戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 nduction, iron deficiency and a hypochromic, microcytic anemia.
2 ssive loss of body (but not facial) hair and microcytic anemia.
3   Heterozygous gammabeta(0) mice suffer from microcytic anemia.
4 ion by red blood cells leads to hypochromic, microcytic anemia.
5 -/- mice represent a new paradigm of genetic microcytic anemia.
6 % CI: -7.5, -1.1% points) and nonthalassemic microcytic anemia (-1.7% points; 95% CI: -3.3, -0.1% poi
7 hianti (cia) mutant manifests a hypochromic, microcytic anemia after the onset of embryonic circulati
8 ere 343 mild anemic males in whom 47.8% were microcytic anemia and 3,323 non-anemic males for the ana
9                 Mice lacking Pcbp1 exhibited microcytic anemia and activation of compensatory erythro
10 ted with lower probability of nonthalassemic microcytic anemia and better adequacy of dietary iron in
11 ns or deletions may be a cause of refractory microcytic anemia and bone marrow iron depletion in pati
12                       It is characterized by microcytic anemia and by iron loading, and can be treate
13 tants associated with human diseases such as microcytic anemia and Charcot-Marie-Tooth are unable to
14 entified in a female with severe hypochromic microcytic anemia and iron overload.
15                  Mice that lack IRP2 develop microcytic anemia and neurodegeneration associated with
16                     bdh2 null mice developed microcytic anemia and tissue iron overload, especially i
17 yndrome (joint contractures, muscle atrophy, microcytic anemia, and panniculitis-induced childhood-on
18 rized by joint contractures, muscle atrophy, microcytic anemia, and panniculitis-induced lipodystroph
19  (cdy), a zebrafish mutant with hypochromic, microcytic anemia, and positioned the mutant gene on lin
20                                 Hypochromic, microcytic anemias are typically the result of inadequat
21   The mutation, zinfandel, has a hypochromic microcytic anemia as an embryo, but later recovers in ad
22 ted with higher odds of anemia, particularly microcytic anemia (asthma: 1.61; 1.09-2.38; P = .02; ecz
23 duction, whereas those who lack IRP2 develop microcytic anemia, believed to result from iron deficien
24 deficiency anemia patients, who present with microcytic anemia caused by hyperhepcidinemia, and of qu
25            These animals exhibit hypochromic microcytic anemia due to impaired intestinal iron absorp
26 plinary guidelines on the management of rare microcytic anemias due to genetic disorders of iron meta
27 ome akin to typhoid fever with splenomegaly, microcytic anemia, extramedullary erythropoiesis, and in
28 derstanding of the pathogenesis of inherited microcytic anemias has gained from the identification of
29 9 nmol/L]), leukocytosis (13 800/mm(3)), and microcytic anemia (hemoglobin level, 7.2 g/dL).
30 cythemia in heterozygotes and a hypochromic, microcytic anemia in homozygotes.
31 yses reveal a mild, congenital, hypochromic, microcytic anemia intrinsic to the hematopoietic system
32                                              Microcytic anemia (mk) mice and Belgrade (b) rats have s
33 notype are similar to those reported for the microcytic anemia (mk) mutation in the mouse.
34 athogenesis of a new recessive, hypochromic, microcytic anemia mouse mutant, nm1054.
35 ient with a history of a 6-month hypochromic microcytic anemia of unknown cause.
36 a genetic locus not previously implicated in microcytic anemia or iron phenotypes.
37 lobin level, i.e. 10.9 g/dL with hypochromic microcytic anemia pattern seen in complete blood count (
38 nhibition of mTORC1 results in macrocytic or microcytic anemia, respectively.
39 e normal recipients of HBD marrow obtained a microcytic anemia similar to the donor.
40  and alpha2-globulin and more frequently had microcytic anemia than those without such deposits (P =
41 ene symbol hbd) is characterized by a severe microcytic anemia that is inherited in an autosomal-rece
42          These Tf-deficient mice have severe microcytic anemia, tissue iron overload, and hepcidin de
43                               A hypochromic, microcytic anemia was present from birth, and platelet c
44 d-specific ALA synthase 2 (ALAS2) gene cause microcytic anemia, whereas mitochondrial DNA deletions a
45          Chronic turpentine treatment led to microcytic anemia, which was prevented by concurrent adm
46 d individuals included liver dysfunction and microcytic anemia, while one had fatal cardiomyopathy wi
47       The Rac1(-/-);Rac2(-/-) mice developed microcytic anemia with a hemoglobin drop of about 20% an
48 Homozygous sublytic mice develop hypochromic microcytic anemia with reduced osmotic fragility of RBCs
49 ice alters actin assembly in RBCs and causes microcytic anemia with reticulocytosis, implicating Rac
50  moderately severe, congenital, hypochromic, microcytic anemia, with an elevated red cell zinc protop