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1 han patients without transplants, suggesting microvascular disease.
2  thrombosis, inflammation, dyslipidemia, and microvascular disease.
3 al infarction, and 26% (14-36; p<0.0001) for microvascular disease.
4 ere from patients with APS with a history of microvascular disease.
5 low (hMBF) is complicated by diffuse CAD and microvascular disease.
6 sure complete limb salvage because of severe microvascular disease.
7 e theory of aging, premature senescence, and microvascular disease.
8  the human immunodeficiency virus (PWH) have microvascular disease.
9 bA1c are at higher risk of macrovascular and microvascular disease.
10  of macrovascular disease and 1,345 cases of microvascular disease.
11 acute manifestations of progressive cerebral microvascular disease.
12 ntolerance, electroretinographic defects, or microvascular disease.
13 he relationship between retinal and systemic microvascular disease.
14 he retinal vasculature in the study of brain microvascular disease.
15 rve mobility in elderly people with cerebral microvascular disease.
16 the kidney, which, in turn, is the result of microvascular disease.
17 >10 years] diabetes) to assess the impact of microvascular disease.
18 sequence more closely associated with strial microvascular disease.
19              Diabetes increases the risk for microvascular disease.
20 ovide evidence of its complicity in diabetic microvascular disease.
21 ts in the genetic contribution to macro- and microvascular disease.
22 einaemia and AD could be linked by stroke or microvascular disease.
23 erence in susceptibility to diabetes-induced microvascular disease.
24 (stent) <2.0, a normal rCVR supported global microvascular disease.
25 vents most end-stage complications caused by microvascular disease.
26  and the management of patients with cardiac microvascular diseases.
27 ssive weight is a well-known risk factor for microvascular diseases.
28  mechanisms for the onset and progression of microvascular diseases.
29  diabetes-related end point (9%, P=0.04) and microvascular disease (24%, P=0.001), and risk reduction
30  trials have reported absolute reductions in microvascular disease (3.5%), such as retinopathy and ne
31                                              Microvascular disease, a characteristic of acute and chr
32 de insights into the temporal development of microvascular disease across other systemic vascular bed
33 ral artery disease or microvascular disease, microvascular disease alone was associated with a 3.7-fo
34                       The role of myocardial microvascular disease among individuals with HDP and lef
35 arvovirus B19 (B19V) is a common pathogen in microvascular disease and cardiomyopathy, owing to infec
36  and software have allowed identification of microvascular disease and introduced new risk categories
37                         The role of coronary microvascular disease and its impact on functional and e
38              The benefits of fenofibrate for microvascular disease and its potential role in combinat
39 s hold promise for the treatment of diabetic microvascular disease and peripheral artery disease but
40                   Future research on retinal microvascular disease and retinal biomarkers might addit
41 lications in women with type 1 or 2 diabetic microvascular disease and the risk factors for worsening
42  an intriguing positive relationship between microvascular disease and treatment with statins and tac
43 t be a useful screening test for generalized microvascular disease and, if detected, might reasonably
44 atherosclerosis, hyperglycemia, which causes microvascular disease, and hypertension.
45 s-related end point, diabetes-related death, microvascular disease, and stroke in the group receiving
46 e of inflammation and the onset of diabetes, microvascular diseases, and periodontal pathologies.
47  transplant patients and the determinants of microvascular disease are not known.
48              Both coronary macrovascular and microvascular disease are prognostically important.
49                                     Cerebral microvascular disease associated with type 2 diabetes ma
50 ion to the pathogenesis of macrovascular and microvascular diseases associated with old age.
51   Recent studies highlight the importance of microvascular disease, autoimmunity, and fibroblast diff
52 oth epicardial (beta = -0.57, p < 0.001) and microvascular disease (beta = -0.60, p < 0.001) on stepw
53                          The effect of total microvascular disease burden on cardiovascular disease r
54 mination showed significant improvement when microvascular disease burden was added to models.
55 on, stroke, peripheral vascular disease, and microvascular disease) by the randomised glycaemic contr
56 emic manifestations (RVCL-S) is an incurable microvascular disease caused by C-terminus truncation of
57 ng to the following classification: coronary microvascular disease (CMD group) if CFR<2.5 and referen
58                                     Coronary microvascular disease (CMD) causes myocardial ischemia i
59                                     Coronary microvascular disease (CMD), characterized by impaired c
60  cardiac disease is associated with coronary microvascular disease (CMD), where narrowing of the smal
61  arteries groups with no MB: 1 with coronary microvascular disease (CMD: coronary flow reserve, <2.5)
62                   Chronic hyperglycaemia and microvascular disease contribute to cognitive dysfunctio
63 llateral blood flow, plaque characteristics, microvascular disease, coronary vasomotor tone, and gene
64                                              Microvascular disease correlated positively with donor a
65                                              Microvascular disease, diabetic foot, cerebrovascular di
66 arge cohort of veterans to determine whether microvascular disease diagnosed in any location increase
67 allel increases in bAPV and hAPV, "acquired" microvascular disease due to distal microembolization or
68 white matter injury associated with cerebral microvascular disease extends well beyond what can be id
69 nd haemodynamics are indicative of potential microvascular diseases for patients with symptoms of cor
70      Pre-diabetes has only a minor impact on microvascular disease; glucose-lowering drugs can delay
71 obin level) is related to the development of microvascular disease; however, the relation of glycosyl
72  biopsy revealed more advanced grade C and D microvascular disease in 45% and 36% of the patients, re
73 ymal mineral deposition and intraparenchymal microvascular disease in addition to previously reported
74 ibition might be a novel strategy to prevent microvascular disease in diabetes and other diseases.
75                                              Microvascular disease in diabetes extends to the brain.
76 impair insulin sensitivity and contribute to microvascular disease in diabetes mellitus.
77 of glomerular, tubulointerstitial, and renal microvascular disease in individuals with sub-Saharan Af
78                         An increased risk of microvascular disease in NPH versus basal after 3 OGLDs,
79       We hypothesized that preexisting brain microvascular disease in patients with diabetes might pa
80 isease and the risk factors for worsening of microvascular disease in pregnancy using a prospective p
81  response, which may lead to exacerbation of microvascular disease in susceptible patients, such as p
82 a, and hence accelerated atherosclerosis and microvascular disease in T2DM, obesity, and related synd
83 lar mechanisms underlying the development of microvascular disease in the memory centers of the brain
84 (NO) and free radicals in the development of microvascular disease in type 1 diabetes remains unclear
85 of age and is commonly associated with other microvascular disease, including nephrosclerosis and dia
86 of traditional risk factors, the presence of microvascular disease increases the risk of amputation a
87                                              Microvascular disease increases with donor age.
88                            Diabetes-specific microvascular disease is a leading cause of blindness, r
89                      We investigated whether microvascular disease is associated with amputation in a
90 al to the development of diabetic macro- and microvascular disease is endothelial dysfunction, which
91  the drug approach will delay development of microvascular disease is in dispute.
92        If one of the key mechanisms of brain microvascular disease is leakage of serum proteins into
93             We then show that dose-dependent microvascular disease is seen in a transgenic mouse mode
94  inflammation in the development of diabetic microvascular diseases is still unclear, it is likely th
95  burden of diabetes as a cause of macro- and microvascular disease linked to the epidemics of obesity
96  Often, overlooked factors, such as coronary microvascular disease, malnutrition, and poor physical p
97 tes, supporting the hypothesis that cerebral microvascular disease may contribute to their observed a
98             To determine the added effect of microvascular disease, measurements of flow reserve were
99 h those without peripheral artery disease or microvascular disease, microvascular disease alone was a
100 miological terms, with a higher incidence of microvascular disease-mostly retinopathy.
101 c cardiomyopathy (HCM), myocyte disarray and microvascular disease (MVD) have been implicated in adve
102 ect, microhemorrhages, white matter changes, microvascular disease (MVD), and volume loss).
103 s, including cardiovascular diseases (CVDs), microvascular diseases (MVDs), and hypoglycemia, were as
104 res of cerebrovascular disease because brain microvascular disease occurs gradually and insidiously.
105 t the importance of BP on the progression of microvascular disease of the brain, which has been assoc
106                       Affected patients have microvascular disease of the kidneys, heart, and brain.
107  and mostly because of an intrinsic cerebral microvascular disease of unknown cause.
108            Such events may arise from spasm, microvascular disease, or other pathways.
109 ion, acute coronary syndrome and stroke) and microvascular disease (peripheral neuropathy, nephropath
110 his model may be applied to conditions where microvascular disease plays a major pathogenic role.
111                                     Cerebral microvascular disease predominantly affects brain white
112    Skin AGEs are robust long-term markers of microvascular disease progression, emphasizing the impor
113             Ischemia in AS is not related to microvascular disease; rather, it is driven by abnormal
114                   The Coronary Slow-flow and Microvascular Diseases Registry [MICAT]; NCT02180178).
115                  The benefits of fibrates on microvascular disease remain to be fully explored.
116                                  Subcortical microvascular disease represented by brain white matter
117 ticipants had preexisting cardiovascular and microvascular disease, respectively; mean HbA1c level wa
118 ression, we analyzed the effect of prevalent microvascular disease (retinopathy, neuropathy, and neph
119 en MRI findings and four findings of retinal microvascular disease: retinopathy, focal arteriolar nar
120                     The cumulative burden of microvascular disease significantly affects the risk of
121 rization, such as restenosis, calcification, microvascular disease, silent embolization, and tools fo
122  To date, the drug approach to prevention of microvascular disease starting with pre-diabetes has not
123      For individuals with one, two, or three microvascular disease states versus none, the multivaria
124 for age, sex, and conditions associated with microvascular disease, such as hypertension, hyperlipide
125 lesterolemia, T2D-macrovascular disease, T2D-microvascular disease, T2D-neuropathy, T2D-carpal-tunnel
126 ress, plays an important role in the retinal microvascular disease that is characteristic of diabetic
127              This limits early evaluation of microvascular diseases that originate in capillaries.
128 r; however, in individuals with diabetes and microvascular disease these cells are dysfunctional.
129                     There was no evidence of microvascular disease until 12 months, when trypsin dige
130                                              Microvascular disease violates the integrity of the bloo
131 combination of peripheral artery disease and microvascular disease was associated with a 22.7-fold (9
132                                     Coronary microvascular disease was classified by light microscopy
133 roups, and no significant risk reduction for microvascular disease was observed for metformin therapy
134 art transplant patients without suspicion of microvascular disease who underwent stress cardiac MRI w
135      The evidence-based reduction in risk of microvascular disease with glucose lowering has resulted
136 ation of serum proteins as a result of brain microvascular disease would account for the perivascular
137                                 First, brain microvascular disease would not be recognized by traditi

 
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