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1 han patients without transplants, suggesting microvascular disease.
2 thrombosis, inflammation, dyslipidemia, and microvascular disease.
3 al infarction, and 26% (14-36; p<0.0001) for microvascular disease.
4 ere from patients with APS with a history of microvascular disease.
5 low (hMBF) is complicated by diffuse CAD and microvascular disease.
6 sure complete limb salvage because of severe microvascular disease.
7 e theory of aging, premature senescence, and microvascular disease.
8 the human immunodeficiency virus (PWH) have microvascular disease.
9 bA1c are at higher risk of macrovascular and microvascular disease.
10 of macrovascular disease and 1,345 cases of microvascular disease.
11 acute manifestations of progressive cerebral microvascular disease.
12 ntolerance, electroretinographic defects, or microvascular disease.
13 he relationship between retinal and systemic microvascular disease.
14 he retinal vasculature in the study of brain microvascular disease.
15 rve mobility in elderly people with cerebral microvascular disease.
16 the kidney, which, in turn, is the result of microvascular disease.
17 >10 years] diabetes) to assess the impact of microvascular disease.
18 sequence more closely associated with strial microvascular disease.
19 Diabetes increases the risk for microvascular disease.
20 ovide evidence of its complicity in diabetic microvascular disease.
21 ts in the genetic contribution to macro- and microvascular disease.
22 einaemia and AD could be linked by stroke or microvascular disease.
23 erence in susceptibility to diabetes-induced microvascular disease.
24 (stent) <2.0, a normal rCVR supported global microvascular disease.
25 vents most end-stage complications caused by microvascular disease.
26 and the management of patients with cardiac microvascular diseases.
27 ssive weight is a well-known risk factor for microvascular diseases.
28 mechanisms for the onset and progression of microvascular diseases.
29 diabetes-related end point (9%, P=0.04) and microvascular disease (24%, P=0.001), and risk reduction
30 trials have reported absolute reductions in microvascular disease (3.5%), such as retinopathy and ne
32 de insights into the temporal development of microvascular disease across other systemic vascular bed
33 ral artery disease or microvascular disease, microvascular disease alone was associated with a 3.7-fo
35 arvovirus B19 (B19V) is a common pathogen in microvascular disease and cardiomyopathy, owing to infec
36 and software have allowed identification of microvascular disease and introduced new risk categories
39 s hold promise for the treatment of diabetic microvascular disease and peripheral artery disease but
41 lications in women with type 1 or 2 diabetic microvascular disease and the risk factors for worsening
42 an intriguing positive relationship between microvascular disease and treatment with statins and tac
43 t be a useful screening test for generalized microvascular disease and, if detected, might reasonably
45 s-related end point, diabetes-related death, microvascular disease, and stroke in the group receiving
46 e of inflammation and the onset of diabetes, microvascular diseases, and periodontal pathologies.
51 Recent studies highlight the importance of microvascular disease, autoimmunity, and fibroblast diff
52 oth epicardial (beta = -0.57, p < 0.001) and microvascular disease (beta = -0.60, p < 0.001) on stepw
55 on, stroke, peripheral vascular disease, and microvascular disease) by the randomised glycaemic contr
56 emic manifestations (RVCL-S) is an incurable microvascular disease caused by C-terminus truncation of
57 ng to the following classification: coronary microvascular disease (CMD group) if CFR<2.5 and referen
60 cardiac disease is associated with coronary microvascular disease (CMD), where narrowing of the smal
61 arteries groups with no MB: 1 with coronary microvascular disease (CMD: coronary flow reserve, <2.5)
63 llateral blood flow, plaque characteristics, microvascular disease, coronary vasomotor tone, and gene
66 arge cohort of veterans to determine whether microvascular disease diagnosed in any location increase
67 allel increases in bAPV and hAPV, "acquired" microvascular disease due to distal microembolization or
68 white matter injury associated with cerebral microvascular disease extends well beyond what can be id
69 nd haemodynamics are indicative of potential microvascular diseases for patients with symptoms of cor
71 obin level) is related to the development of microvascular disease; however, the relation of glycosyl
72 biopsy revealed more advanced grade C and D microvascular disease in 45% and 36% of the patients, re
73 ymal mineral deposition and intraparenchymal microvascular disease in addition to previously reported
74 ibition might be a novel strategy to prevent microvascular disease in diabetes and other diseases.
77 of glomerular, tubulointerstitial, and renal microvascular disease in individuals with sub-Saharan Af
80 isease and the risk factors for worsening of microvascular disease in pregnancy using a prospective p
81 response, which may lead to exacerbation of microvascular disease in susceptible patients, such as p
82 a, and hence accelerated atherosclerosis and microvascular disease in T2DM, obesity, and related synd
83 lar mechanisms underlying the development of microvascular disease in the memory centers of the brain
84 (NO) and free radicals in the development of microvascular disease in type 1 diabetes remains unclear
85 of age and is commonly associated with other microvascular disease, including nephrosclerosis and dia
86 of traditional risk factors, the presence of microvascular disease increases the risk of amputation a
90 al to the development of diabetic macro- and microvascular disease is endothelial dysfunction, which
94 inflammation in the development of diabetic microvascular diseases is still unclear, it is likely th
95 burden of diabetes as a cause of macro- and microvascular disease linked to the epidemics of obesity
96 Often, overlooked factors, such as coronary microvascular disease, malnutrition, and poor physical p
97 tes, supporting the hypothesis that cerebral microvascular disease may contribute to their observed a
99 h those without peripheral artery disease or microvascular disease, microvascular disease alone was a
101 c cardiomyopathy (HCM), myocyte disarray and microvascular disease (MVD) have been implicated in adve
103 s, including cardiovascular diseases (CVDs), microvascular diseases (MVDs), and hypoglycemia, were as
104 res of cerebrovascular disease because brain microvascular disease occurs gradually and insidiously.
105 t the importance of BP on the progression of microvascular disease of the brain, which has been assoc
109 ion, acute coronary syndrome and stroke) and microvascular disease (peripheral neuropathy, nephropath
110 his model may be applied to conditions where microvascular disease plays a major pathogenic role.
112 Skin AGEs are robust long-term markers of microvascular disease progression, emphasizing the impor
117 ticipants had preexisting cardiovascular and microvascular disease, respectively; mean HbA1c level wa
118 ression, we analyzed the effect of prevalent microvascular disease (retinopathy, neuropathy, and neph
119 en MRI findings and four findings of retinal microvascular disease: retinopathy, focal arteriolar nar
121 rization, such as restenosis, calcification, microvascular disease, silent embolization, and tools fo
122 To date, the drug approach to prevention of microvascular disease starting with pre-diabetes has not
123 For individuals with one, two, or three microvascular disease states versus none, the multivaria
124 for age, sex, and conditions associated with microvascular disease, such as hypertension, hyperlipide
125 lesterolemia, T2D-macrovascular disease, T2D-microvascular disease, T2D-neuropathy, T2D-carpal-tunnel
126 ress, plays an important role in the retinal microvascular disease that is characteristic of diabetic
128 r; however, in individuals with diabetes and microvascular disease these cells are dysfunctional.
131 combination of peripheral artery disease and microvascular disease was associated with a 22.7-fold (9
133 roups, and no significant risk reduction for microvascular disease was observed for metformin therapy
134 art transplant patients without suspicion of microvascular disease who underwent stress cardiac MRI w
135 The evidence-based reduction in risk of microvascular disease with glucose lowering has resulted
136 ation of serum proteins as a result of brain microvascular disease would account for the perivascular