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1 and for restricting auricle expansion at the midrib.
2  veins, with the majority localized near the midrib.
3 al signal propagating between both lobes and midrib.
4 ic reinitiation of blade primordia along the midrib.
5 ly from the margins of the leaves toward the midrib.
6                                        Brown midrib 12 (bmr12) encodes the sorghum caffeic acid O-met
7                           In addition, brown midrib 12-ref (bmr12-ref), a nonsense mutation in the so
8 ion of two major CAD isoforms, SbCAD2 (Brown midrib 6 [bmr6]) and SbCAD4, in lignifying tissues of so
9 riven by embolism initiating in petioles and midribs across all species, and Kx vulnerability was str
10 rlier that the electrical stimulus between a midrib and a lobe closes the Venus flytrap upper leaf wi
11 es is to accumulate glucosinolates along the midrib and at the margin.
12 E plants developed wider leaves with thicker midrib and enlarged palisade parenchyma cells.
13          Mild leaf phenotypes have a reduced midrib and may be moderately narrow and furcated; severe
14 also displayed ectopic lignification in leaf midribs and elevated concentrations of soluble phenolic
15                                         Leaf midribs and stems are light green with sectors of dark g
16 city occurred acropetally, with the class I (midrib) and class II veins becoming functional in phloem
17   The wood phenotype resembles that of brown midrib (bm) mutants and some transgenic plants in which
18                   The midribs of maize brown midrib (bm) mutants exhibit a reddish-brown color associ
19                               The four brown midrib (bm) mutants of maize have a reduced content and
20                                        Brown-midrib (bm) mutants of maize have modified lignin of red
21                                    The brown midrib (bm) mutations of maize affect the biosynthesis o
22 ce showing that a set of three allelic brown midrib (bmr) lignin mutants contained mutations in this
23                                        Brown midrib (bmr) mutants in sorghum (Sorghum bicolor (L.) Mo
24                We utilized the sorghum brown midrib (bmr) mutants, which are impaired in monolignol s
25 and one of these traits of interest is Brown midrib (BMR).
26  of the inner layer of the petals and in the midrib by providing a qualitatively different paradigm t
27 evelopment at the ligular region of the leaf midrib by transforming blade to sheath.
28 nsitivity of K(leaf) to damage; severing the midrib caused K(leaf) and gas exchange to decline, with
29 gle electrical charge between a lobe and the midrib causes closure of the trap and induces an electri
30 ity was strongly correlated with petiole and midrib conduit dimensions.
31 y abnormal patterns of cell expansion in the midrib cortex and in the epidermis of the elongation zon
32 sity of primary veins predicted tolerance of midrib damage.
33           One of these cultivars was a brown-midrib double mutant (DM) which had reduced levels of li
34 djustment of endogenous leaf auxin levels on midrib elongation and final leaf size (fresh weight and
35 ential at which 50% of the xylem in the leaf midrib embolized across leaves.
36 NA-based detection strategies utilizing leaf midribs for sampling.
37 ion of inverse bainite, namely the cementite midrib formation, ferrite formation, secondary cementite
38 al tip leads to premature termination of the midrib into a knot and leaf bifurcation.
39  this assertion, we observe that maize brown midrib mutants affected in lignin biosynthesis are hyper
40 s, genetic and biochemical analyses of brown midrib mutants of maize, sorghum and related grasses hav
41               The levels of cad2 mRNA in the midribs of bm1-das1 and bm1-ref are reduced by 91 and 86
42                                          The midribs of maize brown midrib (bm) mutants exhibit a red
43                Support cells within the leaf midribs of mosses deposit cellulose-rich secondary cell
44                      The cellulose-deficient midribs of ppcesa3/8 knockouts provided negative control
45 ered completely recessive, because the brown midrib phenotype is only apparent in plants homozygous f
46 uses a blade-to-sheath transformation at the midrib region of the maize (Zea mays L.) leaf.
47 e, auricle and ligule into sheath around the midrib region.
48             Maule staining of stalk and leaf midrib sections from SbF5H overexpression lines indicate
49 ed strong negative correlations between leaf midrib thickness and pathogenicity level in sorghum and
50 gion (UTR), conferring the spontaneous brown midrib trait and lignin reduction in the sorghum germpla
51                                     When the midrib was severed near the leaf base, the pinnately vei
52                  Thickness of leaf blade and midrib were recorded separately.
53  frequency generation spectra collected from midribs were consistent with cellulose microfibril aggre
54 um (NPLE) type growth kinetics for cementite midrib, whereas a transition in growth kinetics from Par
55 ndary cell wall cellulose deposition in leaf midribs, whereas the two members of each class are redun
56 l turgor, along with higher vein density and midrib xylem per leaf area, and a higher ratio of K(leaf