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1 s was used to assess risk factors for OAG in migraineurs.
2 n increased risk of cardiovascular events in migraineurs.
3 nderlie the increased cardiovascular risk in migraineurs.
4 re three significant risk factors for OAG in migraineurs.
5 reased circulating levels of CGRP in chronic migraineurs.
6 otions was stronger in control subjects than migraineurs.
7 the interaction of CGRP with its receptor in migraineurs.
8 ell as enhanced vulnerability to ischemia in migraineurs.
9 e increased risk for vascular diseases among migraineurs.
10 lved to a greater degree in female than male migraineurs.
11 he heart in 1.437 patients of which 337 were migraineurs.
12 aine with aura occurs in up to 20-30% of all migraineurs.
13               Severe CA occurred in 20.4% of migraineurs.
14 ormal cerebral cortical energy metabolism in migraineurs.
15 nitroglycerin [glyceryl trinitrate (GTN)] to migraineurs.
16 rmed during spontaneous visual auras in four migraineurs.
17 cribed the general health characteristics of migraineurs.
18 iety-related disorders often seen in chronic migraineurs.
19  pain modulation correlated with TRP only in migraineurs.
20 nsidered similar between migraineurs and non-migraineurs.
21 nd structural MRI data were obtained from 20 migraineurs, 20 cluster headache patients, and 15 health
22 ance images of the brain were acquired in 63 migraineurs and 18 matched healthy control subjects.
23 n-based genome-wide analysis including 5,122 migraineurs and 18,108 non-migraineurs, rs2651899 (1p36.
24                      We included 102 chronic migraineurs and 77 age- and sex-matched individuals free
25 nt functional differences in male and female migraineurs and a sex-specific pattern of functional con
26 s delayed migraine attacks when infused into migraineurs and also causes iNOS expression and delayed
27                                     Studying migraineurs and control subjects, we found that lights t
28    H(2)15O-labelled PET was used to study 24 migraineurs and eight healthy controls.
29 estigated changes of after-image duration in migraineurs and healthy controls (HCs) and throughout th
30 la and precuneus cortices compared with male migraineurs and healthy controls of both sexes.
31 emale age-matched interictal (migraine free) migraineurs and matched healthy controls to determine al
32              Cortical abnormalities occur in migraineurs and may represent the results of a balance b
33 ications could be considered similar between migraineurs and non-migraineurs.
34 rmeability of the blood-brain barrier in six migraineurs and six control subjects at rest and during
35 -years) was 1.29 and 1.02, respectively, for migraineurs and the comparison cohort during the 10-year
36  compare the 10-year risk of OAG between the migraineurs and the comparison cohort.
37 lycerin (NTG, a reliable migraine trigger in migraineurs) and found that both Ccl2 and Ccr2 mRNA were
38                                              Migraineurs appeared prone to abnormally high variabilit
39 ordinates of decreased grey matter volume in migraineurs as seed regions to generate resting state fu
40                                              Migraineurs avoid light because it intensifies their hea
41 e affects approximately 50% to 60% of female migraineurs, but knowledge regarding the role of hormone
42 e following thalamic nuclei were observed in migraineurs: central nuclear complex (F(1,233) = 6.79),
43    Forty-seven episodic (EMs) and 39 chronic migraineurs (CMs; interictal) were compared to 34 HCs fo
44 ons on either interictal or ictal changes in migraineurs compared with controls, largely because of m
45 nd to determine if the metrics obtained from migraineurs could be differentiated from controls.
46        This binding pattern was identical in migraineurs during glyceryl trinitrate-induced migraine
47 ay explain the increased frequency of VMS in migraineurs during menopausal transition.
48                                              Migraineurs experience debilitating headaches that resul
49                                              Migraineurs experience hyperresponsivity to visual stimu
50 ine is a subjective phenomenon, and what the migraineur experiences is necessarily inaccessible to ot
51 mes and vibrotactile detection thresholds of migraineurs failed to differentiate them from controls,
52 001) in both migraine groups than in the non-migraineur group.
53                Our results demonstrated that migraineurs had higher TRP compared to controls.
54                                              Migraineurs had more vascular comorbidities than the com
55                                       Female migraineurs had thicker posterior insula and precuneus c
56 iting increases in brain NO concentration in migraineurs have not yet been identified, although, anim
57 rual migraine affects about 20-25% of female migraineurs in the general population, and 22-70% of pat
58                            CA affects 63% of migraineurs in the population and is associated with fre
59 uate if the increased cardiovascular risk in migraineurs is attributed to an increased coronary arter
60 w that regions of grey matter volume loss in migraineurs localize to a common brain network defined b
61           Homocysteine levels were higher in migraineurs [mean (SD) 12.8 (5.6) versus 9.8 (3.4) micro
62 cing physical discomfort, and high-frequency migraineurs most likely.
63                                              Migraineurs (n = 11,388) completed the Allodynia Symptom
64        In this study, we included 61 chronic migraineurs otherwise healthy treated with OnabotA of wh
65 s including 5,122 migraineurs and 18,108 non-migraineurs, rs2651899 (1p36.32, PRDM16), rs10166942 (2q
66 excitatory/inhibitory imbalance that renders migraineurs susceptible to an attack.
67 ons akin to spreading depression predisposes migraineurs to infarction during mild ischemic events, t
68 ed the risk for open angle glaucoma (OAG) in migraineurs using a 10-year follow-up study that employe
69 ive and somatosensory processing pathways in migraineurs versus controls.
70 as used to assess between-group comparisons (migraineurs vs control subjects, the aura effect, the ef
71            The prevalence of allodynia among migraineurs was 63.2%.
72                                          The migraineurs were divided into three groups according to
73 or patients' age and vascular comorbidities, migraineurs were found to have a 1.68-fold (95% confiden
74 reased cerebral vulnerability to ischemia in migraineurs, whereas there is also evidence of blood-bra
75 l functional and structural abnormalities in migraineurs, which may contribute to hyperexcitability a
76 altered excitatory/inhibitory equilibrium in migraineurs, which oscillates over the migraine cycle.
77  is relevant to spontaneous migraine in many migraineurs, whose symptoms of cranial allodynia are res
78                                     Finally, migraineurs with a history of allodynia exhibit signific
79                            We compared adult migraineurs with aura (MA; n = 187), without aura (MO; n
80 ce of high noise levels, with performance of migraineurs with aura significantly poorer (P < 0.05) th
81 te versus the pain-free state when comparing migraineurs with controls.
82  regions, and anterior cingulate compared to migraineurs without allodynia.
83                                              Migraineurs without aura also showed a significant (P <
84 raine patients relative to healthy controls, migraineurs without vestibular symptoms and patients wit