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1  and after their osteogenic differentiation (mineralized nodules).
2 n and alkaline phosphatase, and formation of mineralized nodules.
3 al cells undergo osteoblastogenesis and form mineralized nodules.
4 ent pericytes that were downregulated in the mineralized nodules.
5  by confluent cells and was downregulated in mineralized nodules.
6 and osteocalcin and accelerated formation of mineralized nodules.
7 yer of aplastic enamel with numerous ectopic mineralized nodules.
8 st cultures revealed a dramatic reduction in mineralized nodules, a significant reduction in Runx2, S
9 ed the area of the cell cultures occupied by mineralized nodules after 5 weeks.
10 amin C metabolites enhanced the formation of mineralized nodules and collagenous proteins.
11 ov in ST-2 cells inhibited the appearance of mineralized nodules and decreased alkaline phosphatase a
12 der osteogenic conditions, both lines formed mineralized nodules, and stained for alizarin red and al
13 ription factors and the development of fewer mineralized nodules, as compared with WT mice.
14 eased the proliferation of cementoblasts and mineralized nodules at all concentrations (p < 0.05).
15 how that thiazides increase the formation of mineralized nodules, but loop diuretics do not.
16 DL cell differentiation and the formation of mineralized nodules by PDL cells.
17 from SRC-2 KO mice formed significantly more mineralized nodules (by 3-fold) in the presence of the P
18 antly reduced bone mineral density and fewer mineralized nodules, coincident with increased expressio
19 steopontin were analyzed along with in vitro mineralized nodule formation and calcium accumulation.
20 amp1, Cog6 and Pacs1 significantly increased mineralized nodule formation and expression of osteoblas
21 3-E1 pre-osteoblasts recapitulated increased mineralized nodule formation and osteoblast differentiat
22 oted, whereas that of RCAS-Dlx5RH inhibited, mineralized nodule formation as compared with that in co
23                                  An in vitro mineralized nodule formation assay demonstrated that ove
24 alcin, osteonectin/osteopontin, and in vitro mineralized nodule formation compared to stimulation wit
25  alkaline phosphatase increases 50-fold with mineralized nodule formation enhanced 30-fold.
26 ial- and bone marrow-derived osteoblasts for mineralized nodule formation in vitro showed increased m
27 ressed bone formation parameters in vivo and mineralized nodule formation in vitro similarly to those
28 toward the osteoblast lineage as measured by mineralized nodule formation in vitro.
29 on labeling is a valid method for evaluating mineralized nodule formation on opaque surfaces.
30                                              Mineralized nodule formation on plastic using von Kossa
31  were significantly larger, and the onset of mineralized nodule formation was delayed when compared w
32                                    Increased mineralized nodule formation was suppressed by concurren
33 hatase activity, osteocalcin production, and mineralized nodule formation were increased.
34  alkaline phosphatase, increased matrix, and mineralized nodule formation when compared with untreate
35 amine the effects of P. gingivalis lipids on mineralized nodule formation, cell viability, apoptosis,
36 as reduced alkaline phosphatase activity and mineralized nodule formation.
37 in and alkaline phosphatase mRNA levels, and mineralized nodule formation.
38  phosphatase activity, Runx2 expression, and mineralized nodule formation.
39 ression of alkaline phosphatase and in vitro mineralized nodule formation.
40 nalysis techniques to micro-Raman spectra of mineralized nodules formed in vitro, we reveal cell-sour
41 he ability of the mutant osteoblasts to form mineralized nodules in culture was severely reduced.
42 Bs from TIEG(+/+) calvaria displayed several mineralized nodules in culture, whereas those from TIEG(
43 sphatase (ALP) activity and the formation of mineralized nodules in DPSCs.
44 kout osteoblasts produced significantly more mineralized nodules in ex vivo cell cultures than did wi
45 te (S1P), increased alkaline phosphatase and mineralized nodules in osteoblast cultures.
46 eogenic media revealed significantly reduced mineralized nodules in SCD-IgG-BMSC that was increased i
47 olates were tested for their ability to form mineralized nodules in vitro and to express alterations
48 ocedure and screened for the ability to form mineralized nodules in vitro, a property of cells with o
49 e cells expressed osteoblastic genes, formed mineralized nodules in vitro, and formed bone in an in v
50        Although all PDL cell isolates formed mineralized nodules in vitro, PDL cells from diabetics f
51 es in vitro, PDL cells from diabetics formed mineralized nodules more slowly than did the controls.
52 (BMSC) from 18-kDa TgFGF2 mice produced more mineralized nodules than VTg.
53 rface areas of the cell cultures occupied by mineralized nodules were measured using computerized ima