ライフサイエンスコーパス: mint

1 ch we term mitochondrial inner NEET protein (MiNT).

2 s APP, presenilins, and Ca(2+) channels (all Mints).

3 interaction data from IntAct, DIP, BIND and MINT.

4 erienced when one consumes pungent spices or mint.

5 resulted in altered binding modalities with Mint.

6 hundred plant species, including potato and mint.

7 brane cargo protein that works together with MINTs.

8 Mint 1 also inhibited transactivation by the "precleaved

9 CASK, Velis, and Mint 1 are evolutionarily conserved in Caenorhabditis el

10 d mapping studies revealed that Caskin 1 and Mint 1 bind to the same site on the N-terminal CaM kinas

11 sing knockout mice, we show that deletion of Mint 1 does not impair survival or alter the overall bra

12 ulse depression, suggesting that deletion of Mint 1 impairs the regulation of gamma-aminobutyric acid

13 that bind to synaptic proteins such as CASK (Mint 1 only) and Munc18-1 (Mints 1 and 2) and conserved

14 alternative tripartite complexes with either Mint 1 or Caskin 1 that may couple CASK to distinct down

15 neurons selectively express higher levels of Mint 1 than Mint 2.

16 les was used to assay methylation changes at MINT 1, 2, 3, 12, 17, 25, and 31 in sets of normal, aden

17 The CaM kinase domain of CASK binds to Mint 1, and the region between the CaM kinase and PDZ do

18 unoprecipitations showed that Caskin 1, like Mint 1, is stably bound to CASK in the brain.

19 Unlike CASK and Mint 1, no Caskin homolog was detected in C. elegans.

20 Thus the Mint 1-CASK complex is not required for AMPA- and NMDA-r

21 t an essential developmental function of the Mint 1-CASK complex.

22 aptic plasticity in excitatory synapses from Mint 1-deficient mice and detected no alterations in the

23 t synaptic functions that become apparent in Mint 1-deficient mice in inhibitory interneurons because

24 rons that contain selectively high levels of Mint 1.

25 The approximately 20% surviving Mint 1/2 double knock-out mice exhibit a decrease in wei

26 ression in cultured neurons from conditional Mint 1/2/3 triple knock-in mice also revealed a decline

27 We find that Mints 1 and 2 are similarly expressed in most neurons ex

28 Our data suggest that Mints 1 and 2 but not Mint 3 have a specific effect on A

29 h all Mints were biochemically similar, only Mints 1 and 2 but not Mint 3 strongly inhibited transact

30 among Mints are unexpected, suggesting that Mints 1 and 2 have a brain-specific function related to

31 Our data indicate that Mints 1 and 2 perform redundant synaptic functions that

32 mmunocytochemistry revealed that in neurons, Mints 1 and 2 were colocalized with APP in the trans-Gol

33 ins such as CASK (Mint 1 only) and Munc18-1 (Mints 1 and 2) and conserved C-terminal PTB- and PDZ-dom

34 Mints/X11s are neuron-specific (Mints 1 and 2) and ubiquitous (Mint 3) adaptor proteins

35 lacking both neuron-specific Mint isoforms (Mints 1 and 2) die at birth, whereas mice lacking any ot

36 composed of three isoforms: neuron-specific Mints 1 and 2, and the ubiquitously expressed Mint 3.

37 nscriptional factor, transient expression of MINT(1-812) suppresses the FGF/forskolin-activated OC pr

38 CpG islands in hMLH1, methylated in tumors (MINT) 1, MINT 2, MINT 31, and CDKN2A (p16).

39 The scaffolding protein LIN-10/Mint-1 also regulates GLR-1 abundance in the nerve cord.

40 The MALS/Veli-CASK-Mint-1 complex of PDZ proteins occurs on both sides of t

41 X11alpha/Mint-1 has multiple protein-protein interaction domains,

42 ectionally regulates the abundance of LIN-10/Mint-1 in the ventral cord.

43 CDK-5 phosphorylates LIN-10/Mint-1 in vitro and bidirectionally regulates the abunda

44 but six markers (CACNA1G, IGF2, RUNX3, MGMT, MINT-1, and SOCS1) were differentially clustered with CI

45 4a), CHFR, CRABP1, HIC1, IGF2, IGFBP3, MGMT, MINT-1, MINT-31, MLH1, NEUROG1, p14 (CDKN2A/arf), RUNX3,

46 Methylation at MINT 2, 3, and 31 increased 11-fold (P = .005), 15-fold

47 nds in hMLH1, methylated in tumors (MINT) 1, MINT 2, MINT 31, and CDKN2A (p16).

48 ctively express higher levels of Mint 1 than Mint 2.

49 Our data suggest that Mints 1 and 2 but not Mint 3 have a specific effect on APP function that canno

50 emically similar, only Mints 1 and 2 but not Mint 3 strongly inhibited transactivation by APP-Gal4/VP

51 ron-specific (Mints 1 and 2) and ubiquitous (Mint 3) adaptor proteins composed of isoform-specific N-

52 ints 1 and 2, and the ubiquitously expressed Mint 3.

53 o APP that is not executed by the ubiquitous Mint 3.

54 A yeast two-hybrid screening identified Mint-3 as the EWV-binding protein.

55 siRNA-mediated knockdown of endogenous Mint-3 decreased MT5-MMP activity.

56 Furthermore, Mint-3 significantly increased the level of MT5-MMP on t

57 Mint-3 stimulates MT5-MMP activity when expressed at low

58 for internalized MT5-MMP by interacting with Mint-3, a protein with two type III PDZ domains.

59 MLH1, methylated in tumors (MINT) 1, MINT 2, MINT 31, and CDKN2A (p16).

60 R, CRABP1, HIC1, IGF2, IGFBP3, MGMT, MINT-1, MINT-31, MLH1, NEUROG1, p14 (CDKN2A/arf), RUNX3, SOCS1,

61 yers, which included, in addition to the new MINTs, a pristine SWNTs layer and a MINT(XYLENE) layer.

62 Mint adaptor proteins bind to the amyloid precursor prot

63 Mint adaptor proteins bind to the membrane-bound amyloid

64 In confocal immunofluorescence microscopy, MINT adopts a reticular nuclear matrix distribution that

65 s of MINTs with different functional groups (MINT(ALKENE) (X:-CH = CH(2)), MINT(COOMe) (X:-COOMe), MI

66 Mints (also called X11-like proteins) are adaptor protei

67 n detect certain lipophilic irritants (e.g., mints, ammonia), the epithelium also houses a population

68 mical analyses, this novel protein was named MINT, an acronym for Msx2-interacting nuclear target pro

69 determined that conditional inactivation of Mint, an inhibitor of Notch-RBP-J interaction, resulted

70 O, and protein-protein interaction data from MINT and BIND.

71 hane lactones may contribute individually to mint and coconut odors, sensory studies suggested for th

72 th CSL, and we also delineate the domains of MINT and CSL that are necessary and sufficient for compl

73 OMIM; protein-protein interaction data from MINT and DIP; functional domain information from Pfam; p

74 Recently, the MINT and IntAct databases decided to merge their separat

75 MINT and OC mRNAs are reciprocally regulated during diff

76 have studied functional interactions between MINT and Runx2, a master regulator of osteoblast differe

77 such as IntAct, BioGRID, ChEMBL, iRefIndex, MINT and STRING.

78 sis, and although Munc18-1 can interact with Mint and Sx1a (Syntaxin1a) proteins simultaneously in vi

79 , Mg and Ni were low in laurel and rosemary; mint and thyme showed the highest Na and the lowest Se c

80 Various Mint and/or CASK-containing complexes can be assembled o

81 ted from other databases (IntAct, DIP, BIND, MINT) and are linked to 24,060 experimental evidences (P

82 the Msx2 interacting nuclear target protein (MINT) and Runx2/Cbfa1.

83 ped to real samples (linden, milk powder and mint) and significant results (94.4-100.7%) were obtaine

84 lavored e-cigarettes, with fruit, menthol or mint, and candy, desserts, or other sweets being the mos

85 into 4 flavor categories (tobacco, menthol, mint, and other).

86 Following infection or vaccination, early-minted antibody secreting cells (ASC) or plasmablasts ap

87 Both IntAct and MINT are active contributors to the IMEx consortium.

88 a(V)2 chanels, most genes that interact with Mint are also deeply conserved including amyloid precurs

89 We propose that MINTs are a family of Arf-dependent, vesicle-coat protei

90 Mints are adaptor proteins that directly interact with t

91 Our studies thus indicate that Mints are important regulators of presynaptic neurotrans

92 the endocytic motif of APP, we proposed that Mints are involved in APP intracellular trafficking, whi

93 These findings demonstrate that Mints are necessary for activity-induced APP and PS1 tra

94 similarity, the functional differences among Mints are unexpected, suggesting that Mints 1 and 2 have

95 Munc18 interacting proteins (MINTs) are a family of three proteins implicated in the

96 Munc18-interacting proteins (Mints) are multidomain adaptors that regulate neuronal m

97 e survival advantage over less mature, newly minted ASCs, by enhancing pro-survival cytokine signalin

98 fruit at 59% (95% CI, 55%-63%), followed by mint at 27% (95% CI, 24%-30%) and menthol at 7% (95% CI,

99 teoblasts (with endogenous Runx2 and FGFR2), MINT augments transcription driven by the OCFRE that is

100 We show that MINTs bind Arfs directly, co-localize with Arf and the A

101 We demonstrate that MINTs bind Arfs through a region of the PTB domain and t

102 tion of membrane-bound APP via endosomes and Mints bind directly to the endocytic motif of APP, we pr

103 We now show that all three Mints bind to the cytoplasmic tail of amyloid-beta precu

104 ockout neurosecretory cells, mutation of the Mint-binding site reduces the ability of Munc18-1 to res

105 data indicate that the novel nuclear protein MINT binds the homeoprotein Msx2 and coregulates OC duri

106 Since X11/Mint binds to APP, and abnormal trafficking of APP contr

107 distinct plasma cell subsets, such as early-minted blood antibody-secreting cells and the bone marro

108 Msx2 and MINT both target an information-dense, osteoblast-specif

109 Compared to spearmint, mint bush showed comparable antioxidant capacity, strong

110 diterpene skeletons have been reported from mints, but biosynthetic pathways are known for just a fe

111 e show in cultured cells that this region of MINT can inhibit Notch signaling in transcriptional repo

112 e illustrate with examples how the making of MINTs can contribute to modifying the surface properties

113 aptic proteins showed that acute deletion of Mints caused a selective increase in Munc18-1 and Fe65 p

114 testis library were sequenced to construct a MINT cDNA contig of 11 kb.

115 tative thermodynamic binding analysis of CSL-MINT complexes.

116 NE) (X:-CH = CH(2)), MINT(COOMe) (X:-COOMe), MINT(COOH) (X:-COOH), and MINT(OH) (X:-CH(2)OH)) were sp

117 tional groups (MINT(ALKENE) (X:-CH = CH(2)), MINT(COOMe) (X:-COOMe), MINT(COOH) (X:-COOH), and MINT(O

118 lavors (Fruit Medley, Virginia Tobacco, Cool Mint, Creme Brulee, Cool Cucumber, Mango, and Classic Me

119 All data manually curated by the MINT curators have been moved into the IntAct database a

120 h, and enhanced paired-pulse facilitation in Mint-deficient mice, suggesting a decreased presynaptic

121 ute to the presynaptic phenotype observed in Mint-deficient mice.

122 These results suggest that X11/Mint-dependent endocytosis in dendrites may serve to pro

123 to interesting odorant zones, reminiscent of mint, detected in the studied wine.

124 2 abrogates Runx2-MINT OCFRE activation, and MINT-directed RNA interference reduces endogenous OC exp

125 cellular and molecular mechanisms underlying Mints effect on amyloid production are unclear.

126 Menthol in mints elicits coolness sensation by selectively activati

127 reas Fe65 (which binds to APP as strongly as Mints) enhanced transactivation.

128 Thus, MINT enhances Runx2 activation of multiprotein complexes

129 ste source that can be kept during winter is mint essential oil.

130 Aqueous methanolic extract of the mint exhibited comparable antioxidant capacity to the co

131 Acute ablation of Mint expression in cultured neurons from conditional Min

132 ps such as the sunflower, potato, coffee and mint families, totalling over 80,000 species(3).

133 Members of the mint family (Lamiaceae) accumulate a wide variety of ind

134 al product first discovered in plants of the mint family (Lamiaceae) and is recognized for its wide r

135 (Mentha x piperita) and its relatives in the mint family (Lamiaceae) are accumulated in specialized a

136 The mint family (Lamiaceae) is well documented as a rich sou

137 er's sage, is an ethnomedicinal plant of the mint family (Lamiaceae).

138 rsification of diterpene biosynthesis in the mint family and establish a comprehensive foundation for

139 e genome assemblies for other members of the mint family and offers an important foundation for decod

140 he diversity of interactions mediated by the Mint family and show that Mints may help facilitate a ke

141 lvinorin A, is a hallucinogenic plant in the mint family that has been used in traditional spiritual

142 The Lamiaceae (mint family) is the largest known source of furanoclerod

143 stems of sage and other Lamiaceae (Labiatae; mint family) or Scrophulariaceae (figwort family) specie

144 pecific flavone biosynthetic pathways in the mint family, Lamiaceae, but also will facilitate the dev

145 on of clerodane biosynthetic pathways in the mint family, Lamiaceae, but also will facilitate the pro

146 es (82.7%; 95% CI, 68.9%-91.1%), followed by mint-flavored e-cigarettes.

147 In the MabThera International Trial (MInT) for young good-prognosis patients and the Rituxima

148 l titration calorimetry, we demonstrate that MINT forms a high affinity complex with CSL, and we also

149 the following categories: sugar-free gum and mints, fruit, vegetables, nuts, seeds, legumes, yogurt o

150 Such regions include newly minted gene families in lineage-specific segmental dupli

151 Protein sequence analysis reveals that MINT has three N-terminal RNA recognition motifs (RRMs)

152 However, cultivated mints have complex polyploid genomes and are sterile.

153 Australian native mints have traditionally been used by the aboriginal peo

154 d event-free survival (EFS; hazard ratio for MInT [HRMInT] = 0.4, P > 001; hazard ratio for RICOVER-6

155 taneous release, confirming that deletion of Mints impair presynaptic function.

156 nd Veli which form a tripartite complex with Mint in bilaterians.

157 Mint knock-out neurons, revealing a role for Mints in APP trafficking.

158 Acute conditional deletion of all three Mints in cultured neurons suppresses the accumulation of

159 redox enzymes of monoterpene biosynthesis in mint indicates that these genes arose from different anc

160 Verticillium wilt are top priorities for the mint industry.

161 the PTB domain and the PDZ2 domain, and Arf-MINT interaction is necessary for the increased cellular

162 % identity) to the monoterpene reductases of mint involved in (-)-menthol biosynthesis.

163 show through various in silico analyses that Mint is an animal-specific gene with a highly divergent

164 MINT is freely available at with DOI doi.org/10.5281/zen

165 ken together, our findings reveal that CISD3/MiNT is important for supporting the biogenesis and func

166 port a model whereby one of the functions of Mints is to localize the vesicle fusion protein Munc18 t

167 the inner mitochondrial CISD protein, CISD3/MiNT, is currently unknown.

168 ely 80% of mice lacking both neuron-specific Mint isoforms (Mints 1 and 2) die at birth, whereas mice

169 hereas mice lacking any other combination of Mint isoforms survive normally.

170 show that APP endocytosis was attenuated in Mint knock-out neurons, revealing a role for Mints in AP

171 analyzed neuronal cultures from control and Mint knockout neurons that were treated with either glut

172 ficking and Abeta generation were blocked in Mint knockout neurons.

173 rescue APP internalization and insertion in Mint knockout neurons.

174 The Hawaiian endemic mints (Lamiaceae family) are the second largest plant ra

175 e), dogwoods (Cornaceae), lilies (Liliales), mints (Lamiaceae p.p.), orchids (Orchidaceae), roses (Ro

176 iological studies indicate that knockdown of MiNT leads to increased accumulation of mitochondrial la

177 f curry leaf (EHEC) and the water extract of mint leaf (WEM) showed higher DPPH and ABTS(+) activity.

178 y of different solvent extracts of curry and mint leaf and their effect on colour and oxidative stabi

179 phobic dyes could be incorporated to imitate mint leaf infusions aromatically and visually.

180 Menthol is a cooling compound derived from mint leaves and is extensively used as a flavoring chemi

181 y in peppermint oil, known for their potent, mint-like olfactory properties.

182 in and syndecan and the cytoplasmic proteins Mint/LIN-10 and Veli/LIN-7.

183 king complex composed of calsyntenin/CASY-1, Mint/LIN-10, and kinesin suppresses axon degeneration.

184 ge/zona occludens-1)-containing protein dX11/Mint/Lin-10, which also regulates synapse expansion down

185 thylation of 16 tumor suppressor genes and 3 MINT loci (acknowledged classifiers of CIMP) in 344 blad

186 Increased MI of both MINT loci 1 and 31 was significantly associated with MSI

187 P < 0.0001) between methylation of the three MINT loci and methylation index, although the distributi

188 Methylation levels of specific MINT loci can be used as prognostic variables in patient

189 gned to determine whether the methylation of MINT loci during the progression of adenoma to CRC is re

190 ve assessment of methylated alleles at seven MINT loci in primary CRC with contiguous adenomatous and

191 MI of specific MINT loci may be prognostic indicators of colorectal ade

192 mic hypermethylation of methylated-in-tumor (MINT) loci are both strong prognostic indicators in a su

193 sue at multiple genomic methylated-in-tumor (MINT) loci sequences.

194 Increased MINT locus methylation appears to precede MSI and may ha

195 tudy characterizes JUUL e-cigarette flavors (mint, mango, fruit, and others) most often used by US mi

196 to Alzheimer's disease, deregulation of X11/Mint may be important for Alzheimer's disease pathogenes

197 In osteoblasts, MINT may serve as a nuclear matrix platform that organiz

198 Therefore, Mints may be the adaptor proteins that regulate the traf

199 ns mediated by the Mint family and show that Mints may help facilitate a key trigger point in SNARE (

200 quality attributes of refrigerated fresh-cut mints (Menthaxpiperita and M. spicata) was studied.

201 used for the p16 and hMLH1 genes, and three MINT (methylated in tumor) loci (MINT1, MINT2, and MINT3

202 supervised grouping analyses of quantitative MINT methylation data of TME trial patients demonstrated

203 MINT MI was found to be correlated with mismatch repair

204 nce the selection of one meaning (e.g., coin-mint-money) but failed to emerge when competition betwee

205 aning appropriate to the context (e.g., coin-mint-money) or under conditions of increased competition

206 d was unresolved by the context (e.g., candy-mint-money).

207 olution to a particular meaning (e.g., candy-mint-money).

208 The corepressor MINT (Msx2-interacting nuclear target protein) has been

209 of the Myocardial Ischemia and Transfusion (MINT) multicenter randomized clinical trial was conducte

210 nding of vesicles to the active zone via the Mint.Munc18-1 complex in conjunction with syntaxin 1.

211 ae isolates recovered from sympatric potato, mint, mustard and grasses were characterized genotypical

212 When consumed with foods, mint, mustard, and chili peppers generate pronounced ora

213 es, and strongly enhance defects seen in X11/Mint mutants.

214 Myocardial Ischemia and Transfusion (MINT), NCT02981407.

215 a significant accentuation of freshness and mint notes.

216 Msx2 abrogates Runx2-MINT OCFRE activation, and MINT-directed RNA interferenc

217 COOMe) (X:-COOMe), MINT(COOH) (X:-COOH), and MINT(OH) (X:-CH(2)OH)) were specifically synthesized to

218 Mint oil is a key source of natural flavors with wide in

219 factor Gal4/VP16, we examined the effects of Mints on the proteolytic processing and putative transcr

220 MINT only partially co-localizes with Runx2; however, co

221 andy (578 [36.3%]); fruit (532 [33.4%]); and mint or menthol (321 [20.2%]); similar flavor patterns w

222 tions of coffee, alcohol, flower, plant, and mint or menthol flavors by age group.

223 x odor but not other, nonthreatening, odors (mint or orange) rapidly decreased the number of prolifer

224 Instead, EGL-9 interacts with the Mint orthologue LIN-10, a mediator of GLR-1 membrane rec

225 9, with impaired subcellular localization of Mint orthologue LIN-10, internalization of glutamate rec

226 MINT outperformed expressive machine learning methods in

227 MINT outperformed other interpretable methods in every c

228 In addition, we found that Mint overexpression increased excitatory synaptic activi

229 ased cellular levels of beta-APP produced by MINT overexpression.

230 MINT performed well across tasks, suggesting its assumpt

231 ometimes called "superpostdocs," offer newly minted PhDs instant independence and enable them to unde

232 ic acid-type peptides to grasses, Lowiaceae, mints, pinks, and spurges while demonstrating that large

233 We developed the Methylation INTegration (mint) pipeline to support the comprehensive analysis of

234 improve overall soil quality, fertility and mint plant productivity under high Pb soil concentration

235 ecalis + CB treatment significantly improved mint plant root dry weight (58%), leaves dry weight (32%

236 major active component of the hallucinogenic mint plant Salvia divinorum Epling and Jativa (Lamiaceae

237 Acting as a scaffold protein, MINT potentially exerts both positive and negative regul

238 inal) and approximately 250 kDa (C-terminal) MINT protein fragments accumulate in chromatin and nucle

239 Taken together, our study suggests that the MiNT protein functions in the same pathway as its homodi

240 Mint proteins are detectable only in brain and are compo

241 In brain, Mint proteins are part of a multimeric complex containin

242 Here we report that Drosophila X11/Mint proteins are required for targeting several protein

243 e show that deletion of all three individual Mint proteins delays the age-dependent production of amy

244 es have shown that loss of each of the three Mint proteins delays the age-dependent production of amy

245 n both the specific interactions of neuronal Mint proteins with Munc18-1 as well as their wider inter

246 with regions of similarity to mammalian X11/mint proteins, containing a phosphotyrosine-binding and

247 tus, Garland thorn, honeydew, heather, lime, mint, rapeseed, sage, strawberry tree, sulla flower, sav

248 however, the molecular mechanisms underlying Mint regulation in APP binding and processing remain unc

249 iated by an endocytic motif, and loss of X11/Mint results in a dramatic increase in cell-surface leve

250 ton, UK) IntAct, the Molecular Interactions (MINT, Rome, Italy) database, the Protein-Protein Interac

251 The N-terminal MINT RRM domain represents an authentic dsDNA binding mo

252 In gel shift assays, the MINT RRM domain selectively binds T- and G-rich DNA sequ

253 MINT's computations are simple, scale favorably with inc

254 MINT's performance and simplicity suggest it may be a st

255 on-inscribed nascent transcripts sequencing (MINT-seq), we characterized the landscapes of N6-methyla

256 As in human cells by developing a new method MINT-Seq, which revealed that many classes of RTE RNAs,

257 nction, but it shares homology with the spen/Mint/SHARP family of proteins defined by three amino-ter

258 Additionally, MINT should be competitive with expressive machine learn

259 If those constraints are accurate, MINT should outperform standard methods that explicitly

260 The genus Mentha encompasses mint species cultivated for their essential oils, which

261 Mentha x piperita was the best mint species performing under low nitrate and salt stres

262 development of genomic resources for fertile mint species.

263 for participants with chronic low back pain (Mint study) were conducted in 16 multidisciplinary pain

264 In patients with CKD included in this MINT subgroup analysis, a liberal transfusion strategy w

265 In CV1 cells, MINT synergizes with Runx2 to enhance OCFRE activity in

266 variate Information in Neuroscience Toolbox (MINT) that combines these new methods with statistical t

267 ed in p-menthane monoterpene biosynthesis in mint: the large and small subunits of peppermint (Mentha

268 r, our analyses suggest that the capacity of Mint to bind Ca(V)2 channels predates bilaterian animals

269 family, Spen, which includes the vertebrate MINT transcriptional regulator.

270 , anise/caraway, carrot, eucalyptus, garlic, mint) transmit to and flavor breast milk in a time-depen

271 The MINT trial (Myocardial Ischemia and Transfusion) raised

272 In the MINT trial (Myocardial Ischemia and Transfusion), 3504 p

273 The MINT trial (Myocardial Ischemia and Transfusion; N=3504)

274 In this secondary analysis of the MINT trial, acute anemia was associated with less favora

275 In this prespecified analysis of the MINT trial, patients who underwent revascularization bef

276 In this analysis of the MINT trial, revascularization did not alter the effect o

277 in the Myocardial Ischemia and Transfusion (MINT) trial with nonmissing serum creatinine, we compare

278 of the Myocardial Ischemia and Transfusion (MINT) trial, a randomized clinical trial comparing a lib

279 e selected in the TRICC, FOCUS, REALITY, and MINT trials.

280 s from five primary databases: IntAct, HPRD, MINT, UniProt and the Gene Ontology.

281 structural studies reveal that synergy with MINT uniquely requires Runx2 activation domain 3.

282 Na, P, Pb, S, V, and Zn in beans, basil, and mint using ICP OES and flame photometry.

283 the levels of the CASK-interacting proteins Mints, Veli/Mals, and neurexins are decreased, whereas t

284 ion threshold of this terpene reminiscent of mint was 0.9mug/L in model hydroalcoholic solution and 7

285 One of the zones, reminiscent of mint, was found in red wines with a prune flavor.

286 assayed for packaging and storing fresh-cut mints were adequate to achieve a relatively long shelf l

287 Although all Mints were biochemically similar, only Mints 1 and 2 but

288 The RNA recognition motif domain of MINT (which binds the OCFRE) is required.

289 Four new types of MINTs with different functional groups (MINT(ALKENE) (X:

290 release, suggesting that the interaction of Mints with Munc18-1 may contribute to the presynaptic ph

291 MINT works on Linux, Windows and macOS operating systems

292 ase; however, the biological significance of Mint/X11 binding to APP and their possible role in Abeta

293 Previous studies suggested that Mint/X11 proteins influence APP cleavage and affect prod

294 These results suggest that the three Mint/X11 proteins regulate Abeta production by a novel m

295 ind to synaptic-signaling molecules, such as MINT/X11.

296 Mints/X11s are adaptor proteins composed of three isofor

297 Mints/X11s are neuron-specific (Mints 1 and 2) and ubiqu

298 Mints/X11s are neuronal adaptor proteins that bind to am

299 and conditional knock-out mice for all three Mints/X11s.

300 the new MINTs, a pristine SWNTs layer and a MINT(XYLENE) layer.