ライフサイエンスコーパス: migraine with aura

1 lence of self-reported migraine was 16% (13% migraine with aura).

2 ]) were significantly higher than those with migraine with aura.

3 d analogous to, those found in patients with migraine with aura.

4 mpared to controls, similar to patients with migraine with aura.

5 in migraine pathophysiology, particularly in migraine with aura.

6 1 (FHM1), a rare monogenic subtype of common migraine with aura.

7 miplegic migraine (FHM) is a rare subtype of migraine with aura.

8 hemiplegic migraine and in one patient with migraine with aura.

9 pression (CSD) is a key pathogenetic step in migraine with aura.

10 ma, and seizure phenotype in this variant of migraine with aura.

11 scular neurons that underlie the headache of migraine with aura.

12 cations such as cerebral emboli, stroke, and migraine with aura.

13 ising acute treatment for some patients with migraine with aura.

14 portable sTMS device for acute treatment of migraine with aura.

15 en ovale (PFO) is prevalent in patients with migraine with aura.

16 nce of right-to-left shunts in patients with migraine with aura.

17 between high oestrogen states and attacks of migraine with aura.

18 that cannot easily be controlled, including migraine with aura.

19 association between right-to-left shunts and migraine with aura.

20 ynamic changes during spontaneous attacks of migraine with aura.

21 bands, there appears to be an excess risk of migraine with aura.

22 mong the participants with migraine, 103 had migraine with aura, 180 chronic migraine, and 88 were ic

23 Migraine (59%), migraine with aura (27%), anxiety and depression were co

24 170 subjects were enrolled (56 migraine with aura, 61 migraine without aura, 53 control

25 assified into no history of migraine, active migraine with aura, active migraine without aura, and pa

26 4.7 [7.1] years), among whom 1435 (5.2%) had migraine with aura and 26 423 (94.8%) did not (2177 [7.8

27 tantial external noise-exclusion deficits in migraine with aura and a minor impairment of noise exclu

28 a) was identified in non-hemiplegic familial migraine with aura and advanced sleep phase syndrome.

29 A relation between migraine with aura and cardiac right-to-left shunts has

30 e analyzed 16 families for co-segregation of migraine with aura and chromosome 19p13 markers.

31 We studied patients with migraine with aura and healthy controls with 31P-MRS and

32 t evidence exists on the association between migraine with aura and increased incidence of cardiovasc

33 disorder, appears genetically distinct from migraine with aura and is linked to 22q12.

34 ith two diagnostic types of migraine, termed migraine with aura and migraine without aura, from the I

35 omponents in the two main migraine subtypes: migraine with aura and migraine without aura.

36 ine (FHM) and more common types of migraine, migraine with aura and migraine without aura.

37 bout the role of spreading depolarization in migraine with aura and models of spreading depolarizatio

38 o difference in PFO prevalence in those with migraine with aura and those without (26.8% versus 26.1%

39 by seven individuals who had previously had migraine with aura and three who had previously had migr

40 s a pathophysiological process implicated in migraine with aura and various other brain pathologies,

41 ing headaches of intracranial origin such as migraine with aura and why this therapeutic approach may

42 ld-type mice, providing a novel mechanism in migraine with aura and, by extension, the other neurolog

43 From age 12, she regularly suffered from migraines with auras and photophobia.

44 patients (88.9%) had chronic migraine; 4 had migraine with aura, and 5 had migraine without aura.

45 cutaneous Closure of Patent Foramen Ovale in Migraine With Aura] and PREMIUM [Prospective Randomized

46 e prospective cohort suggest that women with migraine with aura are at increased risk of experiencing

47 ding depolarization to the headache phase of migraine with aura, as it can activate trigeminal nocice

48 Eight others who had had migraine with aura before closure reported improvement i

49 Early treatment of migraine with aura by sTMS resulted in increased freedom

50 Migraine with aura can be associated with allodynia and

51 legic migraine type 1 (FHM1) is a subtype of migraine with aura caused by a gain-of-function mutation

52 Familial history of stroke, migraine with aura, circulating antiphospholipid antibod

53 Willis was significantly more common in the migraine with aura compared to control group (73% vs. 51

54 lysis revealed overlapping ADC elevations in migraine with aura compared with controls (p = 0.0069).

55 Self-reported migraine with aura compared with migraine without aura o

56 ificant decrease in D0, D1, and D2 values in migraine with aura, especially in the deep capillary ple

57 Patients who suffered from migraine with aura, experienced frequent migraine attack

58 ouse model for a severe monogenic subtype of migraine with aura, familial hemiplegic migraine type 3

59 perimeter were significantly enlarged in the migraine with aura group.

60 without aura (Group 1), 45 patients who had migraines with aura (Group 2), and 30 healthy participan

61 essionals aged at least 45 years, women with migraine with aura had a higher adjusted incidence rate

62 Patients with migraine with aura had a higher risk for incident RAO co

63 perience a TIA or stroke, women who reported migraine with aura had adjusted relative risk (95% confi

64 migraine history, women who reported active migraine with aura had multivariable-adjusted hazard rat

65 Migraine with aura has been associated with an adverse c

66 Although migraine with aura has been associated with increased ri

67 Although migraine with aura has many causes (eg, neuronal network

68 tical inflammation in migraine, particularly migraine with aura, has been a subject of considerable i

69 Migraine-specifically migraine with aura-has been identified as a risk factor

70 Individuals with migraine with aura have an elevated vascular risk, neces

71 People with migraine with aura have an increased risk of atrial fibr

72 SS, and simultaneously comparable to that of migraine with aura, highlighting a shared biology betwee

73 This is consistent with the hypothesis that migraine with aura in midlife is associated with late-li

74 Migraine with aura in midlife was associated with late-l

75 associated with both hemiplegic migraine and migraine with aura in patients.

76 d three risk variants that seem specific for migraine with aura (in HMOX2, CACNA1A and MPPED2), two t

77 In a young man with migraine with aura including hemiplegia, we identified a

78 Here, using a rat model of migraine with aura involving cortical spreading depolari

79 Migraine with aura is a common but poorly understood sen

80 These findings support the concept that migraine with aura is a heterogeneous disorder with dist

81 Migraine with aura is a severe debilitating neurological

82 with increased risk of all types of RAO and migraine with aura is associated with increased risk of

83 Migraine with aura is associated with increased risk of

84 Migraine with aura is characterized by recurrent attacks

85 We suggest that migraine with aura is initiated by waves of CSD that lea

86 Migraine with aura is known to increase the risk of card

87 Migraine with aura is often the first manifestation of c

88 of the higher risk of stroke in people with migraine with aura, it is important to identify and modi

89 lations to study the main migraine subtypes, migraine with aura (MA) and migraine without aura (MO).

90 Migraine with aura (MA) is a prevalent neurological cond

91 Migraine with aura (MA) is associated with cardiovascula

92 es showing intergenerational transmission of migraine with aura (MA).

93 ombined hormone contraceptive use in MRM and migraine with aura may decrease both headache frequency

94 Migraine with aura may respond differently to therapies

95 Higher occipital qT2 in migraine with aura might represent either extracellular

96 Compared with migraine with aura, migraine without aura was independen

97 Migraine with aura, migraine without aura, and control s

98 pression (CSD) underlies the neurobiology of migraine with aura (MWA).

99 aine patients ([MIG], n = 25, 15 of whom had migraine with aura [MwA]).

100 ore per month (n = 3243), those with midlife migraine with aura (n = 361) had an increased risk of la

101 OR, 7.01 [95% CI, 4.43-11.09]; P < .001), or migraine with aura (n = 66; 69.7% vs 26.5%; OR, 5.73 [95

102 Migraine with aura occurs in up to 20-30% of all migrain

103 al and non-neurological conditions including migraine with aura (odds ratio = 12.24, 95%CI: 2.54-35.2

104 iplegic migraine type-3 (FHM3), a subtype of migraine with aura, of which CSD is the neurophysiologic

105 thout aura to chromosome 14, and a locus for migraine with aura on chromosome 4.

106 three SNP associations was preferential for migraine with aura or without aura, nor were any associa

107 classified as having migraine without aura, migraine with aura, or nonmigraine headache.

108 K(+) (TRESK) channel has been identified in migraine with aura patients in a large pedigree.

109 Migraine with aura patients showed significantly reduced

110 atients, and compared with recordings from 8 migraine-with-aura patients and 6 normal controls during

111 additional major CVD events attributable to migraine with aura per 10 000 women per year.

112 cremental increase in the incidence rate for migraine with aura ranged from 1.01 additional cases per

113 nd toward increased spatial heterogeneity in migraine with aura, reflected by lower b values.

114 Attacks of migraine with aura represent a phenomenon in which abnor

115 of the burden of circle of Willis variants, migraine with aura subjects had a higher burden of varia

116 ncomplete circle of Willis is more common in migraine with aura subjects than controls, and is associ

117 q)) exhibited a consistent downward shift in migraine with aura, suggesting global architectural simp

118 Ten migraine with aura, ten migraine without aura, and ten a

119 1 (FHM1) is an autosomal dominant subtype of migraine with aura that is associated with hemiparesis.

120 contributes to the genetic susceptibility of migraine with aura that is distinct from the FHM locus.

121 In migraine with aura, the higher qT2 was more widespread a

122 Studies have linked migraine with aura to an increased risk of ischemic stro

123 The absolute contribution of migraine with aura to CVD incidence in relation to other

124 Three (one migraine with aura, two migraine without aura) reported

125 hemiplegic migraine type 1 (FHM1), a severe migraine with aura variant, is caused by mutations in th

126 (prevalence of infarcts 23.0% for women with migraine with aura vs 14.5% for women not reporting head

127 was 3.36 (95% CI, 2.72-3.99) for women with migraine with aura vs 2.11 (95% CI, 1.98-2.24) for women

128 a 19.3% prevalence of infarcts for men with migraine with aura vs 21.3% for men not reporting headac

129 s large, prospective cohort of women, active migraine with aura was associated with increased risk of

130 The incidence rate for women with migraine with aura was significantly higher than the adj

131 duals had to meet international criteria for migraine with aura, with visual aura preceding at least

132 le with migraine is strongly associated with migraine with aura, young age, female sex, use of oral c