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1 educed mitochondrial DNA content and reduced mitochondrial mRNA.
2 rry out guide-RNA-dependent U-insertion into mitochondrial mRNA.
3 ct the insertion and deletion of uridines in mitochondrial mRNAs.
4 s required throughout the life cycle of most mitochondrial mRNAs.
5 ression relies on mitoribosomes to translate mitochondrial mRNAs.
6 r element by Rmd9 confers stability to yeast mitochondrial mRNAs.
7 generate open reading frames in trypanosomal mitochondrial mRNAs.
8 required for U-insertion/deletion editing of mitochondrial mRNAs.
9 he leaderless or nearly leaderless nature of mitochondrial mRNAs.
10 ng and thereby generate mature forms of some mitochondrial mRNAs.
11 for correcting the coding sequences of many mitochondrial mRNAs.
12 affinity to 5'-untranslated regions of yeast mitochondrial mRNAs.
13 ation phenomenon required for translation of mitochondrial mRNAs.
14 ement to facilitate degradation of T. brucei mitochondrial mRNAs.
15 specific for the 5'-untranslated regions of mitochondrial mRNAs.
16 equences in 5'-untranslated regions of yeast mitochondrial mRNAs.
17 is required for 5' end processing of several mitochondrial mRNAs.
18 eletion of uridines at specific sites within mitochondrial mRNAs.
19 eotide sequence present in all S. cerevisiae mitochondrial mRNA 5'-UTLs that is a potential rRNA bind
20 mLRPPRC1 and causes non-uniform increases of mitochondrial mRNAs, accumulation of some unprocessed mi
22 asts showed decreased steady-state levels of mitochondrial mRNAs, although the length of poly(A) tail
23 significantly higher levels of steady-state mitochondrial mRNA and butyrate-induced p21(WAF1/Cip1) a
27 rotein that associates with both nuclear and mitochondrial mRNAs and as such is a potential candidate
30 -dependent degradation pathway for T. brucei mitochondrial mRNAs and reveal an unprecedented role for
32 transport profiles of mitochondrial and non-mitochondrial mRNA, and differences in transport paramet
33 es short and long A-tails typically found in mitochondrial mRNAs, and decreases the abundance of neve
34 ifically to 5'-untranslated regions of yeast mitochondrial mRNAs, and transcripts containing these re
35 rance, perhaps involved in recruiting unique mitochondrial mRNAs; and it has a polypeptide exit tunne
38 l process in kinetoplastid parasites whereby mitochondrial mRNAs are modified through the specific in
39 A-containing cargoes, including granules and mitochondrial mRNA, are transported within neuronal proj
40 RBP SYNJ2BP retains specific nuclear-encoded mitochondrial mRNAs at the OMM during translation stress
42 specifically removes poly(A) extensions from mitochondrial mRNAs both in vitro and in mitochondria of
44 osoma brucei inserts and deletes uridines in mitochondrial mRNAs by a series of enzymatic steps that
45 diting in Trypanosomatids creates functional mitochondrial mRNAs by extensive uridylate (U) insertion
47 ce for binding of 5'-untranslated regions of mitochondrial mRNAs by yeast mitochondrial NADP+-specifi
49 lleviation of catalytic inhibition of IDH by mitochondrial mRNA correlates with the property of allos
50 uding binding and regulating nuclear-encoded mitochondrial mRNAs, coupled with a role in mitigating p
51 ia, or that a mechanistically unique type of mitochondrial mRNA editing has evolved within the dinofl
55 hic Parkinson's disease (IPD), expression of mitochondrial mRNA encoding the ND1 subunit of mitochond
56 despite the previously observed increases in mitochondrial mRNAs encoding electron transport chain (E
57 Brar et al. reveal a novel mechanism where mitochondrial mRNAs escape global translational repressi
61 hen subjected to poly(A) tail-length assays, mitochondrial mRNAs from affected individuals were shown
63 RSF2P95H/+ mutation disrupts the splicing of mitochondrial mRNAs, impairs mitochondrial complex I fun
64 rized the transport of mitochondrial and non-mitochondrial mRNA in differentiated axons and dendrites
68 dence of extensive substitutional editing of mitochondrial mRNAs in the dinoflagellate species Pfiest
70 nd deletion RNA editing generates functional mitochondrial mRNAs in Trypanosoma brucei Editing is cat
75 DNA shuffling, we have mapped the termini of mitochondrial mRNAs in wheat, a monocot, and compared th
76 translational termination of nuclear-encoded mitochondrial mRNAs, including complex-I 30kD subunit (C
77 protein complex called the editosome cleaves mitochondrial mRNA, inserts or deletes uridine nucleotid
82 xamining the developmental timing of altered mitochondrial mRNA levels, we also reveal transcript-spe
84 l disorders, our findings reveal a defect of mitochondrial mRNA maturation associated with human dise
85 uridylate insertion or deletion cycles is a mitochondrial mRNA maturation process catalyzed by multi
86 levels and half-lives of a subset of mature mitochondrial mRNAs: ND2, ND3, CYTB, COX2, and ATP8/6.
92 in vitro but did not affect the stability of mitochondrial mRNAs or alter the expression of nuclear g
93 ase did not affect the number or sequence of mitochondrial mRNA poly(A) tails, where unexpectedly we
95 to stabilize mitochondrial mRNAs, to promote mitochondrial mRNA polyadenylation, and to coordinate mi
97 essing and degradation, miRNA biogenesis and mitochondrial mRNA processing, systematically decoding t
98 Dramatically elevated levels of the COX2 mitochondrial mRNA-specific translational activator prot
100 These findings demonstrate the control of mitochondrial mRNA synthesis by a protein that has an es
102 ocesses such as kinetoplast DNA replication, mitochondrial mRNA synthesis, glycosyl phosphatidylinosi
104 e second pathway results in slow turnover of mitochondrial mRNA (t(1/2) of approximately 3 h) and is
105 increased the levels of mutant COX3 and COX2 mitochondrial mRNAs that were destabilized by mutations
106 brucei editosome catalyzes the maturation of mitochondrial mRNAs through the insertion and deletion o
109 echanistically, CBFB enhanced the binding of mitochondrial mRNAs to TUFM, a mitochondrial translation
110 the post-transcriptional level to stabilize mitochondrial mRNAs, to promote mitochondrial mRNA polya
111 iting is a process that creates translatable mitochondrial mRNA transcripts from cryptogene encoded R
112 ments indicate a role for MRB1590 in editing mitochondrial mRNA transcripts, in particular the transc
114 e caused primarily by inhibition of the COX1 mitochondrial mRNA translation, a finding confirmed by l
117 dine insertion/deletion (U-indel) editing of mitochondrial mRNA, unique to the protistan class Kineto
120 By contrast, depletion of Not1 solubilizes mitochondrial mRNAs, which are rendered insoluble upon N
121 l ribosomes to facilitate the translation of mitochondrial mRNAs, which lack typical anti-Shine-Dalga