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1 nd largest subunit of RNA polymerase II, and minichromosome maintenance protein).
8 n identify colorectal cancer by detection of minichromosome maintenance protein 2 (MCM2) expression i
12 egulated loading of the replicative helicase minichromosome maintenance proteins 2-7 (MCM2-7) onto re
14 ereplication complex that contains a Mcm2-7 (minichromosome maintenance proteins 2-7) double hexamer.
15 lation sites in the carboxyl terminus of the minichromosome maintenance protein 3 (MCM3), a component
21 damage marker gammaH2AX is upregulated under minichromosome maintenance protein 7 (MCM7) knockdown in
22 We then used our mouse model to identify minichromosome maintenance protein 7 (MCM7), an E2F-indu
23 bers, whereas Double-parked protein/Cdt1 and minichromosome maintenance proteins are apparently essen
27 (origin recognition complex [ORC], Cdc6, and minichromosome maintenance proteins) causes a cell cycle
29 asmid DNA template by DNA polymerases (pol), minichromosome maintenance protein complex (Mcm), topois
31 regulate DNA replication; are members of the minichromosome maintenance protein complex family; or en
32 s the Cdc19 (Mcm2) subunit of the six-member minichromosome maintenance protein complex purified from
33 be essential to recruit and load MCM2-7, the minichromosome maintenance protein complex, around DNA a
35 etylation and RNR levels, maintains helicase minichromosome maintenance protein complexes (Mcm2-7) on
42 This modification enables the loading of the minichromosome maintenance protein (MCM) complex onto ch
43 pyrophosphatase/phosphodiesterase (ENPP-1), minichromosome maintenance protein (MCM-3), and double-s
46 ession of E2F-5, centromere protein A and E, minichromosome maintenance proteins (MCM)-2, -3, -5, -6
47 ognition complex, and the elongation factors minichromosome maintenance proteins (MCM)2-7 and prolife
50 ication, involves the loading of six related minichromosome maintenance proteins (Mcm2-7) into prerep
54 body of the mutant, and overexpression of a minichromosome maintenance protein subunit extended life
55 ber of other replication proteins, including minichromosome maintenance proteins, the origin recognit
56 monstrate that uncontrolled DNA unwinding by minichromosome maintenance proteins upon Cdt1 overexpres