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1 in-8 (IL-8) on tumor growth and intratumoral microvascular density.
2 rowth, proliferation (Ki-67 percentage), and microvascular density.
3  systolic dysfunction, fibrosis, and reduced microvascular density.
4 ghly branched vessels, and overall preserved microvascular density.
5                 CBF is correlated with tumor microvascular density.
6 e to insulin, exercise and VEGF-A and reduce microvascular density.
7 ted for Ki-67 proliferative indexes and CD34 microvascular density.
8 reased VEGF and FGF2 BM plasma levels and BM microvascular density.
9 ced distant tumor growth, proliferation, and microvascular density.
10 ed seizures also caused robust reductions in microvascular density.
11 ch was confirmed by histological analysis of microvascular density.
12 t angiogenic factor, VEGF; and (3) decreased microvascular density.
13 proteinase-9 in GFAP-positive astrocytes and microvascular density.
14  with contractile function (wall motion) and microvascular density.
15 ly accompanied by an increase in dental pulp microvascular density.
16 h histopathologic findings for viability and microvascular density.
17 intraepithelial COX-2 and iNOS proteins, and microvascular densities.
18 n (30 min), increased GFR, and reduced renal microvascular density 14 days after injection.
19                                    Likewise, microvascular density, a histologic surrogate of angioge
20     The LVAD unloading resulted in increased microvascular density accompanied by increased fibrosis
21 as-induced murine colon carcinomas increased microvascular densities and vessel sizes.
22 ings were associated with both reduced tumor microvascular density and a reduction in the amount of v
23 alysis also showed significant reductions in microvascular density and actively dividing cells in the
24 tumors and caused a significant reduction in microvascular density and alphavbeta3 integrin expressio
25 ontrast-enhanced intensity corresponded with microvascular density and blood volume.
26 red, resulting in smaller tumors and reduced microvascular density and branching.
27  each patient was compared with the ratio of microvascular density and capillary area (r=0.84 and 0.8
28 s was associated with increased intratumoral microvascular density and enhanced endothelial cell surv
29 SIT and ET are effective in improving muscle microvascular density and eNOS protein content.
30 ed intestines of CRHR1(-/-) mice had reduced microvascular density and expression of vascular endothe
31                      Increased peripapillary microvascular density and flux were detected in a large
32 sults in lethality attributable to decreased microvascular density and hemorrhages.
33                            CCA showed higher microvascular density and higher expression of nuclear f
34 wth inhibition was associated with decreased microvascular density and increased vascular leakage.
35 yS3KO mice had no significant alterations in microvascular density and interstitial fibrosis in remod
36  MCF7-MCT-1 tumors in vivo, we found greater microvascular density and lower apoptosis in the MCF7-MC
37 omplex microvascular changes to preserve the microvascular density and maintain a stable microvascula
38 /- mice had significantly increased cerebral microvascular density and more effective restoration of
39               NTR1-knockout mice had reduced microvascular density and mucosal integrity score compar
40 lar hypertension, normalized stenotic kidney microvascular density and oxygenation, stabilized functi
41                            No differences in microvascular density and sympathetic or pan-neuronal in
42 dentified recovery of function compared with microvascular density and the sole use of peak MCI.
43 try and histomorphometry of the BM to assess microvascular density and to evaluate pan-neuronal and s
44 n nonmetastatic mammary tumor cells elevated microvascular density and vascular recruitment.
45 ile a physically active lifestyle keeps both microvascular density and vasodilator response high.
46 nalyses to assess skin and muscle viability, microvascular density and vessel morphology.
47 gnificant correlations were observed between microvascular density and vessel perimeter and area (r =
48 42, von Willebrand factor (VWF; a measure of microvascular density) and the potent vasoconstrictor en
49 n is associated with increased inflammation, microvascular density, and blood clotting.
50 decreased tumor volume, tumor cell survival, microvascular density, and lung metastasis relative to t
51  therapy also decreased BP and improved GFR, microvascular density, and oxygenation in the stenotic k
52 had reduced cutaneous hair-follicle density, microvascular density, and panniculus adiposus layer thi
53 st area, liver/body weight ratio, pericystic microvascular density, and PCNA expression while increas
54 unostaining indicated decreased intratumoral microvascular density, and TUNEL demonstrated enhanced t
55  overexpression increased tumor VEGF levels, microvascular density, and vessel permeability, whereas
56 pth, proliferation, macrophage infiltration, microvascular density, apoptosis) were assessed after a
57                       Increases in the tumor microvascular density are accompanied by a strong reduct
58                Two weeks after implantation, microvascular density, as measured by capillaries per hi
59 ted growth of BT474 xenografts and decreased microvascular density associated with downregulation of
60  no significant differences were observed in microvascular density between young and aged mice in nor
61 16K) cells was correlated with a decrease in microvascular density by 44%.
62 ls were disorganized and displayed decreased microvascular density by embryonic day 11.5.
63       We can, therefore, conclude that brain microvascular density can be controlled by HIF-independe
64 ation mouse model, based on lowest levels of microvascular density (CD31) and cellular proliferation
65                                        These microvascular density changes were significantly attenua
66 anied by a significant reduction in the mean microvascular density compared to the IgG control group.
67 ease distant tumor growth, proliferation, or microvascular density compared with sham treatment.
68  angiogenic gene signatures and had a higher microvascular density compared with their SOX11-negative
69 c (716/+) mice had significant reductions in microvascular density, consistent with the high expressi
70 r, EGFR and HER2/neu overexpression and high microvascular density correlate with survival.
71 ciated endothelial cell apoptosis, decreased microvascular density, decreased proliferation rate, and
72                                      Reduced microvascular density diminished the ability of the brai
73 uantifying changes in the marrow vascular by microvascular density, do not differentiate between diff
74     Clinical observations of increased colon microvascular density during IBD have been made.
75 ld, endocardium-to-epicardium evaluation for microvascular density, fibrosis, cardiomyocyte size, and
76 tyle keeps both the vasodilator response and microvascular density high.
77  production retained significantly increased microvascular density, improved glucose profiles, and in
78 l microcirculation, characterized by reduced microvascular densities in the capillary plexi, a lower
79 GF signaling does not result in reduction of microvascular density in a variety of tumor models.
80 urthermore, HE4 serum levels correlated with microvascular density in EOC tissue and inversely correl
81     There was a significant reduction of the microvascular density in eyes with exudative vs traction
82  fibrin deposits (5) significantly increased microvascular density in lumbar spinal cord, (6) IgG mic
83         There was a significant reduction in microvascular density in patients with CHF compared with
84  revealed a significant increase in hindlimb microvascular density in response to experimentally indu
85 herapy reduced both the total and functional microvascular density in the brain xenografts.
86 icate some established breast tumors, reduce microvascular density in the remaining tumors, protect a
87                      Similar to PAB animals, microvascular density in the RV was preserved in patient
88 site for breast cancer progression, and high microvascular density in tumors is a poor prognostic ind
89 uced endothelial cell migration in vitro and microvascular density in vivo.
90 losely related to functional (i.e. perfused) microvascular density, independent of arterial blood flo
91 esistance is closely coupled with functional microvascular density, independent of arterial blood flo
92  decreases tumor perfusion, vascular volume, microvascular density, interstitial fluid pressure and t
93                                 Importantly, microvascular density is significantly less, correlating
94 ll lung cancer (NSCLC) tumors have increased microvascular density, localized hypoxia, and high VEGF
95                                   Myocardial microvascular density (MCD) in aFGF-NP+UTMD group was up
96 performed to compare groups 1 and 2 both for microvascular densities (MVD) on histologic sections and
97                                              Microvascular density (MVD) and percentage of microvascu
98 nstrated to be significantly associated with microvascular density (MVD) and unfavorable prognosis of
99 planted into Cav-2 KO mice displayed reduced microvascular density (MVD) determined by IHC with anti-
100 relation 0.610, P = 0.0033) and pretreatment microvascular density (MVD) in all patients (Spearman co
101 was performed through the imaged tissue, and microvascular density (MVD) was determined, together wit
102 rast medium between K(PS) and the histologic microvascular density (MVD), an angiogenesis indicator.
103 l proliferation and differentiation in PBGs, microvascular density (MVD), and hypoxia.
104 sets (i.e., radiomic imaging features, tumor microvascular density (MVD), and vascular endothelial gr
105                                              Microvascular density (MVD), collagen content and cellul
106 in full-thickness left ventricular sections, microvascular density (MVD), myocardial fibrosis, and th
107 helial markers that can be used to calculate microvascular density (MVD).
108 gher RPS15A expression demonstrated a higher microvascular density (MVD).
109 ib alone were stained with CD31 to determine microvascular density (MVD).
110                   Neither reduced functional microvascular density nor major alterations in arterial
111 l CT sections were examined to measure tumor microvascular density, number of luminal vessels, vascul
112                                          The microvascular density of bone marrow did not change sign
113 n the CHF group suggests that a reduction in microvascular density of skeletal muscle may precede oth
114 tudy changes in the marrow vasculature using microvascular density or quantifying changes in the vasc
115  of distant tumor growth, proliferation, and microvascular density (P < .01).
116    This was accompanied by a 33% increase in microvascular density (p = 0.001) and a 36% decrease in
117 inent feature of TSP2-null mice is increased microvascular density, particularly in connective tissue
118  dihydrotetrabenazine, insulin staining, and microvascular density patterns were consistent with isle
119                                              Microvascular density (percentage of vessel area divided
120 ein, acidic, and rich in cysteine) < 5%, and microvascular density quartile 4.
121             CBF was strongly correlated with microvascular density (R = 0.66, P < .001).
122                     Peak MCI correlated with microvascular density (r=0.59, P<0.001) and capillary ar
123 ronary collaterals correlated with increased microvascular density, reduced fibrosis, and decreased l
124 rm neuronal survival, increased peri-infarct microvascular density, reduced microglia/macrophage accu
125                                     Afferent microvascular density +/- standard deviation in LA lesio
126                                           LA microvascular density tended to be lower in HFpEF and in
127 erative phenotype and showed lower pulmonary microvascular density than wild-type rats.
128  of (68)Ga-NODAGA-c(RGDfK) was correlated to microvascular density, vascular morphology, and permeabi
129 not affect the vascular parameters including microvascular density, vascular size, and vascular archi
130              The correlation between CBF and microvascular density was analyzed in specimens stained
131                                              Microvascular density was compared between control (n=6)
132 eated eyes by image processing software, and microvascular density was determined by counting von Wil
133                      Likewise, distant tumor microvascular density was greater for 5-W MWA and RFA (P
134                                              Microvascular density was reduced significantly in CLI c
135                     A significant overlap in microvascular density was seen between segments with and
136                                 Histological microvascular density was significantly correlated to UL
137                                              Microvascular density was significantly diminished by th
138                                   Mean tumor microvascular density was significantly lower in SU5416-
139                                          The microvascular density was significantly reduced in the x
140 epithelial COX-2 and iNOS protein levels and microvascular densities were determined by image analysi
141 cells demonstrated that mast cell number and microvascular density were significantly higher in S-P t
142  endothelial cell apoptosis and reduction of microvascular density within the core of the tumor leadi
143 lls, mononuclear cells), blood clotting, and microvascular density within the tumors produced by subc

 
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