ライフサイエンスコーパス: mitochondrial respiration

1 anti-inflammatory signals depend on enhanced mitochondrial respiration.

2 nflammatory SASP by enhancing glycolysis and mitochondrial respiration.

3 hat venetoclax-sensitive myeloma has reduced mitochondrial respiration.

4 CAF1 or COX7A2L), promoting SCs and enhanced mitochondrial respiration.

5 glucose production associated with increased mitochondrial respiration.

6 Additionally, Hif1a is an inhibitor of mitochondrial respiration.

7 lasmic unfolded protein response and impairs mitochondrial respiration.

8 ability of the complex, as well as decreased mitochondrial respiration.

9 ation and transfer fuel-derived electrons to mitochondrial respiration.

10 with consequent control over glycolysis and mitochondrial respiration.

11 and autophagy under conditions that require mitochondrial respiration.

12 O(2) through AOX (especially AOX1)-dependent mitochondrial respiration.

13 itochondrial superoxide levels, and impaired mitochondrial respiration.

14 transfer into mitochondria, thus stimulating mitochondrial respiration.

15 ic state via histology, gene expression, and mitochondrial respiration.

16 NKA, as an HNF1A target necessary for normal mitochondrial respiration.

17 st hemodynamics, and cerebral and myocardial mitochondrial respiration.

18 Drosophila ovary, which necessitates active mitochondrial respiration.

19 ess so for a subset of genes associated with mitochondrial respiration.

20 perate to enhance ANT transport capacity and mitochondrial respiration.

21 m mice treated with Honokiol showed enhanced mitochondrial respiration.

22 I) hemes, most of which were associated with mitochondrial respiration.

23 Q (CoQ) biosynthesis-a pathway essential for mitochondrial respiration.

24 d lactate production, and partially restored mitochondrial respiration.

25 m-like cells, which is blocked by inhibiting mitochondrial respiration.

26 e is mirrored by a significant impairment in mitochondrial respiration.

27 chondrial membrane potential, and diminished mitochondrial respiration.

28 nergy through glycolysis rather than through mitochondrial respiration.

29 ation of glutamate enables cells to maintain mitochondrial respiration.

30 hosphorylation genes resulting in diminished mitochondrial respiration.

31 insight into the transcriptional control of mitochondrial respiration.

32 ells into brown adipose tissue and increased mitochondrial respiration.

33 ome clearance by the lysosome, and decreased mitochondrial respiration.

34 the host animal, leading to normalisation of mitochondrial respiration.

35 ug doses needed to assess various aspects of mitochondrial respiration.

36 athological remodeling, and improved cardiac mitochondrial respiration.

37 etabolic activity by reducing glycolysis and mitochondrial respiration.

38 s that FAK enhances glycolysis and decreases mitochondrial respiration.

39 se idebenone as an electron donor to support mitochondrial respiration.

40 f cardiomyocytes with 12,13-diHOME increased mitochondrial respiration.

41 nd thermogenesis are two critical outputs of mitochondrial respiration.

42 e of ATP levels under conditions of impaired mitochondrial respiration.

43 lycogen in favor of fatty acid oxidation and mitochondrial respiration.

44 naling that can regulate fuel metabolism and mitochondrial respiration.

45 pentose phosphate pathway flux and increased mitochondrial respiration.

46 ratory chain Complex I that acts to restrain mitochondrial respiration.

47 m CcO and attenuate CO-induced inhibition of mitochondrial respiration.

48 & number; Drp1 GTPase enzymatic activity and mitochondrial respiration (1) in N2a cells treated with

49 , but its impact on longer term survival and mitochondrial respiration-a potential neurotherapeutic t

50 l equation models estimated that PM(2.5) and mitochondrial respiration accounted for 34% (SD = 4%) an

51 Consequently, augmenting mitochondrial respiration affects the survival of hypoxi

52 e the glycolytic utilization of glucose from mitochondrial respiration, allowing for the maintenance

53 vironmental hypoxia suppressed mass-specific mitochondrial respiration and additionally lowered the p

54 Mitochondrial respiration and aerobic capacity are thus

55 ions, Cytc is phosphorylated, which controls mitochondrial respiration and apoptosis.

56 CKMT1 inhibition altered mitochondrial respiration and ATP production, an effect

57 NAD levels may have the potential to improve mitochondrial respiration and attenuate proinflammatory

58 hat an feedforward insulin-Myc loop promotes mitochondrial respiration and biogenesis by boosting the

59 In vitro, beige adipocytes exhibit uncoupled mitochondrial respiration and cAMP-induced lipolytic act

60 itochondrial respiratory chain complexes and mitochondrial respiration and causes a shift to glycolyt

61 Accumulating evidence suggests that mitochondrial respiration and chloroplast photosynthesis

62 Accordingly, affected tissues had defects in mitochondrial respiration and complex I biogenesis that

63 Mitochondrial respiration and content were measured in p

64 Mitochondrial respiration and content were not associate

65 refore sought to determine if alterations in mitochondrial respiration and content within circulating

66 The present study examined skeletal muscle mitochondrial respiration and contractile economy in an

67 Reduced placental mTOR activity may impair mitochondrial respiration and contribute to placental in

68 loroacetate, an inhibitor of PDKs, increased mitochondrial respiration and decreased production of re

69 significantly and positively associated with mitochondrial respiration and density in mothers, but no

70 evealed high enrichment of genes involved in mitochondrial respiration and downstream targets of IL-6

71 T) OGT isoforms is associated with increased mitochondrial respiration and elevated glycolysis, sugge

72 The dual mechanisms of increased mitochondrial respiration and enterohepatic bile acid re

73 ta/Delta mutants are profoundly deficient in mitochondrial respiration and Fe accumulation, both Cu-d

74 ral metabolic pathways including glycolysis, mitochondrial respiration and glutamine metabolism.

75 from macrophages and produced a decrement in mitochondrial respiration and glycolysis compared to tho

76 Further, host mitochondrial respiration and glycolysis were enhanced.

77 increased Drp1 phosphorylation, and impaired mitochondrial respiration and glycolysis.

78 red insulin signaling and insulin-stimulated mitochondrial respiration and glycolysis.

79 TCDD-induced MDSCs had high mitochondrial respiration and glycolytic rate and exhibi

80 Two regulators of mitochondrial respiration and heat production in brown a

81 of mitochondrial disease including impaired mitochondrial respiration and hematopoetic abnormalities

82 n, the patients' B cells exhibited defective mitochondrial respiration and impaired regulation of mit

83 o identify a novel role of VCP in preserving mitochondrial respiration and in preventing the opening

84 ystemic metabolic defects, including reduced mitochondrial respiration and increased glycolysis, ener

85 nduces metabolic reprograming with repressed mitochondrial respiration and increased glycolytic activ

86 nerated rho0 variants that lacked functional mitochondrial respiration and increased glycolytic metab

87 nd contractile weakness, but lacked impaired mitochondrial respiration and increased levels of oxidat

88 Accordingly, loss of GRSF1 impaired mitochondrial respiration and increased the levels of re

89 n metabolism, DMOG treatment decreased iTreg mitochondrial respiration and increased their glycolytic

90 CCI-006 inhibited mitochondrial respiration and induced mitochondrial memb

91 we show here that bcat-1 knockdown increases mitochondrial respiration and induces oxidative damage i

92 maternal CM and mitochondrial bioenergetics (mitochondrial respiration and intracellular mitochondria

93 eserved shortly after parturition to measure mitochondrial respiration and intracellular mitochondria

94 nces whole-animal health parameters, such as mitochondrial respiration and midlife survival, increase

95 n vitro and in vivo rescues their defects in mitochondrial respiration and myogenic commitment.

96 o parallel and synergistic induction of both mitochondrial respiration and nuclear transcription fact

97 POBEC3G can be mimicked by the inhibition of mitochondrial respiration and occurs independently of HI

98 ipocytes, which included positively enriched mitochondrial respiration and oxidation pathways.

99 ix2 and Wt1 expression, as well as decreased mitochondrial respiration and prolonged reliance on glyc

100 acological agent to optimize skeletal muscle mitochondrial respiration and promote healthy aging in m

101 es glycolysis-dependent growth and increases mitochondrial respiration and reactive oxygen species (R

102 metabolites required for the maintenance of mitochondrial respiration and redox homeostasis.

103 g of ovarian cancer cells, causing decreased mitochondrial respiration and reduced ATP levels.

104 tophagy, limiting substrate availability for mitochondrial respiration and reducing gluconeogenesis.

105 ly, these findings integrate HKL action with mitochondrial respiration and shape and substantiate a p

106 llular Cu(1+) abundance and copper-dependent mitochondrial respiration and Sod1 function in the yeast

107 ongs the quality attributes, affecting basic mitochondrial respiration and starch degradation rate.

108 However, the link between mitochondrial respiration and stem cells' behavior is no

109 on homeostasis, and plays a critical role in mitochondrial respiration and synthesis of heme.

110 x mechanisms, including long-term changes in mitochondrial respiration and that patterns of early dev

111 antiretroviral drug zalcitabine to suppress mitochondrial respiration and to force glycolysis in our

112 in which leader cells preferentially utilize mitochondrial respiration and trailing follower cells re

113 ells metabolically vulnerable by attenuating mitochondrial respiration and tricarboxylic acid cycling

114 novel functions of genes-for example, DDT in mitochondrial respiration and WDFY4 in T cell activation

115 l-but not whole-cell-NAD(+) content, impairs mitochondrial respiration, and blocks the uptake of NAD(

116 disrupted mitochondrial structure, decreased mitochondrial respiration, and compromised TCA flux comp

117 llular functions such as cell proliferation, mitochondrial respiration, and epigenetic regulation.

118 phenomena including cell cycle progression, mitochondrial respiration, and learning and memory.

119 h a higher glycolytic rate at the expense of mitochondrial respiration, and led to lactate accumulati

120 olic pathways, including aerobic glycolysis, mitochondrial respiration, and lipid synthesis.

121 aling are associated with ER stress, altered mitochondrial respiration, and mitochondria fragmentatio

122 th carbidopa impairs aspartate biosynthesis, mitochondrial respiration, and reduces exercise performa

123 downregulation of metabolic genes, decreased mitochondrial respiration, and suppression of the serine

124 w-fed mice compromises fatty acid oxidation, mitochondrial respiration, and the abundance of mitochon

125 tary-reducing equivalents by elevated muscle mitochondrial respiration appears to be the mechanism by

126 etabolism, being both a limiting step during mitochondrial respiration as well as a key player in car

127 e effect (the glucose-mediated inhibition of mitochondrial respiration) as most in vitro experiments

128 OS can be generated either as a byproduct of mitochondrial respiration, as a result of the endoplasmi

129 potential, whereas Abeta-CEL16 had increased mitochondrial respiration at complex II.

130 midpoint but suffer from a severe deficit in mitochondrial respiration at the clinical phase of disea

131 t8l-deficient adipocytes exhibited increased mitochondrial respiration, ATP synthesis, and an inducti

132 sal glycolysis, glycolytic capacity, maximal mitochondrial respiration, ATP-linked respiration, and s

133 Because iron deficiency impairs mitochondrial respiration/ATP production, we treated mix

134 Inhibitors of mitochondrial respiration attenuate EMCV replication in

135 TP turnover in cells, NAD(+) regeneration by mitochondrial respiration becomes constrained, promoting

136 We first examined mitochondrial respiration before investigating evidence

137 resuscitation and to compare brain and heart mitochondrial respiration between groups 24 hours after

138 re no significant differences in measures of mitochondrial respiration between legs, but peroxisome p

139 itions were found for maximal ADP-stimulated mitochondrial respiration (both for complex I and comple

140 culum (ER) chaperone that not only regulates mitochondrial respiration but also controls cellular def

141 ressive multiple sclerosis not only impaired mitochondrial respiration but also decreased the bioavai

142 esulting in muscle hypertrophy and increased mitochondrial respiration, but does not improve overall

143 here that HKL administration rapidly reduces mitochondrial respiration by broadly inhibiting ETC comp

144 e 2 and phosphofrucokinase, while decreasing mitochondrial respiration by downregulating respiratory

145 lung interstitial macrophages and increases mitochondrial respiration by glutaminolysis.

146 translocates into mitochondria and inhibits mitochondrial respiration by increasing PTCD1.

147 that elevated non-vacuolar cysteine impairs mitochondrial respiration by limiting intracellular iron

148 MP-treated PBMCs secreted IL-6 that impaired mitochondrial respiration by reducing complex I activity

149 how that the lysosome-like vacuole maintains mitochondrial respiration by spatially compartmentalizin

150 r apoptosis despite a similar attenuation of mitochondrial respiration by the compound.

151 dependent stimulation of glycolysis, but not mitochondrial respiration, by the reproductive neuropept

152 metabolic product in KD, recovers defective mitochondrial respiration bypassing the metabolic failur

153 This was coupled to an enhanced mitochondrial respiration capacity and a preference for

154 chondrial protein-coding mRNAs and decreases mitochondrial respiration capacity.

155 A cycle block reflected by decreased maximal mitochondrial respiration) caused lethal fetal liver hem

156 RBM10 overexpression markedly inhibited mitochondrial respiration, cell migration and proliferat

157 om the pleiotropic effects of CO on cellular mitochondrial respiration, cellular energy utilization,

158 Female newborns showed higher mitochondrial respiration compared to male newborns.

159 FAM173A-deficient cells displayed increased mitochondrial respiration compared with FAM173A-proficie

160 e-aged, obese, insulin-resistant men affects mitochondrial respiration, content and morphology in ske

161 ved anti-helminth drug nitazoxanide (NTZ) on mitochondrial respiration could possess any therapeutic

162 ntricular cardiomyocytes initially activates mitochondrial respiration, coupled with increased mitoch

163 d increased glycolytic activity but impaired mitochondrial respiration, decreased ATP production, and

164 ito-FAP-MG-2I complex led to a rapid loss of mitochondrial respiration, decreased electron transport

165 hibit significantly higher glycolysis, lower mitochondrial respiration, decreased enzymatic activity

166 ith Compound C increased mortality, impaired mitochondrial respiration, decreased OCR, and disrupted

167 We discovered that mitochondrial respiration decreases during starvation in

168 pared to UC separation, to measure decreased mitochondrial respiration, demonstrated in a paradigm of

169 mycin reversed sesamol-induced apoptosis and mitochondrial respiration disorders.

170 They discover that not only is mitochondrial respiration dispensable for embryonic cart

171 ial to various cellular processes, including mitochondrial respiration, DNA repair, and iron homeosta

172 A switch from glycolysis to mitochondrial respiration drives these cells toward diff

173 We determined if improvements in mitochondrial respiration due to exercise training requi

174 iency is associated with inhibited complex I mitochondrial respiration due to lack of NADH for the el

175 Prenatal PM(2.5) exposure influenced mitochondrial respiration during childhood, but this rel

176 Thus, miR-144/451 represses mitochondrial respiration during erythropoiesis by inhib

177 However, the mechanisms that promote mitochondrial respiration during stem cell differentiati

178 Fluoride negatively affected mitochondrial respiration, elicited mitochondrial membra

179 (13)C tracing and mitochondrial respiration experiments map NO-mediated su

180 peroxisomal membrane protein PEX11beta with mitochondrial respiration factors led us to discover an

181 normal fumarate levels but defective maximal mitochondrial respiration) failed to self-renew and disp

182 ysis, macrophages exposed to oxPAPC also use mitochondrial respiration, feed the Krebs cycle with glu

183 tion, unlicensed NK cells depended solely on mitochondrial respiration for cytolytic function, wherea

184 abetes had a significantly decreased rate of mitochondrial respiration fueled by palmitoyl-carnitine

185 er, Mdm30 does not have a dramatic effect on mitochondrial respiration/function, and mRNA export occu

186 trates that the MOC1-dependent modulation of mitochondrial respiration helps control the stromal redo

187 CD31 signals also sustain mitochondrial respiration, however this pathway does not

188 ly, MOF/FAO inhibition acts through reducing mitochondrial respiration (i.e., OXPHOS), which in turn

189 ction, consistent with observed reduction in mitochondrial respiration, impairment in mitochondrial m

190 that genetic or pharmacologic disruption of mitochondrial respiration improves cancer-free survival

191 n Control LCLs and resulted in a increase in mitochondrial respiration in AD-A LCLs.

192 exposure by itself resulted in a decline in mitochondrial respiration in all LCL groups.

193 trate that our method can be used to measure mitochondrial respiration in anatomically defined subfie

194 ated conditional Cox10 mouse mutants lacking mitochondrial respiration in astrocytes, which forces th

195 the oxygen consumption rate as a measure of mitochondrial respiration in B cells from patients with

196 ntal difference in the way idebenone affects mitochondrial respiration in cortical neurons compared w

197 exhibited a reduction in glucose uptake and mitochondrial respiration in darkness.

198 therapy through targeting glutaminolysis and mitochondrial respiration in ESCC with dysregulated Fbxo

199 t it is unknown whether manassantin inhibits mitochondrial respiration in intact mammalian cells and

200 evelopmental Cell, Yao et al. (2019) disrupt mitochondrial respiration in limb skeletal progenitors b

201 nvestigate the effect of Honokiol on cardiac mitochondrial respiration in mice subjected to Dox treat

202 myogenic lineage progression by stimulating mitochondrial respiration in mice.

203 iration and density in mothers, but not with mitochondrial respiration in newborns.

204 osis in patients' lymphocytes, a decrease in mitochondrial respiration in patient fibroblasts with a

205 to investigate the effects of temperature on mitochondrial respiration in permeabilized heart and red

206 eceptor gamma (ERRgamma) negatively controls mitochondrial respiration in prostate cancer cells.

207 We analyzed glycolysis and mitochondrial respiration in resting and activated T cel

208 ory chain proteins may lead to impairment of mitochondrial respiration in the brain.IMPORTANCE Mitoch

209 en electrode, we measured isolated rat liver mitochondrial respiration in the presence and absence of

210 e show that insulin differentially regulates mitochondrial respiration in two human mucosal epithelia

211 rovide genetic evidence for a requirement of mitochondrial respiration in vascular endothelial cells

212 Here, we have studied astrocytes that lack mitochondrial respiration in vitro and in vivo A novel m

213 Mitochondrial respiration increased in aSAT and correlat

214 showed LECs in co-culture exhibited reduced mitochondrial respiration, increased reliance on glycoly

215 h in mice and functions synergistically with mitochondrial respiration inhibitors.

216 Krt6b or Krt16 exhibit elevated ROS, reduced mitochondrial respiration, intracellular distribution di

217 hibition of cellular oxygen usage, for which mitochondrial respiration is an obvious target.

218 We thus propose that partial suppression of mitochondrial respiration is crucial during development

219 Hence, we hypothesized that enhancing mitochondrial respiration is detrimental to the survival

220 Our findings show that mitochondrial respiration is dispensable for survival of

221 Our results demonstrate that mitochondrial respiration is essential for Drosophila IS

222 This indicates that mitochondrial respiration is required for ISG expression

223 Furthermore, mitochondrial respiration is suppressed in WAS patient M

224 of energy, and so iron, a critical player in mitochondrial respiration, is an important component of

225 Here we show that PMI promotes mitochondrial respiration, leading to a superoxide-depen

226 dent mitochondrial proton leak and uncoupled mitochondrial respiration, leading to transient bioenerg

227 Mediation analysis found that mitochondrial respiration linked to energy production ac

228 ons of protein synthesis, energy metabolism, mitochondrial respiration, lipid and carbon metabolism a

229 Organoids were analyzed for mitochondrial respiration, lipid droplet content, and tr

230 These changes are concomitant with impaired mitochondrial respiration, loss of mitochondrial membran

231 e-S protein 1 (NDUFS1), leading to decreased mitochondrial respiration, marked oxidative stress, and

232 of PD, and that strategies aimed at reducing mitochondrial respiration may constitute a surprising ne

233 ateral inhibition of oncogenic signaling and mitochondrial respiration may help enhance the therapeut

234 metabolic profile as displayed by increased mitochondrial respiration, membrane potential, and level

235 a severe complex I assembly defect, impaired mitochondrial respiration, mitochondrial depolarisation,

236 of fission and fusion can cause deficits in mitochondrial respiration, morphology and motility.

237 he restrictive effects of CDK1 inhibition on mitochondrial respiration, NADH turnover, ATP/ADP, and c

238 the metabolic adaption and increased rate in mitochondrial respiration necessary for the differentiat

239 rfused muscle, our data suggest that neither mitochondrial respiration, nor muscle force generation a

240 ng the NAD level.METHODSWe compared the PBMC mitochondrial respiration of 19 hospitalized patients wi

241 ncrease in energy metabolism (glycolysis and mitochondrial respiration) of peripheral blood mononucle

242 Direct inhibition of mitochondrial respiration or ablation of mitochondrial g

243 Increased mitochondrial respiration, or "mitochondrial hyperactivi

244 One mode is to directly inhibit mitochondrial respiration/OXPHOS.

245 lysis provides free fatty acids to support a mitochondrial respiration pathway essential to neutrophi

246 Mitochondrial respiration plays a crucial role in determ

247 rate, hypoxia, and the regulation of cardiac mitochondrial respiration-probing the role of PPARalpha.

248 e, attenuated PEMF-stimulated calcium entry, mitochondrial respiration, proliferation, differentiatio

249 energy phosphates or protein constituents of mitochondrial respiration, promoting the accumulation of

250 Robust mitochondrial respiration provides energy to support phy

251 nd that decreased histone expression induces mitochondrial respiration, raising the question whether

252 NAL displayed a higher baseline mitochondrial respiration rate than SAL.

253 intracellular potassium levels, and reduced mitochondrial respiration rates.

254 ified systematic down-regulation of multiple mitochondrial respiration-related protein and genesets.

255 ffects and mechanisms of Honokiol on cardiac mitochondrial respiration remain unclear.

256 Forcing non-beta cells to derive ATP via mitochondrial respiration (replacing glucose with galact

257 dministration of fatty acids or pyruvate for mitochondrial respiration rescued differentiation in aut

258 h normalized fumarate levels but not maximal mitochondrial respiration) rescued these phenotypes, ind

259 f TAG metabolites for membrane synthesis and mitochondrial respiration, respectively, supported by di

260 matin binding, transcriptional oscillations, mitochondrial respiration rhythms, and late evening acti

261 n of glycolysis (2-deoxy-D-glucose, 2DG) and mitochondrial respiration (rotenone) caused profound act

262 mical inhibition of beta-secretase decreased mitochondrial respiration, suggesting that non-amyloidog

263 ed glucose uptake and lower nutrient-induced mitochondrial respiration than wild-type (WT) cells.

264 The HCL population had lower mitochondrial respiration, than did the control populati

265 d that behavior was indirectly influenced by mitochondrial respiration through neurodevelopment but d

266 submergence, and the lack of oxygen impairs mitochondrial respiration, thus affecting the plant's en

267 RP1) and is important for the maintenance of mitochondrial respiration, thus ultimately protecting th

268 type 2 diabetes medication metformin reduces mitochondrial respiration to control levels and signific

269 y controlling insulin action, lipolysis, and mitochondrial respiration to control the usage of substr

270 nd the mitochondrial Ca(2+) transients boost mitochondrial respiration to restore energy homeostasis.

271 mily domain containing 7C (MFSD7C) uncouples mitochondrial respiration to switch ATP synthesis to the

272 l of NTZ from incubation medium restored the mitochondrial respiration to that of controls.

273 rmogenesis enables cells to match outputs of mitochondrial respiration to their metabolic state and n

274 Hepatic mitochondrial respiration transiently increased and decl

275 epletion reduces mitochondrial integrity and mitochondrial respiration under conditions of cellular s

276 DH/NAD(+), and increased succinate-supported mitochondrial respiration under conditions of low DeltaP

277 M induces Acod1 and itaconate, which reduced mitochondrial respiration via complex II inhibition.

278 ative capacity, but reduced the capacity for mitochondrial respiration via complex II relative to com

279 light, which was accompanied by an increased mitochondrial respiration via the alternative oxidase pa

280 Although light stress stimulated mitochondrial respiration via the energy-conserving cyto

281 Intriguingly, the rate of mitochondrial respiration was 20% to 35% lower in bkdE1a

282 Mitochondrial respiration was acutely decreased in perip

283 Our data show that mitochondrial respiration was decreased in all patient c

284 Muscle biopsies were taken and mitochondrial respiration was evaluated by respirometry.

285 Liver mitochondrial respiration was higher in L rats when usin

286 The Dox-induced impairment of mitochondrial respiration was less pronounced in honokio

287 Thus, synaptic mitochondrial respiration was measured and showed a sign

288 Substrate-induced mitochondrial respiration was reduced in skeletal muscle

289 Unexpectedly, myofibre bundle mitochondrial respiration was reduced, whereas contracti

290 The inhibition of mitochondrial respiration was sufficient to diminish gal

291 Moreover, mitochondrial respiration was up-regulated in myoblasts

292 gher gSAT H(2)O(2) production and lower aSAT mitochondrial respiration were independently associated

293 Secretion of all hormones and mitochondrial respiration were lowered when FFAR1 or fat

294 muscle force, muscle contractile economy and mitochondrial respiration were not affected by acute inf

295 thin minutes, indicating rapid activation of mitochondrial respiration, which coincided with a sharp

296 ce a hypoxic microenvironment known to limit mitochondrial respiration, which is incompatible with lo

297 ing to enhanced sensitivity to inhibition of mitochondrial respiration, which provides a potential th

298 garette smoke also inhibited THP-1 and AMPhi mitochondrial respiration while inducing glycolysis and

299 izing the Seahorse platform showed decreased mitochondrial respiration with age in the hippocampus an

300 ORAI1-deficient LS8 cells showed altered mitochondrial respiration with increased oxygen consumpt