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1 ce-mediated knockdown of factors involved in mitochondrial dynamics.
2 ions of mitochondrial proteins that regulate mitochondrial dynamics.
3 n2, Opa1 and p-Drp1 leading to disruption of mitochondrial dynamics.
4 ciency has detrimental influence on neuronal mitochondrial dynamics.
5 hat TMEM135 is involved in the regulation of mitochondrial dynamics.
6 osomes were identified and found to regulate mitochondrial dynamics.
7 he mitochondria, where it similarly disrupts mitochondrial dynamics.
8 port that Smad2 is a critical determinant of mitochondrial dynamics.
9 , and report an elegant mechanism of shaping mitochondrial dynamics.
10 thology triggered by defective or imbalanced mitochondrial dynamics.
11 her layer of complexity to the regulation of mitochondrial dynamics.
12 ations in GDAP1 impede the protein's role in mitochondrial dynamics.
13 drial-encoded protein synthesis and abnormal mitochondrial dynamics.
14 ss of modified solute transporters linked to mitochondrial dynamics.
15 ve disorders characterized by alterations in mitochondrial dynamics.
16  are critically involved in animal and yeast mitochondrial dynamics.
17  stage-specific programs involved in cardiac mitochondrial dynamics.
18  Ser-637 phosphorylation, both indicators of mitochondrial dynamics.
19 ed levels of OPA1 protein, and impairment of mitochondrial dynamics.
20 ium signaling, metabolism, proteostasis, and mitochondrial dynamics.
21 ion of cancer cell migration and invasion by mitochondrial dynamics.
22 ting a specific role of TDP-43 in regulating mitochondrial dynamics.
23  induced substantial and widespread abnormal mitochondrial dynamics.
24 ng pharmacological and genetic modulators of mitochondrial dynamics.
25 s slingshot phosphatase to modulate neuronal mitochondrial dynamics.
26 embrane, where it functions in mitophagy and mitochondrial dynamics.
27  consequences of Drp1S600 phosphorylation on mitochondrial dynamics.
28 s (ROS) production, and promoted exaggerated mitochondrial dynamics.
29 otes proline synthesis through regulation of mitochondrial dynamics.
30 e thioredoxin reductase 1 TrxR-1 to regulate mitochondrial dynamics.
31          Bif-1 and prohibitin-2 may regulate mitochondrial dynamics.
32 1/2, and its depletion causes dysfunction in mitochondrial dynamics.
33 ofusin 2 (Mfn2) could abolish TDP-43 induced mitochondrial dynamics abnormalities and mitochondrial d
34        Interestingly, these manipulations of mitochondrial dynamics altered the subcellular distribut
35 wth, which was associated with disruption of mitochondrial dynamics and a reduction in mitochondrial
36 lar communication, leading to alterations in mitochondrial dynamics and acquisition of invasive pheno
37 c mechanisms by which gut bacteria influence mitochondrial dynamics and aging, a first step toward an
38 us in this context has been the link between mitochondrial dynamics and amyloid beta toxicity.
39 proximal signaling events can influence both mitochondrial dynamics and apoptosis through phosphoryla
40 vous system, alterations in and the roles of mitochondrial dynamics and associated signaling in micro
41  of mitochondrial quality control, including mitochondrial dynamics and autophagy/mitophagy, under hi
42 ticular vulnerabilities that often implicate mitochondrial dynamics and axon transport mechanisms.
43 cate that IHG-1 is a novel regulator of both mitochondrial dynamics and bioenergetic function and con
44 n of mitochondria, it is not surprising that mitochondrial dynamics and bioenergetics reciprocally in
45 e new thinking in the intersecting fields of mitochondrial dynamics and bioenergetics, as treatment o
46 els of electron transport chain (ETC) genes; mitochondrial dynamics and biogenesis genes; enzymatic a
47                                 Dysregulated mitochondrial dynamics and biogenesis have been associat
48 oscopy, we provide a new correlation between mitochondrial dynamics and bleb vesicle formation using
49 ral immunity and underlay nutrient-triggered mitochondrial dynamics and cell fate determination.
50 oting mitochondrial biogenesis and improving mitochondrial dynamics and clearance.
51     This study confirms the role of TRAK1 in mitochondrial dynamics and constitutes the first report
52 rter (MCU) complex (MCUC) function influence mitochondrial dynamics and contribute to PAH's cancer-li
53 tion for seemingly paradoxical expression of mitochondrial dynamics and death factors in cardiomyocyt
54 cytic mitochondrial Dlp1 is a key protein in mitochondrial dynamics and decreased Dlp1 may interfere
55              Recent studies reported altered mitochondrial dynamics and decreased mitochondria- endop
56 /reverse diabetic retinopathy by maintaining mitochondrial dynamics and DNA stability, and prevent re
57  causes degeneration in neurons by affecting mitochondrial dynamics and energy production.
58                   Given the critical role of mitochondrial dynamics and energy requirements in neuron
59  and mitochondrial-encoded genes involved in mitochondrial dynamics and energy transduction in the ad
60 reduces mitochondrial dysfunction, maintains mitochondrial dynamics and enhances mitochondrial biogen
61 kDa interacting protein 3) pathway modulates mitochondrial dynamics and function and contributes to m
62                               An analysis of mitochondrial dynamics and function in morphogenetic eve
63 hether and how TDP-43 mutations might impact mitochondrial dynamics and function.
64 nd cellular damage, can reprogram and govern mitochondrial dynamics and function.
65                                              Mitochondrial dynamics and functionality are linked to t
66 anisms by which cytoplasmic stimuli modulate mitochondrial dynamics and functions are largely unknown
67 In the present study, we examined changes in mitochondrial dynamics and functions triggered by alpha
68  of DNA methylation in continued compromised mitochondrial dynamics and genomic stability in diabetic
69 fector cells, which respectively depend upon mitochondrial dynamics and ICOS-mTORC2 signaling.
70 sed by hypoxic breast cancer cells reprogram mitochondrial dynamics and induce oncogenic changes in a
71 nonical Wnt ligand, is a potent activator of mitochondrial dynamics and induces acute fission and fus
72                       These processes define mitochondrial dynamics and inextricably link the fate of
73                        Miro GTPases modulate mitochondrial dynamics and interfering with this functio
74 everal pieces of evidence suggested impaired mitochondrial dynamics and its association with the path
75 uccinate on hMSC migration via regulation of mitochondrial dynamics and its related signaling pathway
76  the same pathway and play a crucial role in mitochondrial dynamics and maintenance.
77 othrombotic status, relies on alterations in mitochondrial dynamics and metabolism that may be preven
78             The effects of PDK activation on mitochondrial dynamics and metabolism, RVfib proliferati
79  interorganelle lipid exchange and influence mitochondrial dynamics and mitochondrial DNA maintenance
80 tability, abnormalities in the regulation of mitochondrial dynamics and mitochondrial quality control
81 vasive in a manner dependent on ROS-mediated mitochondrial dynamics and mitochondrial repositioning t
82 ated in aging, but a deeper understanding of mitochondrial dynamics and mitophagy during aging is mis
83                                              Mitochondrial dynamics and mitophagy have been linked to
84  the latest findings regarding the impact of mitochondrial dynamics and mitophagy on the development
85                                     Although mitochondrial dynamics and mitophagy were induced at P1-
86 itochondrial homeostasis by interfering with mitochondrial dynamics and mitophagy.
87 ons, including oxidative phosphorylation and mitochondrial dynamics and morphology, and is essential
88 st study to link the innate immune system to mitochondrial dynamics and morphology.
89 scribe novel functions for NIK in regulating mitochondrial dynamics and motility to promote cell inva
90                                              Mitochondrial dynamics and mtDNA maintenance is another
91 apeutic target against BPA-mediated impaired mitochondrial dynamics and neurodegeneration in the hipp
92 drial and synaptic toxicities, and maintains mitochondrial dynamics and neuronal function in AD neuro
93 drial and synaptic toxicities, and maintains mitochondrial dynamics and neuronal function in AD.
94 ysosomes accumulation, altered expression of mitochondrial dynamics and oxidative phosphorylation reg
95 chondrial Ser/Thr phosphatase that modulates mitochondrial dynamics and participates in both apoptoti
96 ase models to study the relationship between mitochondrial dynamics and peripheral neurodegeneration.
97 ng of PINCH-1, DRP1 and PYCR1 that regulates mitochondrial dynamics and proline synthesis, and sugges
98 ), and increasing evidence suggests abnormal mitochondrial dynamics and quality control as important
99 function, and recent emphasis has focused on mitochondrial dynamics and quality control.
100 gether, our results reveal insights into how mitochondrial dynamics and quality orchestrate T cell an
101 iation represents an additional link between mitochondrial dynamics and recognizable neurological syn
102                                              Mitochondrial dynamics and related cell function require
103  Parkinson's disease, has been implicated in mitochondrial dynamics and removal in cells including ne
104 new function for myoglobin as a modulator of mitochondrial dynamics and reveal a novel pathway by whi
105 ditions (<1% O(2)), the relationship between mitochondrial dynamics and sensitivity to cisplatin (CDD
106  and plant-specific aspects of CL biology in mitochondrial dynamics and the organism response to envi
107 open a new dimension to our understanding of mitochondrial dynamics and the role of miRNA in mitochon
108 gnaling pathway by which myoglobin regulates mitochondrial dynamics and thereby decreases cell prolif
109 ween peroxisomes and mitochondria regulating mitochondrial dynamics and thermogenesis.
110  novel function for NP1 in the regulation of mitochondrial dynamics and trafficking during apoptotic
111 accharide colanic acid (CA), which regulates mitochondrial dynamics and unfolded protein response (UP
112 scular remodeling, mediates the link between mitochondrial dynamics and vascular smooth muscle cell (
113  relationships between parkin gene activity, mitochondrial dynamics, and aging have not been explored
114   We assessed temporal changes in autophagy, mitochondrial dynamics, and bioenergetics in mouse model
115 s biological pathways involved in apoptosis, mitochondrial dynamics, and innate immune response.
116 crosstalk between mitochondrial translation, mitochondrial dynamics, and lysosomal signaling in regul
117 ral proteins involved in electron transport, mitochondrial dynamics, and mitochondrial protein synthe
118 n recent years, evidence linking metabolism, mitochondrial dynamics, and protein homeostasis (proteos
119 ontrol machinery that regulates respiration, mitochondrial dynamics, and protein import.
120 causes mitochondrial depolarization, reduces mitochondrial dynamics, and restricts turnover of cellul
121  a determinative effect in the regulation of mitochondrial dynamics, and therefore neuronal function.
122 ein-protein interactions, (2) alterations in mitochondrial dynamics, and/or (3) post-translational mo
123 sterol, and phospholipid metabolism; altered mitochondrial dynamics; and reduced bioenergetic functio
124                                     Aberrant mitochondrial dynamics are also associated with major me
125                                   Defects of mitochondrial dynamics are emerging causes of neurologic
126                                              Mitochondrial dynamics are generally associated with qua
127                                              Mitochondrial dynamics are regulated by a complex system
128 ress a gap in understanding of how dendritic mitochondrial dynamics are regulated when energy depleti
129 eveal that adrenergically-induced changes to mitochondrial dynamics are required for BA thermogenic a
130                                              Mitochondrial dynamics are required for mitochondrial vi
131  (AD), a condition initiated at the synapse, mitochondrial dynamics are severely impaired.
132             These results implicate DRP1 and mitochondrial dynamics as an important mediator of AKI a
133 dicate the potential value of restoration of mitochondrial dynamics as an innovative therapeutic stra
134                        These findings reveal mitochondrial dynamics as an upstream regulator of essen
135        Increasing evidence suggests abnormal mitochondrial dynamics as important underlying mechanism
136         We demonstrate that genes regulating mitochondrial dynamics, as well as mitophagy are induced
137            In this study we demonstrate that mitochondrial dynamics, as well as mitophagy, is affecte
138 ciency affected ER-mitochondria contacts and mitochondrial dynamics, at least in part, by regulating
139 NK1 action on other substrates, can restrict mitochondrial dynamics before mitophagy.
140 Drp1 interaction, mRNA and protein levels of mitochondrial dynamics, biogenesis and synaptic genes, m
141 evels of SS31, (2) mRNA levels and levels of mitochondrial dynamics, biogenesis proteins and synaptic
142 tion of the FOXO3a-BNIP3 pathway and altered mitochondrial dynamics, biogenesis, and function.
143 ned mechanisms for direct cross talk between mitochondrial dynamics, biogenesis, quality control, and
144               Acute energy depletion impairs mitochondrial dynamics, but how chronic energy insuffici
145 an physiology can be restored by rebalancing mitochondrial dynamics, but this concept remains to be v
146                                     Impaired mitochondrial dynamics by BPA resulted in increased reac
147 type full-length tau (termed htau) disrupted mitochondrial dynamics by enhancing fusion and induced t
148                          Imposing imbalanced mitochondrial dynamics by manipulating the expression le
149  that SLC25A46 may play an important role in mitochondrial dynamics by mediating mitochondrial fissio
150                        Indomethacin impaired mitochondrial dynamics by promoting fissogenic activatio
151               Here we show that HCV perturbs mitochondrial dynamics by promoting mitochondrial fissio
152 hereby disrupts the physiological balance of mitochondrial dynamics by promoting mitochondrial hyper-
153  previously reported, but also by regulating mitochondrial dynamics by targeting OPA1.
154                      Moreover, disruption of mitochondrial dynamics by targeting the DISC1-Miro-TRAK
155 mechanical roles in skeletal muscle and that mitochondrial dynamics can be manipulated to alter muscl
156 rall, this work demonstrates that disrupting mitochondrial dynamics can have opposite effects on resi
157           Our data provide insights into how mitochondrial dynamics can impact germ cell maintenance
158                                   Defects in mitochondrial dynamics can lead to neurodegenerative dis
159 ies and less ATP, and to the deregulation of mitochondrial dynamics, causing in consequence the accum
160 how that a subset of SLC25A46 interacts with mitochondrial dynamics components and the MICOS complex.
161 icating that deregulation of calcineurin and mitochondrial dynamics contributes to high-risk and poor
162             Therefore, we questioned whether mitochondrial dynamics controls T cell metabolism.
163 we investigated the involvement of Parkin in mitochondrial dynamics, distribution, morphology, and re
164 s and protein levels of genes related to the mitochondrial dynamics-Drp1 and Fis1 (fission), Mfn1, Mf
165                             By investigating mitochondrial dynamics during meiotic differentiation in
166 tion between energy production and dendritic mitochondrial dynamics during neuronal development and m
167 c energetic insufficiency and its effects on mitochondrial dynamics during neuronal development.
168                                              Mitochondrial dynamics during nutrient starvation of can
169  highlight the evolutionary context in which mitochondrial dynamics emerged and consider unanswered q
170 ces mitochondrial dysfunction, and maintains mitochondrial dynamics, enhances mitochondrial biogenesi
171 ces mitochondrial dysfunction, and maintains mitochondrial dynamics, enhances mitochondrial biogenesi
172 expand upon previous observations of altered mitochondrial dynamics following alphaherpesvirus infect
173 icular, we found sex-specific alterations of mitochondrial dynamics following cohabitation, with a sh
174 ia splicing defects of export factors and/or mitochondrial dynamics/function, since Sub2 controls mRN
175 emonstrated that it has a profound impact on mitochondrial dynamics (fusion and fission) and clearanc
176 ng evidence has shown that proper control of mitochondrial dynamics (fusion and fission) is required
177 erall, our data unmask an important role for mitochondrial dynamics governed by Mfn1 and Mfn2 in Agrp
178                    Although dysregulation of mitochondrial dynamics has been linked to cellular senes
179                                 Dysregulated mitochondrial dynamics has been reported in various dise
180 (Abeta) and TAU, only the impact of Abeta on mitochondrial dynamics has been studied in detail.
181  and suggest that enhancing SIRT3 to improve mitochondrial dynamics has potential as a strategy for i
182                                              Mitochondrial dynamics has recently become an area of pi
183                          Here we report that mitochondrial dynamics have a profound effect on PME.
184  the molecular players involved in mediating mitochondrial dynamics have been identified, the precise
185                    Recent imaging studies of mitochondrial dynamics have implicated a cycle of fusion
186                                              Mitochondrial dynamics have recently been shown to play
187 , and (4) screening for regulators of muscle mitochondrial dynamics in a high-throughput format.
188 important for regulating signaling-dependent mitochondrial dynamics in astrocytic processes remains u
189 hus, DISC1 acts as an important regulator of mitochondrial dynamics in both axons and dendrites to me
190 e a role for peroxisomal lipid metabolism in mitochondrial dynamics in brown and beige adipocytes.
191                              IDH2 reprograms mitochondrial dynamics in cancer through a HIF-1alpha-re
192 y was to investigate whether insulin affects mitochondrial dynamics in cardiomyocytes.
193  in FECD; however, the mechanism of aberrant mitochondrial dynamics in CE cell loss is poorly underst
194 ondrial biogenesis and its coordination with mitochondrial dynamics in developing and diseased hearts
195 e functional crosstalk between mitophagy and mitochondrial dynamics in Drosophila heart tubes.
196 nd actin assembly, involving Rab11a-mediated mitochondrial dynamics in E4orf4-induced signaling.
197 ty could drive mitochondrial dysfunction via mitochondrial dynamics in ECs.
198 atypical, dimeric phospholipid essential for mitochondrial dynamics in eukaryotic cells.
199 ediated knockdown, 15 of the genes modulated mitochondrial dynamics in human neuronal cultures and fo
200 ls, and very little is currently known about mitochondrial dynamics in mature axons of the mammalian
201 vestigated the role of molecules involved in mitochondrial dynamics in medium spiny neurons (MSNs) fr
202 igenesis, little is known about the roles of mitochondrial dynamics in metastasis, the major cause of
203 embrane protein that plays a pivotal role in mitochondrial dynamics in most tissues, yet mutations in
204 s though to initiate, suggests that impaired mitochondrial dynamics in motor neurons may be involved
205                            The regulation of mitochondrial dynamics in neurite outgrowth by retinoic
206 regulating Milton GlcNAcylation, OGT tailors mitochondrial dynamics in neurons based on nutrient avai
207 inally identified as a negative regulator of mitochondrial dynamics in neurons, is abundantly express
208                     These interactions alter mitochondrial dynamics in neurons, thereby facilitating
209    In this work, we investigated the role of mitochondrial dynamics in organismal stress response.
210  has been investigated in depth, the role of mitochondrial dynamics in regulating early germ cell beh
211 EM reconstruction argues for a major role of mitochondrial dynamics in regulating neuronal survival.
212              Our data also support a role of mitochondrial dynamics in regulating oxidative stress-me
213 rial plasticity has emerged, pointing toward mitochondrial dynamics in regulating stem cell fate deci
214 These observations shed light on the role of mitochondrial dynamics in the biology and drug response
215  studies suggest that Akt3 is a regulator of mitochondrial dynamics in the vasculature via regulation
216 ittle is known about the normal functions of mitochondrial dynamics in these neurons, especially in a
217     Little is known about the involvement of mitochondrial dynamics in tolerance of skeletal muscle a
218         To elucidate the functional roles of mitochondrial dynamics in vivo, we identified genes that
219 al cellular biological process controlled by mitochondrial dynamics in VMH regulation of systemic glu
220 protein Drp1 and factors that cause abnormal mitochondrial dynamics, including GTPase Drp1 enzymatic
221 Drp1 S-palmitoylation accompanied by altered mitochondrial dynamics, increased glycolysis, glutaminol
222 d biochemical studies revealed that impaired mitochondrial dynamics-increased mitochondrial fragmenta
223 is not entirely clear the impact of impaired mitochondrial dynamics induced by alpha-syn on neurodege
224                                              Mitochondrial dynamics is a conserved process by which m
225                        Here we describe that mitochondrial dynamics is a physiological regulator of a
226 the dendritic tree, indicating that abnormal mitochondrial dynamics is an early event in the pathogen
227                          It is believed that mitochondrial dynamics is coordinated with endosomal tra
228                                              Mitochondrial dynamics is crucial for the regulation of
229                 How PTMs contribute to plant mitochondrial dynamics is just beginning to be elucidate
230                          Therefore, abnormal mitochondrial dynamics is likely a common feature of ALS
231                 Another critical function of mitochondrial dynamics is the removal of damaged and dys
232  their fusion and fission, a process termed 'mitochondrial dynamics', is crucial for neurons, given t
233 cterized role for LYCAT in the regulation of mitochondrial dynamics, its involvement in lung cancer,
234 isrupt important cellular processes, such as mitochondrial dynamics, leading to elevated stability an
235    Expanding evidence suggests that impaired mitochondrial dynamics likely contribute to the selectiv
236 apies that target aberrant regulation of the mitochondrial dynamics machinery and characterizing the
237 12V)-mediated cellular transformation on the mitochondrial dynamics machinery and observe a positive
238     Our data indicate that interference with mitochondrial dynamics may be an unappreciated strategy
239                               Disruptions in mitochondrial dynamics may contribute to the selective d
240 ogether, these results suggest that impaired mitochondrial dynamics may contribute to the selective d
241 he hope that pharmacological manipulation of mitochondrial dynamics may have therapeutic benefit.
242 r prognosis in glioblastoma, suggesting that mitochondrial dynamics may represent a therapeutic targe
243 s and protein levels of genes related to the mitochondrial dynamics, mitochondrial biogenesis and syn
244                       To study one aspect of mitochondrial dynamics-mitochondrial fission-in mouse DA
245                                              Mitochondrial dynamics, mitophagy and calcium uptake pro
246 n quality control, mitochondrial DNA repair, mitochondrial dynamics, mitophagy and mitochondrial biog
247 gy demand, are particularly dependent on the mitochondrial dynamics, mitophagy represents a key mecha
248                 Membrane homeostasis affects mitochondrial dynamics, morphology, and function.
249 s, highlighting the association of defective mitochondrial dynamics, mtDNA multiple deletions, and al
250 mitochondrial membrane protein implicated in mitochondrial dynamics, nucleoid organization, protein t
251      We also show that the Wnt-5a effects on mitochondrial dynamics occur with an increase in both in
252                                   Therefore, mitochondrial dynamics of cancer cells adapting to the h
253 and biochemical relationship, the effects of mitochondrial dynamics on skeletal muscle contractility
254                     However, reactivation of mitochondrial dynamics only occurs after transfer to ger
255 vulnerability (Ryr3), resilience (Oxr1), and mitochondrial dynamics (Opa1), suggesting high age-relat
256 now show that tumours reprogram a network of mitochondrial dynamics operative in neurons, including s
257 heart did not result in evidence of abnormal mitochondrial dynamics or heart failure.
258                               In conclusion, mitochondrial dynamics participates in the control of he
259                     Our study indicates that mitochondrial dynamics play a critical role in regulatin
260                                              Mitochondrial dynamics play an important role within sev
261 ur findings support the notion that abnormal mitochondrial dynamics plays an early and causal role in
262 verses PINCH-1 deficiency-induced defects on mitochondrial dynamics, proline synthesis and cell proli
263 dy highlights the limitation of categorizing mitochondrial dynamics proteins based on architecture me
264  (Fis1), mitochondrial fission factor (Mff), mitochondrial dynamics proteins of 49 and 51 kDa (MiD49
265 human diseases that result from mutations in mitochondrial dynamics proteins.
266 eful for revealing noncanonical functions of mitochondrial dynamics proteins.
267       Implicated mechanisms involve impaired mitochondrial dynamics, reduced organelle biogenesis and
268                                   Changes in mitochondrial dynamics regulate stem cell fate decisions
269                        Here, we uncover that mitochondrial dynamics regulates stem cell identity, sel
270 n the absence of Fzo1, which is required for mitochondrial dynamics/respiration.
271                       Our results reveal how mitochondrial dynamics respond to cellular and tissue in
272                                  Deregulated mitochondrial dynamics resulting in defective mitochondr
273 ctivity, oxidative stress detoxification and mitochondrial dynamics, resulting in increased levels of
274     A genetic screen targeting regulators of mitochondrial dynamics revealed that mitochondrial fusio
275 tudy was to determine how PIM kinases impact mitochondrial dynamics, ROS production, and response to
276                          During cell stress, mitochondrial dynamics shift to fission, leading to mito
277   Interference with this pathway deregulates mitochondrial dynamics, shuts off subcellular organelle
278 ent beta-cells, demonstrating that defective mitochondrial dynamics solely affect substrate supply up
279  extracellular vesicles (sEVs) that activate mitochondrial dynamics, stimulate mitochondrial movement
280 ese differential degradation rates depend on mitochondrial dynamics, suggesting a mechanism coupling
281          Thus SLC25A46 is a new component in mitochondrial dynamics that serves as a regulator for MF
282                                             'Mitochondrial dynamics', the processes of mitochondrial
283               We show that KIF1Bbeta affects mitochondrial dynamics through CN-dependent dephosphoryl
284 hese data suggest that mutant TDP-43 impairs mitochondrial dynamics through enhanced localization on
285 is known to participate in the regulation of mitochondrial dynamics through interaction with the mito
286 These results suggest that the regulation of mitochondrial dynamics through TMEM135 is critical for p
287 nism coupling weak physical segregation with mitochondrial dynamics to achieve a distillation-like ef
288 erae Type-III-secreted effector that targets mitochondrial dynamics to dampen host innate immune sign
289                   This pathway may reprogram mitochondrial dynamics to differentially adjust energy p
290 rmed "MECA," that functions in parallel with mitochondrial dynamics to distribute and position the es
291 lar homeostasis ranging from proteostasis to mitochondrial dynamics to energy metabolism.
292                                 Studies link mitochondrial dynamics to the balance between energy dem
293                 Here we show that modulating mitochondrial dynamics toward increased fusion through e
294                                Dysfunctional mitochondrial dynamics, transport, homeostatic control o
295 ative phosphorylation capacity (OXPHOS), and mitochondrial dynamics, turnover, and plasticity.
296 nce of parkin, PINK1, and alpha-synuclein on mitochondrial dynamics uncovers a common function of the
297 Consistent with a role for NIK in regulating mitochondrial dynamics, we demonstrate that Drp1 is requ
298 , its establishment and maintenance requires mitochondrial dynamics, which can be controlled by the m
299 oblasts were characterized by a slow rate of mitochondrial dynamics, which was reversed by expression
300 Understanding how lipid metabolism regulates mitochondrial dynamics will reveal its role in cellular

 
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