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1 te detachment and/or mitotic podocyte death (mitotic catastrophe).
2 empted, it may lead to aberrant mitosis (ie, mitotic catastrophe).
3 ading to apoptotic cell death in S-phase and mitotic catastrophe.
4 h the emergence of disorganized anaphase and mitotic catastrophe.
5 nd a strong Plk1 hypomorph underwent serious mitotic catastrophe.
6 efects, delays cells in mitosis, and induces mitotic catastrophe.
7 nhanced, temozolomide-induced senescence and mitotic catastrophe.
8 agenicity by a combination of senescence and mitotic catastrophe.
9 cytokinesis and, thereby, is responsible for mitotic catastrophe.
10 cells bypassing G(2)-M arrest and undergoing mitotic catastrophe.
11 only transient G(2)-M arrest before death by mitotic catastrophe.
12 emperature-sensitive lethal phenotype termed mitotic catastrophe.
13 tion was modelled as proliferation-dependent mitotic catastrophe.
14 iant, multinucleated cells characteristic of mitotic catastrophe.
15 radiation, which is accompanied by increased mitotic catastrophe.
16 kinase inhibitors by activating BIM-mediated mitotic catastrophe.
17 entrosomes, and vulnerability to anaphase or mitotic catastrophe.
18 istent DNA damage, telomere dysfunction, and mitotic catastrophe.
19 segregation defects, mitotic exit delays and mitotic catastrophe.
20  fork collapse, chromosomal instability, and mitotic catastrophe.
21 sisting replication intermediates can confer mitotic catastrophe.
22            Piezo1 loss in podocytes leads to mitotic catastrophe.
23 of cyclin B1, resulting in G(2)-M arrest and mitotic catastrophe.
24 is, which was associated with aneuploidy and mitotic catastrophe.
25 echanism of action of BETi in TNBC, that is, mitotic catastrophe.
26 r-replicated DNA to enter mitosis leading to mitotic catastrophe.
27 1 induced aberrant mitosis and cell death by mitotic catastrophe.
28 cells eliminates Ras-activated cells through mitotic catastrophe.
29 ssion through mitosis, marked aneuploidy and mitotic catastrophe.
30  cell death, both of which are indicative of mitotic catastrophe.
31 sion before repair is completed resulting in mitotic catastrophe.
32 ycle checkpoints, resulting in cell death by mitotic catastrophe.
33 and avoidance of replication catastrophe and mitotic catastrophe.
34  cycle progression, and both replication and mitotic catastrophe.
35 er hand, forced expression of KLF14 leads to mitotic catastrophe.
36 ugh into mitosis, resulting in cell death by mitotic catastrophe.
37 ed mitotic arrest then led to cell death via mitotic catastrophe.
38 s into mitosis where they ultimately died by mitotic catastrophe.
39  required for cell cycle progression undergo mitotic catastrophe.
40 PS SMCs exhibit prolonged mitosis and die of mitotic catastrophe.
41 on after DNA replication, ultimately causing mitotic catastrophe.
42 n clusters of nuclear vesicles indicative of mitotic catastrophe.
43 nd apoptosis induced in vivo in part through mitotic catastrophe.
44 sing cells from centrosome reduplication and mitotic catastrophe.
45 s (G2/M) checkpoint activation and resultant mitotic catastrophe.
46 reas p53 wild-type tumors arrested, avoiding mitotic catastrophe.
47 , have more than two centrosomes and undergo mitotic catastrophe.
48 increased the frequency of radiation-induced mitotic catastrophe.
49 postmitotic endoreduplication checkpoint and mitotic catastrophe.
50 oint, leading to premature mitotic entry and mitotic catastrophe.
51  mitotic transition and results in increased mitotic catastrophe.
52 damage- and heat shock-induced death, and in mitotic catastrophe.
53 itions of RBM3 downregulation, cells undergo mitotic catastrophe.
54 lity characterized by gross polyploidism and mitotic catastrophe.
55 itochondrial membrane potential, followed by mitotic catastrophe.
56 rther differs from cellular death defined by mitotic catastrophe.
57 t; centrosome overduplication; and, finally, mitotic catastrophe.
58 nase NIMA and suppress its ability to induce mitotic catastrophe [2, 3].
59     Double-strand breaks (DSBs) can initiate mitotic catastrophe, a complex oncosuppressive phenomeno
60 lt cardiac myocytes resulted in induction of mitotic catastrophe, a previously unreported event in ca
61 c progression with 4N DNA content leading to mitotic catastrophe after abrogation of the S or G2 chec
62 c28 and Mre11 appear to cooperate to prevent mitotic catastrophe after DNA replication arrest.
63 s the engagement of apoptosis, probably by a mitotic catastrophe, after 8 Gy of gamma-irradiation in
64 -M checkpoint function, leading to increased mitotic catastrophe and a modest increase in radiation s
65 ately resulting in centrosome reduplication, mitotic catastrophe and abrogation of cell proliferation
66  accompanied by reduced radiotherapy-induced mitotic catastrophe and an accelerated release from G2/M
67 ough telomeric DNA end fusions, resulting in mitotic catastrophe and apoptosis; and 4) induction of c
68 ecociously enter mitosis and ultimately face mitotic catastrophe and apoptotic cell death.
69                Inactivation of SAE2 leads to mitotic catastrophe and cell death upon Myc hyperactivat
70 -E6 cells and similarly enhanced TMZ-induced mitotic catastrophe and cell death.
71 yclinB1 activity, thus preventing cells from mitotic catastrophe and cell death.
72 ear features, all of which are indicative of mitotic catastrophe and chromosome instability.
73 yet uncontrolled replicative stress leads to mitotic catastrophe and inflammatory responses that prom
74 g prolongation, multipolarity, misalignment, mitotic catastrophe and loss of spindle checkpoint, that
75 4 knockdown or overexpression coincided with mitotic catastrophe and multinucleation that are typical
76 on of CX-5461 and radiation occurred through mitotic catastrophe and not apoptosis.
77 icroscopy studies revealed that it induces a mitotic catastrophe and precocious mitotic exit even in
78 e spindle assembly checkpoint, and triggered mitotic catastrophe and reduced cell viability.
79 however, with increases in radiation-induced mitotic catastrophe and senescence and produced only a m
80 wed polyploidy, and features consistent with mitotic catastrophe and senescence.
81 ion abnormalities lead to cell death through mitotic catastrophe and that cell death occurred also fr
82 drive abnormal mitotic spindle formation and mitotic catastrophe and that deregulated FOXM1 and KIF20
83 hat FoxM1 depletion causes cell death due to mitotic catastrophe and that inhibiting FoxM1 represents
84 ing both pathways simultaneously resulted in mitotic catastrophe and tumor apoptosis and markedly red
85 ubulin, leading to sustained mitotic arrest, mitotic catastrophe, and apoptosis while sparing nonmali
86 ss their interplay with cellular senescence, mitotic catastrophe, and autophagy.
87 cancer cells become PARPi sensitive, undergo mitotic catastrophe, and die.
88  xenografts induces premature mitotic entry, mitotic catastrophe, and reduction of tumor growth.
89        Ionizing radiation induces apoptosis, mitotic catastrophe, and senescence-like terminal prolif
90 d cytokinesis defects, eventually leading to mitotic catastrophe/apoptosis.
91 asing the percentage of cells that underwent mitotic catastrophe as a consequence of G(2) checkpoint
92 sels against repeated doses, and introducing mitotic catastrophe (as opposed to arbitrary delayed cel
93              The 14-3-3sigma-/- cells died ('mitotic catastrophe') as they entered mitosis.
94  of EAG2 expression results in G2 arrest and mitotic catastrophe associated with failure of premitoti
95                    PIAS2b depletion promotes mitotic catastrophe at prophase.
96 prevented cell death by selectively limiting mitotic catastrophe, but not apoptosis.
97 odocyte injury, MDM2 drives podocyte loss by mitotic catastrophe, but the function of MDM2 in resting
98 oducing DL78, which induces cancer-selective mitotic catastrophe by disrupting Myc's interaction with
99 ces multiple effects of its loss: G2 arrest, mitotic catastrophe, chromosome mis-segregation / aneupl
100  repair and suppresses the radiation-induced mitotic catastrophe compared to control cells.
101 ncreased DNA damage, chromosomal breaks, and mitotic catastrophe compared with monotherapy.
102 ide an appraisal of the in situ pathology of mitotic catastrophe compared with other proposed types o
103 utated but not in wild-type cells leading to mitotic catastrophe, defective cell division and apoptos
104 e alignment, which have been shown to induce mitotic catastrophe-dependent senescence.
105 human homolog of Xenopus gene which prevents mitotic catastrophe), directly binds to the EpRE and int
106 y of the SLX4 complex is critical to prevent mitotic catastrophe following common fragile site expres
107 53-dependent activation of Snk/Plk2 prevents mitotic catastrophe following spindle damage.
108 abditis elegans ATR) and rad-5/clk-2 prevent mitotic catastrophe, function in the S-phase checkpoint
109 human homolog of Xenopus gene which Prevents Mitotic Catastrophe (hPMC2).
110                                              Mitotic catastrophe implies that mitotic podocytes event
111 omosome segregation, which, in turn, induces mitotic catastrophe in cancer cells and enhances their v
112 ubject to inhibitory phosphorylation induced mitotic catastrophe in cells lacking p53, the p53-expres
113 eads to centrosome depletion, which triggers mitotic catastrophe in cells that exhibit amplicon-direc
114  with accumulation of DNA damage that led to mitotic catastrophe in differentiated podocytes.
115 leads to aberrant cell cycle progression and mitotic catastrophe in epidermal keratinocytes, which is
116 actor 1; Xenopus laevis XPMC2, prevention of mitotic catastrophe in fission yeast; Drosophila melanog
117 ere we investigate replication stress-driven mitotic catastrophe in human cells and identify that rep
118 multaneous Chk1- and MK2 inhibition leads to mitotic catastrophe in KRAS-mutant cells.
119 repair inhibition resulted in cell death via mitotic catastrophe in MGMT-producing cells.
120 ur studies show centrosome reduplication and mitotic catastrophe in osteosarcoma cells inducibly expr
121 hromosomal abnormalities, ultimately causing mitotic catastrophe in PARP inhibitor treated HR-profici
122  that discusses the clinical implications of mitotic catastrophe in podocyte health during diabetic k
123 dc2/cyclin B complexes, specifically induced mitotic catastrophe in pRb-deficient myocytes, consisten
124 y a high level of spontaneous DNA damage and mitotic catastrophe in the absence of the master checkpo
125 g an explanation for the spindle failure and mitotic catastrophe in the MAD2 knockdown cells.
126    siRNA-mediated reduction of Msi-1 lead to mitotic catastrophe in tumor cells.
127 ion stabilizes APC substrates and results in mitotic catastrophe including centrosome overduplication
128 ffect was ATM-dependent and mediated through mitotic catastrophe independently of apoptosis.
129 amage, cell cycle checkpoint activation, and mitotic catastrophe-induced apoptotic cell death.
130 more than two centrosomes and, indicative of mitotic catastrophe, irregularly shaped nuclei or multip
131                                              Mitotic catastrophe is a broad descriptor encompassing u
132 he actin-related mode of cell death known as mitotic catastrophe is an important pathway involved in
133 lear whether the termination of mitosis in a mitotic catastrophe is regulated by DNA damage checkpoin
134                                              Mitotic catastrophe is the response of mammalian cells t
135 olyploidy are hallmarks of radiation-induced mitotic catastrophe (MC), a common phenomenon occurring
136             Higher eukaryotes have evolved a mitotic catastrophe mechanism for eliminating mitosis-in
137 nuclear bridging, nuclear fragmentation, and mitotic catastrophe, mirroring the effects of HDACi on c
138 cytes to re-enter the cell cycle and undergo mitotic catastrophe, modulated via the Hippo pathway but
139 reated with nocodazole were found to undergo mitotic catastrophe more readily when endogenous caspase
140 tion during metaphase, ultimately leading to mitotic catastrophe, multinucleation, and the loss of st
141 d protein (XORC2) by its ability to rescue a mitotic-catastrophe mutant of the fission yeast Schizosa
142                                              Mitotic catastrophe occurs when cells enter mitosis with
143                  These alterations result in mitotic catastrophe of neuronal progenitors, which would
144 ng triggers multipolar spindle formation and mitotic catastrophe, offering an attractive therapeutic
145 , and DNA damage before cells display severe mitotic catastrophe or apoptosis.
146  complete cell division, resulting in either mitotic catastrophe or endoreduplication.
147 s because apoptosis-resistant cells die from mitotic catastrophe or undergo senescence-like terminal
148 iation (IR) by inducing apoptosis, necrosis, mitotic catastrophe, or permanent growth arrest.
149  DSB load, serves as a reliable predictor of mitotic catastrophe outcomes following genome damage.
150 s originally isolated as a suppressor of the mitotic catastrophe phenotype of a cdc2-3w wee1-50 doubl
151 ase and cytokinesis and ultimately underwent mitotic catastrophe, phenotypes that were rescued by het
152 dition, filamin-A-deficient M2 cells undergo mitotic catastrophe-related nuclear fragmentation after
153  new mechanistic model for the phenomenon of mitotic catastrophe, relying on a combination of unregul
154             In brief, ZIKV infection induces mitotic catastrophe resulting from unscheduled mitotic e
155 tinucleated cells that then arrest or die by mitotic catastrophe, revealing a new role for Myc in the
156         DmChk2 is therefore essential for a "mitotic catastrophe" signal that disrupts centrosome fun
157 a demonstrate how replication stress-induced mitotic catastrophe signals cell death with implications
158 cell death that resembles a necrotic form of mitotic catastrophe suggesting that CD95 protects cancer
159  proliferation, inhibition of apoptosis, and mitotic catastrophe, suggesting an important potential m
160                                          Six mitotic catastrophe suppressor (mcs1-6) genes were ident
161                         A mutation of mcs4+ (mitotic catastrophe suppressor) was originally isolated
162 environmental stresses as the product of the mitotic catastrophe suppressor, mcs4.
163 ant role of UNC5B in small-T antigen-induced mitotic catastrophe that also requires PP2A.IMPORTANCE U
164 ving into an aberrant mitosis, indicative of mitotic catastrophe that may contribute to increased sen
165  attenuates mitotic transition and increases mitotic catastrophe, thereby inhibiting glioma developme
166 tylation-phosphorylation permits escape from mitotic catastrophe, thus allowing early precursor lesio
167 s to incomplete cytokinesis and irreversible mitotic catastrophe via actomyosin ring malformation.
168                                 Importantly, mitotic catastrophe was also confirmed in isolated adult
169 uction of a further mechanism of cell death, mitotic catastrophe, was observed in the more sensitive
170  arrested state, which displays hallmarks of mitotic catastrophe, was prevented by expression of eith
171 a screen designed to discover suppressors of mitotic catastrophe, we identified the Xenopus ortholog
172             PEAMOtecan kills glioma cells by mitotic catastrophe with p53 mutant/knockdown cells bein

 
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