ライフサイエンスコーパス: mitochondrial gene

1 fragments (amplicons <200 base pairs) of the mitochondrial gene.

2 dinucleotide dehydrogenase subunit IV (ND4) mitochondrial gene.

3 mains, and coopted the promoter of a genuine mitochondrial gene.

4 l benefits via transcriptional activation of mitochondrial genes.

5 -S and cms-T cytotypes is linked to chimeric mitochondrial genes.

6 FKBIA, for activated pathways and RICTOR for mitochondrial genes.

7 expression, whereas having little effect on mitochondrial genes.

8 l benefits via transcriptional activation of mitochondrial genes.

9 tem for the proper expression of nuclear and mitochondrial genes.

10 rial NADH kinase, is a mutator, specific for mitochondrial genes.

11 ere assigned to key metabolic and regulatory mitochondrial genes.

12 gulating alternative splicing alterations of mitochondrial genes.

13 sed oxygen consumption and the expression of mitochondrial genes.

14 n of some stress-related and nuclear-encoded mitochondrial genes.

15 ete and seven partial sets of protein coding mitochondrial genes.

16 itochondrial DNA hinder the transcription of mitochondrial genes.

17 titive nuclear genomic elements (LINE-1) and mitochondrial genes.

18 ypes in rare diseases caused by mutations in mitochondrial genes.

19 mino acid homology of 91-97% among the seven mitochondrial genes.

20 receptor-coactivator 1alpha (PGC-1alpha) and mitochondrial genes.

21 assess potential biases, primer sets of two mitochondrial genes, 12S and 16S rRNA, were used alongsi

22 lar molecule of 16,065 bp and encodes the 37 mitochondrial genes (13 protein-coding, 22 tRNAs, and tw

23 Mitochondrial genes accumulate substitutions more quickl

24 Finally, more variable mitochondrial genes also exhibit a lower identity with n

25 ation and aggregation, splicing changes of a mitochondrial gene and mitochondrial defects.

26 ng a platform for introduction of almost any mitochondrial gene and perhaps even allowing insertion o

27 All of them displayed 37 typical animal mitochondrial genes and an A+T-rich region.

28 receptor, the glucocorticoid receptor, p11, mitochondrial genes and cannabinoids are bringing new pe

29 We explore the possibility of re-engineering mitochondrial genes and expressing them from the nucleus

30 tion was also critical for the expression of mitochondrial genes and for the processing of self-splic

31 We observe a marked suppression of mitochondrial genes and function across cohorts in activ

32 red by TNF as evidenced by downregulation of mitochondrial genes and increased free radical productio

33 on the nucleotide sequences of COI and COII mitochondrial genes and ITS2 spacer of nuclear ribosomal

34 ism and exercise capacity by using different mitochondrial genes and mechanisms in a tissue-specific

35 g that compatibility between nuclear-encoded mitochondrial genes and minicircle-encoded guide RNA gen

36 ssimilation were up-regulated, while diverse mitochondrial genes and other metabolic enzymes were dow

37 over, these cells have reduced expression of mitochondrial genes and reduced mitochondrial activity,

38 al enhancers in proximity to nuclear-encoded mitochondrial genes and that many of these binding sites

39 Parallel transfer of mitochondrial genes and their functional replacement by

40 nts, and DNA methylation analysis identified mitochondrial genes and their transcriptional regulators

41 e barcode region in the cytochrome oxidase I mitochondrial gene, and analyzed representative BMSB sam

42 e mostly and abundantly been described using mitochondrial genes, and in particular cytochrome b (cyt

43 le, decreasing expression of nuclear-encoded mitochondrial genes, and increasing extracellular matrix

44 loroplast integrants exist apart from native mitochondrial genes, and only a few, involving chloropla

45 on gene expression of glucose transporter 4, mitochondrial genes, and PGC-1alpha itself are almost en

46 ore of the membrane arm, are translated from mitochondrial genes, and the remaining subunits, the pro

47 rsal GRR include ribosomal, translation, and mitochondrial genes, and those with negative GRR include

48 Given that six mitochondrial genes are located within 22q13, including

49 Because mitochondrial genes are transcribed by a dedicated RNA p

50 regulation, and that several nuclear-encoded mitochondrial genes are under strong translational contr

51 Considerable evidence supports mutations in mitochondrial genes as the cause of maternally inherited

52 tional mechanisms for expressing nuclear and mitochondrial genes as well as unusual subcellular local

53 ociated with energy metabolism, particularly mitochondrial genes, as well as alterations in the expre

54 revealed over a dozen previously overlooked mitochondrial genes at the level of eukaryotic supergrou

55 single-nucleotide polymorphism (SNP) in the mitochondrial gene, atp1.

56 which lineage sorting has taken place in the mitochondrial genes but not yet in the nuclear alleles.

57 of recurrent conversion of short patches of mitochondrial genes by chloroplast homologs during angio

58 genus Encope, based on one nuclear and four mitochondrial genes, calibrated with fossils at multiple

59 ranscriptional regulation of nuclear-encoded mitochondrial genes can result in experimental glomerula

60 tatively linking male sterility to orf293, a mitochondrial gene causing homeotic conversion of anther

61 ed the barriers to transfer of yeast COX2, a mitochondrial gene coding for a subunit of cytochrome c

62 r, the putative pseudogene DeltaybgF and the mitochondrial gene COI, respectively.

63 alpha-F1, EF1alpha-F2, and Wingless) and one mitochondrial gene (COI) sampled from populations in Flo

64 In the present study, we use three mitochondrial genes (COI, ND1 and ND5) and nine microsat

65 A large number of these mitochondrial genes contain binding sites for the transc

66 The mitochondrial gene content included a majority of N. tab

67 he altered dosage of one or several of these mitochondrial genes contributing to 22qDS etiology and/o

68 ve was to determine the relationship between mitochondrial gene copy number and transcript abundance

69 Mitochondrial gene copy number decreased from 3 to 8-DPA

70 ication of these sublimons allows individual mitochondrial gene copy numbers to vary independently be

71 d processed to produce DNA barcodes from the mitochondrial gene cytochrome c oxidase I (COI).

72 ty (h) and nucleotide diversity (pi) for the mitochondrial gene cytochrome c oxidase subunit I (cox1)

73 bfamilies of Chinese skippers based on three mitochondrial genes (cytochrome b (Cytb), the NADH dehyd

74 species delimitation techniques based on two mitochondrial genes (cytochrome c oxidase I and 16S rRNA

75 We show that the barcoding region of the mitochondrial gene, cytochrome oxidase subunit I (COI),

76 mparative approach in which we sequenced two mitochondrial genes, cytochrome b (cob) and cytochrome o

77 of the remaining patients (2.2% overall) had mitochondrial gene deletions consistent with Pearson mar

78 Phylogenetic analysis of plastid and mitochondrial genes demonstrated that the three endosymb

79 nduction is accompanied by the expression of mitochondrial genes (e.g., TFAM, MtCO1) and increased mi

80 Because mitochondrial genes encode proteins essential for aerobi

81 By mutating or deleting mitochondrial genes encoded in the mtDNA [NADH dehydroge

82 visual loss when caused by a mutation in the mitochondrial gene encoding NADH:ubiquinone oxidoreducta

83 forms using the nucleotide sequences of the mitochondrial gene encoding the cytochrome c oxidase sub

84 itochondrial targeting sequence to carry the mitochondrial gene encoding the human NADH ubiquinone ox

85 howed a higher abundance of transcripts from mitochondrial genes encoding oxidative phosphorylation p

86 s display aberrant expression of nuclear and mitochondrial genes encoding subunits of complex V resul

87 LPv and F-measure as well as between LPv and mitochondrial gene enrichment p-values were 0.90 and 0.9

88 Some mitochondrial genes exhibit atypically high degrees of n

89 d sequences of at least three intron-lacking mitochondrial genes exhibit elevated nucleotide diversit

90 A more detailed mitochondrial gene expression analysis revealed that in

91 ly of mTERF proteins whose members influence mitochondrial gene expression and DNA replication.

92 ) gamma agonist, by systematically analyzing mitochondrial gene expression and function in two mouse

93 and saturated fatty acids decrease PGC-1 and mitochondrial gene expression and function via p38 MAPK-

94 Mitochondrial gene expression and function were not indu

95 However, PPARdelta agonism does not induce mitochondrial gene expression and function.

96 are accompanied by correspondingly increased mitochondrial gene expression and higher expression of P

97 down of YY1 caused a significant decrease in mitochondrial gene expression and in respiration, and YY

98 o statistically significantly down-regulates mitochondrial gene expression and induces oxidative stre

99 equired for the full CR-induced increases in mitochondrial gene expression and mitochondrial density

100 s role in post-transcriptional regulation of mitochondrial gene expression and more recently because

101 Akt3, but not Akt1, results in a decrease in mitochondrial gene expression and mtDNA content.

102 A new study reveals that decreased mitochondrial gene expression and reduced lipid biosynth

103 ard to the mechanism and regulation of human mitochondrial gene expression and the potential multi-fu

104 Defects in mitochondrial gene expression are associated with aging

105 our work reinforces the idea that changes in mitochondrial gene expression are key players in the pre

106 Similar effects on PGC-1beta and mitochondrial gene expression are seen in vivo; fraction

107 encoding a variety of proteins required for mitochondrial gene expression at both the transcriptiona

108 translational regulation of nuclear-encoded mitochondrial gene expression by 4E-BP plays an importan

109 Taken together, ClpXP regulates mitochondrial gene expression by changing the protein le

110 cin-like enhancer of split 3 (TLE3) inhibits mitochondrial gene expression in beige adipocytes.

111 In addition, we observed 40% residual mitochondrial gene expression in FRDA patient blood.

112 oteins that interact with POLRMT to regulate mitochondrial gene expression in humans.

113 ated pattern of reduction in nuclear-encoded mitochondrial gene expression in insulin resistance.

114 vulgaris an excellent model for the study of mitochondrial gene expression in plants.

115 down of PPARdelta affects PGC-1alpha-induced mitochondrial gene expression in primary myotubes.

116 d cannot defend their ATP levels or increase mitochondrial gene expression in response to reduced oxi

117 ck cycles may provide switch-like control of mitochondrial gene expression in response to the metabol

118 Here, we examine T. cruzi mitochondrial gene expression in the mammalian intracell

119 plicing isoform NT-PGC-1alpha, which control mitochondrial gene expression in the nucleus.

120 genome-wide transcriptomic study to examine mitochondrial gene expression in the prefrontal cortex (

121 Mitochondrial gene expression in Trypanosoma brucei invo

122 RNA editing are essential for regulation of mitochondrial gene expression in Trypanosoma brucei.

123 h the synthetic ligand, GW501516, on FAO and mitochondrial gene expression in vitro and in vivo.

124 uced inflammation and surprisingly increased mitochondrial gene expression in white adipose tissue al

125 ription-PCR was used to determine changes in mitochondrial gene expression induced by MMC treatment.

126 Mitochondrial gene expression is a fundamental process t

127 nder strong evolutionary constraint and that mitochondrial gene expression is highly conserved across

128 In ANT1-deficient skeletal muscle, mitochondrial gene expression is induced in association

129 The induction of mitochondrial gene expression is more dependent on DMF's

130 Mitochondrial gene expression is necessary for proper mi

131 egulation of SERCA2 (0.77-fold, P=0.009) and mitochondrial gene expression levels and upregulation of

132 Consequent abnormal mitochondrial gene expression may contribute to the deve

133 heir interactions with other elements of the mitochondrial gene expression pathway.

134 Outbreak strains share a unique mitochondrial gene expression profile and an increased a

135 s recently emerged as a central regulator of mitochondrial gene expression through the function of an

136 of the molecular processes mediating altered mitochondrial gene expression to dysfunction associated

137 Mitochondrial gene expression was increased in adipose t

138 e analysis revealed that YY1 is required for mitochondrial gene expression, and ultrastructural analy

139 n complexes that regulate different steps in mitochondrial gene expression, at least one of which doe

140 patterns, but not global levels, and altered mitochondrial gene expression, copy number, and oxygen r

141 Post-transcriptional control of mitochondrial gene expression, including the processing

142 Concurrent with these changes in mitochondrial gene expression, lower O(2) consumption wa

143 positive U343MG glioblastoma line suppressed mitochondrial gene expression, reduced mitochondrial mem

144 increase is accompanied by downregulation of mitochondrial gene expression, similar to that observed

145 ction, parallel to other factors controlling mitochondrial gene expression, such as NRF1 and NRF2/GAB

146 cts are independent of the downregulation of mitochondrial gene expression, suggesting novel mechanis

147 at Cyp-D could play a key role in regulating mitochondrial gene expression.

148 volved in post-transcriptional regulation of mitochondrial gene expression.

149 on, we examined whether Cyp-D could regulate mitochondrial gene expression.

150 proach is likely to be useful for studies of mitochondrial gene expression.

151 factor is an essential protein necessary for mitochondrial gene expression.

152 s known about translational control of plant mitochondrial gene expression.

153 tics suggested that ABCC6 deficiency affects mitochondrial gene expression.

154 RNA polymerase, is essential for maintaining mitochondrial gene expression.

155 putative higher order structures coordinate mitochondrial gene expression.

156 f acetyl-CoA, and which in turn may regulate mitochondrial gene expression.

157 tion and/or ribosome biogenesis during human mitochondrial gene expression.

158 tutes one of the key regulatory steps during mitochondrial gene expression.

159 ed as key post-transcriptional regulators of mitochondrial gene expression.

160 to the relevance of RNA pseudouridylation in mitochondrial gene expression.

161 eutics by manipulating nuclear regulation of mitochondrial gene expression.

162 tribute to between-individual variability in mitochondrial gene expression.

163 N, including higher STAT1 levels and reduced mitochondrial gene expression.

164 f post-transcriptional mRNA modification for mitochondrial gene expression.

165 al or nuclear genes able to cause defects in mitochondrial gene expression.

166 Comparison of the mitochondrial gene flanking sequences from normal fertil

167 Conversely, mitochondrial gene flow is more extensive between non-Af

168 redicted genes, our data identified nine new mitochondrial genes, four of which encode proteins that

169 dendritic maturation, altered expression of mitochondrial genes, fragmented mitochondria, impaired m

170 lus nasutus is due to interactions between a mitochondrial gene from M. guttatus and two tightly link

171 quence the full complement of protein coding mitochondrial genes from pooled samples using the 454/Ro

172 s from glass sponges and examined individual mitochondrial genes from three others.

173 NA targets in cells and the brain, including mitochondrial genes, genes involved in glutathione metab

174 n of both positive and negative selection on mitochondrial genes has been put forward, and the comple

175 pathogenic mutations in both mtDNA and nDNA mitochondrial genes have been identified in the past 21

176 Patterns of amino-acid polymorphism in human mitochondrial genes have been interpreted as evidence fo

177 ene content, yet it is not known why not all mitochondrial genes have been transferred to the nuclear

178 dministration up-regulated the expression of mitochondrial genes in AML-12 hepatocytes, with or witho

179 The rate of evolution of mitochondrial genes in Encope has been ten times slower

180 es differ by 11%-15% from putative thylacine mitochondrial genes in GenBank, with one of our samples

181 ed a significant downregulation of essential mitochondrial genes in glomeruli from diabetic D2 mice,

182 sequencing revealed altered expression of 41 mitochondrial genes in Gq hearts, with normalization of

183 Most foreign mitochondrial genes in Lophophytum are highly transcribe

184 in reduced expression of the thermogenic and mitochondrial genes in mice housed at ambient temperatur

185 g to decreased expression of nuclear-encoded mitochondrial genes in muscle and adipose tissue.

186 s necessary for transcriptional induction of mitochondrial genes in muscle with both RSV and SRT trea

187 pression analysis highlights the function of mitochondrial genes in oxidative phosphorylation, DNA re

188 Nuclear-encoded mitochondrial genes in particular correlate with protein

189 ns show a diminished capacity to up-regulate mitochondrial genes in response to hypoosmotic stress.

190 that activate expression of nuclear-encoded mitochondrial genes in response to hypoxia, and by loss

191 from controls in the expression profiles of mitochondrial genes in the blood, but not in liver or br

192 st study to explore genetic variation in the mitochondrial genes in the context of AIWG.

193 target of rapamycin (mTOR) did not suppress mitochondrial genes in YY1mKO mice.

194 ficantly decreased protein expression of key mitochondrial genes including cytochrome C oxidase coppe

195 ber of TCA cycle enzymes and nuclear-encoded mitochondrial genes including genes involved in oxidativ

196 transcription factors encoded by nuclear and mitochondrial genes, including mitochondrial transcripti

197 A number of nuclear encoded mitochondrial genes, including those in Complex I and IV

198 Evidence of functional changes in mitochondrial genes indicated adaptation to environmenta

199 d compensatory expression of nuclear-encoded mitochondrial genes, indicating mitochondrial biogenesis

200 n nuclear genes than in maternally inherited mitochondrial genes, indicating that dispersal by males

201 Linezolid AUC0-24 best explained the mitochondrial gene inhibition, with 50% inhibition at 94

202 The technology of introducing human mitochondrial genes into the mouse germ line has never b

203 cts are particularly puzzling given that the mitochondrial genes involved are under strong evolutiona

204 Mutations in nDNA-encoded mitochondrial genes involved in energy metabolism, antio

205 transcriptional induction of thermogenic and mitochondrial genes involved in energy metabolism.

206 s, the expression levels and copy numbers of mitochondrial genes involved in energy production and re

207 set in filamentous fungi expanded to include mitochondrial genes involved in the tricarboxylic acid (

208 Of note, downregulation of mitochondrial genes is associated with the worst clinica

209 In yeast, expression of mitochondrial genes is controlled by a group of gene-spe

210 eart, regulation of fatty acid oxidation and mitochondrial genes is controlled by the PPARgamma coact

211 Consistently, suppression of mitochondrial genes is identified as a key metabolic sig

212 uclear genes, yet the impact of selection on mitochondrial genes is significantly reduced relative to

213 As many mitochondrial genes lack introns, there is a critical ne

214 To date, two mitochondrial gene markers, Cytochrome Oxidase I (COI) a

215 Several mechanisms could be operating: mitochondrial genes, maternal genes, or fetal genes expr

216 on of in vitro evolution strategies to other mitochondrial genes might ultimately lead to yeast entir

217 howed association of NEIL2 and PNKP with the mitochondrial genes MT-CO2 and MT-CO3 (cytochrome c oxid

218 ntioxidant protection contribute to elevated mitochondrial gene mutagenesis in cells lacking the mito

219 ctional evidence for the role of a bona fide mitochondrial gene mutation in cancer.

220 However, the development of mice harboring mitochondrial gene mutations is permitting demonstration

221 One of these processes involves genomic and mitochondrial gene mutations, mitochondrial protein expr

222 e chain reaction (ddPCR) assay targeting the mitochondrial gene nad1 to diagnose schistosomiasis japo

223 Two mitochondrial genes, namely, cytochrome b (CYT B) and NA

224 e mitochondrial genome in Tachycineta, three mitochondrial genes (ND2, ND5, and CYTB) contain regions

225 indromic repeats (CRISPR) screen to identify mitochondrial genes necessary for the growth of acute my

226 These changes in mitochondrial gene networks were validated by quantitati

227 Here, we analyse the cytochrome oxidase II mitochondrial gene of 250 Sabethes albiprivus B mosquito

228 reviously appreciated and creates functional mitochondrial genes of chimeric origin.

229 Phylogenetic analysis of mitochondrial genes of H. oligactis and other cnidarians

230 sequences of Prostriata, and for nuclear and mitochondrial genes of two species of Rhipicephalus, whi

231 port the discovery, in two lineages of plant mitochondrial genes, of novel gene combinations that aro

232 ects on the transcription of nuclear-encoded mitochondrial genes or through direct effects of the mut

233 h, we identified macroevolutionary shifts in mitochondrial gene order among the freshwater mussels (U

234 Mitochondrial gene order within Decemunciger was similar

235 An overview of mitochondrial gene orders (MGOs) reveals a high level of

236 stigated the hypothesis that nuclear-encoded mitochondrial genes, particularly those involved in oxid

237 eprogramming of inflammatory, metabolic, and mitochondrial gene pathways in old mice that appeared to

238 us population sources that likely shaped the mitochondrial gene pool of ancient Umbri over time, sinc

239 The mitochondrial gene pool thus may contain signals of loca

240 We found that various mitochondrial genes possessed shorter and less structure

241 we identified mutations in a nuclear-encoded mitochondrial gene, ppr, a homolog of human LRPPRC.

242 te provides predictive RNA editors for plant mitochondrial genes (PREP-Mt), for chloroplast genes (PR

243 how that when there is a benefit to having a mitochondrial gene present in the nucleus, but absent in

244 .004) and within nuclear-encoded genes with mitochondrial gene products (odds ratio, 1.075; 95% CI,

245 rix, and assembled into the complex with the mitochondrial gene products ATP6 and ATP8.

246 Many mitochondrial gene products exhibit extraordinary levels

247 Translation of mitochondrial gene products in Saccharomyces cerevisiae

248 biogenesis in which Cox1p and the two other mitochondrial gene products, Cox2p and Cox3p, constitute

249 Unlike Atp9p, the other mitochondrial gene products, including ATPase subunits A

250 To further our understanding of mitochondrial gene regulation, we identified proteins th

251 complex that coordinates various aspects of mitochondrial gene regulation.

252 y rescues myocyte triglyceride accumulation, mitochondrial gene regulatory derangements, and contract

253 eby coordinate the expression of nuclear and mitochondrial genes relevant to mitochondrial biogenesis

254 Potential clinical applications include mitochondrial gene replacement therapy to prevent transm

255 Mitochondrial gene reprogramming induced by CaMKIIdelta

256 Here we introduced a mutant human mitochondrial gene responsible for Leber hereditary opti

257 Phylogenetic trees inferred from mitochondrial genes retrieve Xenacoelomorpha as an early

258 us nocturnus and Pachycerianthus magnus, the mitochondrial gene sequences could not be assembled into

259 zed using isozyme analysis and ribosomal and mitochondrial gene sequences.

260 atter of which was based on both nuclear and mitochondrial gene sequences.

261 t elevation in expression of nuclear-encoded mitochondrial gene sets and a mitonuclear protein imbala

262 he hypothesis of resistance exercise-induced mitochondrial gene-shifting in muscle containing satelli

263 and second codon positions) across multiple mitochondrial genes strongly supports a radically differ

264 Both uncouplers increased the expression of mitochondrial genes such as Tfam and COXIV while inducin

265 e expression of a cluster of nuclear-encoded mitochondrial genes, suggesting a role for VEGF in the r

266 chondrial number and deregulation of several mitochondrial genes, suggesting towards a specific role

267 Up-regulated mitochondrial genes support in vitro evidence associatin

268 ative stress, unlike many other mutations in mitochondrial genes that are associated with elevated ox

269 iption factor (ETS) that binds and activates mitochondrial genes that are required for electron trans

270 intracellular domain (NICD1) to nuclear and mitochondrial genes that encode respiratory chain compon

271 highly enriched (1.2 x 10(-9)) with nuclear mitochondrial genes that play a role in oxidative phosph

272 requires the expression of both nuclear and mitochondrial genes, the import of proteins, cofactor bi

273 on, and the up-regulation of nuclear-encoded mitochondrial genes, thus enhancing the capacity of the

274 (106 and 120-bp) and buffalo (90 and 138-bp) mitochondrial genes to discriminate beef and buffalo in

275 A recent study marshals mitochondrial genes to place that root between the enigm

276 Experimentally relocating mitochondrial genes to the nucleus for functional expres

277 The transfer of mitochondrial genes to the nucleus is a recurrent and co

278 Functional transfer of mitochondrial genes to the nucleus is very common in som

279 al and self-fertilizing plants transfer more mitochondrial genes to their nuclei than do outcrossing

280 nt enrichment of ancestry at nuclear-encoded mitochondrial genes towards the source populations contr

281 Using microarray technology, adipose mitochondrial gene transcription was studied in db/db, h

282 pression of stress response genes, exuberant mitochondrial gene transcription, and increased expressi

283 Nuclear-encoded mitochondrial gene transcripts are also found to decreas

284 mption and altered expression of many muscle mitochondrial gene transcripts.

285 ement of native by foreign genes, functional mitochondrial gene transfer to the nucleus, and/or loss

286 urther characterize factors that control the mitochondrial gene transfer/retention process, we have i

287 lve existing differences between nuclear and mitochondrial gene trees.

288 Early upregulation of mitochondrial genes (Ucp3 and Cpt1) and downregulation o

289 Most modern eukaryotes transmit mitochondrial genes uniparentally, often without recombi

290 A survey of 362 C-to-U editing sites in 33 mitochondrial genes was conducted on RNA extracted from

291 he rate of evolution for the nuclear-encoded mitochondrial genes was greater in post-WGD species than

292 The effects of RSG on adipose mitochondrial genes were confirmed by quantitative RT-PC

293 n volumes (>/=100 mul) and PCR targeting the mitochondrial genes were demonstrated.

294 a set of accessory gland-specific genes and mitochondrial genes were downregulated in males with res

295 Surprisingly, we found that when mitochondrial genes were highly transcribed, they also h

296 Importantly, nuclear-encoded mitochondrial genes were identified as the major functio

297 f mitochondrial complexes I, II, and III and mitochondrial genes were observed in rats given TNF.

298 We show that transcripts from many mitochondrial genes were similar to those from uninfecte

299 92% of 5,471 exons from 524 nuclear-encoded mitochondrial genes were successfully amplified from gen

300 o sustain the transcription of metabolic and mitochondrial genes, whose levels are unchanged compared