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1 re, neutrophils directly bind to immobilized mindin, and mindin matrix mediates neutrophil migration
2                                    Moreover, mindin binds directly to bacteria and their components a
3                                              Mindin comprises an N-terminal F-spondin (FS) domain and
4                                              Mindin(-/-) DCs had reduced expression of Rac1/2 and imp
5 xpression restored the priming capability of Mindin(-/-) DCs.
6  influenza virus particles directly and that mindin-deficient macrophages exhibited impaired activati
7 ed ability to clear bacterial infection, and mindin-deficient macrophages show defective responses to
8        We found that intranasal infection of mindin-deficient mice by influenza virus resulted in dra
9 ontrast, lungs from intratracheally infected mindin-deficient mice contained similar influenza virus
10  MMP-10 and the extracellular matrix protein mindin (encoded by Spon2) in the diseased FVB/N Cd151 (-
11 rt we show that mice lacking the ECM protein mindin exhibit severely impaired recruitment of neutroph
12                                          The mindin FS domain therefore represents a new integrin lig
13 trated that the extracellular matrix protein mindin functions as a pattern recognition molecule for b
14                                Additionally, mindin functions as a pattern recognition molecule for i
15 Gene expression analysis showed that a human mindin homologue, mindin/RG-1, is expressed selectively
16       In this study, we examined the role of mindin in influenza virus infection.
17                  Given the dispensability of Mindin in normal tissue physiology, targeting this prote
18 n serves as a novel ligand for integrins and mindin-integrin interactions are critical for inflammato
19                   These results suggest that mindin-integrin interactions play a key role in regulati
20                               We showed that mindin interacted with influenza virus particles directl
21               The adhesion of neutrophils to mindin is blocked by anti-integrin alpha4, anti-integrin
22                                        Thus, mindin is essential in the initiation of the innate immu
23                                              Mindin is produced by SNAI1 transgenic skin keratinocyte
24                  Given the known function of mindin, its early expression in the diseased GBM could r
25 rins exhibit enhanced binding to immobilized mindin matrix and the increased binding can be blocked b
26 ils directly bind to immobilized mindin, and mindin matrix mediates neutrophil migration in vitro.
27     Furthermore, we show that DC adhesion to mindin matrix was blocked by antibodies to alpha4, alpha
28 cking beta1 integrin had reduced adhesion to mindin matrix, decreased expression of Rac1/2 and impair
29 o influenza virus infection and suggest that mindin may be used as an immune-enhancing agent in influ
30                                              Mindin(-/-) mice displayed defective CD4+ T-cell priming
31     Together, these results demonstrate that mindin plays an essential role in the host innate immune
32 dual interactions, the FS and TSR domains of mindin promote activation of both adaptive and innate im
33                       We further showed that mindin recognizes lipopolysaccharide (LPS) through its T
34 e show that the extracellular matrix protein mindin regulates the expression of Rho GTPases in DCs.
35                                 In contrast, mindin/RG-1 expression in other normal tissues is signif
36  human antibody, 19G9, was generated against mindin/RG-1 protein and was shown to accumulate at high
37                                              Mindin/RG-1 protein expression is maintained in >80% of
38 alysis showed that a human mindin homologue, mindin/RG-1, is expressed selectively in prostate tissue
39                     Our results suggest that mindin serves as a novel ligand for integrins and mindin
40 re, intranasal administration of recombinant mindin significantly enhanced the clearance of influenza
41  critical role for the matricellular protein Mindin (SPON2) in fibrogenesis.
42                   Mice lacking expression of mindin (spondin 2), a highly conserved ECM protein, have
43                                              Mindin (spondin-2) is an extracellular matrix protein of
44                               Interestingly, mindin was also detected in urinary samples of FVB/N Cd1