ライフサイエンスコーパス: methylmercury

1 toxic solutes (e.g., monomeric aluminum and methylmercury).

2 ry and convert the oxidized Hg to neurotoxic methylmercury.

3 t can be subsequently transformed into toxic methylmercury.

4 s bioavailability and thus the production of methylmercury.

5 are consuming fish heavily contaminated with methylmercury.

6 ment of the fluorescence quenching caused by methylmercury.

7 ich these microorganisms produce and degrade methylmercury.

8 sediments containing coal ash was present as methylmercury.

9 particularly if the metal is in the form of methylmercury.

10 consumption of fish high in PUFAs and low in methylmercury.

11 ffect of PUFA on MI risk was counteracted by methylmercury.

12 uptake that leads to the formation of toxic methylmercury.

13 be the single significant dietary source of methylmercury.

14 al moderating effect of prenatal exposure to methylmercury.

15 ansform reactive ionic mercury to neurotoxic methylmercury.

16 ict the conversion of Hg(II) to bioavailable methylmercury.

17 have sulfate-reducing bacteria that produce methylmercury.

18 organisms is a key step in the production of methylmercury, a biomagnifiable toxin.

19 re a complex process, with the production of methylmercury, a potent human neurotoxin, repeatedly dem

20 The pollutant methylmercury accumulates within lean tissues of birds a

21 ock, 6-10% of total mercury was converted to methylmercury after one day.

22 A detection limit of 5.9 nM methylmercury and a repeatability expressed as a relativ

23 e found between carapace length and mercury, methylmercury and cadmium concentrations, and between fa

24 oil and cardiovascular risk, (2) effects of methylmercury and fish oil on early neurodevelopment, (3

25 lean tissue during flight which may mobilize methylmercury and increase circulating levels of this ne

26 Hydrophobicity of methylmercury and its ability to facilitate a nonradiati

27 0.1, and 0.6 parts per million (ppm) dietary methylmercury and measured changes in blood total mercur

28 fish consumption have frequently focused on methylmercury and omega-3 fatty acids, not persistent po

29 r nutrients, but because of contamination by methylmercury and other toxicants, higher fish intake of

30 Uniform depth distributions of methylmercury and oxygen encountered in the poorly strat

31 PCBs may account for the adverse effects of methylmercury and the degree to which co-exposure to doc

32 tudies incorporating information on both the methylmercury and the docosahexaenoic acid contained wit

33 ty of environmental factors such as alcohol, methylmercury, and maternal seizure activates HSF1 in ce

34 blood (1999-2004) predominantly represented methylmercury, and urine (1999-2002) represented inorgan

35 omponent of the mercury cycle that maintains methylmercury at low concentrations in natural waters.

36 Methylmercury, at low levels generally considered safe,

37 For example, methylmercury bioaccessibility decreased significantly a

38 Methylmercury bioaccessibility varied between 10 (octopu

39 Methylmercury bioaccumulation in zooplankton is higher t

40 sk to the toxicological effects of increased methylmercury burdens.

41 al of total mercury by 600 nmol m(-2) and of methylmercury by 214 nmol m(-2) in the Gotland Deep, pro

42 events and human activities and converts to methylmercury by anaerobic organisms.

43 tion, thus affecting the production of toxic methylmercury by PCA cells.

44 The production of methylmercury by some bacteria is a key first step in th

45 Here we show for the first time that methylmercury can be produced in particles sinking throu

46 er, depended on water constituents that bind methylmercury cations.

47 We investigated microbial methylmercury (CH(3)Hg) production in sediments from the

48 procedure for inorganic mercury (Hg(IN)) and methylmercury (CH(3)Hg) was developed.

49 Methylmercury (CH3Hg(+)) is the common form of organic m

50 d is present in either inorganic [Hg(II)] or methylmercury (CH3Hg) form.

51 y in foods, in inorganic form [Hg(II)] or as methylmercury (CH3Hg), can have adverse effects.

52 Inorganic Hg(II) and methylmercury ([CH3Hg(II)](+)) are commonly complexed wi

53 , which was approximately 3 times higher for methylmercury chloride than for thimerosal.

54 for the three species mercury(II) chloride, methylmercury chloride, and thimerosal after intoxicatio

55 In contrast, methylmercury-chloride complexes, which are dominant in

56 ansformations likely contributed to the high methylmercury concentration found in settling particles.

57 y (0.236+/-0.1001mg/kg(-1)), and the highest methylmercury concentration was found at the Labe - Obri

58 thers, we detected significantly higher mean methylmercury concentrations and higher proportions of s

59 Methylmercury concentrations decreased by 77% in underly

60 of total mercury concentrations to estimate methylmercury concentrations in bird eggs.

61 Methylmercury concentrations in clams and snails also de

62 south arm decreased by approximately 81% and methylmercury concentrations in deep waters decreased by

63 mercury in bird eggs have actually measured methylmercury concentrations in eggs.

64 centrations in eggs are a reliable proxy for methylmercury concentrations in eggs.

65 Total mercury and methylmercury concentrations in surface waters were decr

66 eviously implied connection between elevated methylmercury concentrations in the deep brine layer and

67 high-spatial-resolution depiction of surface methylmercury concentrations in this area.

68 ent geochemical data suggested that elevated methylmercury concentrations occurred in regions where n

69 and settling particles were not significant, methylmercury concentrations were about ten-fold greater

70 Methylmercury concentrations were determined by gas chro

71 cana) and Forster's terns (Sterna forsteri), methylmercury concentrations were highly correlated (R(2

72 Total mercury and methylmercury concentrations were measured in the settli

73 stratification, exacerbating high biological methylmercury concentrations.

74 r study and that co-exposure to nutrients in methylmercury-contaminated fish may have obscured and/or

75 for AIE-based fluorescence imaging study on methylmercury-contaminated live cells and zebrafish for

76 sed in mercury monitoring programs to assess methylmercury contamination and toxicity to birds.

77 f this work was to determine the mercury and methylmercury content in muscle tissue of chub (Leuciscu

78 Similarly, methylmercury content in the Emory and Clinch River sedi

79 Methylmercury cycling in the Pacific Ocean has garnered

80 Methylmercury demethylation rate constants (kd) were of

81 or inorganic mercury (Hg) to be converted to methylmercury depends, in part, on the chemical form of

82 tion of rice with fish and the food trade in methylmercury exposure assessments, and anthropogenic bi

83 imental neurocognitive effects from prenatal methylmercury exposure from maternal fish consumption du

84 Their prenatal methylmercury exposure had been assessed from the mercur

85 Although prenatal methylmercury exposure has been linked to poorer intelle

86 may predate clinical disease by years; thus, methylmercury exposure may be relevant to future autoimm

87 We developed equations to estimate methylmercury exposure of piscivorous birds and sport fi

88 However, prenatal methylmercury exposure seemed to attenuate these positiv

89 In 2013, globalization caused 9.9% of human methylmercury exposure via the international rice trade

90 In the group with lower prenatal methylmercury exposure, a 1 SD increase in VO2Max was as

91 tion of cord tissue as a measure of prenatal methylmercury exposure, data on 247 single-nucleotide po

92 a significant global dietary source of human methylmercury exposure, especially in South and Southeas

93 in delta(13)C over time and delta(15)N with methylmercury exposure, year remained a significant inde

94 d their reproductive success is sensitive to methylmercury exposure.

95 fect neurodevelopmental outcomes of prenatal methylmercury exposure.

96 e compared groups with low and high prenatal methylmercury exposure.

97 was observed in the group with high prenatal methylmercury exposure.

98 Better models included filtered water methylmercury (FMeHg) or unfiltered water methylmercury

99 oil on early neurodevelopment, (3) risks of methylmercury for cardiovascular and neurologic outcomes

100 ether the percentage of total mercury in the methylmercury form differed among species.

101 The mean percentage of total mercury in the methylmercury form in eggs was 97% for American avocets

102 The percentage of total mercury in the methylmercury form ranged from 63% to 116% among individ

103 The net potential for methylmercury formation, assessed by the ratio between t

104 above the maximum allowable limit with toxic methylmercury found as the dominant mercury species with

105 Exposure to methylmercury from fish consumption has been linked to a

106 In practice, contamination by methylmercury from fish consumption is assessed by measu

107 Exposure to methylmercury from fish has been associated with increas

108 Methylmercury from seafood, ethylmercury used as a bacte

109 e morphology, genes, or proteins involved in methylmercury generation.

110 H(sebenzim(Me)) also reacts with methylmercury halides, but the reaction is accompanied b

111 thylation of inorganic mercury to neurotoxic methylmercury has been attributed to the activity of ana

112 ) as optical nanoprobes for the detection of methylmercury has been developed.

113 rovide a basis for assessing and remediating methylmercury hotspots in the environment.

114 combination of cadmium chloride (CdCl2) and methylmercury (II) chloride (CH3HgCl) (0, 0.125, 0.5, or

115 l heart attacks are related to the intake of methylmercury in 2010.

116 Health effects of low-level methylmercury in adults are not clearly established; met

117 A first step toward bioaccumulation of methylmercury in aquatic food webs is the methylation of

118 Elevated levels of neurotoxic methylmercury in Arctic food-webs pose health risks for

119 hey may play a key role in the production of methylmercury in biofilms.

120 t step in the production and accumulation of methylmercury in biota.

121 o examine the relationship between total and methylmercury in eggs of two species, and (2) reviewing

122 spill, with a specific focus on mercury and methylmercury in sediments.

123 The production of the neurotoxic methylmercury in the environment is partly controlled by

124 that cause the production and degradation of methylmercury in the environment is ultimately needed to

125 There was no evidence of methylmercury in the fish or DOM within the 10% uncertai

126 layer in the accumulation and persistence of methylmercury in the Great Salt Lake.

127 the bioaccumulation of the potent neurotoxin methylmercury in the marine food web.

128 vity of anaerobic bacteria, the formation of methylmercury in the oxic water column of marine ecosyst

129 ic sea ice can harbour a microbial source of methylmercury in the Southern Ocean.

130 The production of methylmercury in these areas is a concern because this n

131 pected from reservoir creation will increase methylmercury inputs to the estuary by 25-200%, overwhel

132 important and changing source of mercury and methylmercury into the Arctic Ocean marine ecosystem.

133 Methylmercury is a global pollutant of aquatic ecosystem

134 Methylmercury is a neurotoxin and endocrine disruptor an

135 Methylmercury is a potent neurotoxin produced in natural

136 sediments where NOM can be reduced and toxic methylmercury is formed.

137 Our findings indicate that methylmercury is mobilized from lean tissues during prot

138 Methylmercury is one of the more toxic forms of mercury

139 Methylmercury is the environmental form of neurotoxic me

140 s with organic matter and, when converted to methylmercury, is a potent neurotoxicant.

141 son collections coincided with uniformly low methylmercury levels along the river downstream from the

142 gnificant independent covariate with feather methylmercury levels among the albatrosses.

143 Information on ocean scale drivers of methylmercury levels and variability in tuna is scarce,

144 he Pacific basin exhibit temporal changes in methylmercury levels consistent with historical global a

145 Methylmercury levels declined gradually to 200 km downst

146 Methylmercury levels in larvae, however, were much less

147 In general, the methylmercury levels in plankton and fishes downstream f

148 We investigated the seasonal variation of methylmercury levels in the Balbina reservoir and how th

149 ratification of the reservoir influenced the methylmercury levels in the reservoir and in the river d

150 Higher methylmercury levels observed 200-250 km downstream from

151 fied, and anoxic hypolimnion water with high methylmercury levels was exported downstream.

152 e dramatic mercury loss from deep waters and methylmercury loss from underlying sediment in response

153 DHA appears beneficial for, and low-level methylmercury may adversely affect, early neurodevelopme

154 rcury in adults are not clearly established; methylmercury may modestly decrease the cardiovascular b

155 Methylmercury (MeHg(+) ) is one of the most potent neuro

156 sted for removal of mercury species [Hg(2+), methylmercury (MeHg(+)), ethylmercury (EtHg(+)), and phe

157 In this study, the biodilution hypothesis of methylmercury (MeHg) accumulation was examined in a Hg-c

158 m, respectively, to study the effect on the methylmercury (MeHg) accumulation.

159 Methylmercury (MeHg) affects wildlife and human health m

160 of the highest concentrations of neurotoxin methylmercury (MeHg) among marine top predators.

161 Human exposure to methylmercury (MeHg) and elemental mercury vapor (Hg(0)(

162 stem-scale study examining the production of methylmercury (MeHg) and greenhouse gases from reservoir

163 posed here to compare toxicity mechanisms of methylmercury (MeHg) and inorganic mercury (iHg) in musc

164 atographic method was developed to determine methylmercury (MeHg) and inorganic mercury (iHg) levels

165 calibrated modeled hydrology, we calculated methylmercury (MeHg) and total mercury (THg) mass balanc

166 rcumpolar Arctic, elevated concentrations of methylmercury (MeHg) are accumulated in Arctic marine bi

167 enyls (PCBs), organochlorine pesticides, and methylmercury (MeHg) are environmentally persistent with

168 orption data especially for mercury (Hg) and methylmercury (MeHg) at the low porewater concentrations

169 THg) concentrations in nettles, with only 1% methylmercury (MeHg) being detected, while concentration

170 divalent mercury (Hg(II)) is transformed to methylmercury (MeHg) by anaerobic microbes.

171 The production of methylmercury (MeHg) by anaerobic microorganisms depends

172 Cellular uptake of dissolved methylmercury (MeHg) by phytoplankton is the most import

173 ct of the seasonal inundation of wetlands on methylmercury (MeHg) concentration dynamics in the Amazo

174 er delta to quantify total mercury (THg) and methylmercury (MeHg) concentrations and export.

175 position, along with total mercury (THg) and methylmercury (MeHg) concentrations and fluxes, to decre

176 Methylmercury (MeHg) concentrations can increase by 1000

177 Rapid growth could significantly reduce methylmercury (MeHg) concentrations in aquatic organisms

178 To better understand the source of elevated methylmercury (MeHg) concentrations in Gulf of Mexico (G

179 We present a case study comparing metrics of methylmercury (MeHg) contamination for four undeveloped

180 a: Anisoptera: Gomphidae) as biosentinels of methylmercury (MeHg) contamination.

181 Methylmercury (MeHg) determinations in hake, its food-ch

182 isease (FMD), which is caused by exposure to methylmercury (MeHg) during development, many neurons ar

183 s-independent fractionation (MDF and MIF) of methylmercury (MeHg) during trophic transfer into fish.

184 Developmental methylmercury (MeHg) exposure alters dopamine neurotrans

185 Seafood consumption is the primary route of methylmercury (MeHg) exposure for most populations.

186 Maternal transfer is a predominant route of methylmercury (MeHg) exposure to offspring.

187 202)Hg decreased) with a strongly decreasing methylmercury (MeHg) fraction.

188 lved gaseous mercury (Hg(0)((aq))) and toxic methylmercury (MeHg) govern mercury bioavailability and

189 limination of inorganic mercury [Hg(II)] and methylmercury (MeHg) in a marine fish, Terapon jarbua.

190 The presence of toxic methylmercury (MeHg) in Arctic freshwater ecosystems and

191 nterest in measuring total mercury (THg) and methylmercury (MeHg) in dried blood spots (DBS) though m

192 Elevated concentrations of methylmercury (MeHg) in freshwater ecosystems are of maj

193 s of high concentrations of mercury (Hg) and methylmercury (MeHg) in mangroves, in conjunction with t

194 Net formation of methylmercury (MeHg) in sediments is known to be affecte

195 Rice is known to accumulate methylmercury (MeHg) in the rice grains.

196 As a ubiquitous environmental pollutant, methylmercury (MeHg) induces toxic effects in the nervou

197 d dietary fish consumption, from which daily methylmercury (MeHg) intake was estimated.

198 Methylmercury (MeHg) is a bioaccumulative neurotoxin pro

199 Methylmercury (MeHg) is a bioaccumulative toxic contamin

200 Methylmercury (MeHg) is a known neuro-toxicant.

201 The formation of the potent neurotoxic methylmercury (MeHg) is a microbially mediated process t

202 Methylmercury (MeHg) is a neurotoxic compound that threa

203 Methylmercury (MeHg) is a potent neurotoxin that has bee

204 Methylmercury (MeHg) is a potent neurotoxin that is biom

205 Methylmercury (MeHg) is an environmental contaminant of

206 Methylmercury (MeHg) is an environmental neurotoxin with

207 Methylmercury (MeHg) is an established neurotoxicant of

208 Fluvial methylmercury (MeHg) is attributed to methylation in up-

209 conversion of inorganic mercury (Hg(II)) to methylmercury (MeHg) is central to the understanding of

210 production of the bioaccumulative neurotoxin methylmercury (MeHg) is stimulated in newly flooded soil

211 Methylmercury (MeHg) is the only species of mercury (Hg)

212 The neurotoxicity of high levels of methylmercury (MeHg) is well established both in humans

213 Methylmercury (MeHg) is well known to induce auditory di

214 on the fatty acid profile, mercury (Hg), and methylmercury (MeHg) levels of salmon was studied.

215 Methylmercury (MeHg) may affect fetal growth; however, p

216 Methylmercury (MeHg) production by ND132 increased linea

217 gions where sloughed Cladophora accumulates, methylmercury (MeHg) production is enhanced.

218 lack of estimation and comparison of the net methylmercury (MeHg) production or degradation in these

219 the resulting short and long-term changes in methylmercury (MeHg) production.

220 c conditions in the hypolimnion that promote methylmercury (MeHg) production.

221 atic systems and accumulated as highly toxic methylmercury (MeHg) represents a threat to wildlife and

222 Inorganic Hg is readily converted to toxic methylmercury (MeHg) that bioaccumulates in aquatic food

223 To understand the biochemistry of methylmercury (MeHg) that leads to the formation of merc

224 Maternal transfer of methylmercury (MeHg) to eggs is an important exposure pa

225 tion exists on their potential as sources of methylmercury (MeHg) to freshwaters.

226 proposed to investigate inorganic (iHg) and methylmercury (MeHg) trophic transfer and fate in a mode

227 urrent frequency caused by the neurotoxicant methylmercury (MeHg) was examined in Purkinje cells of c

228 ion, and magnitude of hydrological fluxes of methylmercury (MeHg), a bioavailable Hg species of ecolo

229 Mercury (Hg) is of particular interest as methylmercury (MeHg), a neurotoxin which bioaccumulates

230 fate-reducing bacterium capable of producing methylmercury (MeHg), a potent human neurotoxin.

231 educing bacterium (SRB) capable of producing methylmercury (MeHg), a potent human neurotoxin.

232 the predominant source of human exposure to methylmercury (MeHg), a potent neurotoxic substance.

233 portant link between the global contaminant, methylmercury (MeHg), and human exposure through consump

234 ey step in microbial formation of neurotoxic methylmercury (MeHg), but the mechanisms remain largely

235 cosystems are contaminated with highly toxic methylmercury (MeHg), but the specific sources and pathw

236 ed in the environment, and its organic form, methylmercury (MeHg), can extensively bioaccumulate and

237 dology for the simultaneous determination of methylmercury (MeHg), ethylmercury (EtHg), and inorganic

238 ations of both total Hg (THg) and especially methylmercury (MeHg), the species of Hg having the highe

239 tu amendments for remediation of mercury and methylmercury (MeHg), using a study design that combined

240 The formation of methylmercury (MeHg), which is biomagnified in aquatic f

241 al into the highly bioaccumulated neurotoxin methylmercury (MeHg).

242 es leading to significant bioaccumulation of methylmercury (MeHg).

243 o the atmosphere, including mercury (Hg) and methylmercury (MeHg).

244 norganic Hg into bioaccumulative, neurotoxic methylmercury (MeHg).

245 ing steps in the formation of the neurotoxin methylmercury (MeHg).

246 onstituents and often display high levels of methylmercury (MeHg).

247 AOSR) of Canada contain elevated loadings of methylmercury (MeHg; a neurotoxin that biomagnifies thro

248 The sources of methylmercury (MeHg; the toxic form of mercury that is b

249 speciation of inorganic mercury, Hg(II) and methylmercury, MeHg(I) in water and fish tissue samples.

250 atmospheric pollutants, epilimnetic aqueous methylmercury (MeHgaq) and mercury in small yellow perch

251 Mercury (Hg) methylation and methylmercury (MMHg) demethylation activity of periphyto

252 dy of a method for the determination of mono methylmercury (MMHg) in foodstuffs of marine origin by g

253 lue mussel, killifish, eider) to investigate methylmercury (MMHg) sources and exposure pathways in fi

254 ion energy of the carbon-mercury bond on the methylmercury molecule6-7 and subsequently increased rea

255 nd efflux of inorganic HgII (as HgCl(2)) and methylmercury or MeHg (as CH(3)HgCl) in EA.hy926 endothe

256 Chronic exposure to low levels of methylmercury (organic) and inorganic mercury is common,

257 5 years of age were estimated for chemicals (methylmercury, organophosphate pesticides, lead) and a v

258 able for only three environmental chemicals (methylmercury, organophosphate pesticides, lead), the re

259 Our results explain methylmercury photodecomposition rates that are relative

260 icals as developmental neurotoxicants: lead, methylmercury, polychlorinated biphenyls, arsenic, and t

261 Elevated human exposure to methylmercury primarily results from consumption of estu

262 y anaerobic microbes; however, the amount of methylmercury produced varies greatly, as Hg methylation

263 orthern ecosystems and reservoir flooding on methylmercury production and bioaccumulation through a c

264 ing bacteria are responsible for the rate of methylmercury production and thus bioaccumulation in mar

265 3) Se inhibits Hg bioavailability to, and/or methylmercury production by, microbial communities.

266 However, net methylmercury production in cultures exposed to nanopart

267 y from the wastes but also the potential for methylmercury production in receiving waters.

268 s offer new insights into the role of DOM in methylmercury production in the environment.

269 Methylmercury production rates are generally related to

270 r regional information on vertical habitats, methylmercury production, and/or Hg inputs are needed to

271 o risk-benefit model on the basis of data on methylmercury, PUFA, and MI risk has yet been presented.

272 at studies on bioaccumulation should measure methylmercury rather than total mercury when using museu

273 be how exposure to both marine n-3 PUFAs and methylmercury relates to MI risk by using data from Finl

274 levels ( approximately 40,000 ng . g(-1)) of methylmercury relative to prior time points, suggesting

275 swamp, serving as net sources and a sink for methylmercury respectively.

276 be 0.2 and 0.1 ng g(-1) for mercuric ion and methylmercury, respectively.

277 s characterized for the Hg-C protonolysis of methylmercury rule out the direct protonation mechanism

278 benthic sediment was thought to be the main methylmercury source for coastal fish.

279 and modeling show that currently the largest methylmercury source is production in oxic surface seawa

280 vations in freshwater lakes) applied only to methylmercury species bound to organic sulfur-containing

281 ects of prenatal fish consumption as well as methylmercury suggest there are benefits from prenatal f

282 may experience greater surges in circulating methylmercury than demonstrated here as a result of thei

283 ons may also generate a higher proportion of methylmercury than more oligotrophic environments.

284 total mercury and, for a subset of samples, methylmercury (the bioaccumulated form of mercury) in mu

285 enhanced transfer of accumulated mercury and methylmercury to the planktonic food chain and finally t

286 males, because breeding females can depurate methylmercury to their eggs.

287 er methylmercury (FMeHg) or unfiltered water methylmercury (UMeHg), whereas filtered total mercury di

288 stantially to models explaining the observed methylmercury variation.

289 ish can also be used to estimate the loss of methylmercury via photoreduction in aquatic ecosystems.

290 A filter-passing methylmercury vs DOC relationship was also developed usi

291 Exposure to methylmercury was associated with increased risk of MI,

292 For the first step, methylmercury was extracted into 1-undecanol and for the

293 Methylmercury was measured in 90% of samples with concen

294 While the low-level prenatal exposure to methylmercury was not associated with child cognition, p

295 A finding of enhanced interaction with methylmercury was replicated in this study for the minor

296 Exposure to methylmercury was shown to decrease neural stem cell pop

297 itions, the calibration graphs of Hg(II) and methylmercury were linear in the range of 0.83-8.0 ug L(

298 humans and wildlife is the net production of methylmercury, which occurs mainly in reducing zones in

299 dual Hg species (inorganic Hg, ehtylmercury, methylmercury) with inductively coupled plasma mass spec

300 ve been caused by production of bioavailable methylmercury within those wetlands.

301 een assay is achieved for quick detection of methylmercury without the use of tedious sample preparat