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1 rasitica (P.p.) races Emoy2 and Emwa1 (downy mildew).
2 stresses (watermelon mosaic virus and downy mildew).
3 h levels of quantitative resistance to downy mildew.
4 s upon inoculation with a nonadapted powdery mildew.
5 interactions between Arabidopsis and powdery mildew.
6 R1, and fully enhanced resistance to powdery mildew.
7 asal and RPW8-mediated resistance to powdery mildew.
8 6S(l)#3, which confer resistance to powdery mildew.
9 a role that has been appropriated by powdery mildew.
10 confer broad-spectrum resistance to powdery mildew.
11 in penetration success by the barley powdery mildew.
12 to NHR in Arabidopsis against wheat powdery mildew.
13 rimary foliar diseases, Botrytis and powdery mildew.
14 ild type in response to infection by powdery mildew.
15 siphe cichoracearum, causal agent of powdery mildew.
16 that confers enhanced resistance to powdery mildew.
17 ated, EHM-focused resistance against powdery mildew.
18 entified as a susceptibility factor to downy mildew.
19 hesis and cell wall modification for powdery mildew.
20 NLS to it resulted in resistance to powdery mildew.
21 he corresponding avirulence genes in powdery mildew.
22 ng and extent of outbreaks of cucurbit downy mildew.
23 exhibit biological activity against powdery mildews.
24 s of uninucleate zoospores in multiple downy mildews.
25 re-invasive immune responses against powdery mildews.
26 putative chromatin regulator Enhanced Downy Mildew 2 (EDM2) as a cellular antisilencing factor and r
28 many angiosperms, the RESISTANCE TO POWDERY MILDEW 8 (RPW8)-CC domain containing NLR (RNL) subclass
32 Arabidopsis thaliana RESISTANCE TO POWDERY MILDEW 8.2 (RPW8.2) is specifically targeted to the EHM
33 and molecular epidemiology of wheat powdery mildew, a disease caused by the biotrophic fungus Blumer
36 a forward genetic screen with barley powdery mildew and consequently characterized it by pathogen ass
37 uding enhanced disease resistance to powdery mildew and enhanced ethylene-induced senescence; thus, t
39 silenced plants were more resistant to downy mildew and Soybean mosaic virus compared with vector con
41 eronospora parasitica (causal agent of downy mildew), and encodes an NBS-LRR type R protein with a pu
42 us Podosphaera pannosa, which causes powdery mildew, and the hemibiotrophic fungus Diplocarpon rosae,
43 es, those living in houses with pets, pests, mildew, and water damage, those whose parents had asthma
50 fer durable and robust resistance to powdery mildew (Blumeria graminis), a biotrophic fungal leaf pat
53 enetration resistance against barley powdery mildew, Blumeria graminis f. sp. hordei, and we isolated
54 resent the genome analysis of barley powdery mildew, Blumeria graminis f.sp. hordei (Blumeria), as we
55 ex, presence of water damage or visible mold/mildew, born in winter, breastfeeding, and being exposed
57 dozyma flocculosa for the control of powdery mildew by two Canadian research programs is presented.
58 escribe two barley (Hordeum vulgare) powdery mildew candidate secreted effector proteins, CSEP0105 an
61 des Dm3 which determines resistance to downy mildew caused by the oomycete Bremia lactucae carrying t
64 ly elusive 5' sequence of the barley powdery mildew chitin synthase gene, BgChs2, which includes a my
66 O2, MLO6 and MLO12 not only restrict powdery mildew colonization, but also affect interactions with a
68 associated with defense responses to powdery mildew disease but function in different signaling pathw
70 ment provided primed defence against powdery mildew disease caused by the biotrophic fungal pathogen,
71 of Arabidopsis confers resistance to powdery mildew disease caused by the fungus Erysiphe cichoracear
73 lex inheritance of resistance to the powdery mildew disease in the model plant Arabidopsis thaliana,
74 e rwa2 and tbr mutants also suppress powdery mildew disease resistance in pmr6, a mutant defective in
75 study suggests that the Arabidopsis powdery mildew disease will be a suitable model system in which
81 such as a recent outbreak of severe powdery mildew disease; furthermore, allergy to hazelnuts is an
83 and accession Ms-0 was resistant, to powdery mildew diseases caused by Erysiphe cruciferarum UEA1 and
88 otic and abiotic stresses, among which downy mildew (DM) is a severe biotic stress that is detrimenta
89 s sativus) is resistant to oomyceteous downy mildew (DM), bacterial angular leaf spot (ALS) and funga
93 rabidopsis thaliana is a host to the powdery mildew Erysiphe cichoracearum and nonhost to Blumeria gr
95 o the nonadapted pea (Pisum sativum) powdery mildew Erysiphe pisi However, PLC2-silenced plants displ
96 phenotype, conferring resistance to powdery mildew (Erysiphe cichoracearum) and enhancing ethylene-i
97 enhanced sensitivity to the pathogen powdery mildew (Erysiphe cruciferarum) and fails to induce SA bi
98 itional inappropriate biotrophs, pea powdery mildew (Erysiphe pisi) and potato late blight (Phytophth
101 I 1.14-2.04, and work environments with mold/mildew exposures [environments with possible exposures t
102 enic tomato variety resistant to the powdery mildew fungal pathogen using the CRISPR/Cas9 technology.
104 naling in nonhost resistance against powdery mildew fungi and put PLDdelta forward as the main isofor
105 on phenotype upon challenge with the powdery mildew fungi Golovinomyces orontii and Erysiphe pisi, th
106 ial and quality, the three rusts and powdery mildew fungi have historically caused major crop losses
109 s for viruses and for rust, smut and powdery mildew fungi that infect 473 plant species naturalized t
117 ed resistance against the biotrophic powdery mildew fungus (Blumeria graminis f.sp. hordei), while MO
118 a wide spectrum of resistance to the powdery mildew fungus (PM), Erysiphe necator (Schw.) Burr., but
120 nteraction of barley with the barley powdery mildew fungus Blumeria graminis f. sp. hordei (Bgh).
121 NHR in Arabidopsis against the wheat powdery mildew fungus Blumeria graminis f. sp. tritici (Bgt).
122 Arabidopsis thaliana) and the barley powdery mildew fungus Blumeria graminis f.sp. hordei (Bgh) was i
124 o enhance resistance of wheat to the powdery mildew fungus Blumeria graminis f.sp. tritici, a novel e
125 conferring resistance to the oomycete downy mildew fungus Bremia lactucae map to the major resistanc
126 responses to a host-adapted virulent powdery mildew fungus but were impaired in preinvasive defenses
127 he mitochondrial genome of the grape powdery mildew fungus Erysiphe necator and a high-quality mitoch
129 uring epiphytic growth, the cucurbit powdery mildew fungus Podosphaera xanthii expresses a family of
130 ntify AVR(a10) and AVR(k1) of barley powdery mildew fungus, Blumeria graminis f sp hordei (Bgh), and
134 also mediates defense to an adapted powdery mildew fungus, thus regulating a durable type of defense
135 ating Arabidopsis infection with the powdery mildew fungus, we find that the Arabidopsis atypical E2F
142 netration resistance to the virulent powdery mildew Golovinomyces cichoracearum due to enhanced callo
146 We evaluated the effects of grape powdery mildew (GPM) as supplemental food, and habitat structura
149 hronized with a higher expression of powdery mildew haustorial effectors, a sharp decline in the phot
150 PP2B confer recognition of Arabidopsis downy mildew (Hyaloperonospora arabidopsidis [Hpa]) isolate Ca
151 he 3221 mutant exhibited resistance to downy mildew (Hyaloperonospora arabidopsidis) and green peach
153 in R-gene-mediated resistance against downy mildew in Arabidopsis and their regulatory control by th
156 confers resistance to Bremia lactucae (downy mildew) in 1033 accessions of Lactuca serriola (prickly
157 ts exhibited a dramatic reduction in powdery mildew-induced chloroplast TAGs, attributable to decreas
158 ertion allele in DRP1E did not cause powdery mildew-induced lesions, suggesting that this phenotype i
159 protected wheat systemically against powdery mildew infection by affecting multiple steps in the life
160 rsensitive response (HR) to restrict powdery mildew infection via the salicylic acid-dependent signal
161 ransgenic lines during both types of powdery mildew infection, and neither the salicylic acid-depende
162 ) gene confer enhanced resistance to powdery mildew infection, enhanced senescence, and enhanced prog
168 ts profound effect on the outcome of powdery mildew infections: when the appropriate MLO protein is a
169 penetration by Bgh during the barley-powdery mildew interaction but is independent of gene-for-gene r
171 ude that edr1-mediated resistance to powdery mildew is mediated, in part, by enhanced ABA signaling.
172 e necator, the causal agent of grape powdery mildew, is a destructive pathogen of grapevines worldwid
173 ergent alleles of ATR1NdWsB from eight downy mildew isolates and demonstrate that the ATR1NdWsB allel
176 e logistic regression to investigate whether mildew/musty odour and increased concentrations of Alter
180 f Hyaloperonospora arabidopsidis (Hpa, downy mildew of Arabidopsis) contains at least 134 candidate R
183 errations and altered sensitivity to powdery mildews of rop6(DN) plants result from perturbations tha
185 l as from 5 related species that cause downy mildew on other hosts, the number of different bands det
187 iated enhanced disease resistance to powdery mildew or spontaneous lesions, indicating that these phe
188 te to pearl millet defense against the downy mildew pathogen by activating the expression of defense
189 d enhanced disease resistance to the powdery mildew pathogen Erysiphe cichoracearum and identified th
193 ced immunity (iri) mutants against the downy mildew pathogen Hyaloperonospora arabidopsidis, yielding
196 Hyaloperonospora arabidopsidis, a downy mildew pathogen of the model plant Arabidopsis, has been
197 Interactions between barley and the powdery mildew pathogen, Blumeria graminis f. sp. hordei, (Bgh)
206 solving among phylogenetically related downy mildew pathogens dramatically point out the need for sim
214 W8.2) is specifically induced by the powdery mildew (PM) fungus (Golovinomyces cichoracearum) in the
215 wth and reproduction of the virulent powdery mildew (PM) Golovinomyces orontii on Arabidopsis (Arabid
216 exhibited altered expression at the powdery mildew (PM) infection site, with subsets of these playin
220 standing genetic variation in wheat powdery mildew prior to the Pm17 introgression, thereby paving t
221 ntified and mapped three independent powdery mildew quantitative disease resistance loci, which act a
222 e MAP kinases AtMPK6 and AtMPK3, the powdery mildew R proteins RPW8.1 and RPW8.2, EDS1 and PR protein
223 inoculations with non-host and host powdery mildews relative to non-inoculated control plants accoun
224 report the map-based cloning of the powdery mildew resistance allele Pm5e from a Chinese wheat landr
225 tivar C.I. 16151 (harboring the Mla6 powdery mildew resistance allele) and its fast neutron-derived B
227 tive regulation on HR cell death and powdery mildew resistance by limiting the transcriptional amplif
228 omain is responsible for the gain of powdery mildew resistance function of Pm5e, an allele endemic to
229 barley (Hordeum vulgare), the Mla13 powdery mildew resistance gene confers Rar1-dependent, AvrMla13-
230 nctional characterization of Pm41, a powdery mildew resistance gene derived from WEW, which encodes a
232 , we report the map-based cloning of powdery mildew resistance gene Pm24 from Chinese wheat landrace
234 -mediated resistance to TMV and some powdery mildew resistance genes in barley provide the first exam
235 plant defense response initiated by powdery mildew resistance genes in chromosomes 3S(l)#2 and 6S(l)
238 osa might harbor new stripe rust and powdery mildew resistance genes, respectively, therefore, they c
240 that RPW8.2 from A. lyrata conferred powdery mildew resistance in A. thaliana, suggesting that RPW8.2
244 om Ethiopian landraces and nowadays controls mildew resistance in the majority of cultivated European
248 l resistance and effector-triggered, powdery mildew resistance locus A12-mediated resistance against
249 abidopsis thaliana), NORTIA, a member of the MILDEW RESISTANCE LOCUS O (MLO) family of proteins, play
251 enes encoding protein homologs of the barley mildew resistance locus o (MLO) protein biochemically sh
253 netically related Arabidopsis thaliana genes MILDEW RESISTANCE LOCUS O 4 (MLO4) and MLO11, encoding h
255 of the Mla locus can dictate similar powdery mildew resistance phenotypes yet still require distinct
257 Here, we report that the broad-spectrum mildew resistance protein RPW8.2 from Arabidopsis thalia
258 t region from the orthologous barley powdery mildew resistance protein, MLA10, but is similar to the
259 essions Wa-1, Kas-1, Stw-0 and Su-0, powdery mildew resistance was encoded by a semi-dominant allele.
261 n direct contrast to their increased powdery mildew resistance, both pmr5 and pmr6 plants are highly
269 nations of host resistance genes, designated Mildew-resistance locus (Ml), and cognate pathogen aviru
272 indicated that four loci, designated powdery mildew resistant 1-4 (pmr1-4), are defined by this colle
273 Surprisingly, here we found that powdery mildew resistant 4 (pmr4), a mutant lacking pathogen-ind
275 ection phenotypes of two independent powdery mildew-resistant triple mutant lines with a range of mic
276 genic Arabidopsis lines that express POWDERY MILDEW RESISTANT4 (PMR4), which encodes a stress-induced
281 endered Arabidopsis resistant to the powdery mildew species Erysiphe cichoracearum and Erysiphe oront
283 Erysiphe vaccinii is an emerging powdery mildew species that is impacting the blueberry industry.
286 o E. orontii, a very closely related powdery mildew, suggesting that a very specific resistance mecha
287 10) define a core set conserved in all three mildews, suggesting that most effectors represent specie
288 ed pmr5 seed selecting for increased powdery mildew susceptibility identified two previously characte
289 susceptible to the fungal pathogen, powdery mildew, than wild type as measured by conidiophore produ
290 pairs studied in detail using DNA from downy mildews that attack maize (P. maydis & P. philippinensis
291 ant surfaces among distantly related powdery mildews that is based on KCS6-derived wax components.
292 For plant pathogenic fungi, such as powdery mildews, that survive only on a limited number of host p
293 gen but rather a biocontrol agent of powdery mildews; this relationship makes it unique for the study
294 inoculated with the non-host barley powdery mildew to those inoculated with a virulent, host powdery
295 ommon parasite Peronospora parasitica (downy mildew) to study the evolution of resistance specificity
298 ell death and enhanced resistance to powdery mildew via the salicylic acid-dependent signaling pathwa
300 of three Phytophthora species and one downy mildew were identified and could serve as useful phyloge