戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 nhanced stabilization of NANOG protein after mitosis.
2 o functional nuclear pores toward the end of mitosis.
3  EBV episomes to the host chromosomes during mitosis.
4 lishing long-range chromatin interactions in mitosis.
5 loped a computational model of fission-yeast mitosis.
6 th MRE11A inhibitor PFM39 modestly prolonged mitosis.
7  centromere organization and function during mitosis.
8 e promoting complex/cyclosome (APC/C) during mitosis.
9 l for faithful chromosome segregation during mitosis.
10 ical consequences of failed DSB responses in mitosis.
11  nucleus migrates apically before undergoing mitosis.
12 K1delta and CK1epsilon during interphase and mitosis.
13 le, enabling sister chromatid segregation in mitosis.
14 mmetric distribution of gamma-tubulin during mitosis.
15 lular mechanisms adopted by single cells for mitosis.
16 er complex (CPC) is a conserved regulator of mitosis.
17 sis partitions the cell contents to complete mitosis.
18 uclei, during telophase of a DNA elimination mitosis.
19  interphase but stiffen more upon entry into mitosis.
20 pha-tubulin interacts with galectin-8 during mitosis.
21 ed to the centrosomes and chromosomes during mitosis.
22 kinetochores and spindle microtubules during mitosis.
23 ionally active RNA polymerase II (Pol II) in mitosis.
24 ic changes due to cortical stiffening around mitosis.
25 lls that harbor only one SPB delay exit from mitosis.
26 s, switching cell fate from apoptosis toward mitosis.
27 ly after nuclear envelope reformation during mitosis.
28 symmetric segregation of extra centrioles in mitosis.
29 rical and mechanically stiffen as they enter mitosis.
30 urora B kinase (AURKB) and Survivin in early mitosis.
31  is either repressed or highly active during mitosis.
32  division, despite rounding up as they enter mitosis.
33  nuclear position into a signal to exit from mitosis.
34  the upstream centrosome defects or extended mitosis.
35 chment point for spindle microtubules during mitosis.
36 parental genomes into a single nucleus after mitosis.
37 ilized as kinetochores come under tension in mitosis.
38 showed chromosome segregation defects during mitosis.
39 MEN signaling, enabling the timely exit from mitosis.
40 ation of chromosomes with the INM throughout mitosis.
41     Aurora B kinase plays essential roles in mitosis.
42 pronounced spindle abnormalities and delayed mitosis.
43 mentalization of eukaryotic cells after open mitosis.
44 ism of licensing of chromosome compaction in mitosis.
45  S-phase entry and promotes progression into mitosis.
46  that MRN may also impact on the duration of mitosis.
47 lation necessary to ensure proper entry into mitosis.
48 n that were generationally conserved through mitosis.
49 ortant role in promoting efficient exit from mitosis.
50 icus, which breaks and reforms the NE during mitosis.
51 P/p25 alters microtubule dynamics and delays mitosis.
52 to complete the replication of CFSs in early mitosis.
53 s then sharply downregulated at the onset of mitosis.
54 ow acute depletion of these proteins affects mitosis.
55  inheritance during Saccharomyces cerevisiae mitosis.
56 lin B:CDK1 is the master kinase regulator of mitosis.
57 in cells, especially before their entry into mitosis.
58 , modulation of actomyosin contractility, or mitosis.
59 mitted from mother to daughter cells through mitosis.
60  this endocytic interaction mechanism during mitosis.
61 esin that peaks in protein expression during mitosis.
62 o ensure repression of macroautophagy during mitosis.
63 NA repair, and chromosome segregation during mitosis.
64  future study of autophagy and mTORC1 during mitosis.
65 at phosphorylates histone H3 at Thr-3 during mitosis.
66 ric chromatin to spindle microtubules during mitosis.
67 some segregation defects in cells undergoing mitosis.
68 atid compaction and their segregation during mitosis.
69 iquitination of cell cycle regulators during mitosis.
70 wing this delay, cells entered and completed mitosis.
71 dentity is determined shortly after terminal mitosis.
72 esin from chromatin as cells transit through mitosis.
73 e necessary for catenation resolution during mitosis.
74  and function of the inner centromere during mitosis.
75 vere locus instability and missegregation in mitosis.
76 ion at S227 by PKCepsilon also occurs during mitosis.
77 nterference between apical contractility and mitosis.
78  incomplete dissociation of DAs and cilia in mitosis.
79  during interphase, ahead of Rcd4 and before mitosis.
80 aryotic chromosomes in interphase and during mitosis.
81 to specific transcription start sites during mitosis.
82 rominent role in spindle organization during mitosis.
83 apture and segregation of chromosomes during mitosis.
84  and impairs chromosome biorientation during mitosis.
85 ial for proper chromosome segregation during mitosis.
86 ntil the breaks are repaired before entering mitosis.
87  branched species at the very late stages of mitosis.
88 is critical for EWSR1/FLI1 interference with mitosis.
89 n important kinase that regulates DAs before mitosis.
90 tial for chromosome bridge resolution during mitosis.
91 enance of adhesions and retraction fibers in mitosis [1-6], which are thought to act as local cues th
92 s and then dividing it into two in a 'closed mitosis'(2,3).
93 y mirrors nuclear envelope breakdown in open mitosis(3), revealing an unexpectedly high conservation
94 aromyces pombe (a classical model for closed mitosis(5)), genetics, live-cell imaging and electron to
95 led in G1 and disassembled sometime prior to mitosis [6-9].
96 molecular brushes during the early stages of mitosis(8), but during mitotic exit the brushes collapse
97  protein kinase Czeta (PKCzeta) and never in mitosis A (NIMA)-related kinase 9 (NEK9).
98 te within a commonly phosphorylated motif in mitosis (a non-canonical target of Cyclin B-Cdk1), and o
99 e/threonine-protein kinase-like (MASTL) is a mitosis-accelerating kinase with emerging roles in cance
100 to promote attachment turnover, and in later mitosis, activity decreases to ensure attachment stabili
101  chromatin and recruits membrane proteins in mitosis, also facilitates nuclear membrane repair in int
102 ation, abnormal ciliogenesis, and changes in mitosis and cell cycle progression.
103 ons are central to many processes, including mitosis and chemotropism.
104 its components changes at distinct stages of mitosis and cytokinesis has not been addressed.
105 es pombe AMR proteins at different stages of mitosis and cytokinesis using an approach we have develo
106  Plk1 levels resulted in multiple defects in mitosis and cytokinesis, supernumerary centrosomes, and
107 e stability relies on proper coordination of mitosis and cytokinesis, where dynamic microtubules capt
108 arasite attachment and facilitation of rapid mitosis and cytokinesis.
109 etic studies imply that NTMT1 regulates cell mitosis and DNA damage repair.
110 he inner kinetochore protein CENP-C in early mitosis and does not require either the Bub1/pH2A/Sgo1 o
111 e-organizing centers required for error-free mitosis and embryonic development.
112 spatiotemporal cues about the progression of mitosis and ensures that release of Cdc14 phosphatase oc
113 l, we blocked cells from progressing through mitosis and found that prolonged metaphase delay is suff
114 oordinating gamma-tubulin recruitment during mitosis and illuminates the mechanism by which signaling
115 e activity causes cell morphology defects in mitosis and impairs epithelial tissue integrity.
116 of Bim1-binding by Cik1-Kar3 delays cells in mitosis and impairs microtubule bundle organization and
117                         Furthermore, delayed mitosis and increased mitotic cell death was observed fo
118 play an absence of the H3R17me2a mark during mitosis and increased signs of DNA damage.
119  decreases by approximately 50% during early mitosis and increases back to G2 levels during cytokines
120 tion of centrosome proteins in NPCs prolongs mitosis and increases TP53-mediated apoptosis.
121  actin network organization and mechanics in mitosis and is required for successful cell division in
122 s promote chromosome segregation during both mitosis and meiosis and regulate chromosomal and genomic
123 ctive segregation patterns of chromosomes in mitosis and meiosis are dictated in part by the kinetoch
124 he control of chromosome segregation between mitosis and meiosis II.
125 e kinases (PLKs) have numerous roles in both mitosis and meiosis, including functions related to chro
126 o link the chromosome to microtubules during mitosis and meiosis.
127 r of oogonia_4 is the critical state between mitosis and meiosis.
128  for the proper separation of chromosomes in mitosis and meiosis.
129 inetochores direct chromosome segregation in mitosis and meiosis.
130  variant of the cell cycle that differs from mitosis and occurs in specific tissues of different orga
131 nes that were rate-limiting for the onset of mitosis and of the S-phase, that were responsible for th
132 trosome genes cause microcephaly by delaying mitosis and pathologically activating the mitotic survei
133             Cells lacking cyclin B can enter mitosis and phosphorylate most mitotic proteins, because
134 genes from non-treatment studies are part of mitosis and play vital role in DNA repair and cell-cycle
135 ish parallels between microtubule-sliding in mitosis and post-mitotic neurons.
136 nges in the intracellular redox state during mitosis and provide insights into how novel small-molecu
137 und factors in pluripotent stem cells during mitosis and reveal that PRC2, DNA methylation and Mecp2
138  protein levels increase before the onset of mitosis and sharply decrease during mitosis exit.
139 s related to gamete fusion, oocyst shedding, mitosis and spermiogenesis.
140  the chromosome arms and then centromeres in mitosis and subsequently the central spindle in anaphase
141 chinery that ensures the proper execution of mitosis and the accurate segregation of chromosomes to d
142 er (MCF-7) cells from the interphase through mitosis and then to the cytokinesis stages of their grow
143      Smc5 cKO NPCs formed DNA bridges during mitosis and underwent chromosome missegregation.
144 porary inactivation of CDK4/6 activity after mitosis, and a progressively increasing persistence in C
145 ht, undergo rapid cycles of DNA replication, mitosis, and cell division, producing up to 16 daughter
146  nocodazole, which inhibits DNA replication, mitosis, and cell division; this suggests some aspect of
147 typically form a single, round nucleus after mitosis, and failures to do so can compromise genomic in
148 n of the TRe response leads to DNA damage in mitosis, and promotes chromosome instability and cell de
149 sses, and mTORC1 itself, is different during mitosis, and this has remained an area of significant co
150 fold greater, with preferential induction of mitosis- and interferon-related genes.
151 karyotes, but their specialised functions in mitosis are incompletely understood.
152 for recruitment of dynein to kinetochores in mitosis, as a key factor required for dynein-dependent m
153 downregulation of cell-cycle progression and mitosis-associated proteins.
154  low proliferative and show a delay in early mitosis at prometaphase, associated with chromosome-alig
155 seems to transpose only at the second pollen mitosis because all dRemp insertion mutants are nonconco
156 at inactivating TRIM37 improves acentrosomal mitosis because TRIM37 prevents PLK4 from self-assemblin
157 e interruption of not only the second pollen mitosis but also the movement of siRNA from the vegetati
158 cking Cmp7 have compromised NE sealing after mitosis but are viable.
159 somal localization of Cdc13 and the onset of mitosis but does not prevent S phase.
160 bition of ATR kinase significantly prolonged mitosis but this effect was mostly independent of RAD50
161 s of 5-ITu eliminates the H3T3ph mark during mitosis, but does not affect the mode or the outcome of
162 occurs late in the cell cycle to prepare for mitosis, but in CHK1i-sensitive cells, high activity can
163 e qualitatively similar to those observed in mitosis, but quantitatively far less prevalent.
164 ds of a cell are believed to increase during mitosis, but the rates of ATP synthesis and consumption
165         DAs are released from centrosomes in mitosis by an undefined mechanism.
166 these fate decisions can be determined after mitosis by mitochondrial remodeling.
167 de cyclin B1, cumulating in the cell exiting mitosis by mitotic slippage.
168 overning spindle assembly during meiosis and mitosis by releasing the inhibitory effects of importin
169  both NPCs in interphase and kinetochores in mitosis can generate anaphase inhibitors to efficiently
170 t kinetochore-microtubule attachments during mitosis can lead to chromosomal instability, a hallmark
171 nsure faithful chromosome segregation during mitosis, cell polarization and migration, and primary ci
172                  To segregate chromosomes in mitosis, cells assemble a mitotic spindle, a molecular m
173                                At the end of mitosis, cells must remodel their nuclear envelope to pr
174              However, when DSBs occur during mitosis, cells no longer arrest but prioritize completio
175                                       During mitosis chromosomes reorganise into highly compact, rod-
176                                       During mitosis, chromosomes are compacted in length by more tha
177              We observed that in contrast to mitosis, Cin8 and Kip3 together are indispensable for me
178                                       During mitosis, CRL4 dissociates from RepID and replaces it wit
179 interphase and chromosome segregation during mitosis, demonstrating that active retrotransposons are
180 n of mature ribosomes from the nucleus after mitosis depends on Ki-67-regulated chromosome clustering
181 endent mRNA translation is maintained during mitosis despite mTORC1 activity being repressed.
182  to reproduce the cardinal features of human mitosis determined experimentally by numerous laboratori
183                   Microtubules (MTs) mediate mitosis, directional signaling, and are therapeutic targ
184                    Removal of TOP2A prior to mitosis does not affect prophase timing or the initiatio
185 ent only on the rate of random movements vs. mitosis-driven advection.
186 ng neurogenic divisions results in prolonged mitosis due to extension of early mitotic stages.
187 ment is intimately dependent upon progenitor mitosis duration and uncover a crucial post-transcriptio
188 s dynamic behavior is mediated by asymmetric mitosis, ERK activity, cell-cycle duration, and DNA repa
189                                At the end of mitosis, eukaryotic cells must segregate the two copies
190 ex to autophagic puncta, is repressed during mitosis, even when mTORC1 is inhibited.
191 onset of mitosis and sharply decrease during mitosis exit.
192                                        After mitosis, fate-committed daughter cells delaminate from t
193 progression and control, particularly during mitosis for which the role of kinases has been extensive
194 uantify mitochondrial ATP synthesis rates in mitosis from the single-cell time-dynamics of mitochondr
195 sequent burst of DNA replication in the next mitosis generates extensive DNA damage.
196 e physical separation of daughter cells once mitosis has been completed, is executed in fungal and an
197                                              Mitosis has been studied in a wide variety of eukaryotes
198                The primary roles of TOP2A in mitosis have been difficult to decipher due to its multi
199 ates of ATP synthesis and consumption during mitosis have not been quantified.
200                                       During mitosis, however, the nuclear envelope in animal and pla
201 , myb81-1 microspores fail to undergo pollen mitosis I (PMI) and arrest at polarized stage with a sin
202 d subsequently complete locus duplication in mitosis in a process known as 'MiDAS'.
203 d colocalize on condensed chromosomes during mitosis in C. elegans embryos.
204 cumulation of reactive oxygen species during mitosis in cancer by disturbing mitotic progression and
205 is important for chromosome alignment during mitosis in cancer cell lines.
206 lants, it remains present in later stages of mitosis in ccs52a2-1 mutant plants, marking it as a puta
207 serves as a dynamic structural framework for mitosis in eukaryotic cells.
208                                During closed mitosis in fission yeast, growing microtubules push onto
209                Despite an equivalent rate of mitosis in hepatocytes of differing ploidies, we found n
210 nce nuclear envelope breakdown occurs during mitosis in metazoan cells, it has been proposed that mac
211 meiotic chromosomes, and unusual restitution mitosis in tapetum cells.
212  (CRK5), is a critical regulator of atypical mitosis in the gametogony and is required for mosquito t
213 c24, and Spc25) has well-documented roles in mitosis including 1) connecting mitotic chromosomes to s
214 over-elongation, especially during prolonged mitosis induced by various factors, importantly includin
215 ange of cancers and their important roles in mitosis, inhibitors targeting Aurora kinases have attrac
216 s to proliferate and that transitioning from mitosis into G1 phase is delayed in galectin-8-knockout
217                          The transition from mitosis into the first gap phase of the cell cycle in bu
218  cycle in tumor cells, cells are driven into mitosis irrespective of DNA damage and unattached/misatt
219                                              Mitosis is a dramatic process that affects all parts of
220                Chromosome segregation during mitosis is antagonistically regulated by the Aurora-B ki
221                       CDK phosphorylation in mitosis is compromised for approximately half of all mit
222 plain how centromere targeting of the CPC in mitosis is coupled to its movement to the central spindl
223 oreplication in which the genome replicates, mitosis is omitted, and cells grow in size.
224  are both mediated by Scc1-cohesin, which in mitosis is released from chromosomes by Wapl and separas
225  which affect lamina depolymerization during mitosis, is sufficient to prevent the mixing of the pare
226 links and DNA-protein crosslinks, whereas in mitosis it triggers disassembly of all replisomes that r
227  nuclear rim in interphase cells, but during mitosis it was absent from kinetochores and enveloped ch
228 eria (age at diagnosis, histologic category, mitosis-karyorrhexis index (MKI), grade of differentiati
229                                       During mitosis, Kif11, a kinesin motor protein, promotes bipola
230                                     Early in mitosis, kinase activity at kinetochores is high to prom
231                                Inappropriate mitosis-like chromosome segregation in meiosis leads to
232 n an undetectable paternal allele and, after mitosis, loss of one or both chromosomal arms.
233 nitor of sperm) to promote the second pollen mitosis, mediates siRNA movement to reinforce heterochro
234                             When cells enter mitosis, membrane-chromatin contacts must be broken to a
235 .10% accuracy with only 3300 samples for the Mitosis (microscopic) dataset.
236 ensures proper chromosome segregation during mitosis not only by controlling KT-MT attachment but als
237                   We found that GOF disrupts mitosis of radial-glia neural progenitors (RGCs), inside
238 the two parental pronuclei fuse in the first mitosis of the embryo are poorly understood.
239 by promoting cell rounding and stiffening in mitosis, oncogenic Ras(V12) enables cells to proliferate
240 se in each cell cycle, and that ensured that mitosis only took place when S-phase was properly comple
241 and that CDK4/6 activity can be active after mitosis or inactive for variable time periods.
242 eassembling the nuclear envelope in an 'open mitosis' or by reshaping an intact nucleus and then divi
243 waves play an essential role in coordinating mitosis over large distances, but what determines the sp
244 progression and chromatid segregation during mitosis, possibly in a nontranscriptional manner.
245  the establishment of centriole asymmetry in mitosis primes biased interphase MTOC activity, necessar
246                                As cells exit mitosis, promoters regain H3K27ac, which correlates with
247            Indeed, mTORC1 is inactive during mitosis, reflecting its failure to localize to lysosomes
248 it prevents NuMA from binding chromosomes at mitosis, regulates its nuclear mobility, and is essentia
249 otein cysteine oxidation is increased during mitosis relative to other cell cycle phases suggests tha
250 bution of Mad1-C-Mad2 as cells progress into mitosis remain elusive.
251 However, repression of macroautophagy during mitosis remains controversial and mechanistic detail lim
252           Accurate chromosome segregation in mitosis requires sister kinetochores to bind to microtub
253 kout hearts display widespread cardiomyocyte mitosis, sarcomere disassembly and improved left ventric
254  Topoisomerase II (Topo II) is essential for mitosis since it resolves sister chromatid catenations.
255 rgoes extensive remodeling in interphase and mitosis, so mechanisms that seal NE holes and protect it
256                                       During mitosis, spatiotemporal regulation of phosphorylation at
257  and autosomal genes independently influence mitosis such that their phenotypic consequences can be c
258 cle entry, thus interfering with S-phase- or mitosis-targeting agents.
259 ll division, specifically the final stage of mitosis termed abscission.
260 y, ATP levels and ATP synthesis are lower in mitosis than in G2 in synchronized cell populations.
261 interphase, reaching a maximal size prior to mitosis that declined steadily over the course of develo
262  to study spindle dynamics in cells entering mitosis, the chromatin-based signal was found to prefere
263                               As cells enter mitosis, the genome is restructured to facilitate chromo
264                                       During mitosis, the genome is transformed from a decondensed, t
265                                        After mitosis, the majority of daughter cells extend a long, b
266                              At the onset of mitosis, the position of the astral MT network, specific
267                                       During mitosis, the Spindle Assembly Checkpoint (SAC) maintains
268 gene expression is globally inhibited during mitosis, the transcriptional programs that define cell i
269  accumulate on the daughter centriole during mitosis, thereby generating molecularly distinct mother
270 local nuclear envelope breakdown in a closed mitosis therefore closely mirrors nuclear envelope break
271 cient MRE11A (p.W210C) tended to show slower mitosis, though by far not as significant as RAD50-defic
272 s by maintaining centrosome integrity during mitosis through interacting with alpha-tubulin.
273 disassembly allows cells that undergo closed mitosis to achieve nuclear division.
274 hrin heavy chain (CHC) are repurposed during mitosis to directly recruit the microtubule-stabilizing
275 gulates the RZZ-Spindly-dynein module during mitosis to ensure the fidelity of chromosome segregation
276 pression as cells transition from G2 through mitosis to G1.
277 synchronized cells at different stages after mitosis to generate genome-wide maps of histone modifica
278        The asynchrony of the transition from mitosis to meiosis results in heterogeneity in the femal
279 allows the MEN to couple the final stages of mitosis to spindle position.
280 scriptomes undergo dynamic changes after the mitosis-to-meiosis transition and have been subject to e
281                                    After the mitosis-to-meiosis transition in male mice, specific ERV
282 bursts in germline gene expression after the mitosis-to-meiosis transition.
283                                       During mitosis, transcription of genomic DNA is dramatically re
284 he master regulator of macroautophagy during mitosis, uncoupling autophagy regulation from nutrient s
285                 Most metazoan cells entering mitosis undergo characteristic rounding, which is import
286 d to organize the spindle microtubule during mitosis using an additional microtubule binding site in
287                                We found that mitosis was commonly initiated at sperm-derived nuclei a
288                   Cell death after a delayed mitosis was rescued by inactivation of the mitotic surve
289 re precisely understand the role of TOP2A in mitosis, we used the auxin-inducible degron (AID) system
290             DSBs are especially dangerous in mitosis when cells go through the complex process of equ
291 al for adequate tumor cell engagement during mitosis, when tumor cells are vulnerable, for efficacy.
292 pletion of DNA replication prior to entering mitosis, which ensures accurate chromosome segregation.
293 formation in the apical cell at the onset of mitosis, which occurs one to two cell cycles before a br
294 er birth via endoreplication and interrupted mitosis, which persists through adulthood.
295 otes Bub3 loading onto kinetochores in early mitosis, which, in turn, facilitates Bub1 and BubR1 kine
296 e kinesin-5 motors and crosslinkers in early mitosis, while interkinetochore stretch becomes importan
297                                           In mitosis, while the importance of kinetochore (KT)-microt
298 taphase, and ultimately causes cells to exit mitosis without chromosome segregation occurring.
299 ope is split into two at the end of a closed mitosis without compromising the impermeability of the n
300      Mitotic slippage involves cells exiting mitosis without proper chromosome segregation.
301  same basic organization persists throughout mitosis, without helical coiling.

 
Page Top