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1 or of maximal oxygen uptake rate than muscle mitochondrial volume.
2 ss, this study examined the effect of DZX on mitochondrial volume.
3 ) and Bmal1(-/-) mice had a 40% reduction in mitochondrial volume.
4 might increase its sensitivity to changes in mitochondrial volume.
5 hondrial proteins and a general reduction of mitochondrial volume.
6 letes have an increased ratio of surface per mitochondrial volume.
7      In addition, the oxidative capacity per mitochondrial volume (0.22 +/- 0.042 vs. 0.32 +/- 0.015
8  cells, the feedback responses that maintain mitochondrial volume across generations of dividing cell
9 chondrial phenotype that includes (1) a high mitochondrial volume and (2) specific mitochondrial arch
10           In 1alpha,25(OH)2D3-treated cells, mitochondrial volume and branching and expression of the
11 otentially due to mdivi-1-induced changes in mitochondrial volume and compression of myofibrils.
12 in, porin, nNOS, p-nNOS, and Tfam as well as mitochondrial volume and cristae abundance were signific
13 muscle mitochondrial respiration relative to mitochondrial volume and cristae density across training
14  individuals may be explained by both higher mitochondrial volume and cristae density in active indiv
15 area density); and (2) no sex differences in mitochondrial volume and cristae density or mass-specifi
16 6-9) and males (n = 15-18) did not differ in mitochondrial volume and cristae density, OXPHOS, or whe
17  time periods presynaptic terminals optimize mitochondrial volume and density to meet power demand.SI
18 ose of this study was to define the roles of mitochondrial volume and distribution in axonal degenera
19 uilibrate just before division, and that the mitochondrial volume and DNA-containing nucleoids instea
20  present a mechanism by which variability in mitochondrial volume and functionality, along with cell
21 e (5HD) at 100 or 300 microM, also increased mitochondrial volume and inhibited respiration.
22  with an osmoregulatory function controlling mitochondrial volume and ion homeostasis.
23  have inhibited or reversed the reduction in mitochondrial volume and numbers caused by PD.
24 ptic terminals as well as a decrease in mean mitochondrial volume and numbers in DA projections.
25        However, the developmental profile of mitochondrial volumes and subsynaptic distribution at th
26                    Subsequently, we measured mitochondrial volumes and subsynaptic distributions at t
27     Based on these findings, we propose that mitochondrial volumes and synaptic localization developm
28 ial morphology and physiology, a decrease in mitochondrial volume, and a severe suppression of cellul
29 ochondrial "membrane mass," has no effect on mitochondrial volume, and does not affect the permeabili
30 d significantly increased calcium, increased mitochondrial volume, and increased numbers of synaptic
31 rophages correlates with necrotic core area, mitochondrial volume, and oxidative damage to DNA.
32 ature and mature labeled calyces reveal that mitochondrial volumes are increased to support high firi
33 d of transported tracer label showed reduced mitochondrial volumes as well as decreased active zone n
34 so being reflected by a relative increase in mitochondrial volume, as shown by quantitative fluoresce
35 that all cells ultimately contained the same mitochondrial volume at cell division.
36 s conveyed by its individual fibers; and (4) mitochondrial volume/axon length rises >/=d(2).
37                            A new study links mitochondrial volume control with growth and cell divisi
38 e demonstrate an increase in skeletal muscle mitochondrial volume density (Mito(VD)) over equivalent
39   Skeletal muscle biopsies were analysed for mitochondrial volume density (Mito(VD)), capillarity, fi
40 ch was the result of a 1.6-fold reduction in mitochondrial volume density and altered substrate oxida
41                                              Mitochondrial volume density and capillary density were
42                                              Mitochondrial volume density and capillary density were
43 nsity, OXPHOS, or when normalising OXPHOS to mitochondrial volume density and muscle cristae surface
44 cle biopsy sample permitted determination of mitochondrial volume density and the contribution of the
45 een active groups and UT when normalising to mitochondrial volume density and were lost when normalis
46 e to extreme high altitude (>5500 m), muscle mitochondrial volume density falls, with a particular lo
47    The XC and RUN groups demonstrated higher mitochondrial volume density than the RA and UT groups w
48 c analyses demonstrated that cardiac myocyte mitochondrial volume density was increased in insulin-re
49                                              Mitochondrial volume density was significantly lower in
50 iber cross-sectional areas, capillarity, and mitochondrial volume density were not different between
51 XPHOS) can change independently of shifts in mitochondrial volume density, which may be attributed to
52                            DZX did not alter mitochondrial volume during CPG; however, it was associa
53                   5-Hydroxydecanoate reduced mitochondrial volume during exposure to both stresses wi
54 wever, it was associated with an increase in mitochondrial volume during MI.
55 itochondrial CO therapy, and connect cardiac mitochondrial volume expansion with the inducible networ
56  localization to mitochondria by controlling mitochondrial volume fraction (influencing diffusive sea
57                If, as found for optic nerve, mitochondrial volume fraction is constant with axon diam
58 stsynaptic density length and curvature, and mitochondrial volume fraction were similar for axosomati
59 lity of the synaptosomal preparations or the mitochondrial volume fraction.
60  yeast cells and observed that cell size and mitochondrial volume grew exponentially during the cell
61 f anions and is postulated to be involved in mitochondrial volume homeostasis in conjunction with the
62 el (IMAC) that is believed to be involved in mitochondrial volume homeostasis.
63 dictating cellular growth rates and ensuring mitochondrial volume homeostasis.
64       Early cure of infection also increased mitochondrial volume in Tmem compared with Teff, support
65                                              Mitochondrial volume in vastus lateralis biopsies increa
66 lated the mitochondrial respiration rate and mitochondrial volume in winter.
67                      We show that the axonal mitochondrial volume increase following acute demyelinat
68                            Both cellular and mitochondrial volume increased during exposure to MI and
69                                              Mitochondrial volume increased notably in the metamorpho
70 ors are suppressed by control at two levels: Mitochondrial volume is actively distributed throughout
71 r cell activity is not required for the high mitochondrial volume, it shapes the mitochondrial archit
72 gene expression rather than gross changes in mitochondrial volume or mitochondrial turnover.
73           We examined ontogenetic changes in mitochondrial volume, oxidative capacity, oxygen consump
74  a constant fraction of axonal volume--thus, mitochondrial volumes rise as the diameter squared.
75 because of a reduction in the total cellular mitochondrial volume, suggesting that intermitochondrial
76 als had higher citrate synthase, a proxy for mitochondrial volume, than Nuttall's individuals.
77 ddition of DZX did not alter the response of mitochondrial volume to CPG (P=0.912) but increased swel
78  mPT onset was assessed by measuring in situ mitochondrial volume using the 'thinness ratio' and the
79  of synaptic boutons (SBs), e.g., bouton and mitochondrial volume, vesicle pool size, as well as post
80 strate that Letm1 is involved in maintaining mitochondrial volume via potassium/proton exchange acros
81                                              Mitochondrial volume was a strong predictor of bouton si
82                                              Mitochondrial volume was measured.