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1 after birth and evaluated them regularly for middle-ear effusion.
2 days of age and evaluated them regularly for middle-ear effusion.
3 days of age and evaluated them regularly for middle-ear effusion.
4  Gly-Gly peptide-encoding gene in chinchilla middle ear effusions.
5 ET) in infants limits or delays clearance of middle ear effusions.
6 o and persistence in the planktonic phase in middle-ear effusions.
7 y culture, may be the source of endotoxin in middle-ear effusions.
8 ime with middle ear effusion and episodes of middle ear effusion and AOM in children.
9 genetic component to the amount of time with middle ear effusion and episodes of middle ear effusion
10 bacteria exist in culture-negative pediatric middle-ear effusions and that experimental infection wit
11  of MGAS5005 Deltasrv were isolated from the middle ear effusion, and MGAS5005 Deltasrv was found ran
12 e healthy young children who have persistent middle-ear effusion, as defined in our study, prompt ins
13 n impactions to eustachian tube dysfunction, middle ear effusion, cochlear malformations, and conduct
14                                              Middle ear effusion disappeared 2.0 weeks (13.7 days) ea
15 significant percentage of culturally sterile middle ear effusions, establishing the presence of viabl
16                                              Middle-ear effusion fluid was obtained from 12 patients
17  from the nasopharynx of healthy children or middle ear effusions from patients with otitis media, re
18 raxella catarrhalis (detected by PCR) in 106 middle-ear effusions from pediatric patients with chroni
19 ients still have persistent symptoms such as middle ear effusion in COME after intensive antibiotic t
20  children have been attributed to persistent middle-ear effusion in their early years of life.
21 f antimicrobial treatment on the duration of middle ear effusion (MEE) and concomitant hearing impair
22 o detect the smaller amount and variation of middle ear effusion (MEE) and middle ear bacterial biofi
23  composition (microbiota) present in matched middle ear effusion (MEE) samples, external ear canal (E
24 hamella) catarrhalis, which were detected in middle-ear effusion (MEE) samples taken from children wi
25  low-passage NTHi clinical isolates from the middle ear effusions of patients with chronic otitis med
26 f AOM; the estimated proportion of time with middle ear effusion; or the utilization of selected heal
27  younger than 3 years of age with persistent middle-ear effusion, prompt as compared with delayed ins
28  to explain the failure to culture NTHi from middle-ear effusions, recalcitrance to antibiotics and i
29 t, the estimate of heritability of time with middle ear effusion was 0.73 (P<.001).
30 tes of discordance for 3 or more episodes of middle ear effusion were 0.04 for monozygotic twins and
31                      Bacterial counts of the middle ear effusions were lower in the vaccine groups th
32                                              Middle ear effusions were recovered by epitympanic tap a
33 e years of age, 429 children with persistent middle-ear effusion were randomly assigned to have tympa
34 in the chinchilla, inducing culture-positive middle ear effusions, whereas pgm and siaB mutants were
35  than three years of age who have persistent middle-ear effusion within the duration of effusion that