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1 dium difficile toxin B significantly reduced microtubular acetylation and the delivery of viral DNA t
2 acellular transport in plant cells occurs on microtubular and actin arrays.
3                     We hypothesized that the microtubular and actin cytoskeletons influence the expre
4 w insights into interaction between both the microtubular and microfilament cytoskeleton and cellular
5 cl-2 phosphorylation following disruption of microtubular architecture, serving a role similar to p53
6  relationship of motile cilia with the 9 + 2 microtubular arrangement have helped explain some of the
7 sts, mitochondria were observed close to the microtubular array and displayed both short- and long-ra
8                     The mitotic spindle is a microtubular assembly required for chromosome segregatio
9 hic inversion on 17q21, sometimes called the microtubular associated protein tau (MAPT) inversion, is
10 ion of Beclin 1, and increased conversion of microtubular-associated protein 1 light chain 3-I to -II
11 y distributed throughout the cytosol without microtubular association, C19ORF5C specifically accumula
12 is an essential ciliary protein required for microtubular attachment of ODAs in the axoneme.
13 y cilia are sensory organelles composed of a microtubular axoneme and a surrounding membrane sheath t
14 lar (interchangeable terms) membrane and the microtubular axoneme of motile and sensory cilia.
15                             In addition to a microtubular axoneme, the flagellum contains a crystalli
16 tes the individualization machinery with the microtubular axonemes.
17                             Tau proteins are microtubular binding proteins localized in the axonal co
18 ealization that targeting various aspects of microtubular biology with small molecules might offer ne
19 hieves its effects by acting on the neuronal microtubular content, which is involved with growth, sta
20 d to detect the presence of alpha-tubulin, a microtubular cytoskeletal component, in isolated nuclear
21 t C19ORF5 mediates communication between the microtubular cytoskeleton and mitochondria in control of
22 dles at the leading margin direct the distal microtubular cytoskeleton as growth cones turn to avoid
23 lexes and immunofluorescence analyses of the microtubular cytoskeleton of mitotic cells using wild-ty
24  demonstrated that iso-2 associates with the microtubular cytoskeleton that underlies the cell body m
25                            Disruption of the microtubular cytoskeleton with colchicine did not affect
26 y, and an intact actin cytoskeleton, but not microtubular cytoskeleton, are required for disruption o
27  TubA1 resulted in an incorporation into the microtubular cytoskeleton, demonstrating the effectivene
28 bacterial motility structures and became the microtubular cytoskeleton, including the mitotic apparat
29 rating neuronal migration by influencing the microtubular cytoskeleton.
30 overn tubulin transport to build the ciliary microtubular cytoskeleton.
31 ogenesis, and components of the cortical and microtubular cytoskeleton.
32 (ts) alp4 and alp6 mutants show two types of microtubular defects.
33  Electron microscopy revealed characteristic microtubular deposits with a diameter of 14-60 nm, hollo
34 ways are involved with cellular detection of microtubular disarray and subsequent activation of JNK/S
35 r dynein arm, central apparatus defects, and microtubular disorganization (IDA/CA/MTD) (n = 41) were
36  with IDA and central apparatus defects with microtubular disorganization (IDA/CA/MTD; n = 40).
37                                        Thus, microtubular disruption provides a noninvasive method fo
38                  To test the hypothesis that microtubular disruption should promote transgene persist
39 ry stages of infection such as modulation of microtubular dynamics, movement of virus in the cytoplas
40 gnaling cascades in apoptosis resulting from microtubular dysfunction induced by paclitaxel, we have
41 unrelated phenotypes have now been linked to microtubular dysfunction, especially in systems dependen
42 We show that it involved an interaction with microtubular elements, required activation of the kinase
43 on characteristics and optimization of these microtubular engines are described, along with their eff
44                          These mass-produced microtubular engines are only 8 mum long, are self-prope
45                          MIP-based catalytic microtubular engines are prepared by electropolymerizati
46                   Highly efficient catalytic microtubular engines are synthesized rapidly and inexpen
47 ential for efficient propulsion of catalytic microtubular engines.
48 ustion yields freestanding CNT or reduced GO microtubular fibers.
49  the production of carbon nanomaterial-based microtubular fibers.
50 ed perinuclear positioning of the convergent microtubular framework.
51 ate gene for phenotypic expansion related to microtubular function (DNAH5) was identified in 1 case (
52 ing shared pathogenic mechanisms in terms of microtubular function and interaction with microtubule-a
53 eroallergen exposure, implicating epithelial microtubular functions in the pathogenesis of Th2-mediat
54 alization is an aggregation of extracellular microtubular-like structures found within the sclerad re
55 ific differences in the hypertrophy of these microtubular-like structures may be related to inherent
56 ith the motor protein is responsible for the microtubular localization of PP5 in vivo.
57  underlying axonal neurofilament lattice and microtubular loss.
58 cyte microparasol, composed of a perinuclear microtubular/melano-phagolysosomal complex, protects the
59 reen targeting genes potentially involved in microtubular motility.
60  polarity proteins with microtubules and the microtubular motor KIF3/Kinesin-II.
61 ytoplasmic factor that does not seem to be a microtubular motor or a kinase/phosphatase.
62 nding sites for cellular proteins needed for microtubular movement and actin tail formation.
63 ly, but occasionally they bind to the cell's microtubular network and perform directed migration, whi
64       In contrast, structural changes of the microtubular network explain perceived eradication of di
65     To study the possible involvement of the microtubular network in the alpha-synuclein-dependent tr
66        Cargo transport along the cytoplasmic microtubular network is essential for neuronal function,
67 4 amino acids from the C-terminal, reveals a microtubular network localization by confocal microscopy
68                                          The microtubular network represents another potential regula
69 esulted in almost complete disruption of the microtubular network, abolished the adaptive increases i
70 ns help tether incoming viral capsids to the microtubular network, thus promoting cytoplasmic traffic
71 f a Stau-bcd mRNA complex through a nonpolar microtubular network, which confines the bcd mRNA to the
72 uorescent confocal microscopy to disrupt the microtubular network.
73 oward the nuclei of infected cells using the microtubular network.
74 ed continued protein synthesis and an intact microtubular network.
75 ining endosomes to the autophagosome via the microtubular network.
76 es EspG and EspG2, known to disrupt the host microtubular network.
77 ctivity, by tethering the transporter to the microtubular network.
78 MV MP, which, instead, was redirected to the microtubular network.
79 t CD155-containing endocytic vesicles to the microtubular network.
80  with decreased free tubulin or a diminished microtubular network.
81 a-tubulin, and the intermediate filament and microtubular networks of the transfected cells appeared
82 on microscopy revealed that the manchette, a microtubular organelle essential for sperm head and flag
83 e primary cilium is a ubiquitous, non-motile microtubular organelle lacking the central pair of micro
84  withdrawal of leading processes, changes in microtubular organization and, in some instances, to det
85 ently translocated into mLRs, mobilizing the microtubular organizing center and lytic granules to the
86  active Nercc has important functions at the microtubular organizing center during cell division.
87 frequency was used to transfer energy to the microtubular pacemaker for electrical stimulation.
88 design that can be rolled into a lightweight microtubular pacemaker for intravascular implantation an
89                                   Thus, this microtubular pacemaker paves the way for the minimally i
90 consanguineous families with PCD and central-microtubular-pair abnormalities.
91 oma, are transduced by the primary cilium, a microtubular projection found on many cells.
92           Excitatory amino acids may promote microtubular proteolysis observed in ischemic neuronal d
93  link between excitotoxic neurotransmission, microtubular proteolysis, and neuronal degeneration in f
94 ulin binding sites spaced 8 nm apart along a microtubular protofilament.
95       It works by altering the intracellular microtubular reorganization and, based on this mechanism
96                             Such acid-driven microtubular rockets offer considerable potential for di
97   Here, we introduce a submillimeter bundled microtubular (SBMT) flow battery cell configuration that
98 r compartment and anchored into the axonemal microtubular scaffold via the ODA docking complex (ODA-D
99 cyte aging and improves the integrity of the microtubular spindle apparatus in young and old oocytes.
100                   We tested the effects of a microtubular stabilizer (Taxol) in liver cell preservati
101  of isolated tubulin in vitro, disrupted the microtubular structure in MCF-7 cells as visualized by c
102               We hypothesize that this novel microtubular structure is involved in transporting mater
103 using this nuclear shaping is generated by a microtubular structure termed the manchette, which attac
104 igher concentrations of colchicine disrupted microtubular structure, but also caused increased actin
105  and FKBP1b-mediated restoration of neuronal microtubular structure.
106                        We propose that these microtubular structures contribute to a checkpoint contr
107 ound to be required to generate late-mitotic microtubular structures located at the division plane, a
108                                   When these microtubular structures were disrupted, the actin ring m
109 centrations <1.0 microM caused disruption of microtubular structures, but had little effect on either
110 tion of an F-actin meshwork, associated with microtubular structures, is actively involved in formati
111 that taurocholate-mediated changes involve a microtubular system.
112 ient tubulin for assembly and maintenance of microtubular systems.
113 ese photoelectrodes are composed of a porous microtubular top layer and an interlayer between the por
114 known to be transported to the nucleus along microtubular tracks by cytoplasmic dynein.
115  the latest stage of organelle traffic along microtubular tracks in the proplatelet shafts as shown b
116 nein, a molecular motor that processes along microtubular tracks to the nucleus.
117 g cellular transport of specific cargo along microtubular tracks via kinesin motor proteins.
118 ate of mass transfer is optimally related to microtubular transport and clustering properties of vesi
119                            At the same time, microtubular transport and vesicle clustering were model
120 stand the relationship of mass transfer with microtubular transport and vesicle clustering, we varied
121 he best representation of diffusion, whereas microtubular transport is accurately modeled with fracti
122                                        While microtubular transport is crucial for the proper functio
123 mation of a zone around the centrosome where microtubular transport of lysosomes is suppressed, resul
124                   Any perturbation to either microtubular transport or vesicle aggregation led to red
125                           Disruption of cell microtubular transport system by colchicine blocked the
126                           Disruption of cell microtubular transport system by colchicine inhibited th
127 om viral factories at speeds consistent with microtubular transport to the peripheries of ATIs, where
128 o other polyomaviruses, trafficking required microtubular transport, acidification of endosomes, and
129 a-lumicolchicine, which does not affect cell microtubular transport, did not inhibit the stimulatory
130 articles with a double membrane that enables microtubular transport, exocytosis, and actin polymeriza
131 tional double-membrane envelope that enables microtubular transport, exocytosis, and actin polymeriza

 
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