ライフサイエンスコーパス: migration

1 een proposed as a mechanism driving directed migration.

2 e are multiple ways to generate variation in migration.

3 tribute to the regulation of collective cell migration.

4 its Rho activity to enhance directional cell migration.

5 nasal mucosa and subsequent GnRH-1 neuronal migration.

6 ivation of the GTPase and for efficient cell migration.

7 o metabolize stored lipids and fuel invasive migration.

8 itic cells perform a random walk by amoeboid migration.

9 wave and influences the foraging benefits of migration.

10 way activated by eotaxin-1 during eosinophil migration.

11 in intergroup relations in this age of mass migration.

12 atform for progenitor cell proliferation and migration.

13 cal GAP activity rescues cell morphology and migration.

14 when birds stopover during spring and autumn migration.

15 ncluding the regulation of cell adhesion and migration.

16 emporal mobilization of Ca(2+) to drive cell migration.

17 ive CP state and exhibit directional lateral migration.

18 r the RAP1/RAC1 signaling axis in HNSCC cell migration.

19 ed to pose a problem to efficient DNA bubble migration.

20 d as a regulator of mechanically guided cell migration.

21 followed drought-altered green waves during migration.

22 salmon in common domains during their marine migration.

23 ranging from enzymatic reactions to species migration.

24 phery localized FMN2 is required for spindle migration.

25 tive tool for characterization of neutrophil migration.

26 d high-content time-resolved imaging of cell migration.

27 -linked, ras-like protein that promotes cell migration.

28 nd various aspects of cell proliferation and migration.

29 eristics reminiscent of interkinetic nuclear migration.

30 DPP4 blocked dexamethasone-induced THP1-MPhi migration.

31 f neuroepithelia during interkinetic nuclear migration.

32 tial for cell survival, differentiation, and migration.

33 ing cell's neurosphere-forming capacity, and migration.

34 ed BMP6 expression in ECs was involved in EC migration.

35 onclassical carbocation followed by anti-1,2-migration.

36 e slowed by disrupting the machinery of cell migration.

37 ociated with increased HSC death and reduced migration.

38 essary and sufficient to orient both nuclear migrations.

39 l to many concepts in ecology [1], including migration [2, 3].

40 her regions, which indicated past and recent migration across regions.

41 xin-1-induced TrkA activation and eosinophil migration, additively with 1-NM-PP1, indicating a role f

42 mportant developmental gene involved in cell migration, adhesion, and morphogenesis.

43 sity to migrate, orientation and distance of migration all map to a small number of genomic regions t

44 yclic changes in pressure and unsteady fluid migration along faults.

45 address this, we use Drosophila border cell migration, an invasive, collective migration that occurs

46 The combination with cell migration analysis and traction force microscopy shows a

47 tein-analysis method (protein topography and migration analysis platform) to attempt to identify subs

48 y in vivo via regulation of HSC survival and migration and affect the immune microenvironment, especi

49 the innovative strategies of restricting the migration and agglomeration of single metal sites into m

50 or Rho-GAP activity and for suppressing cell migration and anchorage-independent growth.

51 sorting through concentration dependent cell migration and apoptosis, independent of the morphogen tr

52 tion to couple phagocytic processing to cell migration and facilitate space exploration by immune cel

53 ht on the interplay between tectonics, basin migration and faunal change on the one hand and the fate

54 gate the case of population subdivision with migration and find that the common claim that conformity

55 tivity is required in vivo for directed cell migration and for maintaining normal levels of F-actin [

56 lly, we found preliminary evidence of recent migration and gene flow in one of the largest persisting

57 Cell migration and glucose uptake assays combined with protei

58 T cell effector function, migration and glycolytic responses were amplified in LXA

59 Further, silencing furin reduced tumor cell migration and invasion in vitro and tumor growth and met

60 demonstrated to reduce viability and inhibit migration and invasion of 4T1 murine breast cancer cells

61 sis, and we showed that TTCC blockers reduce migration and invasion rates because of autophagy blocka

62 1 (AQP1) dual water and ion channels enhance migration and invasion when upregulated in leading edges

63 strong inhibitory effects on cell mobility (migration and invasion) through anti-glycolysis and pro-

64 es with increased epithelial to mesenchymal, migration and invasion, and metastasis.

65 ured gene expression, binding activity, cell migration and invasion, and transcriptional activity of

66 and Lgals3bp, directly involved in leukocyte migration and invasion, were significantly upregulated a

67 rate that glutamine deprivation promotes CAF migration and invasion, which in turn facilitates the mo

68 atenin nuclear translocation to promote cell migration and invasion.

69 and is necessary and sufficient for RMS cell migration and invasion.

70 cytoskeletal dynamics to promote tumor cell migration and invasion.

71 pean blackcap exhibits enormous variation in migration and is renowned for research on its evolution

72 hat JNK signaling facilitates the tangential migration and laminar deposition of cortical interneuron

73 To describe migration and life history timing, we deployed light-lev

74 erized organic heterostructures shows energy migration and light-harvesting across the interfaces of

75 In the absence of Mitf function, cNCC migration and localization in the optic cup are perturbe

76 cells after lung metastasis have the fastest migration and lowest stiffness, with a significantly hig

77 lity to urban settings with high rates of in-migration and mobility and to Zambia and SA.

78 Only VEGF-A stimulated HUVEC migration and proliferation whereas both GW0742 and VEGF

79 tion of organogenesis, differentiation, cell migration and proliferation.

80 importance of the interfacial oxygen vacancy migration and redistribution in controlling the electron

81 ed to the health system challenges of forced migration and refugee-related health including the healt

82 alysis revealed poverty reduction, increased migration and remittances, food security, and maternal n

83 ce that FASN activity directly promotes cell migration and supports FASN as a potential therapeutic t

84 demonstrate that Ift20 is required for HF-SC migration and their contribution to epidermal regenerati

85 structures that are resistant to both charge migration and unfolding.

86 characterized by a complex history of human migrations and population interactions.

87 stance founders were particularly suited for migration, and 3) whether recently established nonmigran

88 ce unique factors prior to migration, during migration, and after migration that shape their health.

89 e known to influence cellular proliferation, migration, and angiogenesis.

90 ies, increasing at higher latitudes and with migration, and decreasing with territoriality.

91 -sexual dichromatism, age and sex-structured migration, and delayed plumage maturation.

92 ntal processes such as neural proliferation, migration, and differentiation.

93 Cellular integrity, migration, and genome-wide gene expression changes were

94 significant factor in cell differentiation, migration, and in the case of cancer, metastatic progres

95 ) play important roles in cell polarization, migration, and invasion and are markedly upregulated in

96 ls (NSCs) proliferation and differentiation, migration, and maturation of their progenies were evalua

97 the vDG showed a prolonged differentiation, migration, and maturation.

98 nts of the household, including composition, migration, and mortality.

99 ty is important for neuritogenesis, neuronal migration, and neurodevelopment.

100 gation during mitosis, cell polarization and migration, and primary cilia formation.

101 diated the glucocorticoid-induced macrophage migration, and siRNA-mediated knockdowns of GR and DPP4

102 gnaling promotes keratinocyte proliferation, migration, and survival.

103 ignaling pathways to coordinate cell growth, migration, and the formation of the extracellular matrix

104 proliferation, development, differentiation, migration, and treatment efficacy.

105 +) ECs demonstrated decreased proliferation, migration, and tubule formation, and Cept1Lp/LpCre (+) m

106 stimulation of MoS(2) on integrins, cellular migration, and wound healing.

107 dly contractile, some polarize to facilitate migration, and yet others exhibit mixtures of these resp

108 The pre- and post-division migrations are dependent on microtubules and actin, resp

109 k measures, and the coverage of a single out-migration area of origin.

110 FCW66 were shown to inhibit MDA-MB-231 cell migration as effectively as ST3GALIII-gene knockdown did

111 ht into its effect on cellular adherence and migration, as well the basis of TMTC3-associated Cobbles

112 ow two-state micropatterns provide a dynamic migration assay with distinct dwell times and relative c

113 d 1-octanol were detected by sniffers, after migration assay, with high modified frequency (between 5

114 layers of different cells in an antagonistic migration assay.

115 everal advantages over traditional transwell migration assays and its design is amenable to future in

116 ained by genuine Neanderthal ancestry due to migrations back to Africa, predominately from ancestral

117 rent and expected stages, aiming to quantify migration behavior and decision mode for different migra

118 ical confinement can variably influence cell migration behaviors, and it is presently unclear whether

119 -dependent swimming and actin-dependent cell migration, both of which are used by animal cells and un

120 d glycolysis accompanies collective cellular migration but show, further, that this shift occurs thro

121 the results suggest that Abi1 regulates cell migration by affecting Pfn-1 and N-WASP, but not pVASP,

122 nd produces a drastic reduction of Cu(+) ion migration by nearly two orders of magnitude.

123 ibited eotaxin-1 (CCL11)-mediated eosinophil migration, calcium flux, cell polarization, and ERK1/2 a

124 en dubbed "superspreaders" of infection, but migration can also reduce parasite burdens within host p

125 recognized as an important regulator of cell migration, cancer cell invasion, and vesicular content r

126 VEGF inhibition, including genes involved in migration, chemotaxis, and cell adhesion as well as indu

127 However, cell migration chromatography, the sorting of large populatio

128 We apply the model to Chinese labor migration data at the current and expected stages, aimin

129 l, and knockdown of Cep85l causes a neuronal migration defect in mice.

130 rexpression of Pfn1 successfully rescued the migration defects of HuR-deleted neurons.

131 Despite this, migration defects of WNK1-deficient thymocytes do not ac

132 cholesterol depletion (-27.8%) lowered VSMC migration distance on a fibronectin (FN)-coated surface

133 n timing rather than latitude, elevation, or migration distance such that sympatric ecotypes often sh

134 idual plasticity (individuals decrease their migration distances over their lifetime) and generationa

135 Changes in outdoor PM(2.5) exposure due to migration drove 137.1 (95%CI: 93.2, 179.4) premature dea

136 l activation and neutrophil transendothelial migration during AAA formation.

137 ses the question of the significance of bird migration during past periods with different patterns of

138 blation of IFT20 in vitro slows keratinocyte migration during wound healing.

139 mmigrants experience unique factors prior to migration, during migration, and after migration that sh

140 , in turn determining excitonic behavior and migration dynamics.

141 combinant CXCL11 significantly induced tumor migration, epithelial-to-mesenchymal transition, and mat

142 ivergence times, effective population sizes, migration events) to the msprime coalescent simulator by

143 adhesion is important for many developmental migration events.

144 The concentration of these compounds in migration exceeded the maximum level established by Regu

145 ich in turn, mirrors within-China population migration flows to a large extent.

146 Typically, ion migration for crystal deformation or connection with oth

147 workers globally as forced displacement and migration from countries with high rates of this practic

148 nsient absorption spectra reveal the carrier migration from low n to high n species with different ki

149 nced cytolytic potential and requires T cell migration from lymph nodes for therapeutic efficacy.

150 ric origin of the OFG and active groundwater migration from onshore are inferred.

151 It was more challenging to trace migrations from metastases back to primary tumors.

152 Unexpectedly, concomitant to a poleward migration, future niche models suggest a considerable re

153 ,5-H shift and instead activates 1,5-methoxy migration, giving methoxy-migrated indenone, with the 1,

154 ntly unclear whether the mechanisms used for migration in 2D unconfined environments are relevant in

155 ificantly reduced cell growth and suppressed migration in A549 and NCI-H1944 cells, accompanied by re

156 alt homeostasis, also regulates adhesion and migration in CD4(+) T cells.

157 ing cell proliferation, differentiation, and migration in developing organs.

158 sion and positively regulates CXCL12-induced migration in DN thymocytes.

159 d functional changes including, adhesion and migration in EOC-derived fibroblasts that suggest the de

160 nd their receptors are orchestrators of cell migration in humans.

161 S data and utilize it to quantify excitation migration in LHCII trimers.

162 at different MYH9-RD mutations suppressed MK migration in the BM without compromising bipolar filamen

163 Natural gas migration in the subsurface can have environmental impli

164 nd gives fresh insights into controlling ion migration in these ionic-transport-dominated materials.

165 Other drivers (e.g. human demography and migration in tropical and subtropical regions) were also

166 and immune cells based on their spontaneous migration in two-dimensional and three-dimensional micro

167 taurin or bosutinib significantly suppressed migration in vitro and in zebrafish models.

168 related to calcification, proliferation, and migration in VSMCs isolated from 151 multiethnic heart t

169 Macrophage migration inhibitory factor (MIF) has been recognized as

170 al role of the versatile cytokine macrophage migration inhibitory factor (MIF) in regulating a metabo

171 The alternative endogenous ligand macrophage migration inhibitory factor behaves opposite to CXCL12 i

172 s) but also to increase their activation and migration into lymphoid tissues and the pancreas.

173 ortion of organic matter to survive vertical migration into saturated and anaerobic zones of peatland

174 arge fast-growing population by impeding its migration into the patch, while a small fast-growing pop

175 is is required for proper GABAergic neuronal migration into the substantia nigra pars reticulata.

176 Effects on cell migration, invasion, and cytoskeletal organization in vi

177 ithelial-to-mesenchymal transition, cellular migration, invasion, angiogenesis, stemness, transcripti

178 metastasis by regulating cell proliferation, migration, invasion, drug resistance, and epithelial-mes

179 Cell migration involves dynamic changes in cell shape.

180 Windborne migration is a key strategy enabling exploitation of eph

181 Temporary labor migration is an increasingly important mode of migration

182 Collective cell migration is central to many developmental and pathologi

183 Successful apical nuclear migration is critical for planar-orientated cell divisio

184 Cell migration is driven by local membrane protrusion through

185 Smooth muscle cell migration is essential for many diverse biological proce

186 is highly expressed in thymocytes, and since migration is important for thymocyte maturation, we inve

187 Although T cell migration is most frequently defined in the context of t

188 e gap closure model in which collective cell migration, large-scale actin-network fusion, and purse-s

189 hibitors of metastasis must account for each migration mode.

190 Mammalian cells developed two main migration modes.

191 ve been used extensively for tracking animal migrations, most commonly small birds.

192 infection-related mechanisms that can favour migration: moving to escape versus recover from infectio

193 We find the type-specific migration network differs significantly for migrants wit

194 actively on their destination, the resulting migration network emerges naturally as an Nash equilibri

195 ormation, existing theories suggest that gas migration occurs via capillary invasion and/or initiatio

196 te moiety was performed by N -> O phosphinyl migration of aminophosphines borane by heating at 50 deg

197 We reported the EF-directed migration of both rat Schwann cells (SCs) and oligodendr

198 3A did not have any effect on the growth and migration of cancer cells nor did it induce receptor dow

199 Coordinated directional migration of cells in the mesoderm layer of the early em

200 of mutations in subcellular pathways on the migration of cells within the colorectal crypt.

201 en found in other parts of the world, due to migration of chronically infected patients.

202 ic double layer, which blocks the long-range migration of Cu(+) and produces a drastic reduction of C

203 The migration of each of these radionuclide analogs (RAs) wa

204 atory cytokine production, and intracellular migration of early-stage inflammatory elements.

205 ey player in promoting the proliferation and migration of ECs that are critical in the pathogenesis o

206 stance (336.7 Omega cm(2)) that promotes the migration of electrons and interfacial charge separation

207 hibition of ABC transporter activity reduces migration of GSCs towards low concentrations of S1P in v

208 Nevertheless, maturation and migration of LC lacking E-cadherin was not altered, neit

209 uid interface, it was possible to follow the migration of live M. bovis Bacille Calmette-Guerin (BCG)

210 phorylation, the expression of Vcan, and the migration of lung mesenchyme fibroblasts, and suggest th

211 ng hair placode morphogenesis and diminished migration of melanoblasts.

212 estricting motility and preventing efficient migration of microglia to sites of neuronal damage.

213 The temperature-dependent migration of molecular weight protein size standards and

214 lls, facilitating increased transendothelial migration of monocytes.

215 pressure-driven flow and DC electric field, migration of particles inside microfluidic channels exhi

216 monocytes, and their activation promotes the migration of phagocytes to sites of infection.

217 the importance of Pfn1 in proliferation and migration of RCC cells and in soluble Pfn1's involvement

218 eurite outgrowth of diabetic DRG neurons and migration of Schwann cells challenged by high glucose.

219 bserved with C(5)F(5)N, which results in the migration of the fluorinated pyridine moiety to the carb

220 ovides geological evidence for the northeast migration of the Hadar Basin, extending the record of th

221 Wolves may follow the winter migration of their staple prey, moose (Alces alces), to

222 neuroblastoma, potentiates the invasion and migration of transformed sympathetic neuroblasts, and dr

223 mposition influenced LC and caused selective migration of triglycerides, resulting in higher proporti

224 tion, which then decreased proliferation and migration of tumor cells in different in vitro 2D and 3D

225 ice optimally (1) enhances the sprouting and migration of tumor spheroids, (2) promotes angiogenesis,

226 limb-innervating motor neurons and abnormal migration of V2a interneurons.

227 of this compound coupled with uniquely easy migration of water molecules between the layers allows f

228 ely related and underlies the idea that cell migration operates by a common set of physical principle

229 lation composition as a consequence of these migrations, or with changes in climate.

230 vices leads to a steric effect to impede the migration pathways of ions to increase the activation en

231 and raise questions concerning understudied migration pathways, such as the Sicilian route.

232 gnificantly contributed to a decrease in the migration pattern of patient-derived GBM cells by modula

233 re, we investigated the origin, division and migration patterns of the adult-born neural progenitor (

234 dies, we document climatic influences on the migration phenology of eagles, geographic differences in

235 ese results suggest that the consequences of migration policy should play a greater part in deliberat

236 that enables precise control of the C=C bond migration position, in both cyclic and acyclic systems,

237 Knowledge about migration potential is key to forecasting species distri

238 RhoU and Cdc42 that directly influences cell migration potential.

239 sed platform alters cell behavior, including migration, proliferation, and paracrine activity, which

240 echanisms governing PKA activity during cell migration remain largely unknown.

241 tic architecture and neurogenetics of animal migration remain poorly understood.

242 nd maintenance of EGFR signaling during cell migration remains limited.

243 he deep sea, potentially mirroring the great migrations so well characterized in terrestrial systems.

244 ally reveals an intercellular variability in migration speeds.

245 luated the association between family member migration status and cognitive decline; future work shou

246 arverius) to investigate the effects of sex, migration strategy, population density, weather, year an

247 lements are as follows: the "treatment stage migration" strategy, which allows moving to another trea

248 tly different FAD mean lifetimes and greater migration than monocultures at 24, 48, and 72 hours post

249 u(V) sources exhibited significantly greater migration than previous studies using Pu(IV) or Pu(III)

250 but also suggest alternative routes of virus migration that are plausible within the epidemiological

251 gration is an increasingly important mode of migration that generates substantial remittance flows, b

252 sis of cell protrusion formation during cell migration that is regulated by subcellular mitochondrial

253 rder cell migration, an invasive, collective migration that occurs during Stage 9 of oogenesis.

254 or to migration, during migration, and after migration that shape their health.

255 s displayed defects in polarization and cell migration that were rescued by uncoupling VE-cadDEE from

256 ology-dependent pai transmission and exciton migration; these key fundamental pai functions are uniqu

257 and cultured samples, and we propose nuclear migration through bridges maintains this nuclear heterog

258 ht-sheet microscope geometry, including cell migration through confined spaces within a microfluidic

259 examine their effects on breast cancer cell migration through exosome-mediated delivery.

260 induces leader cell activity and collective migration through the engagement of the adenosine recept

261 key parameter in comparison to the relative migration time (RMT) approach.

262 tage of the host brain and is limited by the migration time and the increasing size of the developing

263 The average migration times and %RSD for K(+), Na(+), and Li(+) were

264 based on federal criteria, was influenced by migration timing rather than latitude, elevation, or mig

265 We found novel progenitor migrations, timings, dynamic cell-cell interactions, sig

266 Finally, VR23 effectively reduces neutrophil migration, TNF-alpha secretion, and tissue inflammation

267 led over into wild birds and spread via bird migration to countries in Europe, Africa, and North Amer

268 l physiology, ranging from cell polarity and migration to cytokinesis.

269 The aryl group undergoes 1,2-migration to give tert-alpha-arylated aldehydes (as acet

270 ays chemotactic signals to direct neutrophil migration to inflamed sites through its receptor BLT1.

271 Tumor cell migration to LECs was inhibited by blocking CXCL11 where

272 e oxidative metabolism during the process of migration to metabolize stored lipids and fuel invasive

273 which influences the amount and type of fat migration to particle surface resulting in varying wetta

274 d phase separation but rather facilitates Y6 migration to the BHJ film top surface, changes the PBDB-

275 ord sheath segmentation, altering osteoblast migration to the developing spine, and increasing sensit

276 otoreceptor states cause microglial dominant migration to the subretinal space as a protective respon

277 UVPD fragments enabled monitoring of charge migration to the unfolded regions.

278 mouse and human neutrophils inhibited their migration toward CXCL2 and CXCL8, respectively, and this

279 signaling axis coordinates neighboring cell migration toward oncogenic cells and is required for onc

280 s (TCRs) show increased cytokine production, migration toward tumor cells, and tumor cell killing.

281 m, designed to mimic the tight extracellular migration tracts in brain parenchyma, allowed high-conte

282 rtfolio, maintaining a broad distribution in migration traits still increased adult production and re

283 Here, we quantify dendritic cell migration under well-defined 2-dimensional confinement a

284 eta1 integrin in focal adhesions during cell migration using confocal microscopy and total internal r

285 cytotoxicity, proliferation, redox state and migration using mouse embryonic fibroblast Balb/3T3, hum

286 Through real-time tracking of Li(+) migration using operando electron energy-loss spectrosco

287 changes in cell shapes, traction forces, and migration velocities have yet to penetrate.

288 f cell shape and polarity, cytokinesis, cell migration, vesicle trafficking, and receptor signaling.

289 HIV migration was more frequent from San Diego county toward

290 orticoid-induced eosinopenia and bone marrow migration were consistent with those of the induction of

291 some instances of potential tangential cell migration were noted.

292 bundler T-Plastin to promote protrusions and migration when adhesion is spatially-gapped.

293 polycrystalline perovskites suffer from ion migration, which causes overshoot of luminance over time

294 ons to increase the activation energy of ion migration, which is demonstrated through multiple theore

295 hRNA reduced human airway smooth muscle cell migration, which was restored by Abi1 rescue.

296 se resource gain, many herbivores pace their migration with the flush of nutritious plant green-up th

297 ransepithelial resistance but increased cell migration, with increased expression of extracellular ma

298 ns to simulate the past 50,000 years of bird migration worldwide, a period encompassing the transitio

299 es cellular proliferation, colony formation, migration, wound healing, tumor growth, and lung metasta

300 enotype that nevertheless undergo retrograde migration, yet remain durably committed to the residency