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1 is-N7G-BD adduct amounts in liver tissues of mice exposed to 0.5, 1.0, and 1.5 ppm BD for 2 weeks wer
2 ide (TG) levels, were higher in conventional mice exposed to 1 ppm arsenic, while arsenic exposure di
6 r-activated receptor-gamma and CD36 in those mice exposed to 16alphaOHE and protein derived from HPAH
7 y and used it to isolate RGCs from wild type mice exposed to 2 weeks of IOP elevation generated by th
8 quantify TETS concentration in the serum of mice exposed to 2x LD50 dose of TETS and to monitor kine
11 d pulmonary vascular remodeling in wild-type mice exposed to 3 weeks of hypoxia; this beneficial acti
14 in the spleen of adult male and female CD-1 mice exposed to 4 to 40,000 mug/kg/day BPA or 0.02 to 2
15 ounced effects were seen in 2.7-3.2-year-old mice exposed to 40% caloric restriction starting at 0.3
16 parenchyma and the bronchoalveolar space of mice, exposed to 48 hours of hyperoxia together with nor
17 ously demonstrated that PHDi prevents OIR in mice exposed to 5 days of sustained 75% oxygen followed
23 well as the brain and blood transcriptome in mice exposed to 9 weeks of unpredictable chronic mild st
24 wborn severe combined immunodeficiency-beige mice exposed to 90% O2 from birth; sham controls receive
25 High-dose i.p. MSC administration to newborn mice exposed to 90% O2 resulted in the restoration of no
28 ary damage and inflammation in Cftr-knockout mice exposed to a dextran sodium sulfate-induced portal
29 n-1, by human airway epithelial cells and in mice exposed to A fumigatus spores or secreted fungal fa
32 and CAST/EiJ, both in unexposed mice and in mice exposed to a model DNA-damaging chemical, 1,3-butad
34 encing, we characterized lung neutrophils in mice exposed to a pro-allergic low dose of lipopolysacch
36 recordings of single units in behaving male mice exposed to a rat to investigate the encoding of pre
39 the colonic mucosa-associated microbiota in mice exposed to a social stressor (called social disrupt
40 ented the acquisition of social avoidance in mice exposed to a social threat, but did not affect a pr
41 lly induced demyelination is enhanced in old mice exposed to a youthful systemic milieu through heter
42 spectrometry, in liver and kidney tissues of mice exposed to AA-I, at doses ranging from 0.001 to 1 m
43 e, miR-146a delivery or Akt2 silencing in WT mice exposed to acid resulted in suppression of iNOS in
45 were continuously measured for three days in mice exposed to ad libitum and restricted feeding condit
47 n protein of the BAFF-receptor, BAFFR-Fc, in mice exposed to air or CS for 24 weeks and evaluated sev
48 administration of anti-CXCL13 antibodies in mice exposed to air or CS for 24 weeks, and several hall
50 e examination of adenosine receptor-knockout mice exposed to AKI demonstrated that renal protection b
52 fected social recognition in adulthood: only mice exposed to all three hits showed deficits in this a
55 basis for these effects, we determined that mice exposed to an HFD combined with MGF exhibited a sub
56 ments were performed using NZM and apoE(-/-) mice exposed to an IFNalpha-containing or empty adenovir
61 Leishmania donovani was serially passaged in mice exposed to arsenic in drinking water at environment
64 cells and in bronchoalveolar lavage fluid of mice exposed to B20 were approximately 20-30% higher tha
66 ges in vascular remodeling were monitored in mice exposed to bleomycin in conjunction with genetic re
74 TcdB) in sera and body fluids of piglets and mice exposed to C. difficile to investigate the relation
75 mined the F2 generation and F3 generation of mice exposed to caffeine from E10.5-13.5, as this coinci
76 l ventricle dilation) preferentially in male mice exposed to CAPs, and it persisted through young adu
79 le and immune related gene sets in Lcn2(-/-) mice exposed to CCl(4), along with decrease in Timp1 and
80 r the colonization of pneumococcus in BALB/c mice exposed to cholera toxin 4 weeks prior to challenge
81 ngly, the reduced plasma insulin in M4K4 iKO mice exposed to chronic (16 weeks) HFD was not observed
82 ne the role of FGF21 in hepatic steatosis in mice exposed to chronic alcohol treatment and to discern
83 MS/MS quantification of the hepatic ECM from mice exposed to chronic carbon tetrachloride (CCl4); rec
84 t CS exposure.Measurements and Main Results: Mice exposed to chronic CS or to elastase, and patients
87 hypoxia-pulmonary arterial hypertension, and mice exposed to chronic hypoxia expressed increased StAR
89 vo in miR-143-/- and anti-miR-143-3p-treated mice exposed to chronic hypoxia in both preventative and
91 developed less severe PH than did wild-type mice exposed to chronic hypoxia, with less distal pulmon
97 ook an unbiased plasma proteomic analysis of mice exposed to chronic social stress and observed dysre
99 trol subjects (n = 19/group) and of C57BL/6J mice exposed to chronic stress or control conditions (n
100 Finally, LAC had no effect on mGlu2 knockout mice exposed to chronic unpredictable stress, and a sing
101 ffect in Flinders Sensitive Line rats and in mice exposed to chronic unpredictable stress, which, res
106 The development of emphysema in humans and mice exposed to cigarette smoke is promoted by activatio
107 d the expression and function of miR-135b in mice exposed to cigarette smoke or nontypeable Haemophil
108 Importantly, NTHI-specific T cells from mice exposed to cigarette smoke produced lower levels of
113 ly ameliorated acute renal tubular damage in mice exposed to cisplatin insult, associated with enhanc
115 , developed by Celldex Therapeutics Inc., in mice exposed to Co-60 gamma total body irradiation (TBI)
116 tine on the metabolic profiles in tissues of mice exposed to cobalt-60 total-body gamma-radiation.
122 ighly induced ATFs in thermogenic tissues of mice exposed to cold temperatures or treated with the be
127 Neither CD4(+) nor CD8(+) T cells from donor mice exposed to CS alone are sufficient to cause inflamm
128 was acetylated and degraded in the lungs of mice exposed to CS and in patients with chronic obstruct
129 ze the activation of 2'-5' OAS in lungs from mice exposed to CS and viral pathogen-associated molecul
132 nd conventional natural killer (NK) cells in mice exposed to CS over 4 days and examined the contribu
134 icroglia isolated from the frontal cortex of mice exposed to CUS show elevated CSF1 receptor expressi
135 were produced quickly in the lungs of naive mice exposed to cysteine proteases, such as bromelain an
136 on were evaluated in wild-type and Mdr2(-/-) mice exposed to darkness or melatonin treatment or in ma
137 ive abnormalities were prevented in DN-DISC1 mice exposed to Delta(9)-THC by simultaneous adolescent
140 s, eyelids from normal young and old mice or mice exposed to desiccating stress were evaluated by imm
141 mbrane conductance regulator (Cftr) knockout mice exposed to dextran sodium sulfate and in vitro in p
143 livers of long-lived Snell dwarf mice and in mice exposed to drugs that have been shown to extend lif
144 aily PZ-235 treatment of filaggrin-deficient mice exposed to dust mite allergens for 8 weeks signific
148 pocampal BDNF was detected in APPSWE /PS1dE9 mice exposed to EE, however, no changes were detected in
149 ole-genome microarray analysis of lungs from mice exposed to either 24 hours hypoxia or normoxia.
150 tent antidepressant-like effect in Ghsr-null mice exposed to either CSDS or caloric restriction, whil
153 ned the effects of spontaneous withdrawal in mice exposed to either nicotine (6.3 or 18 mg/kg/day) or
154 rozygotes and wild type (Hhip (+/+)) C57/BL6 mice exposed to either room-air or CS for six months.
156 donovani parasites were serially passaged in mice exposed to environmentally relevant concentrations
158 When compared with control mice, female mice exposed to ES displayed decreased social behavior a
161 s potential role in synaptic dysfunctions in mice exposed to ethanol during early brain development i
162 onse curves were generated in naive mice and mice exposed to ethanol in a model of voluntary consumpt
166 a was endogenously increased) and from young mice exposed to exogenous TNF-alpha exhibited significan
175 in insulin receptor mutant (Insr(P1195L/+)) mice exposed to HFD (Insr(P1195L/+)/HFD mice) revealed i
177 RF1 in vivo and in vitro, whereas MuRF1(-/-) mice exposed to high CO2 did not develop muscle atrophy.
180 dative cytosine modification accumulation in mice exposed to high-fat diet (HFD), injected with strep
183 characterize the microbiota in intestines of mice exposed to hyperbaric oxygen, human rectal biopsy a
184 of autophagy in smooth muscle cells of young mice exposed to hyperlipidemia led to increased aortic P
185 ly, we reported that 14-week-old young adult mice exposed to hyperoxia as newborns had spatial and le
189 revented alveolar simplification in neonatal mice exposed to hyperoxia.Conclusions: Cell therapy invo
190 d in human PAH, and the deletion of Arrb1 in mice exposed to hypoxia led to worse PAH with a loss of
192 terine arteries (UtA) isolated from pregnant mice exposed to hypoxia or normoxia from gestational day
193 ulation, miR410 is downregulated in lungs of mice exposed to hypoxia-induced pulmonary hypertension (
198 d to apoE(-/-) mice, while NZM and apoE(-/-) mice exposed to IFNalpha developed accelerated thrombosi
203 otection following BBT-059 administration in mice exposed to lethal and supralethal doses of total bo
205 lucose tolerance was measured in (nocturnal) mice exposed to light-dark stimulus patterns simulating
208 nses in wild-type BALB/c and Il21r-deficient mice exposed to local airway challenge with house dust m
210 with their wild-type littermates, BAP1(+/-) mice exposed to low-dose asbestos fibers showed signific
211 e also measured these markers in midbrain of mice exposed to maternal immune activation (MIA) during
213 performed a global analysis on the brains of mice exposed to MeHg by magnetic resonance imaging, an i
214 ency and decreases in threshold in the IC of mice exposed to MeHg for 4 weeks compared with vehicle m
215 ion increased in skeletal muscles from young mice exposed to metabolic stress and in muscles from hea
216 : group 1, untreated mice (n = 15); group 2, mice exposed to metformin treatment (750 mg/kg/d) for th
219 cortex (PFC) and nucleus accumbens (NAc) of mice exposed to multimodal chronic restraint stress.
222 at uric acid was increased in the airways of mice exposed to NO2 and that administration of uricase i
223 nvestigated colitis development in Il10(-/-) mice exposed to normal or 3SL-deficient milk during lact
226 the risk of acute and subchronic toxicity to mice exposed to OSPW-OF at environmentally relevant conc
238 Coupled with increased FR response rates, mice exposed to postnatal CAPS displayed increased FR re
244 u-P responses were seen in WT and CRFR2 null mice exposed to repeated stress, which were sustained at
249 in body weight between days 4 and 9, whereas mice exposed to SEB and also treated with abatacept show
250 ected in TNFR KO mice as well as in C57BL/6J mice exposed to SF but treated with TNF-alpha neutralizi
254 ysema and antigen-presenting cells (APCs) of mice exposed to smoke or nanoparticulate carbon black (n
258 ally identified DRN GABA and 5-HT neurons in mice exposed to social defeat, a model that induces long
260 (15 nM) previously measured in the serum of mice exposed to social stress significantly increased pr
261 or antagonist, is antihyperalgesic in primed mice exposed to spinal administration of a D1/D5 agonist
263 behavioral and neurophysiological studies in mice exposed to stress or to intracerebroventricular inj
264 in the nucleus accumbens was reduced in all mice exposed to stress, whereas decreased myelin thickne
268 systematically characterize CIPN recovery in mice exposed to the antitubulin cancer drugs eribulin, i
270 ons of socially isolated versus group-housed mice exposed to the contextual fear conditioning test, e
271 tihistamine-resistant scratching behavior in mice exposed to the haptens, oxazolone and urushiol, the
272 hFVIII-specific antibodies and inhibitors in mice exposed to the live-attenuated measles-mumps-rubell
273 agmentation and increased sleep duration for mice exposed to the more robust light-dark pattern.
274 early demonstrated that P2X(7)(-/-) mice and mice exposed to the P2X(7) antagonist, brillian blue G,
278 our bases and using DNA from liver tissue of mice exposed to the tobacco-specific nitrosamine 4-(meth
280 the fate of CD8 T cells from transgenic TCR mice exposed to their cognate Ags as self or in the cont
281 pared to H(2)O and a novel odorant, vanilla, mice exposed to TMT in the home cage showed increased av
282 cotinine concentrations in serum samples of mice exposed to tobacco smoke for 12 or 24 weeks and fou
283 othesized that metabolites in the urine from mice exposed to total body radiation (TBI) would predict
284 e the auditory pathology of adult male CBA/J mice exposed to traumatic noise (2-20 kHz; 106 dB; 2 h).
285 T8-deficient hepatocytes and GLUT8-deficient mice exposed to trehalose resisted trehalose-induced AMP
286 showed that great-great-grandsons of female mice exposed to tributyltin (TBT) throughout pregnancy a
288 quencing to characterize DCs from skin LN of mice exposed to two different Th2 stimuli: the helminth
289 eolar lavage fluids (BALf) from male C57BL/6 mice exposed to ultrafine carbon black nanoparticles, a
292 two etiologically distinct models of autism: mice exposed to valproic acid in utero and Fmr1 knockout
293 zed lipidomics to analyze serum samples from mice exposed to various percentages of neutrons and X-ra
297 c analysis was performed on lung tissue from mice exposed to virulent (Francisella tularensis ssp tul
300 on and assessed tumor formation in humanized mice exposed to wild-type virus and a viral mutant (Delt