ライフサイエンスコーパス: microaneurysm

1 opathy lesions (retinal blot hemorrhages and microaneurysms).

2 els and penetrating arterioles with numerous microaneurysms.

3 l Neural Network (Mask-RCNN) for quantifying microaneurysms.

4 r to those seen in human DR including ME and microaneurysms.

5 tailed characterization of perfused diabetic microaneurysms.

6 n sensitivities of new, static and regressed microaneurysms.

7 infiltrated with macrophages, forming pseudo-microaneurysms.

8 hemosiderin deposits, vessel tortuosity, and microaneurysms.

9 ppearance of pericyte ghosts or formation of microaneurysms.

10 ntrol) and was preceded by mesangiolysis and microaneurysms.

11 ll death, leukocyte plugging of vessels, and microaneurysms.

12 etic retinopathy of both eyes with scattered microaneurysms.

13 ers (15/173; 9%) and the outermost extent of microaneurysms (113/173; 68%) were localized to the oute

14 dataset comprising 148 images annotated with microaneurysms, 118 (75%) and 30 (25%) of which were use

15 0) for any retinopathy, 3.11 (1.71-5.65) for microaneurysms, 3.08 (1.42-6.68) for soft exudates, 2.55

16 Over half of closed microaneurysms (45/86, 52.3%) left hyperreflective spots

17 as able to identify a mean (SD) of 6.4 (4.0) microaneurysms (95% CI, 4.4-8.5), while FA identified a

18 while FA identified a mean (SD) of 10 (6.9) microaneurysms (95% CI, 6.4-13.5).

19 cantly inhibiting the development of retinal microaneurysms, acellular capillaries, and pericyte ghos

20 examination revealed multiple mid-peripheral microaneurysms and a wreath-like type 3 arteriovenous an

21 ng clinical pathologic fundus lesions (e.g., microaneurysms and focal edema), were markedly delayed (

22 ading of secondary vascular effects, such as microaneurysms and hemorrhages, by clinical examination

23 lar to those observed in diabetes, including microaneurysms and increased vascular permeability, sugg

24 Zonal assessments of macular microaneurysms and macular leakage index values revealed

25 bx3 in ECs results in glomerular hypoplasia, microaneurysms and regressed fenestrations leading to fi

26 a significant decrease in the mean number of microaneurysms and retinal hemorrhages on UWF CFP at M3

27 Smaller microaneurysms and those with heterogeneous lumen were p

28 as significantly correlated to the number of microaneurysms and to the FAZ surface.

29 n nodular glomerulosclerosis, mesangiolysis, microaneurysms, and arteriolar hyalinosis associated wit

30 tivity, manifesting as dilation, tortuosity, microaneurysms, and decreased cerebral blood flow, as ob

31 sangial matrix expansion, mesangiolysis with microaneurysms, and Kimmelstiel-Wilson nodules.

32 illary length, width, density, the number of microaneurysms, and the percent of capillary length invo

33 Microaneurysms are biomarkers of microvascular injury in

34 e superior field than in the inferior field, microaneurysms are more frequent in the superior than in

35 Microaneurysms are the key hallmark of the early stage o

36 Retinal thickness through microaneurysms as well as the presence of adjacent hypor

37 External diameter of microaneurysms averaged 104 mum (range 43-266 mum).

38 Closure rate of microaneurysms by both FA and SD-OCT was 69.9% (84/123),

39 iabetic retinopathy (DR) is characterized by microaneurysms, capillary nonperfusion, and ischemia wit

40 Some microaneurysm centers (15/173; 9%) and the outermost ext

41 reflectivity were positively associated with microaneurysm closure at 12 months (P < .0001, P < .001,

42 Microaneurysm closure rate increased at 6 and 12 months

43 eneous lumen were positively associated with microaneurysm closure.

44 or DRIL extent, cysts, hyperreflective foci, microaneurysms, cone outer segment tip visibility, and e

45 f DME with macular leakage index and macular microaneurysm count (P < 0.01).

46 ith macular leakage index and posterior pole microaneurysm count (P = 0.0002 and P = 0.03, respective

47 eyes with DME showed a significantly higher microaneurysm count (P = 0.001) and leakage index (P < 0

48 (mean = 9.53%); P<2x10(-16)], and panretinal microaneurysm count [mild NPDR (mean = 36), moderate NPD

49 Additionally, microaneurysm count and ischemic index were quantified i

50 x, panretinal ischemic index, and panretinal microaneurysm count are associated with DR severity.

51 Quantitative measures of leakage index and microaneurysm count in the posterior pole on UWFA images

52 Panretinal leakage index, microaneurysm count, and ischemic index were not signifi

53 addition to posterior pole leakage index and microaneurysm count, DME was associated with older age (

54 anretinal leakage index, ischemic index, and microaneurysm count.

55 Down-trending leakage indices and microaneurysm counts were demonstrated over 1 year of an

56 Microaneurysm dimensions, percent depth within the retin

57 ing of atheromatous plaques, atherosclerotic microaneurysms extending into periaortic vascular channe

58 Characterization of microaneurysms following focal laser photocoagulation re

59 ce of pericyte ghosts, vascular leakage, and microaneurysm formation.

60 Microaneurysms found in regions without nonperfusion wer

61 tworks using ensembling for the detection of microaneurysms from OCT angiography en face images from

62 of ensembled U-nets to automatically segment microaneurysms from OCT angiography fundus projections.

63 ejection with glomerulitis, microthrombosis, microaneurysms, glomerular hypertrophy, podocyte loss, g

64 To evaluate detection of hemorrhage and/or microaneurysm (H/Ma) using ultrawide field (UWF) retinal

65 Most microaneurysms had an internal lumen with homogeneous re

66 d from another 15 dogs after 31 months, when microaneurysms had previously been observed to develop.

67 For retinal hemorrhages and/ or microaneurysms, hard exudates, new vessels, fibrous prol

68 with diabetes and without signs of DR (e.g., microaneurysms, hemorrhages), plus three control eyes an

69 presence of pathologic conditions, including microaneurysms, hemorrhages, exudates, neovascularizatio

70 On UWF-CF images, the number of microaneurysms, hemorrhages, neovascularizations, and ar

71 tio [OR] 2.47 [95% CI 1.42-4.31]) or retinal microaneurysms/hemorrhages (2.28 [1.24-4.18]) were signi

72 r narrowing, arteriovenous (AV) nicking, and microaneurysms/hemorrhages were evaluated on digital ret

73 etinal microvascular lesions (AV nicking and microaneurysms/hemorrhages) are more likely to have mult

74 orithms determined VRA and hemorrhage and/or microaneurysm (HMA) counts.

75 , and histological analysis showed incipient microaneurysms in retinas of gal-fed marmosets.

76 significantly correlated with the number of microaneurysms in the DCP, the surface of capillary non

77 sms in the superficial vascular complex, and microaneurysms in the deep vascular complex (DVC) (p = 0

78 SSPiM may be correlated with microaneurysms in the DVC and a poor anatomical response

79 After multivariate logistic analysis, microaneurysms in the DVC were the only different factor

80 pared to the non-DME eyes, DME eyes had more microaneurysms in the SCP and the DCP (p = 0,039 and p =

81 se-free duration, previous injection number, microaneurysms in the superficial vascular complex, and

82 Microaneurysms, intraretinal microvascular abnormalities

83 Microaneurysms, intraretinal microvascular abnormalities

84 lammation was not a significant predictor of microaneurysm leakage.

85 tion at month 1 and again every 3 months for microaneurysm leakage.

86 signaling leads to the formation of abundant microaneurysms, leaky capillaries, and retinal hemorrhag

87 The worst changes histologically were microaneurysms, leukocyte and platelet plugging of aneur

88 FA metrics, including leakage, ischemia, and microaneurysm (MA) burden.

89 An automated system for the measurement of microaneurysm (MA) turnover was developed and compared w

90 All individual DR lesions (hemorrhage [H], microaneurysm [ma], cotton wool spot [CWS], intraretinal

91 Microaneurysms (MAs) are one of the earliest clinically

92 Microaneurysms (MAs) have distinct, oval-shaped, hyperre

93 To correlate the appearance of microaneurysms (MAs) on structural spectral-domain optic

94 rochannels designed to mimic saccular-shaped microaneurysms (microaneurysm-on-a-chip, or MAOAC), whic

95 haracterized by microangiopathies, including microaneurysms, microhemorrhages, and nerve layer infarc

96 Leaking microaneurysms (n = 123) were analyzed in eyes (n = 29)

97 s manifested by an increase in the number of microaneurysms, neovascular tufts, and preretinal nuclei

98 The exact intraretinal depth of microaneurysms on OCTA was localized in all cases (100%)

99 ned to mimic saccular-shaped microaneurysms (microaneurysm-on-a-chip, or MAOAC), which signify common

100 nal hemorrhages only, 3) presence of retinal microaneurysms only, and 4) presence of moderate or wors

101 nts with diabetes had retinal defects (e.g., microaneurysms or exudates or both) within the field of

102 age, larger haemorrhage, fibrinoid necrosis, microaneurysms, perivascular space dilation, perivascula

103 Increased FA leakage of diabetic microaneurysms positively correlated with perianeurysm f

104 The DR and microaneurysm scores from the first nonreferable DR (NRD

105 Furthermore, DR and microaneurysm scores generated from low- and high-resolu

106 Perfused microaneurysms seen by SD-OCT were localized deeper than

107 Microaneurysms showed relatively slower flow, IRMAs show

108 here was not complete correspondence between microaneurysms shown on FA and PV-OCT images.

109 It also mitigated vascular remodeling and microaneurysms significantly.

110 erfusion, inflammation, and pericyte loss on microaneurysm size and leakage in DR through three-dimen

111 In addition, microaneurysm size was a significant predictor of leakag

112 s were found to be independent predictors of microaneurysm size.

113 Almost all microaneurysms spanned more than 1 retinal layer (157/17

114 layers (DRIL), cysts, epiretinal membranes, microaneurysms, subretinal fluid, and outer layer disrup

115 It identified fewer microaneurysms than FA, but located their exact intraret

116 l vascular integrity regarding the number of microaneurysms, the number of IRMA, the surface of capil

117 ted system to investigate some properties of microaneurysm turnover, in particular the differing dete

118 DR through three-dimensional analysis of 636 microaneurysms using high-resolution confocal scanning l

119 Closure of diabetic microaneurysms was characterized in detail following foc

120 and F1-score values for detecting individual microaneurysms were 0.786, 0.615, and 0.690, respectivel

121 A total of 173 microaneurysms were analyzed in 50 eyes (14 mild nonprol

122 inal microvascular abnormalities (IRMAs) and microaneurysms were associated with the areas of nonperf

123 The characteristics of microaneurysms were evaluated by 2 masked observers usin

124 Microaneurysms were first classified as perfused if flow

125 blocked the development of mesangiolysis and microaneurysms, whereas tubulointerstitial injury was no

126 teriopathy include the "string of beads" and microaneurysms which are indistinguishable from those of

127 es were segmented for ischemia, leakage, and microaneurysms with manual correction as needed.