コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 nhanced stabilization of NANOG protein after mitosis.
2 o functional nuclear pores toward the end of mitosis.
3 EBV episomes to the host chromosomes during mitosis.
4 lishing long-range chromatin interactions in mitosis.
5 loped a computational model of fission-yeast mitosis.
6 th MRE11A inhibitor PFM39 modestly prolonged mitosis.
7 centromere organization and function during mitosis.
8 e promoting complex/cyclosome (APC/C) during mitosis.
9 l for faithful chromosome segregation during mitosis.
10 ical consequences of failed DSB responses in mitosis.
11 nucleus migrates apically before undergoing mitosis.
12 K1delta and CK1epsilon during interphase and mitosis.
13 le, enabling sister chromatid segregation in mitosis.
14 mmetric distribution of gamma-tubulin during mitosis.
15 lular mechanisms adopted by single cells for mitosis.
16 er complex (CPC) is a conserved regulator of mitosis.
17 sis partitions the cell contents to complete mitosis.
18 uclei, during telophase of a DNA elimination mitosis.
19 interphase but stiffen more upon entry into mitosis.
20 pha-tubulin interacts with galectin-8 during mitosis.
21 ed to the centrosomes and chromosomes during mitosis.
22 kinetochores and spindle microtubules during mitosis.
23 ionally active RNA polymerase II (Pol II) in mitosis.
24 ic changes due to cortical stiffening around mitosis.
25 lls that harbor only one SPB delay exit from mitosis.
26 s, switching cell fate from apoptosis toward mitosis.
27 ly after nuclear envelope reformation during mitosis.
28 symmetric segregation of extra centrioles in mitosis.
29 rical and mechanically stiffen as they enter mitosis.
30 urora B kinase (AURKB) and Survivin in early mitosis.
31 is either repressed or highly active during mitosis.
32 division, despite rounding up as they enter mitosis.
33 nuclear position into a signal to exit from mitosis.
34 the upstream centrosome defects or extended mitosis.
35 chment point for spindle microtubules during mitosis.
36 parental genomes into a single nucleus after mitosis.
37 ilized as kinetochores come under tension in mitosis.
38 showed chromosome segregation defects during mitosis.
39 MEN signaling, enabling the timely exit from mitosis.
40 ation of chromosomes with the INM throughout mitosis.
41 Aurora B kinase plays essential roles in mitosis.
42 pronounced spindle abnormalities and delayed mitosis.
43 mentalization of eukaryotic cells after open mitosis.
44 ism of licensing of chromosome compaction in mitosis.
45 S-phase entry and promotes progression into mitosis.
46 that MRN may also impact on the duration of mitosis.
47 lation necessary to ensure proper entry into mitosis.
48 n that were generationally conserved through mitosis.
49 ortant role in promoting efficient exit from mitosis.
50 icus, which breaks and reforms the NE during mitosis.
51 P/p25 alters microtubule dynamics and delays mitosis.
52 to complete the replication of CFSs in early mitosis.
53 s then sharply downregulated at the onset of mitosis.
54 ow acute depletion of these proteins affects mitosis.
55 inheritance during Saccharomyces cerevisiae mitosis.
56 lin B:CDK1 is the master kinase regulator of mitosis.
57 in cells, especially before their entry into mitosis.
58 , modulation of actomyosin contractility, or mitosis.
59 mitted from mother to daughter cells through mitosis.
60 this endocytic interaction mechanism during mitosis.
61 esin that peaks in protein expression during mitosis.
62 o ensure repression of macroautophagy during mitosis.
63 NA repair, and chromosome segregation during mitosis.
64 future study of autophagy and mTORC1 during mitosis.
65 at phosphorylates histone H3 at Thr-3 during mitosis.
66 ric chromatin to spindle microtubules during mitosis.
67 some segregation defects in cells undergoing mitosis.
68 atid compaction and their segregation during mitosis.
69 iquitination of cell cycle regulators during mitosis.
70 wing this delay, cells entered and completed mitosis.
71 dentity is determined shortly after terminal mitosis.
72 esin from chromatin as cells transit through mitosis.
73 e necessary for catenation resolution during mitosis.
74 and function of the inner centromere during mitosis.
75 vere locus instability and missegregation in mitosis.
76 ion at S227 by PKCepsilon also occurs during mitosis.
77 nterference between apical contractility and mitosis.
78 incomplete dissociation of DAs and cilia in mitosis.
79 during interphase, ahead of Rcd4 and before mitosis.
80 aryotic chromosomes in interphase and during mitosis.
81 to specific transcription start sites during mitosis.
82 rominent role in spindle organization during mitosis.
83 apture and segregation of chromosomes during mitosis.
84 and impairs chromosome biorientation during mitosis.
85 ial for proper chromosome segregation during mitosis.
86 ntil the breaks are repaired before entering mitosis.
87 branched species at the very late stages of mitosis.
88 is critical for EWSR1/FLI1 interference with mitosis.
89 n important kinase that regulates DAs before mitosis.
90 tial for chromosome bridge resolution during mitosis.
91 enance of adhesions and retraction fibers in mitosis [1-6], which are thought to act as local cues th
93 y mirrors nuclear envelope breakdown in open mitosis(3), revealing an unexpectedly high conservation
94 aromyces pombe (a classical model for closed mitosis(5)), genetics, live-cell imaging and electron to
96 molecular brushes during the early stages of mitosis(8), but during mitotic exit the brushes collapse
98 te within a commonly phosphorylated motif in mitosis (a non-canonical target of Cyclin B-Cdk1), and o
99 e/threonine-protein kinase-like (MASTL) is a mitosis-accelerating kinase with emerging roles in cance
100 to promote attachment turnover, and in later mitosis, activity decreases to ensure attachment stabili
101 chromatin and recruits membrane proteins in mitosis, also facilitates nuclear membrane repair in int
105 es pombe AMR proteins at different stages of mitosis and cytokinesis using an approach we have develo
106 Plk1 levels resulted in multiple defects in mitosis and cytokinesis, supernumerary centrosomes, and
107 e stability relies on proper coordination of mitosis and cytokinesis, where dynamic microtubules capt
110 he inner kinetochore protein CENP-C in early mitosis and does not require either the Bub1/pH2A/Sgo1 o
112 spatiotemporal cues about the progression of mitosis and ensures that release of Cdc14 phosphatase oc
113 l, we blocked cells from progressing through mitosis and found that prolonged metaphase delay is suff
114 oordinating gamma-tubulin recruitment during mitosis and illuminates the mechanism by which signaling
116 of Bim1-binding by Cik1-Kar3 delays cells in mitosis and impairs microtubule bundle organization and
119 decreases by approximately 50% during early mitosis and increases back to G2 levels during cytokines
121 actin network organization and mechanics in mitosis and is required for successful cell division in
122 s promote chromosome segregation during both mitosis and meiosis and regulate chromosomal and genomic
123 ctive segregation patterns of chromosomes in mitosis and meiosis are dictated in part by the kinetoch
125 e kinases (PLKs) have numerous roles in both mitosis and meiosis, including functions related to chro
130 variant of the cell cycle that differs from mitosis and occurs in specific tissues of different orga
131 nes that were rate-limiting for the onset of mitosis and of the S-phase, that were responsible for th
132 trosome genes cause microcephaly by delaying mitosis and pathologically activating the mitotic survei
134 genes from non-treatment studies are part of mitosis and play vital role in DNA repair and cell-cycle
136 nges in the intracellular redox state during mitosis and provide insights into how novel small-molecu
137 und factors in pluripotent stem cells during mitosis and reveal that PRC2, DNA methylation and Mecp2
140 the chromosome arms and then centromeres in mitosis and subsequently the central spindle in anaphase
141 chinery that ensures the proper execution of mitosis and the accurate segregation of chromosomes to d
142 er (MCF-7) cells from the interphase through mitosis and then to the cytokinesis stages of their grow
144 porary inactivation of CDK4/6 activity after mitosis, and a progressively increasing persistence in C
145 ht, undergo rapid cycles of DNA replication, mitosis, and cell division, producing up to 16 daughter
146 nocodazole, which inhibits DNA replication, mitosis, and cell division; this suggests some aspect of
147 typically form a single, round nucleus after mitosis, and failures to do so can compromise genomic in
148 n of the TRe response leads to DNA damage in mitosis, and promotes chromosome instability and cell de
149 sses, and mTORC1 itself, is different during mitosis, and this has remained an area of significant co
152 for recruitment of dynein to kinetochores in mitosis, as a key factor required for dynein-dependent m
154 low proliferative and show a delay in early mitosis at prometaphase, associated with chromosome-alig
155 seems to transpose only at the second pollen mitosis because all dRemp insertion mutants are nonconco
156 at inactivating TRIM37 improves acentrosomal mitosis because TRIM37 prevents PLK4 from self-assemblin
157 e interruption of not only the second pollen mitosis but also the movement of siRNA from the vegetati
160 bition of ATR kinase significantly prolonged mitosis but this effect was mostly independent of RAD50
161 s of 5-ITu eliminates the H3T3ph mark during mitosis, but does not affect the mode or the outcome of
162 occurs late in the cell cycle to prepare for mitosis, but in CHK1i-sensitive cells, high activity can
164 ds of a cell are believed to increase during mitosis, but the rates of ATP synthesis and consumption
168 overning spindle assembly during meiosis and mitosis by releasing the inhibitory effects of importin
169 both NPCs in interphase and kinetochores in mitosis can generate anaphase inhibitors to efficiently
170 t kinetochore-microtubule attachments during mitosis can lead to chromosomal instability, a hallmark
171 nsure faithful chromosome segregation during mitosis, cell polarization and migration, and primary ci
179 interphase and chromosome segregation during mitosis, demonstrating that active retrotransposons are
180 n of mature ribosomes from the nucleus after mitosis depends on Ki-67-regulated chromosome clustering
182 to reproduce the cardinal features of human mitosis determined experimentally by numerous laboratori
187 ment is intimately dependent upon progenitor mitosis duration and uncover a crucial post-transcriptio
188 s dynamic behavior is mediated by asymmetric mitosis, ERK activity, cell-cycle duration, and DNA repa
193 progression and control, particularly during mitosis for which the role of kinases has been extensive
194 uantify mitochondrial ATP synthesis rates in mitosis from the single-cell time-dynamics of mitochondr
196 e physical separation of daughter cells once mitosis has been completed, is executed in fungal and an
201 , myb81-1 microspores fail to undergo pollen mitosis I (PMI) and arrest at polarized stage with a sin
204 cumulation of reactive oxygen species during mitosis in cancer by disturbing mitotic progression and
206 lants, it remains present in later stages of mitosis in ccs52a2-1 mutant plants, marking it as a puta
210 nce nuclear envelope breakdown occurs during mitosis in metazoan cells, it has been proposed that mac
212 (CRK5), is a critical regulator of atypical mitosis in the gametogony and is required for mosquito t
213 c24, and Spc25) has well-documented roles in mitosis including 1) connecting mitotic chromosomes to s
214 over-elongation, especially during prolonged mitosis induced by various factors, importantly includin
215 ange of cancers and their important roles in mitosis, inhibitors targeting Aurora kinases have attrac
216 s to proliferate and that transitioning from mitosis into G1 phase is delayed in galectin-8-knockout
218 cycle in tumor cells, cells are driven into mitosis irrespective of DNA damage and unattached/misatt
222 plain how centromere targeting of the CPC in mitosis is coupled to its movement to the central spindl
224 are both mediated by Scc1-cohesin, which in mitosis is released from chromosomes by Wapl and separas
225 which affect lamina depolymerization during mitosis, is sufficient to prevent the mixing of the pare
226 links and DNA-protein crosslinks, whereas in mitosis it triggers disassembly of all replisomes that r
227 nuclear rim in interphase cells, but during mitosis it was absent from kinetochores and enveloped ch
228 eria (age at diagnosis, histologic category, mitosis-karyorrhexis index (MKI), grade of differentiati
233 nitor of sperm) to promote the second pollen mitosis, mediates siRNA movement to reinforce heterochro
236 ensures proper chromosome segregation during mitosis not only by controlling KT-MT attachment but als
239 by promoting cell rounding and stiffening in mitosis, oncogenic Ras(V12) enables cells to proliferate
240 se in each cell cycle, and that ensured that mitosis only took place when S-phase was properly comple
242 eassembling the nuclear envelope in an 'open mitosis' or by reshaping an intact nucleus and then divi
243 waves play an essential role in coordinating mitosis over large distances, but what determines the sp
245 the establishment of centriole asymmetry in mitosis primes biased interphase MTOC activity, necessar
248 it prevents NuMA from binding chromosomes at mitosis, regulates its nuclear mobility, and is essentia
249 otein cysteine oxidation is increased during mitosis relative to other cell cycle phases suggests tha
251 However, repression of macroautophagy during mitosis remains controversial and mechanistic detail lim
253 kout hearts display widespread cardiomyocyte mitosis, sarcomere disassembly and improved left ventric
254 Topoisomerase II (Topo II) is essential for mitosis since it resolves sister chromatid catenations.
255 rgoes extensive remodeling in interphase and mitosis, so mechanisms that seal NE holes and protect it
257 and autosomal genes independently influence mitosis such that their phenotypic consequences can be c
260 y, ATP levels and ATP synthesis are lower in mitosis than in G2 in synchronized cell populations.
261 interphase, reaching a maximal size prior to mitosis that declined steadily over the course of develo
262 to study spindle dynamics in cells entering mitosis, the chromatin-based signal was found to prefere
268 gene expression is globally inhibited during mitosis, the transcriptional programs that define cell i
269 accumulate on the daughter centriole during mitosis, thereby generating molecularly distinct mother
270 local nuclear envelope breakdown in a closed mitosis therefore closely mirrors nuclear envelope break
271 cient MRE11A (p.W210C) tended to show slower mitosis, though by far not as significant as RAD50-defic
274 hrin heavy chain (CHC) are repurposed during mitosis to directly recruit the microtubule-stabilizing
275 gulates the RZZ-Spindly-dynein module during mitosis to ensure the fidelity of chromosome segregation
277 synchronized cells at different stages after mitosis to generate genome-wide maps of histone modifica
280 scriptomes undergo dynamic changes after the mitosis-to-meiosis transition and have been subject to e
284 he master regulator of macroautophagy during mitosis, uncoupling autophagy regulation from nutrient s
286 d to organize the spindle microtubule during mitosis using an additional microtubule binding site in
289 re precisely understand the role of TOP2A in mitosis, we used the auxin-inducible degron (AID) system
291 al for adequate tumor cell engagement during mitosis, when tumor cells are vulnerable, for efficacy.
292 pletion of DNA replication prior to entering mitosis, which ensures accurate chromosome segregation.
293 formation in the apical cell at the onset of mitosis, which occurs one to two cell cycles before a br
295 otes Bub3 loading onto kinetochores in early mitosis, which, in turn, facilitates Bub1 and BubR1 kine
296 e kinesin-5 motors and crosslinkers in early mitosis, while interkinetochore stretch becomes importan
299 ope is split into two at the end of a closed mitosis without compromising the impermeability of the n