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1 formed tumors, and prevented both macro- and micrometastasis.
2 because of the associated increased risk of micrometastasis.
3 be overlooked during SLND as they may harbor micrometastasis.
4 th cisplatin to enhance suppression of liver micrometastasis.
5 mb to overwhelmingly numerous and widespread micrometastasis.
6 ften by just one growth-restricted dangerous micrometastasis.
7 trigel plug angiogenesis, and uveal melanoma micrometastasis.
8 ecurrence of established cancer, and to find micrometastasis.
9 androgen-independent growth, accompanied by micrometastasis.
10 (subsequent outgrowth) of lung experimental micrometastasis.
11 the effect of inhibiting VEGF on tumor cell micrometastasis.
12 n aid in the visualization of ovarian cancer micrometastasis.
13 nt IFN-alpha 2b results in decreased hepatic micrometastasis and increased survival time through incr
15 owing at various secondary sites to generate micrometastasis and metastatic colonization than control
17 but not parental cells) were capable of lung micrometastasis and of binding exogenously-added hyaluro
18 p120 isoforms, is predictive of renal tumor micrometastasis and systemic progression, following neph
19 ts of cancer progression (local invasion and micrometastasis) and may no longer be required once meta
21 layer or small cluster of cells simulating a micrometastasis, and little effect in a sphere analogous
22 rognosis similar to that associated with SLN micrometastasis, and the number of positive qRT biomarke
23 ualization of intraperitoneal ovarian cancer micrometastasis as small as 100 mum with optimal resolut
24 el of PI in mice can promote lymph node (LN) micrometastasis, as well as head and neck metastasis of
25 implanted human PC3-M PCa cells formed lung micrometastasis by 4 weeks in >80% of inbred athymic mic
26 que has shown great potential for predicting micrometastasis by capturing the dynamic activation stat
28 l cancers, perhaps because of more effective micrometastasis eradication and reduced risk of incomple
29 -mediated signaling, LUAD cell survival, and micrometastasis expansion in hyaluronan-rich microenviro
30 atic potential of tumor cells by quantifying micrometastasis formation within the ovarioles of adult
36 for the first time small, apparently dormant micrometastasis in the liver of patients with uveal mela
37 The procedure has improved the diagnosis of micrometastasis in the regional tumor-draining lymph nod
43 ection of high-risk breast cancer, including micrometastasis, is critical in tailoring appropriate an
44 olvement was 7% when the sentinel node had a micrometastasis (< or =2 mm), compared with 55% when the
46 it equally as clear when to perform ALND if micrometastasis (Mi) or isolated tumor cells (ITCs) are
50 ingly used to control local tumor spread and micrometastasis of pancreatic ductal adenocarcinoma (PDA
52 ls to predict nanoparticle delivery based on micrometastasis physiology could enable personalized tre
54 be the underlying determinant of spontaneous micrometastasis produced by these cells when compared wi
56 turnover rate, antigen expression level, and micrometastasis size on antibody penetration and retenti
60 or primary tumor outgrowth but that promotes micrometastasis to the lungs at the very earliest stages
61 tients with primary tumors < or = 2.0 cm and micrometastasis to the SLN had remaining axillary lymph
62 Their findings suggest that FOXF2 promotes a micrometastasis-to-macrometastasis transition in bones t
63 ate that PEG tuning can provide control over micrometastasis tracking with high tumor-to-background c
64 010 vs 18 for years 1992-2001, P < .001), LN micrometastasis vs macrometastasis (20 vs 19, P = .005),
68 us 1.0, P = 0.02); however, the incidence of micrometastasis was statistically similar in both HSA an
69 f the ocular tumor and the number of hepatic micrometastasis were compared between the mice inoculate
70 he ocular melanoma and the number of hepatic micrometastasis were decreased and microvessel density w
71 ctin receptor in mouse cancer cells enhances micrometastasis, while treatment with the COX-2 inhibito
72 ng patients with melanoma and regional nodal micrometastasis who may benefit from full nodal basin re
73 icellular spheroids are an in vitro model of micrometastasis whose adhesive abilities have not been e
74 capability of CREKA-Tris(Gd-DOTA)3 to detect micrometastasis with MRI in co-registration with high-re