induced dopamine release, reward, and re

138 amine-induced impulsivity, without affecting methamphetamine-induced hyperactivity.

139 lacking both UCP1 and 3 (UCPDK) fail to show methamphetamine-induced hyperthermia, and have a markedl

140 rum-prefrontal cortex pathway also regulates methamphetamine-induced impulsivity, suggesting a critic

141 inhibition of the CLA-PFC pathway prevented methamphetamine-induced impulsivity, without affecting m

142 to methamphetamine, whereas WT mice showed a methamphetamine-induced increase in synaptosomal mitocho

143 these findings, sigma1R activation decreases methamphetamine-induced locomotion, motivated behavior,

144 rnph1, Rufy1) that was necessary for reduced methamphetamine-induced locomotor activity in C57BL/6J c

145 quantitative trait gene underlying decreased methamphetamine-induced locomotor activity in mice.

146 anti-MA immune response in mice, decreasing methamphetamine-induced locomotor activity.

147 Exercise attenuates methamphetamine-induced neurochemical damage in the rat

148 The role and mechanisms for TAAR1 in methamphetamine-induced neurotoxicity are not known.

149 wn that Hnrnph1 mutants also exhibit reduced methamphetamine-induced reward, reinforcement, and dopam

150 ent and induces dynamic changes in basal and methamphetamine-induced synaptic mitochondrial function.

151 We found that sigma1R activation prevents methamphetamine-induced, DAT-mediated increases in firin

152 ss for social interaction (6 d) and then for methamphetamine infusions (12 d).

153 into CeL and trained them to lever press for methamphetamine infusions (12 d).

154 parison, effects of removing food pellets or methamphetamine injections and FR response requirement m

155 SPR-Cas9 to test the causal role of Taar1 in methamphetamine intake and a genetically-associated ther

156 Both methamphetamine intake and the thermal response mapped t

157 accounts for 60% of the genetic variance in methamphetamine intake in mice selectively bred for high

158 Importance: Methamphetamine is a common illicit drug used worldwide.

159 Released from neurons when methamphetamine is self-administered, dopamine binds to

160 The high prevalence and severity of methamphetamine (MA) abuse demands greater neurobiologic

161 Methamphetamine (MA) addiction is a serious public healt

162 The psychostimulants amphetamine (AMPH) and methamphetamine (MA) are widely abused illicit drugs.

163 Methamphetamine (MA) is highly addictive and neurotoxic,

164 Methamphetamine (MA) use disorder is a serious psychiatr

165 Methamphetamine (MA) use is a major public health proble

166 Continued methamphetamine (MA) use is dependent on a positive MA e

167 Methamphetamine (MA) users are assumed to have a high bu

168 , male Wistar rats were exposed to vaporized methamphetamine (MA), 3,4-methylenedioxypyrovalerone (MD

169 MA, compared with the prototypical stimulant methamphetamine (MA), on two measures of social behavior

170 d by chronic exposure to the psychostimulant methamphetamine (MA), the current study sought to use co

171 the serotonergic enhancer 3,4-methylenedioxy-methamphetamine (MDMA) modulates behavior and its neural

172 analysis of the effect of 3,4-methylenedioxy-methamphetamine (MDMA) on cooperative behavior during in

173 affinities to cannabinol, 3,4-methylenedioxy methamphetamine (MDMA/Ecstasy) and cocaine hydrochloride

174 HIV-1 infection and methamphetamine (METH) abuse frequently occur simultaneo

175 Methamphetamine (Meth) abuse has reached epidemic propor

176 Methamphetamine (METH) abuse is a serious social and hea

177 to examine oxytocin as a pharmacotherapy for methamphetamine (meth) addiction.

178 hypothesized that Glycoprotein 120 (gp120), methamphetamine (METH) and nicotine (NT) can enhance amy

179 rhythmically pairing 2 reinforcing stimuli [methamphetamine (Meth) and running wheel (RW)] restores

180 Methamphetamine (METH) competes with dopamine uptake, in

181 electively disrupts memories associated with methamphetamine (METH) days after learning, without retr

182 Methamphetamine (Meth) exacerbates HIV-1 pathobiology by

183 Prevalence of methamphetamine (meth) injection and associated human im

184 to segregate rats that reduce or stop their methamphetamine (METH) intake (nonaddicted) from those t

185 gonist can reduce high caloric diet (HCD) or methamphetamine (METH) intake and response perseveration

186 (nor-BNI) would attenuate the escalation of methamphetamine (METH) intake in an extended-access self

187 Methamphetamine (METH) is a drug with a high addictive p

188 Methamphetamine (METH) is a potent stimulant that induce

189 Methamphetamine (METH) is a powerfully addictive psychos

190 Methamphetamine (Meth) is a psychomotor stimulant strong

191 Methamphetamine (METH) is a substrate for the dopamine t

192 Recent trends in methamphetamine (METH) misuse and overdose suggest socie

193 Rats that have self-administered methamphetamine (meth) under long access, but not short

194 The prevalence of methamphetamine (METH) use is estimated at ~35 million p

195 ing that storage of memories associated with methamphetamine (METH), but not memories for fear or foo

196 as previously demonstrated in male rats that methamphetamine (Meth), when administered concurrently w

197 clades to exert differential effects and the methamphetamine (METH)-associated dopaminergic dysfuncti

198 ve therapy is available for the treatment of methamphetamine (METH)-induced neurotoxicity, aerobic ex

199 use in rats, the hypothesis was tested that methamphetamine (Meth)-taking concurrently with socio-se

200 nds such as cocaine, amphetamine (AMPH), and methamphetamine (METH).

201 nt relapse to the use of stimulants, such as methamphetamine (METH).

202 under normal conditions to those exposed to methamphetamine (Meth).

203 ntribute to relapse to cocaine, amphetamine, methamphetamine, morphine, heroin, nicotine, or alcohol

204 ine, cannabidiol, cocaine, codeine, heroine, methamphetamine, morphine, phentermine, L-phenylepherine

205 ochondrial dysfunction is a key component in methamphetamine neuropathology.

206 es to other abused drugs, including cocaine, methamphetamine, nicotine, and alcohol, and is also obse

207 cal intervention to attenuate the effects of methamphetamine on nighttime activity under well-control

208 ted; many PWID concurrently used opioids and methamphetamine or cocaine.

209 sses before and after self-administration of methamphetamine or saline.

210 ified as injection drug use (IDU) (opioid or methamphetamine) or non-IDU (opioid, methamphetamine, or

211 five substances: alcohol, nicotine, cocaine, methamphetamine, or cannabis.

212 ioid or methamphetamine) or non-IDU (opioid, methamphetamine, or cocaine).

213 mine, the psychostimulants d-amphetamine and methamphetamine, or to cocaine and cocaine analogues.

214 gm in which they received a moderate dose of methamphetamine paired with one stimulus and placebo wit

215 injection of opioids (including fentanyl) or methamphetamine predominated; many PWID concurrently use

216 ing exon of Hnrnph1 (H1(+/-)) showed reduced methamphetamine reinforcement and intake and dose-depend

217 nence blocked compulsive-like context-driven methamphetamine reinstatement.

218 ls, methamphetamine users chose to view more methamphetamine-related images on one task, with a simil

219 The methamphetamine-related traits were rescued, converting

220 nd slowest-reacting pollutants (morphine and methamphetamine, respectively) were always transport- an

221 role for the RNA binding protein hnRNP H in methamphetamine reward and reinforcement.

222 ent and intake and dose-dependent changes in methamphetamine reward as measured via conditioned place

223 tion, we show new evidence that both ELS and methamphetamine SA alter the expression of the epigeneti

224 genome-wide transcriptional consequences of methamphetamine SA and footshocks in the rat brain.

225 core MeCP2 may be recruited by both ELS and methamphetamine SA and promote the development of certai

226 gether, functional interactions between ELS, methamphetamine SA, and the expression of MeCP2 in the N

227 of MeCP2 expression in the NAc core reduces methamphetamine SA, as well as saccharin intake.

228 We then assessed relapse to methamphetamine seeking after 1 and 15 abstinence days.

229 We then evaluated relapse to methamphetamine seeking after 1 and 15 days of voluntary

230 upenthixol, SCH39166, and raclopride reduced methamphetamine seeking after 15 days of abstinence.

231 SCH39166 or raclopride selectively decreased methamphetamine seeking after 21 abstinence days.

232 s induction, decreased incubated cue-induced methamphetamine seeking after prolonged withdrawal.

233 Incubated methamphetamine seeking after voluntary abstinence was a

234 In both sexes, methamphetamine seeking in the relapse tests was higher

235 AZD8529 decreased cue-induced methamphetamine seeking on day 21 but not day 1 of force

236 DMS on abstinence day 18 decreased incubated methamphetamine seeking on day 21.

237 Cue-induced methamphetamine seeking progressively increases after wi

238 We then assessed relapse to methamphetamine seeking under extinction conditions afte

239 Methamphetamine seeking was higher after 21 d of volunta

240 s, and DMS neuronal ensembles in "incubated" methamphetamine seeking, using selective dopamine recept

241 ocaine, and alcohol seeking, but not yet for methamphetamine seeking.

242 olonged maternal separation, increases adult methamphetamine self-administration (SA) in male rats as

243 bred Wistar rats experienced extended access methamphetamine self-administration and individual diffe

244 In the present paper, we used a model of methamphetamine self-administration during which we used

245 tigraphy-based sleep parameters disrupted by methamphetamine self-administration in non-human primate

246 We found no sex differences in methamphetamine self-administration or in the strong pre

247 Subsequently, methamphetamine self-administration rats were punished b

248 y comorbid and we have previously shown that methamphetamine self-administration significantly disrup

249 longed voluntary abstinence from intravenous methamphetamine self-administration, to demonstrate that

250 ased learning were reduced in rats following methamphetamine self-administration, which was due to a

251 g prior to any drug use predicted subsequent methamphetamine self-administration; rats with lower mod

252 nimals that continue to press a lever to get methamphetamine (shock-resistant) and those that signifi

253 s revealed that sigma1R activation decreases methamphetamine-stimulated dopamine efflux without affec

254 Methamphetamine stimulates TAAR1, a G protein-coupled re

255 , reversed the neurotoxic effects of HIV and methamphetamine, suggesting a novel approach to ameliora

256 ures of marijuana, cocaine, heroin, ecstasy, methamphetamines, synthetic marijuana, and prescription

257 play a role in the regulation of compulsive methamphetamine taking by rats.

258 -contingent punishment significantly reduced methamphetamine taking in some rats (shock-sensitive, SS

259 ats with lower model-free behavior took more methamphetamine than rats with higher model-free behavio

260 of 195 qualified neonates were evaluated (36 methamphetamine/tobacco exposed, 32 tobacco exposed, and

261 Conclusions and Relevance: Prenatal methamphetamine/tobacco exposure may lead to delays in m

262 Only methamphetamine/tobacco- and tobacco-exposed girls showe

263 ly less coherent ACR fibers were observed in methamphetamine/tobacco- and tobacco-exposed girls, poss

264 Only methamphetamine/tobacco-exposed boys had lower FA (group

265 Methamphetamine/tobacco-exposed infants showed delayed d

266 Rats self-administered saline or methamphetamine under extended-access conditions.

267 ood (6 sessions) and then to self-administer methamphetamine under two conditions: 12 sessions (9 hou

268 sex partners (11 vs. 8; P < 0.001) and more methamphetamine use (15% vs. 8%; P < 0.001) than men who

269 -1 acquisition included MSM status (79%) and methamphetamine use (21%).

270 ss was negatively associated with cumulative methamphetamine use and craving for the drug.

271 tion, non-treatment seeking individuals with methamphetamine use disorder (N=23; 74% male, mean age=3

272 Methamphetamine use disorder is associated with striatal

273 to an inpatient behavioral intervention for methamphetamine use disorder reverses deficits in striat

274 risk those who have opioid use disorder and methamphetamine use disorder.

275 Methamphetamine use has emerged as a risk factor for int

276 As methamphetamine use increases, it is critical to identif

277 Chronic methamphetamine use poses potentially devastating conseq

278 A 2-fold increase of methamphetamine use was detected, associated with a chea

279 Methamphetamine use was not an independent predictor of

280 In multivariate analysis, methamphetamine use, unstable housing, current/recent sk

281 In multivariate analysis, methamphetamine use, unstable housing, current/recent sk

282 receptors in cortical adaptation to chronic methamphetamine use.

283 ), men who have sex with men (P < .001), and methamphetamine users (P < .001).

284 1), men who have sex with men (p<0.001), and methamphetamine users (p<0.001).

285 Thirteen of the methamphetamine users and 10 controls also underwent [(1

286 Fifteen recently abstinent methamphetamine users and 15 healthy controls completed

287 availability has been observed in striata of methamphetamine users as compared with controls, but an

288 nalyses showed that, on both tasks, the more methamphetamine users chose to view methamphetamine imag

289 nalyses showed that, compared with controls, methamphetamine users chose to view more methamphetamine

290 post-mortem material, with no differences in methamphetamine users from controls in the caudate nucle

291 eliorate striatal D2/D3 receptor deficits in methamphetamine users, and warrants further evaluation a

292 , insula, hippocampus and temporal cortex in methamphetamine users, but not in control participants (

293 e among participants who reported ever using methamphetamine using bivariate and multivariable logist

294 about the neuropharmacological mechanisms of methamphetamine vs food choice.

295 Here, for methamphetamine, we observed no significant change in su

296 e for female participants who reported using methamphetamine were 4 times than that for male particip

297 Morphine, cocaine, and methamphetamine were chosen as test analytes for this st

298 Low levels of methamphetamine were detected in 41% of the samples.

299 Thereafter, lever-presses for methamphetamine were punished by mild footshocks for 5 d

300 drial proteins that decreased in response to methamphetamine, whereas WT mice showed a methamphetamin

301 coupling to Homer scaffolding proteins after methamphetamine withdrawal, nor did elevation of mGlu1 t