コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 nd the activity required Ti(III) citrate and methyl viologen.
2 as 2,6-dichloro-1,4-benzoquinone (DCBQ) and methyl viologen.
3 is facilitated by the general redox mediator methyl viologen.
4 was evidenced from the electron transfer to methyl viologen.
5 d caffeyl-CoA-dependent oxidation of reduced methyl viologen.
6 persensitive to the oxidative stress reagent methyl viologen.
7 stress sensitivity of this E. coli mutant to methyl viologen.
8 achieved by using a surfactant derivative of methyl viologen.
9 xtreme sensitivity to the redox-active agent methyl viologen.
10 reduction of BSO by either NADPH or reduced methyl viologen.
11 ot electron transfer with electron-accepting methyl viologens.
12 sured decay factors for electron transfer to methyl viologen (0.18 angstrom(-1)) and hole transfer to
13 cetonitrile electrolyte were quantified with methyl viologen [1,1'-(CH(3))(2)-4,4'-bipyridinium](PF(6
14 as photocathodes in contact with an aqueous methyl viologen(2+/+) electrolyte, energy-conversion eff
15 en the enzyme was assayed in the presence of methyl viologen (4 mM) and inhibited by 70% when the enz
16 tiated electron transfer to generate reduced methyl viologen, a species that persists in the presence
17 d reductive electron transfer reactions with methyl viologen and 10-methylphenothiazine occur with ne
18 ferent redox behavior was also observed when methyl viologen and benzyl viologen were used as reducta
19 nd Km values of 29 and 15 microM for reduced methyl viologen and biotin sulfoxide reductase, respecti
20 affinity for the heteroternary complex with methyl viologen and CB[8] (MVCB[8]) within a vast pool o
21 uced Fe(II) cores (Fe(II)-HoSF), prepared by methyl viologen and CO, also reduces M-HoSF and M-AvBF s
22 t all mutants were completely devoid of both methyl viologen and formate-linked thiosulfate reductase
24 type, fib4 KD apples were more sensitive to methyl viologen and had higher superoxide levels during
26 e fraction of chloroplasts were increased by methyl viologen and ozone, but not by high-light treatme
27 lementation line C1 rescued the tolerance to methyl viologen and salinity and recovered the growth an
30 ayed a soxS-dependent induction by paraquat (methyl viologen), and the fldB gene is preceded by two o
32 e kinetic properties of the (de)insertion of methyl viologen are excellent with 60% of capacity retai
33 Upon partial reduction with dithionite using methyl viologen as a mediator, a signal at g(ave) = 1.9
34 , in a proof-of-principle investigation with methyl viologen as electron acceptor, the population of
35 alyzed the reduction of NADP(+) with reduced methyl viologen as electron donor at a rate of 385 U/mg.
36 uction with Ti(III)-citrate as reductant and methyl viologen as mediator were similar to those obtain
40 urthermore, a newly designed high-throughput methyl viologen-based photometric microtiter plate assay
41 e binding constants K(G2) of cucurbit[8]uril-methyl viologen-based ternary complexes (CB8-MV(2+)-G2)
42 e herbicides fosmidomycin, phosphonothrixin, methyl viologen, benzyl viologen, clomazone, 2-(dimethyl
43 ngsten-grown cells yielded decreased reduced methyl viologen:BSO reductase, NADPH:BSO reductase, and
44 rosative stress and the superoxide generator methyl viologen but remarkably resistant to hydrogen per
45 itive to an oxidative stress-inducing agent, methyl viologen, but the sarA sodA double mutant was mor
46 de or the reactive oxidative species inducer methyl viologen can induce macroautophagy in Arabidopsis
47 aracterization reveals that the insertion of methyl viologen causes phase transformation of the organ
48 mical characterization of these Hg tips with methyl viologen, cobalt sepulchrate trichloride, and hex
49 SDS-PAGE for covalently bound haem, but the methyl-viologen-dependent nitrite reductase activities a
52 uniquely different binding pocket wherein a methyl viologen dication is stabilized by interacting wi
53 (CN)6(3-/4-), Ru(NH3)6(3+/2+), IrCl6(2-/3-), methyl viologen, dopamine, ascorbic acid, Fe(3+/2+), and
56 henol plus ascorbate as an electron donor to methyl viologen, however, was the same as observed in th
57 ent pi-radicals such as MV(+*) (MV refers to methyl viologen, i.e., N,N'-dimethyl-4,4'-bipyridinum) e
61 ble insertion of a large molecular dication, methyl viologen, into the crystal structure of an aromat
63 d with the WT, gr3 was sensitive to salt and methyl viologen; it showed inhibited growth, decreased m
65 ond hydrogenase gene cluster, hyd, exhibited methyl viologen-linked hydrogenase enzymatic activity, b
66 e to transcribe nar and subsequently express methyl viologen-linked nitrate reductase activity under
68 und to catalyze the reduction of NO(2)(-) by methyl viologen monocation radical (MV(red)), displaying
70 ronously realize the regeneration of reduced methyl viologen (MV(*+)) and NADH for the nitrogenase an
72 of a 1:1 inclusion complex between the guest methyl viologen (MV(2+)) and the host cucurbit[7]uril (C
73 ometric reduction of nitrate to nitrite used methyl viologen (MV(2+)) as the electron transfer mediat
75 finement of electron mediators (for example, methyl viologen (MV(2+))) within the hybrids enables the
76 a reversible electron acceptor and mediator, methyl viologen (MV(2+)), accelerates the extraction of
77 irradiation with 505 nm, 3 is able to reduce methyl viologen (MV(2+)), an electron acceptor commonly
78 he C-Dots, we followed the photoreduction of methyl viologen (MV(2+)), which acts as a molecular redo
81 der oxidative stress conditions generated by methyl viologen (MV) added during the early exponential
83 oxidative stress generated by the herbicide methyl viologen (MV) and exhibited transcriptional hyper
87 butyrate) (PHB) synthesis, or treatment with methyl viologen (MV), effectively increases glycogen acc
88 es was measured by three electron acceptors, methyl viologen (MV), potassium ferricyanide, or dichlor
89 lasts resulted in an increased resistance to methyl viologen (MV)-induced oxidative stress, documente
90 re essential for superoxide stress response, methyl viologen (MV)-sensitive mutants of S. mutans were
94 n by a photosystem I (duraquinol [DQH(2)] to methyl viologen [MV]) proton pumping partial reaction wa
95 odic reduction of the other electrocatalyst (methyl viologen, [MV](2+)) generates a negative fuel tri
96 s (ruthenium bipyridine [Ru-(bipy)3(2+)] and methyl viologen [MV2+]) was quantified in a U-tube perme
99 y was observed on recombinant CbR module and methyl viologen nitrate reduction by holo-NaR, suggestin
100 ties NADH:cytochrome c reductase and reduced methyl viologen:NR, closely paralleled the appearance an
101 ron-sulfur component oxidized CO and reduced methyl viologen or a ferredoxin isolated from M. thermop
102 Furthermore, the effects of exposure to methyl viologen or H2O2 on intracellular NADH and NAD(+)
105 n sulfoxide reductase activity using reduced methyl viologen or reduced benzyl viologen as artificial
106 re selectively sensitive to peroxide but not methyl viologen or Rose Bengal, and GPXs, APX, and MSRA2
107 m neutral TCNQ, I(2), and fullerene to ionic methyl viologen, organometallic like nickelcarborane, et
108 sed to different oxidative stress conditions-methyl viologen, ozone, and high light-differences were
109 the antioxidant depletion in the presence of methyl viologen (paraquat), a known agent of oxidative s
110 ethal levels of the ROS-generating herbicide methyl viologen (paraquat), suggesting a common protecti
113 1,1'-Dimethyl-4,4'-bipyridinium dichloride (methyl viologen; paraquat), an herbicide that causes dep
114 s bisradical dicationic redox state with the methyl viologen radical cation (MV(*+)) to give a 1:1 in
115 we report the efficient encapsulation of two methyl viologen radical cations (MV(.+) ) in a size-matc
116 t transitions, high light, and the herbicide methyl viologen, rapidly activated GCN2 kinase, whereas
118 valuated using both aqueous (Fe(CN)(6)3-/4-, methyl viologen, Ru(NH3)(6)3+/2+, and IrCl(6)2-/3-) and
119 ccurs in the sarA background, can rescue the methyl viologen-sensitive phenotype observed in the abse
122 uests simultaneously and, in the presence of methyl viologen, to recognize N-terminal tryptophan over
124 G18a transgenic plants are more sensitive to methyl viologen treatment than wild-type plants and accu
126 n (MOP) 3 which bears 24 covalently attached methyl viologen units on its external surface, as eviden
128 of Q8 to aromatic peptides in the absence of methyl viologen was studied by isothermal titration calo
129 g time as well as its resistance to H2O2 and methyl viologen were indistinguishable from those of the
130 with Mito-Bodipy-TOH in cells stressed with methyl viologen, whereas no enhancement was observed in
131 trary to 4,4'-dipyridinium (i.e., archetypal methyl viologen), which is reduced by two single-electro
132 of the artificial electron transfer mediator methyl viologen, whose redox potential lies negative of