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1 these cases the focus should be on using the minimum effective dose.
3 rved for thyrotropin-releasing hormone (TRH, minimum effective dose: 1.0 nM), to stimulate prolactin
5 we demonstrate the low potency of the PrRPs (minimum effective dose: 100 nM), compared to that observ
7 ation only at doses >25-fold higher than the minimum effective dose (3.1 mg/kg) in the Vogel "conflic
8 urther studies are required to establish the minimum effective dose and shortest necessary duration o
9 (total dose: 0.6 mg/kg = 1.8 mg/m2) and the minimum effective dose in the most sensitive model (SF-2
11 in both amphetamine-induced hyperlocomotion (minimum effective dose (MED) = 3 mg/kg, p.o.) and MK-801
12 atio was calculated as the ratio between the minimum effective dose (MED) for significant impairment
15 luid (CSF) 2.5x above the in vitro IC(50) at minimum effective doses (MEDs) of 3 mg/kg in preclinical
20 ayed magnificent antioxidant property with a minimum effective dose of 66 muM during the biochemical
26 n demonstrates that combination therapy with minimum effective doses of 7E3+efegatran provided enhanc
28 he antithrombotic effects of combinations of minimum effective doses of the glycoprotein IIb-IIIa rec
32 ommend stepping down asthma treatment to the minimum effective dose to achieve symptom control, preve