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1 ses can induce injury in MPO-ANCA-associated microscopic polyangiitis.
2 merly known as Wegener's granulomatosis) and microscopic polyangiitis.
3 ents with either Wegener's granulomatosis or microscopic polyangiitis.
4 d make this condition a separate entity from microscopic polyangiitis.
5 of vasculitis as Wegener granulomatosis and microscopic polyangiitis.
6 matosis with polyangiitis, but are absent in microscopic polyangiitis.
7 d patients with diagnosed Wegener disease or microscopic polyangiitis.
8 groups (47 Wegener's granulomatosis [WG], 12 microscopic polyangiitis, 16 Churg-Strauss syndrome [CSS
9 mely granulomatosis with polyangiitis (GPA), microscopic polyangiitis and eosinophilic GPA (EGPA), ar
10 l cytoplasmic autoantibody (ANCA)-associated microscopic polyangiitis and glomerulonephritis (excludi
11 nd treatment resistance in 107 patients with microscopic polyangiitis and necrotizing and crescentic
12 nd systemic small vessel vasculitis, such as microscopic polyangiitis and Wegener granulomatosis.
13 wly diagnosed AAV (Wegener granulomatosis or microscopic polyangiitis) and were aged 18 to 75 years a
14 th granulomatosis with polyangiitis, 23 with microscopic polyangiitis, and 5 with renal-limited ANCA-
15 ener's granulomatosis, polyarteritis nodosa, microscopic polyangiitis, and Churg-Strauss syndrome.
17 seases, granulomatosis with polyangiitis and microscopic polyangiitis, and remains among the most dev
19 of children with Wegener granulomatosis and microscopic polyangiitis are derived by extrapolating fr
21 e histological features of kidney disease in microscopic polyangiitis have been associated with clini
22 is nodosa demonstrates greater similarity to microscopic polyangiitis in relapse rate than previously
26 ation and management of renal involvement in microscopic polyangiitis is discussed, with emphasis on
27 granulomatosis, Churg-Strauss syndrome, and microscopic polyangiitis, is impeded by the small number
28 itis that includes Wegener's granulomatosis, microscopic polyangiitis (microscopic polyarteritis), th
29 ises granulomatosis with polyangiitis (GPA), microscopic polyangiitis (MPA) and eosinophilic granulom
30 ses: granulomatosis with polyangiitis (GPA), microscopic polyangiitis (MPA) and eosinophilic granulom
35 with polyangiitis (GPA, formerly Wegener's), microscopic polyangiitis (MPA), and eosinophilic granulo
36 omatosis with polyangiitis (Wegener's; GPA), microscopic polyangiitis (MPA), and eosinophilic granulo
37 with granulomatosis with polyangiitis (GPA), microscopic polyangiitis (MPA), and eosinophilic granulo
38 matosis with polyangiitis (GPA) (Wegener's), microscopic polyangiitis (MPA), and kidney-limited disea
39 tis has diverse patterns of injury including microscopic polyangiitis (MPA), granulomatosis with poly
40 ith neuropathy include polyarteritis nodosa, microscopic polyangiitis (MPA), rheumatoid vasculitis, C
42 04 consecutive patients (64 with WG, 14 with microscopic polyangiitis [MPA], and 526 others) and 45 h
44 years) received a KTX for ESRD secondary to microscopic polyangiitis (n=43) or Wegener's granulomato
45 It was concluded that most patients with microscopic polyangiitis or necrotizing and crescentic g
46 sis (granulomatosis with polyangiitis versus microscopic polyangiitis), or new diagnosis (versus rela
47 relapsing granulomatosis with polyangiitis, microscopic polyangiitis, or renal-limited ANCA-associat
48 emic lupus erythematosus (P = 2.7 x 10(-8)), microscopic polyangiitis (P = 2.9 x 10(-4)) and Wegener'
49 antineutrophil cytoplasmic antibody-positive microscopic polyangiitis, resulting in pulmonary hemorrh
51 between granulomatosis with polyangiitis and microscopic polyangiitis that are associated with ANCA s
52 076) when controlling for age, ANCA pattern, microscopic polyangiitis versus glomerulonephritis alone
54 ulonephritis, including 69 with evidence for microscopic polyangiitis, were evaluated for this study.