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1 he number of emergency visits and dosages of methylprednisolone.
2 buted to epidural injections of contaminated methylprednisolone.
3 unosuppressed after MI with cyclosporine and methylprednisolone.
4 rade, level of injury, and administration of methylprednisolone.
5 or 3 days or placebo as an add-on therapy to methylprednisolone.
6 anti-CD52 monoclonal antibody alemtuzumab or methylprednisolone.
7 those resulting from treatment with topical methylprednisolone.
8 oval of doxycycline and were unresponsive to methylprednisolone.
9 blinding, and directly compared Hyalgan with methylprednisolone.
10 hromatography for levels of prednisolone and methylprednisolone.
11 renal failure when compared with intravenous methylprednisolone.
12 Acute rejection and SCAR were treated by methylprednisolone.
13 hemotherapy, and 2 improved with intravenous methylprednisolone.
14 bclinical rejections were treated with bolus methylprednisolone.
15 te outbreak due to injection of contaminated methylprednisolone.
16 5-2005, Leiden cohort, n = 153) treated with methylprednisolone.
17 ne or in combination with the corticosteroid methylprednisolone.
18 in inhibitor, short-course methotrexate, and methylprednisolone.
19 andomly assigned: 30 to gentamicin and 30 to methylprednisolone.
20 ng pain following injections of contaminated methylprednisolone.
21 ns associated with injection of contaminated methylprednisolone.
22 on 20 March 2020 using early, short-course, methylprednisolone 0.5 to 1 mg/kg/day divided in 2 intra
23 5% and 16 patients were treated with topical methylprednisolone 0.5% twice daily for 10 weeks, in add
25 /6 mice, without or with topical therapy, 1% methylprednisolone, 0.025% doxycycline, or physiologic s
26 opical formulations containing 5% IL-1Ra, 1% methylprednisolone, 0.05% cyclosporin A, and a vehicle c
27 f the patients were treated with intravenous methylprednisolone 1 g daily for 3 days and then tapered
28 aper therapy (PCT) consisting of intravenous methylprednisolone 1 gm daily for 1 week followed by ora
30 e prospectively randomized to receive either methylprednisolone 1,000 mg followed by a 3-month steroi
32 lind, placebo-controlled study to receive IV methylprednisolone (15 mg/kg of ideal body weight/day) o
33 lly numbered envelopes to receive placebo or methylprednisolone 16 mg/kg/day (</=1000 mg) for 3 days.
35 ght atriotomy with antiinflammatory therapy (methylprednisolone 2 mg/kg per day) (antiinflammatory gr
36 ion at the site of injection of contaminated methylprednisolone, 21% had an abnormal MRI, and all but
37 of cardiopulmonary bypass to receive either methylprednisolone (250 mg at anaesthetic induction and
39 mbination including IVIG 1 g/kg, intravenous methylprednisolone 30 mg/kg, Vinca alkaloids (VCR 0.03 m
40 double-blind randomized controlled trial of methylprednisolone (30 mg/kg) or placebo after the induc
41 ere randomly assigned to receive intravenous methylprednisolone, 30 mg per kilogram of body weight (1
43 pants were randomized to receive intravenous methylprednisolone (4 mg/kg/d for 2 weeks) and oral pred
44 mization, patients in the VSE group received methylprednisolone (40 mg) and patients in the control g
45 groups (37 patients each) received 80 mg of methylprednisolone, 40 mg of methylprednisolone, or plac
46 surgery were 73%, 81%, and 92% in the 80-mg methylprednisolone, 40-mg methylprednisolone, and placeb
47 Corticosteroid therapy consisted of either methylprednisolone, 500 mg intravenously for 3 days, or
48 (1:1) by a block design to two intratympanic methylprednisolone (62.5 mg/mL) or gentamicin (40 mg/mL)
52 third-stage larvae and treated for 6 wk with methylprednisolone acetate (MPA), a synthetic glucocorti
55 meningitis due to injections of contaminated methylprednisolone acetate can present with vascular seq
56 0 units daily for 3 days and 1gr intravenous methylprednisolone acetate for 3 days followed by 1 mg/k
58 ucocorticoid injections of preservative-free methylprednisolone acetate prepared by a single compound
59 ssociated with the injection of contaminated methylprednisolone acetate produced by the New England C
60 ingitis linked to contaminated injections of methylprednisolone acetate produced by the New England C
62 ociated with injections of preservative-free methylprednisolone acetate that was purchased from a sin
63 ll persons potentially exposed to implicated methylprednisolone acetate was conducted by federal, sta
70 50 mg of cyclophosphamide, and 100-250 mg of methylprednisolone, administered on 2 occasions 2 weeks
72 cocorticoid resistance and whether prolonged methylprednisolone administration accelerates the suppre
73 into a randomized trial evaluating prolonged methylprednisolone administration in unresolving ARDS ha
75 ntinued to show superior efficacy over pulse methylprednisolone alone for treatment of lupus nephriti
76 with simvastatin alone or with high dose of methylprednisolone alone or in combination with simvasta
79 as greater in patients who received 80 mg of methylprednisolone and 40 mg of methylprednisolone than
80 Of these, 65 were treated with anakinra and methylprednisolone and 55 were untreated historical cont
82 0-increased MMP-9 secretion was inhibited by methylprednisolone and also by compound A, a novel nonst
83 BALB/c mice underwent immunosuppression with methylprednisolone and antibodies specific for CD4 T cel
86 7-5.24), and treatment with a combination of methylprednisolone and intravenous immunoglobulin (OR 2.
89 steroids (betamethasone, triamcinolone, and methylprednisolone) and three local anesthetics (lidocai
90 nary support (including dialysis), high-dose methylprednisolone, and an anti-interleukin-2 receptor a
91 Her symptoms stabilized with intravenous methylprednisolone, and her cancer was treated with carb
92 d 92% in the 80-mg methylprednisolone, 40-mg methylprednisolone, and placebo groups, respectively.
93 improvement several days later, tocilizumab, methylprednisolone, and therapeutic anticoagulation were
94 l, other particulate steroids (prednisolone, methylprednisolone, and triamcinolone) caused often imme
95 d only after treatment with 5% IL-1Ra and 1% methylprednisolone, and were absent after cyclosporin A
97 d dexamethasone, spironolactone, and 6-alpha-methylprednisolone as major contributors to corticostero
106 Glucocorticoids, including dexamethasone, methylprednisolone, budesonide, and fluticasone, potenti
107 after Exserohilum rostratum contamination of methylprednisolone by the New England Compounding Center
108 er one cycle of chemotherapy with etoposide, methylprednisolone, cisplatin, and cytarabine in patient
109 At 3 mo, 33 (49%) of 67 after intravenous methylprednisolone compared with 48 (69%) or 70 after pl
110 tial inflammatory response, the acute use of methylprednisolone compared with placebo decreased treat
113 spinal cord injury is controversial; however methylprednisolone continues to be widely employed in th
122 te rejection or antirejection treatment with methylprednisolone did not increase the risk of BKV repl
124 cretion of nasal polyp patients treated with methylprednisolone, doxycycline, anti-IL-5, or placebo.
125 N), number of emergency visits and dosage of methylprednisolone during a 16-week period were compared
126 jections with a cumulative dose of 3.24 g of methylprednisolone during a 9-week period with good tole
127 allograft had a lower risk of resistance to methylprednisolone during AR (odds ratio, 0.29; 95% conf
128 essors, combined vasopressin-epinephrine and methylprednisolone during CPR and stress-dose hydrocorti
129 nistration of a 3-day regimen of intravenous methylprednisolone either in an outpatient clinic (n=69)
130 L exposed to plasma samples collected during methylprednisolone exhibited significant progressive inc
134 ents were initially commenced on intravenous methylprednisolone for 3 days, followed by oral predniso
135 ne sodium succinate equivalent to 1000 mg of methylprednisolone for 5 days, 0.4 mg/kg/d of intravenou
136 ostratum conidia preexposed to 0.32 mg/mL of methylprednisolone for 7 days in immunocompetent flies l
139 ch, we evaluated the efficacy of intravenous methylprednisolone for HCPS treatment, through a paralle
141 Patients were treated with alemtuzumab and methylprednisolone for induction, followed by tacrolimus
143 Subjects received concurrent 2 mg/kg per day methylprednisolone for more than or equal to 10 days.
144 RS trial does not support the routine use of methylprednisolone for patients undergoing cardiopulmona
145 se results do not support the routine use of methylprednisolone for persistent ARDS despite the impro
146 he efficacy of a single preoperative dose of methylprednisolone for preventing postoperative complica
147 or paraspinal glucocorticoid injection with methylprednisolone from a single compounding pharmacy.
148 as admitted to the hospital and treated with methylprednisolone, furosemide, and C1 esterase inhibito
150 idence of postoperative complications in the methylprednisolone group (31.2% vs. 47.3%; p = 0.042).
151 ss treatment failure among patients from the methylprednisolone group (8 patients [13%]) compared wit
152 on) and from 16.4 (12.5) to 1.6 (3.4) in the methylprednisolone group (90% reduction; mean difference
153 0.3 to 38.6 percent) and 29.2 percent in the methylprednisolone group (95 percent confidence interval
155 of 67 and 32 of 67 (48%) in the intravenous methylprednisolone group and 51 (73%) of 70 and 35 of (5
156 lycemia occurred in 11 patients (18%) in the methylprednisolone group and in 7 patients (12%) in the
157 etween the 2 groups (6 patients [10%] in the methylprednisolone group vs 9 patients [15%] in the plac
161 surgery incorporated, both the 80- and 40-mg methylprednisolone groups had lower likelihood of surger
162 thylprednisolone, </=240 mg/d) or high-dose (methylprednisolone, >240 mg/d) groups based on CS dosage
163 ving placebo, patients receiving intravenous methylprednisolone had a somewhat shorter initial period
168 ith Therapeutic Plasma Exchange (TPE) and IV methylprednisolone improved the condition of the patient
169 tudy was to determine whether intraoperative methylprednisolone improves post-operative recovery in n
172 mus and mycophenolate in all three arms, and methylprednisolone in groups A and C only (standard clin
174 ficial effect of pulse high-dose intravenous methylprednisolone in patients with allergic bronchopulm
176 Topical tacrolimus was more effective than methylprednisolone in reducing the CFS score at week 10
177 on of IL-1 receptor antagonist anakinra plus methylprednisolone in severe COVID-19 pneumonia with hyp
178 exchange was more effective than intravenous methylprednisolone in the achievement of renal recovery
179 us (SLE) was the use of cyclophosphamide and methylprednisolone in the treatment of lupus nephritis.
181 flow cytometry, and ELISA demonstrated that methylprednisolone increased the expression of miRNA-98
183 adult rats with the antiinflammatory steroid methylprednisolone increases the activity of matrix meta
185 cluded rituximab infusions, cyclophosphamide/methylprednisolone infusions, prednisone and mycophenola
187 d thrombin-induced platelet aggregation, and methylprednisolone inhibited ADP-induced aggregation to
190 chronic tension-type headaches per month vs methylprednisolone injections (SMD, -2.5; 95% CI, -3.5 t
194 early intervention (EI) with intraarticular methylprednisolone into all synovitic joints or to conse
196 Patients were grouped into lower-dose (methylprednisolone, </=240 mg/d) or high-dose (methylpre
203 0 patients with AR received immediate pulsed methylprednisolone (MP) and one untreated patient develo
205 loped an animal model that demonstrates that methylprednisolone (MP) can block PV IgG-induced acantho
208 ls from healthy donors were exposed to 1 muM methylprednisolone (MP) in vitro and then subjected to m
211 ) promote recovery in animal models, whereas methylprednisolone (MP) promotes neurological recovery i
212 rt 1, matched unrelated donor transplant and methylprednisolone (MP) T-cell depletion (TCD) of donor
213 d to compare preoperative and intraoperative methylprednisolone (MP) to intraoperative MP alone with
219 ized controlled trial of tri-iodothyronine+/-methylprednisolone [MP] therapy) undergoing PAC-guided a
222 travenous bolus of 0.5 mg/kg per 12 hours of methylprednisolone (n = 61) or placebo (n = 59) for 5 da
224 e for all 7507 patients randomly assigned to methylprednisolone (n=3755) and to placebo (n=3752).
225 Of the 190 subjects enrolled, 176 (n = 81 methylprednisolone, n = 95 placebo) were included in thi
226 Proadrenomedullin levels were decreased with methylprednisolone on day 3 in patients with infectious
229 fect of donor treatment with simvastatin and methylprednisolone on microvascular dysfunction and immu
233 ived either intensive enteral nutrition plus methylprednisolone or conventional nutrition plus methyl
234 ent of EDE with the anti-inflammatory agents methylprednisolone or doxycycline preserves apical corne
236 s were given aggressive therapy (intravenous methylprednisolone or oral prednisone 5-30 mg/kg/day; n
237 least seven days' duration to receive either methylprednisolone or placebo in a double-blind fashion.
240 Mice were treated daily with dexamethasone, methylprednisolone, or PBS from days 0 to 14 or days 10
244 pical treatment with 5% IL-1Ra (P < .01), 1% methylprednisolone (P < .01), and 0.05% cyclosporin A (P
246 y inhibited by 74% and 90% after exposure to methylprednisolone (P<0.05), 11% and 24% after exposure
247 antly in both groups (tacrolimus, P = 0.003; methylprednisolone, P = 0.008), whereas HLA-DR expressio
248 atment with high-dose pulse intravenous (IV) methylprednisolone permits a shorter course of therapy.
250 tions supported by phase II clinical trials (methylprednisolone plus alemtuzumab or ibrutinib) seem b
251 ylprednisolone premedication, or intravenous methylprednisolone premedication plus oral prednisone fo
252 either placebo glucocorticoids, intravenous methylprednisolone premedication, or intravenous methylp
253 ular RhoA GTPase pathway activation, whereas methylprednisolone prevented activation of innate immune
254 reatment in combination with simvastatin and methylprednisolone prevents IRI and has beneficial effec
255 ngal meningitis associated with contaminated methylprednisolone produced by a compounding pharmacy ha
256 more aggressive approaches such as high-dose methylprednisolone pulse therapy are used to provide tra
257 lative glucocorticoid doses (daily dose plus methylprednisolone pulse) during the first 6 months both
258 ections (cRR, 0.05 [0.02-0.6]; P = 0.01) and methylprednisolone pulses negatively affected the outcom
259 ympanic administration of the corticosteroid methylprednisolone reduces vertigo compared with gentami
260 were desmopressin use, triiodothyronine and methylprednisolone replacement, fluid management, vasopr
261 steroid injections (ESIs) with contaminated methylprednisolone resulted in an outbreak of fungal men
263 CI, 1.44-7.59; P<0.001), and treatment with methylprednisolone (RR, 2.31; 95% CI, 1.07-4.94; P=0.03)
264 alone or in combination with simvastatin and methylprednisolone significantly reduced cardiac troponi
267 ppressive therapy, consisting of intravenous methylprednisolone sodium succinate equivalent to 1000 m
270 lly administration of levothyroxine (T4) and methylprednisolone (steroid, i.e., the "T4 Protocol") in
272 sion treatment with antithymyocyte globulin, methylprednisolone, tacrolimus, mycophenolate mofetil, a
273 ved 80 mg of methylprednisolone and 40 mg of methylprednisolone than in those who received placebo (d
279 nt (placebo, oral prednisone, or intravenous methylprednisolone), time, and treatment x time interact
281 ition of a single pulsed dose of intravenous methylprednisolone to conventional intravenous immune gl
282 etermine whether the addition of intravenous methylprednisolone to conventional primary therapy for K
284 e hypothesis of inferiority of intratympanic methylprednisolone to oral prednisone for primary treatm
285 of 30 placebo-treated patients and 11 of 30 methylprednisolone-treated patients progressed to the pr
288 id, and the particulate steroids cortivazol, methylprednisolone, triamcinolone, and prednisolone.
289 .21, 0, and 0 capillaries per millimeter for methylprednisolone, triamcinolone, or prednisolone, resp
291 iratory distress syndrome, administration of methylprednisolone was associated with improvement in im
292 h anti-CD154 mAb, mycophenolate mofetil, and methylprednisolone was associated with persistently low
296 eceived placebo, those who received 80 mg of methylprednisolone were less likely to have surgery (odd
297 The number of emergency visit and doses of methylprednisolone were significantly reduced compared t
299 e patient responded to high-dose intravenous methylprednisolone, which resulted in improvement of ren
300 -1.47), and two large trials of intravenous methylprednisolone with altogether 467 participants, in