1                                              Microiontophoretic activation of GABA(A) receptors (GABA

2 (2)(+) from internal stores, following focal microiontophoretic activation of the alpha3beta4 nAChRs.

3                                              Microiontophoretic application of a selective mu-opioid

4 ractor muscle was either stimulated by local microiontophoretic application of acetylcholine or the m

5                         Here, we address how microiontophoretic application of ACh modulates SSA in t

6 -evoked responses of PSNs were attenuated by microiontophoretic application of AP-5 (n = 12, P < 0.00

7                       This was combined with microiontophoretic application of arginine analogs that

8 at some sites were prevented by simultaneous microiontophoretic application of kynurenic acid or at o

9 ulation of the superior sagittal sinus or by microiontophoretic application of l-glutamate.

10 arge of most MSNs was selectively reduced by microiontophoretic application of the non-NMDA receptor

11 ion was based on earlier findings that local microiontophoretic application of the putative LC transm

12 vation of VP afferents were monitored during microiontophoretic application of treatment ligands in c

13                Here, we tested the effect of microiontophoretic applications of ACh on the neuronal r

14 enhanced as were responses to juxtacellular, microiontophoretic applications of glutamate.

15 nduced in serotonergic neurons with bath and microiontophoretic applications of NMDA to activate loca

16                      We investigated whether microiontophoretic ejection of beta antagonists could in

17                      By searching with focal microiontophoretic NMDA applications, GABAergic and glut