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1 lagen-activated NPY-rich platelets were more mitogenic (~2-fold vs. ~1.6-fold increase) for human mic
2 rb14 as a physiological repressor of insulin mitogenic action in the liver and further supports that
3 retinoic acid, correlated with sustained pro-mitogenic action of PACAP beyond the developmental switc
4  distinct pathways that produce pro- or anti-mitogenic actions.
5 ch mimicked the glucoregulatory, but not the mitogenic, actions of insulin.
6 tenin signaling and to cooperate with MYC in mitogenic activation and liver transformation.
7 or tyrosine kinases (RTKs) and/or downstream mitogenic activation is almost ubiquitous; thus tailored
8 unction and they produce IFN-gamma following mitogenic activation.
9 activity of FGF1 can be dissociated from its mitogenic activity and is mediated predominantly via FGF
10 secretory activity in the lacrimal gland and mitogenic activity at the corneal epithelium.
11  the increase in tumor engraftment and serum mitogenic activity in mice pretreated with a chemotherap
12 val of mice after partial hepatectomy, FGF19 mitogenic activity is associated with liver carcinoma.
13 and protein assays, we further show that the mitogenic activity of TWEAK on primary astrocytes requir
14                                 However, its mitogenic activity was delayed compared with that of EGF
15 endent of its protease activity, FXII exerts mitogenic activity with implications for angiogenesis.
16 stigating factors that limit YAP's postnatal mitogenic activity, we found that the CM-enriched TEAD1
17          However, the molecular basis of its mitogenic activity, which is currently unknown, must be
18 s with the capacity to withstand an aberrant mitogenic activity, with a profound impact on the geneti
19 we demonstrate that-in contrast to its known mitogenic activity-Rspo3 induces differentiation of basa
20 ination of IRS-2 enhances IGF signalling and mitogenic activity.
21 m of microorganisms and also had the highest mitogenic activity.
22 ve-like signaling properties with negligible mitogenic activity.
23 o-terminal microheterogeneity does not alter mitogenic activity.
24 proliferation, corresponding to their higher mitogenic activity.
25 ncers in which steroid hormones are powerful mitogenic agents.
26             We further demonstrated that the mitogenic and adipogenic effect of ghrelin were mainly d
27 receptor substrates 1 and 2 (IRS1/2) mediate mitogenic and antiapoptotic signaling from insulin-like
28 y as a C-terminal fragment homologous to the mitogenic and bactericidal region in human lacritin, sug
29 demonstrate that tropoelastin elicits strong mitogenic and cell-attractive responses, both as an immo
30 syndecan-1 binding is necessary for lacritin mitogenic and cytoprotective activities, TGM2 cross-link
31 sic 1-10 nM dose optimum, the same domain is mitogenic and cytoprotective for epithelia via a syndeca
32 ERalpha-mediated gene expression involved in mitogenic and developmental processes in MCF7 breast can
33 scaffold proteins that interact with various mitogenic and developmental signals, the genetic role of
34 3, dedicated hubs integrate the antagonistic mitogenic and DNA damage signals, regulating the stoichi
35                         We show that Gas6 is mitogenic and increases schwannoma cell-matrix adhesion
36 meostasis and critical to the integration of mitogenic and metabolic signaling pathways.
37  hedgehog (HH) pathway is well known for its mitogenic and morphogenic functions during development,
38  propose that the potent, growth factor-like mitogenic and motogenic abilities of tropoelastin are bi
39             The surprising magnitude and the mitogenic and mutagenic nature of the effect exerted by
40 eta-catenin pathways effectively blocked the mitogenic and neurogenic effects of ETH.
41 growth, which were mediated by activation of mitogenic and oncogenic molecules.
42 ayers and growth factors alone, we sustained mitogenic and osteogenic signals with these growth facto
43 oxygenase-2 (COX-2) expression is induced by mitogenic and proinflammatory factors.
44                        The interplay between mitogenic and proinflammatory signaling pathways plays k
45                            Activation of the mitogenic and prosurvival transcription factor, STAT5b,
46                                          The mitogenic and second-messenger signals that promote cell
47   We assessed functional T cell responses to mitogenic and viral antigenic stimulation by the express
48 s studies, we find that E-peptide signaling, mitogenic, and motogenic effects are dependent upon IGF-
49 renal ischemia-reperfusion injury, mainly by mitogenic, angiogenic, and anti-inflammatory mechanisms.
50 ture of murine CD3epsilon complexed with the mitogenic anti-CD3epsilon antibody 2C11 enabled the firs
51  role in this relationship, due to insulin's mitogenic/antiapoptotic activity.
52                                              Mitogenic assays in cultured cells showed an age-depende
53 ased upregulation of the CSF1R-dependent pro-mitogenic cascade, correlating with disease severity.
54 hibited C-RAF BxB-mediated activation of the mitogenic cascade.
55 rs (GEFs) of the Dbl family are activated by mitogenic cell surface receptors and activate the Rho fa
56  whose activity results in proliferative and mitogenic changes in the cell.
57 e function as a new class of human beta cell mitogenic compounds.
58 these data suggest mTOR signaling-dependent, mitogenic conditioning of AECII is a determinant of host
59 l state identity better than culture in high mitogenic conditions.
60 venting cell proliferation under unfavorable mitogenic conditions.
61 rexpression of Dsg2 increased EV release and mitogenic content including epidermal growth factor rece
62 t-ratio silica microrods loaded with soluble mitogenic cues and coated with liposomes of defined comp
63 ed via the fluid, synthetic membranes, while mitogenic cues are released slowly from the microrods.
64                 In confluent monolayers, low mitogenic culture conditions preserved corneal endotheli
65  assumption that metastatic cells are in the mitogenic cycle.
66 rowth factors (FGF) act as proangiogenic and mitogenic cytokines in multiple myeloma.
67                                         This mitogenic effect can be neutralized by RNA interference,
68                                    While the mitogenic effect of (E)3-(3-phenylbenzo[c]isoxazol-5-yl)
69  between in vivo and in vitro studies on the mitogenic effect of estrogen and raises questions regard
70                             We show that the mitogenic effect of Ex-4 requires calcineurin/nuclear fa
71 endent of keratinocyte proliferation and the mitogenic effect of FGF signalling, which are only requi
72 c cell types offers new understanding of the mitogenic effect of IL-17 on tissue repair and cancer.
73 f toll-like receptor 4, was essential to the mitogenic effect of NE on HMECs.
74 which abrogates STAT3 signaling, reduces the mitogenic effect of supernatants in CRC cells.
75 the WNT signaling pathway is known to have a mitogenic effect on cells, but relatively little is know
76 l properties against 11 human pathogens, and mitogenic effect on hamster spleen lymphocytes were also
77                       Active neurons exert a mitogenic effect on normal neural precursor and oligoden
78  tumors and cell lines, leading to a similar mitogenic effect through activation of the MAP kinase pa
79                                         This mitogenic effect was abrogated by pharmacological inhibi
80 y smooth muscle cell proliferation, and this mitogenic effect was greatly pronounced in PAH-pulmonary
81 ion of KLF4, which mediates estrogen-induced mitogenic effect.
82       Both forms of IGFBP-3 are also without mitogenic effects alone or in combination with IGFs.
83 ese findings provide evidence that insulin's mitogenic effects are mediated by a subpopulation of IRs
84                     Interestingly, BRAFV600E mitogenic effects in astrocytes were restricted, in part
85 three members of the MAPK family mediate the mitogenic effects of catecholoestradiols in the endothel
86                   Moreover, we show that the mitogenic effects of estradiol require the presence of i
87 age dependent and critical for promoting the mitogenic effects of estradiol via ERalpha.
88 e endometrial epithelial cells to direct the mitogenic effects of estradiol.
89 etic progestins were found to antagonize the mitogenic effects of estrogens.
90 n of direct from indirect and motogenic from mitogenic effects of genes and molecules affecting wound
91 ration, and clinical trials that exploit the mitogenic effects of IL-7 have achieved success in treat
92  only IRA transmits the growth-promoting and mitogenic effects of insulin-like growth factor 2.
93  that elafin is a counterbalance against the mitogenic effects of NE in G0 HMECs.
94                    We tested mAbs neutralize mitogenic effects of SEB in vitro and in vivo with T-cel
95                                But while the mitogenic effects of Shh signaling have been confirmed i
96 clear why some tissues are refractory to the mitogenic effects of the oncogene Myc.
97          PRD1-BF1 does not contribute to the mitogenic effects of VEGF, but directly represses genes
98  analogs like alfacalcidol had rapid, potent mitogenic effects on embryonic and adult cardiomyocytes
99 se BPA exerted c-Myc-dependent genotoxic and mitogenic effects on ERalpha-negative mammary cells.
100 producing IL-22 and IL-17, which have direct mitogenic effects on hepatocytes and promote a regenerat
101 Neuregulin1 (Nrg1), previously shown to have mitogenic effects on mammalian cardiomyocytes, is sharpl
102                                     PcRH had mitogenic effects on rat splenic lymphocytes.
103 iferation, we investigated whether insulin's mitogenic effects result from activation of Ca(2+)-signa
104 olypeptide hormones with potent anabolic and mitogenic effects that regulate cell growth and differen
105 ough E-cadherin, which prevents secretion of mitogenic epidermal growth factors (EGFs) by repressing
106 (q/11) proteins and preferentially inhibited mitogenic ERK signaling rather than canonical phospholip
107       The present study examined the initial mitogenic events marking the regenerative response of th
108 s strains have genes encoding IgA proteases, mitogenic factor deoxyribonucleases, nickel/cobalt uptak
109 wth factor (EGF) is a potent chemotactic and mitogenic factor for epidermal keratinocytes, and these
110       This study demonstrates that GDNF is a mitogenic factor promoting self-renewal that is conserve
111                  Although FGF1 is known as a mitogenic factor, FGF1 knockout mice develop insulin res
112 moattractant protein-1 and hypoxia-inducible mitogenic factor, in the murine model of hypoxic pulmona
113 ar, but involve secretion of an unidentified mitogenic factor.
114 ascades lead to the downstream production of mitogenic factors and the proliferation of neighboring s
115 ated fates and rendering them insensitive to mitogenic factors such as Notch.
116       In parallel, the levels of several ASM mitogenic factors, including the PAR-2 ligands, mast cel
117 f fenestrations and expression of growth and mitogenic factors, leading to proliferative changes and
118 t and regeneration hinges on the activity of mitogenic factors.
119 s and skeletal muscle satellite cells and is mitogenic for both cell types under normal conditions.
120           Our findings identify vitamin D as mitogenic for cardiomyocytes and other cell types in zeb
121         We conclude that although insulin is mitogenic for intestinal tumour cells in vitro, impaired
122 asal and activity-dependent that produce the mitogenic form of neuroligin-3.
123 indicate that NANOG has a lineage-restricted mitogenic function in stratified epithelia.
124 wnregulates CCN1 and alters cytoskeletal and mitogenic gene expression.
125            Quiescent beta-cells exposed to a mitogenic glucose stimulation require 8 h to enter the G
126   Contact inhibition is often antagonized by mitogenic growth factor signaling.
127 sm, inhibition of Hippo pathway signaling by mitogenic growth factors.
128 e expression program, and estrogen-dependent mitogenic growth.
129 lation of which is stimulated in response to mitogenic, growth, and nutritional stimuli, and proteins
130 insulin-sensitizing action for exogenous non-mitogenic human FGF1 with therapeutic potential for the
131 subthreshold TCR signaling in the context of mitogenic IL-2 signals, thereby rendering CD8 T cells ex
132 lls, suggesting that integration of multiple mitogenic inputs may alter the minimal requirement for T
133  mechanism responsible for the generation of mitogenic IR-A and provides a novel interplay between IR
134 ntrol of ENaC activity and the activation of mitogenic kinase pathways; (b) provide evidence for a Cy
135 s a proximal target for EET-activated ERK1/2 mitogenic kinases, (c) characterize a mechanistic common
136 ed by activation of Sestrin2, which impaired mitogenic mammalian target of rapamycin (mTOR) signaling
137 FR through early endosomes, thereby limiting mitogenic MAPK signals.
138 tor through early endosomes are accelerated, mitogenic MAPK-ERK1/2 signals are rapidly terminated, an
139 motes maturation of endosomes and shuts down mitogenic MAPK-ERK1/2 signals from endosomes.
140 ur data demonstrate that IL-1 functions as a mitogenic mediator of encephalitogenic Th17 cells rather
141 LOX12) expression and elevated levels of its mitogenic metabolite, 12-(S)-hydroxy-5,8,10,14-eicosatet
142                 Moreover, the IGF-1-mediated mitogenic microglia response was reduced by N-glycosylat
143 tant roles of the TNF-alpha ligand Eiger and mitogenic molecules in mediating these interactions duri
144 atocyte growth factor, Met signaling confers mitogenic, morphogenic, and motogenic activity to variou
145 ges in expression of the proinflammatory and mitogenic neurokinin-1 receptor (NK-1R).
146 ) SCLC cells, an increased expression of the mitogenic neuropeptide receptors for gastrin-releasing p
147 nt kinase (CDK) complexes to be activated by mitogenic/oncogenic pathways.
148  of a bivalent signaling node that is either mitogenic or proapoptotic.
149 iferation through a novel extracellular cAMP mitogenic pathway and further support MRP4 inhibition as
150 axon guidance; 2) some very highly expressed mitogenic pathway genes (e.g., Map2k1, Igf1r, Rara, Runx
151 ctor Receptor (PDGFR) signaling is a central mitogenic pathway in development, as well as tissue repa
152                       Loss of these critical mitogenic pathways induces cell cycle arrest and cell de
153 by converting a transcriptional repressor of mitogenic pathways into a transcriptional activator.
154 tion 5a (Stat5a), factors that influence key mitogenic pathways that regulate normal mammary gland de
155 n intestinal epithelial cells and stimulates mitogenic pathways upon activation.
156 rs, cytokines, angiogenic factors, and other mitogenic pathways.
157 of adrenomedullin (Adm, gene; AM, protein)-a mitogenic peptide hormone required for normal cardiovasc
158  for producing active endothelin-1 (ET-1), a mitogenic peptide implicated in the aetiology of a numbe
159            In contrast, DLX3 loss promotes a mitogenic phenotype associated with constitutive activat
160  cardiomyocyte (CM) proliferation, but YAP's mitogenic potency declines postnatally.
161          Herein, we further characterize the mitogenic potential of TWEAK on central nervous system c
162 tion of furin reduces IRA maturation and its mitogenic potential without altering the insulin effects
163  exhibiting autocrine/paracrine activity and mitogenic potential.
164 al mediator of FGF19 action on metabolic and mitogenic programs; thus, the involvement of mTORC1 in F
165       However, the detailed mechanism behind mitogenic properties of PMT is unknown.
166 hat controls endocytosis, down-regulation of mitogenic receptors and cell migration.
167 reased hepatocyte proliferation; however, no mitogenic response in hepatocytes to T3 was evident in t
168 r-beta-arrestin complex stabilization in the mitogenic response of AT1R.
169 demonstrate that neuronal activity elicits a mitogenic response of neural progenitor cells and OPCs,
170 ily of Ca(2+)-regulated PDEs, also induced a mitogenic response to AVP in NHK cells, similar to the e
171     We have identified T3-induced hepatocyte mitogenic response to be mediated by PKA-dependent beta-
172 V3 by RNA interference markedly impaired the mitogenic response to CD3/CD28 stimulation.
173 y reflect the requirement for an exceptional mitogenic response to growth factor signalling in the af
174 re rapid VEGFR2 clustering and a more potent mitogenic response triggered by VEGF-A in respect to gre
175 ation of PDGF-beta receptor, and an enhanced mitogenic response, after PDGF-BB stimulation.
176 teractions of RNase L and TTP to attenuate a mitogenic response.
177 strating its functional role in limiting the mitogenic response.
178 mary cilia shortening and attenuation of the mitogenic response.
179  weakly acting phytoestrogen that mimics the mitogenic responses produced by E2 in an ERalpha-depende
180  cholinergic neurons, revealing an important mitogenic role for the planarian hh signaling molecule i
181 rn of events given the pathway's established mitogenic role, and they show that Hedgehog pathway atte
182  prominent prosecretory, cytoprotective, and mitogenic role.
183 inases and transcription factors with myriad mitogenic roles in diverse cell types.
184 phingolipids ceramide and sphingosine to the mitogenic S1P, thereby determining the susceptibility of
185 port that the ER-transiting and functionally mitogenic secreted proenzyme (pCatD) form of cathepsin D
186  Previously, we found that PACAP was an anti-mitogenic signal from embryonic day 13.5 (E13.5) onward
187 ator, mediates this differential response to mitogenic signal gradients.
188                        Although insulin is a mitogenic signal in proliferative cells, whether compone
189                        However, although the mitogenic signal resulted in a more sustained mRNA respo
190 kinase impaired ERBB3 (HER3) create a potent mitogenic signal, but the phosphorylation of ERBB2 in th
191 gulated and extended the transduction of the mitogenic signal, whereas silencing of LNK produced the
192  haplotype, thus leading to a higher ERalpha mitogenic signal.
193 mplying there are mechanisms that resist the mitogenic signal.
194 broblasts function cooperatively to activate mitogenic signaling activities and to induce their trans
195 y reported that, in cancer cells, heightened mitogenic signaling allows TGF-beta-activated Smad3 to i
196 e-independent timer to limit the response to mitogenic signaling and aberrant cycling in terminally d
197 , we propose that cilia decapitation induces mitogenic signaling and constitutes a molecular link bet
198 Cav-1 may provide a mechanism for regulating mitogenic signaling and modulating the cell-surface pres
199  feedback potently suppress ligand-dependent mitogenic signaling and Ras function.
200                       These SMAPs attenuated mitogenic signaling and triggered apoptosis in KRAS-muta
201 how IL-17-mediated inflammatory response and mitogenic signaling are exploited to equip its cancer-pr
202 (SHH) signaling, but downstream effectors of mitogenic signaling are still being elucidated.
203 cell proliferation by induction of paracrine mitogenic signaling between heterogeneous malignant cell
204 ng membrane potential to control the gain in mitogenic signaling circuits.
205 /PDGF/FGF receptors is sufficient to inhibit mitogenic signaling in EC but not in fibroblasts.
206 nockdown or small-molecule targeting reduced mitogenic signaling in non-small cell lung cancer cell l
207 oviding novel insight into how cells inhibit mitogenic signaling in response to cell contact.
208 y in the G(1) cell cycle phase, we find that mitogenic signaling is temporally integrated throughout
209 ation, but how membrane potential influences mitogenic signaling is uncertain.
210 expression activates PI3K, a key node in the mitogenic signaling network known to promote malignancie
211 hinery is a major recipient of the principal mitogenic signaling networks involving Raf-ERK1/2 and ph
212 ction as a tumor suppressor and regulator of mitogenic signaling networks such as the Ras/rac, Akt, a
213 ry of knowledge for beta-cell researchers on mitogenic signaling pathways and to emphasize how little
214  findings show that an important activity of mitogenic signaling pathways is to inactivate the growth
215 increased hHSC proliferation through several mitogenic signaling pathways such as EGFR, PI3K and p38.
216 contain known binding sites for effectors of mitogenic signaling pathways such as Yorkie and Notch.
217 nduces hHSC fibrogenic activity via multiple mitogenic signaling pathways, and is upregulated in muri
218                           Moreover, multiple mitogenic signaling pathways, including ERK MAPK, Wnt an
219 as a signaling hub that negatively regulates mitogenic signaling pathways, like the ERK1/2 MAP kinase
220  cell proliferation, tissue growth, and many mitogenic signaling pathways; we investigated its role i
221  Taken together, these results indicate that mitogenic signaling regulates the miRNA effector machine
222 ogenitors that are driven to proliferate via mitogenic signaling that affects translation.
223 cellular desmosomal adhesion, Dsg2 regulates mitogenic signaling that may promote cancer development
224                     These mutations increase mitogenic signaling through the RhoA axis and, therefore
225 ling, contraction, intracellular Ca(2+), and mitogenic signaling were determined in vivo and in vitro
226 uce the maturation of IRA and its associated mitogenic signaling without altering the signals emanati
227         Reduced tumor growth, proliferation, mitogenic signaling, and apoptosis induction were observ
228 pletion reduces smooth muscle proliferation, mitogenic signaling, and extracellular matrix deposition
229 nic demonstrated RTK blockade, inhibition of mitogenic signaling, and proapoptotic signal induction i
230 in receptor, allowing aberrant activation of mitogenic signaling, promotes aberrant splicing and expr
231 ermal growth factor receptor (EGFR)-mediated mitogenic signaling, promoting tumor cell survival throu
232 hophysiologies associated with dysfunctional mitogenic signaling.
233 C complexes that lack GW182 are regulated by mitogenic signaling.
234 ctors induces apoptosis passively by lack of mitogenic signaling.
235 lls are more responsive to Pten null-induced mitogenic signaling.
236 tivates the GRM1 receptor and its downstream mitogenic signaling.
237 d in liver regeneration and their downstream mitogenic signaling.
238 iency as a potential inhibitor of overactive mitogenic signaling.
239           Well-characterized germinal zones, mitogenic signals and cell types make the cerebellum an
240 late cell proliferation, but how fluctuating mitogenic signals are converted into proliferation-quies
241 hai or Galphas, Akt signaling is suppressed, mitogenic signals are enhanced due to delayed transit ti
242 and implicate tumor-associated DCs and their mitogenic signals as auspicious therapeutic targets.
243 77-eGFP, c-Myc protein expression integrates mitogenic signals downstream of both IL-2 and the TCR, y
244 pathway plays a critical role in transducing mitogenic signals from receptor tyrosine kinases.
245 vels allows effective integration of diverse mitogenic signals in ISCs to adapt their proliferative a
246 ral progenitor groups to broadly distributed mitogenic signals in the embryonic environment.
247 alpha(q) and its coupled receptors transduce mitogenic signals is still unclear because of the comple
248               The ultimate receptor of these mitogenic signals is the retinoblastoma (Rb) family of p
249                                          The mitogenic signals of IGF-1 are predominantly transduced
250  the connection between oxidative stress and mitogenic signals remains obscure.
251 to permeate neurofibroma tissue, mediate key mitogenic signals that contribute to vascular ingrowth,
252 r cells, as well as to normal cells, sending mitogenic signals that greatly enhance tumor growth.
253                                              Mitogenic signals that regulate cell division often proc
254 tal for oncogenic signaling as it integrates mitogenic signals to amplify production of pro-growth an
255 mature myometrial or leiomyoma cells to send mitogenic signals to neighboring tissue stem cells in re
256 clin D-dependent kinases sense a plethora of mitogenic signals to orchestrate specific transcriptiona
257                  MEKK2 integrates stress and mitogenic signals to the activation of NF-kappaB, JNK1/2
258 cking as to how developing neurons integrate mitogenic signals with microenvironment cues to control
259 permanent G0 in which they are refractory to mitogenic signals.
260   We show that Activin directly inhibits the mitogenic sonic hedgehog pathway in a Gli3-dependent man
261 by regulating splicing to suppress fibrosis, mitogenic splicing, and EMT.
262 rs can contribute to the proinflammatory and mitogenic status of resident mural cells.
263 ptor using short-hairpin RNA abolished PACAP mitogenic stimulation at E10.5.
264                        Furthermore, in vitro mitogenic stimulation demonstrated that defective blasti
265                                  Conversely, mitogenic stimulation of Ewing sarcoma cells with IGF1 i
266                                     In vitro mitogenic stimulation of PBMCs resulted in upregulation
267 -associated HIV-1 RNA was detected following mitogenic stimulation of peripheral blood CD4(+) T cells
268                                       During mitogenic stimulation of primary lymphocytes, MYC promot
269 an invariant minimal threshold of cumulative mitogenic stimulation required for cell division.
270 e protein complexes that can be activated by mitogenic stimulation to repress mitogen-stimulated targ
271                                              Mitogenic stimulation triggers the binding of Tiam1 to t
272                                We found that mitogenic stimulation with keratinocyte growth factor (K
273 ents were isolated and subjected to in vitro mitogenic stimulation with PMA and ionomycin.
274                               The effects of mitogenic stimulation with teduglutide in patients with
275         These cells respond to antigenic and mitogenic stimulation, but are distinct from IL-9(+) Th2
276 and temporarily dissociated from mTORC1 upon mitogenic stimulation, suggesting a mechanism underlying
277 vels that permit rapid cell cycle entry upon mitogenic stimulation.
278 ted EAE monkeys were also less responsive to mitogenic stimulation.
279 sence of exogenous stimuli and in the T cell mitogenic stimulation.
280  recruited into RISC complexes subsequent to mitogenic stimulation.
281 als with the mTOR inhibitor rapamycin before mitogenic stimulation.
282 ls were capable of producing cytokines after mitogenic stimulation.
283 is required for cell division in response to mitogenic stimulation.
284 IL-2, IFN-gamma, and TNF-alpha) responses to mitogenic stimulations prior to infection.
285 s protein degradation that is antagonized by mitogenic stimulations.
286 f cord blood mononuclear cells to innate and mitogenic stimuli (P = .0009), with an average 1.7- to 2
287 lar B lymphocytes were expanded by providing mitogenic stimuli and then challenged with KSHV-specific
288 lucose oxidation and an enhanced response to mitogenic stimuli in comparison to hAFSCs.
289 itutes a central node in the transmission of mitogenic stimuli to the cell cycle machinery.
290  proliferation depends on the integration of mitogenic stimuli with environmental conditions.
291 hese conditions, hPSC-CMs were refractory to mitogenic stimuli, and we found that key proliferation p
292 tion of immediate early genes in response to mitogenic stimuli.
293 he mTOR-S6K signaling pathway in response to mitogenic stimuli.
294 ulation thereby evoking sustained and robust mitogenic stimuli.
295 irus 8 (HHV-8) is believed to contribute via mitogenic, survival, and angiogenic activities to HHV-8-
296 nd cytokine secretion after stimulation with mitogenic, TLR, and T-cell stimuli by cytometric bead ar
297 ing growth factor-beta1 (TGF-beta1), LRG1 is mitogenic to endothelial cells and promotes angiogenesis
298 owing that old beta cells can respond to the mitogenic trigger of enhanced glycolysis.
299 regenerative potential and cannot respond to mitogenic triggers.
300 the forebrain, gut and breast by suppressing mitogenic WNT signaling in mouse.

 
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