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1 red for efficient gamma-ray damage repair or mitotic recombination.
2  spontaneous and double-strand break-induced mitotic recombination.
3 f heterozygosity events presumed to arise by mitotic recombination.
4 WC, reversion of KRT1 mutations occurred via mitotic recombination.
5 bility that these variants were generated by mitotic recombination.
6 reaks (DSBs) and can occur by deletion or by mitotic recombination.
7  WTK1 cells could be attributed, in part, to mitotic recombination.
8 ecombination substrates have been studied in mitotic recombination.
9 n young larvae were rendered null for Khc by mitotic recombination.
10 t, Rad1/Rad10 endonuclease also functions in mitotic recombination.
11 m these mice show elevations in the rates of mitotic recombination.
12                The remaining cases exhibited mitotic recombination.
13 of the DAP(r) variants were a consequence of mitotic recombination.
14 RAD52 are required for alternate pathways of mitotic recombination.
15 t telomeres also impose a position effect on mitotic recombination.
16 target site preferences, or increase cDNA or mitotic recombination.
17  rec14 mutations had no detectable effect on mitotic recombination.
18 he possibility of heterokaryon formation and mitotic recombination.
19 match repair genes MSH2 and MSH3 function in mitotic recombination.
20 -mediated single-strand annealing pathway of mitotic recombination.
21  double-strand break repair, and meiotic and mitotic recombination.
22 leotide excision repair and they also act in mitotic recombination.
23 tive mutants are impaired in Alu-IRs-induced mitotic recombination.
24 ng lox-like sequences can exhibit almost 20% mitotic recombination.
25 sion yeast strains also exhibited defects in mitotic recombination.
26 loss of heterozygosity on chromosome 17q via mitotic recombination.
27 ation and elevated rates of both meiotic and mitotic recombination.
28 llicular lymphoma (FL) and is usually due to mitotic recombination.
29 is the major strand-transferase required for mitotic recombination.
30 ely minor role in regulating the fidelity of mitotic recombination.
31 ted with cancer susceptibility and increased mitotic recombination.
32 genes have conserved function in suppressing mitotic recombination.
33 combination events at intervals of preferred mitotic recombination.
34 ty in desmoids involved deletion rather than mitotic recombination.
35 o whole-chromosome deletion and regain or to mitotic recombination.
36 f Mph1-MHF and exerts a procrossover role in mitotic recombination.
37 of TNR expansion, of TNR contraction, and of mitotic recombination.
38 interstitial LOH on 8p12 may be generated by mitotic recombination.
39 oss of ATRX results in increased centromeric mitotic recombination, a high frequency of sister chroma
40 ingle or double homologous and nonhomologous mitotic recombinations, a translocation, chromosome brea
41 cases, representing about 80% of all events, mitotic recombination accounted for all loss of heterozy
42                 We examined the frequency of mitotic recombination across an MRS of average size (app
43 nificant bias in apparent nondisjunction vs. mitotic recombination among male vs. female patients or
44 xhibits a 5-fold higher rate of spontaneous, mitotic recombination and a greater than 10-fold increas
45                                     Elevated mitotic recombination and cell cycle delays are two of t
46                               Frequencies of mitotic recombination and chromosome loss are elevated i
47 ave approximately 80-fold increased rates of mitotic recombination and chromosome loss.
48 of DNA translocases required for meiotic and mitotic recombination and DNA repair.
49  instability phenotypes, including increased mitotic recombination and elongated telomeres.
50  pcr1, spc1, or wis1 and was undetectable in mitotic recombination and in meiotic recombination when
51 y of tumors showing allele loss occurring by mitotic recombination and induced by oxidative damage.
52  BALB/c-Trp53(+/-) mammary tumors occurs via mitotic recombination and is a dominant genetic trait th
53 instability, resulting in increased rates of mitotic recombination and mutagenesis.
54 or rad1 mutant strains that are defective in mitotic recombination and removing unpaired DNA single s
55                                         Most mitotic recombination and repair genes of Saccharomyces
56         hsk1 cells suffer increased rates of mitotic recombination and require recombination proteins
57                Overall, the distributions of mitotic recombination and whole-chromosome LOH in the MS
58        Thus, chromosomal divergence inhibits mitotic recombination and, consequently, may act as a mo
59                  These results indicate that mitotic recombination and, to a much lesser extent, dele
60 h complete LOH; 1-2 terminal regions of LOH (mitotic recombination); and 1 interstitial region of LOH
61 ients, homozygous in a subset as a result of mitotic recombination, and arose in a multipotent progen
62 ones with increased rates of point mutation, mitotic recombination, and chromosome loss.
63 ed reduction in cell proliferation following mitotic recombination, and generate few viable gene conv
64 e findings indicate that WRN plays a role in mitotic recombination, and that a loss of WRN function m
65  a mosaic of each parental species following mitotic recombination, and two chromosomes are trisomic.
66           However, the genetic modulators of mitotic recombination are not well understood.
67 ains displayed increased chromosome loss and mitotic recombination, as well as defective DNA damage r
68  dicentrics in Saccharomyces cerevisiae by a mitotic recombination assay.
69                                              Mitotic recombination assays suggest that these elongati
70 e present study we sequenced the products of mitotic recombination between 94%-identical substrates i
71 monstrate that MMR proteins strongly inhibit mitotic recombination between diverged sequences in Sacc
72                               We report that mitotic recombination between homologous chromosomes occ
73 a demonstrate that copy number variation and mitotic recombination between homologous chromosomes und
74 fraction of LOH events appears to arise from mitotic recombination between homologous chromosomes, re
75 e to an elevated rate of crossover following mitotic recombination between homologous chromosomes.
76 mical treatments for stimulating genome-wide mitotic recombination between human and chimpanzee chrom
77  genes confers defects in the suppression of mitotic recombination between similar but nonidentical (
78  introduced into the same cell and joined by mitotic recombination between the vector arms and the ho
79 of the effect of the MRS on a similar event, mitotic recombination between two chromosome homologs.
80 se rearrangements was measured by monitoring mitotic recombination between two his3 fragments, his3-D
81 is+ prototrophs that result from homologous, mitotic recombination between two truncated his3 genes,
82 rand DNA breaks, indicating the formation of mitotic recombination break points.
83 ely to structural alterations rather than to mitotic recombination, break-induced replication, or gen
84                                              Mitotic recombination, but not point mutation, partly ac
85  expression also strongly induces intragenic mitotic recombination, but only in a strain possessing u
86  rad27 mutants showed similar high levels of mitotic recombination, but varied in their growth rate a
87                            Here we show that mitotic recombination can be reproducibly induced in mou
88  into four classes: apparent point mutation, mitotic recombination, chromosome loss, and large inters
89                                Unexpectedly, mitotic recombination clones point to JO neuron self-rep
90 r such regions, which are the consequence of mitotic recombination, contain homozygous mutations in g
91                           This suggests that mitotic recombination contributes to the survival and th
92 osome 5 is the first large-scale analysis of mitotic recombination done in C. albicans and indicates
93 y, these phenomena suggest that dysregulated mitotic recombination drives the genomic instability und
94         Very few crossovers occurred in ade6 mitotic recombination, either allelic or ectopic.
95                                    Increased mitotic recombination enhances the risk for loss of hete
96 mic sequencing, we mapped the breakpoints of mitotic recombination events and other chromosome rearra
97                                              Mitotic recombination events between homologous chromoso
98 oduction of EDSBs and of ensuing spontaneous mitotic recombination events can account for a substanti
99 uman chromosome 7 and found evidence of rare mitotic recombination events in human lymphocytes.
100 orders of magnitude higher than observed for mitotic recombination events involving single-copy genes
101                                              Mitotic recombination events resulting in extended loss
102                                              Mitotic recombination events were found to occur through
103 gion we have searched for small deletions or mitotic recombination events, neither of which would be
104                                              Mitotic recombination experiments in the germline show S
105                             This analysis of mitotic recombination frequencies across a large portion
106                    Our results indicate that mitotic recombination frequencies across the MRS were no
107 ation done in C. albicans and indicates that mitotic recombination frequencies are similar to the rat
108  but their near-diploid karyotypes concealed mitotic recombination frequencies similar to those of MS
109    In contrast to meiotic recombination, the mitotic recombination frequency did not decline near the
110                 Loss of the JAK2 mutation by mitotic recombination, gene conversion, or deletion was
111 ible mechanisms may be used in this process, mitotic recombination, gene conversion, or loss of one c
112     Additionally, in some revertant patches, mitotic recombination generated areas with homozygous no
113            Mutations in REC103 do not affect mitotic recombination, growth, or repair.
114 oss-of-heterozygosity (LOH), suggesting that mitotic recombination has a significant impact on geneti
115 ion of segmental uniparental disomy (UPD) by mitotic recombination has been reported in 15% to 20% of
116      Techniques to produce mosaic animals by mitotic recombination have been extensively developed in
117 me (an inverted repeat of 80 bp) conferred a mitotic recombination hotspot relative to a control nonp
118                 We found that an Alu-IR is a mitotic recombination hotspot requiring MRE11/RAD50/XRS2
119 ences and inverted Alu repeats (Alu-IRs) are mitotic recombination hotspots, requiring the repair fun
120                                  By inducing mitotic recombination in a subclass of eye progenitors,
121      Here we have exploited the high rate of mitotic recombination in Bloom's syndrome protein (Blm)-
122                                 We exploited mitotic recombination in developing Drosophila imaginal
123                    To this end, we performed mitotic recombination in Drosophila melanogaster, RNAi-m
124                                 In contrast, mitotic recombination in ES cells appeared to be suppres
125  direct evidence for a role of Mus81-Eme1 in mitotic recombination in higher eukaryotes and support t
126 ulation of Mus81 by RNA interference reduces mitotic recombination in human somatic cells.
127 enesis and offer a rare opportunity to study mitotic recombination in nonneoplastic cells.
128 hromosomal homology by backcrossing restores mitotic recombination in offspring.
129                Although DNA breaks stimulate mitotic recombination in plants, their effects on meioti
130 epresses not only Ty1 transcription but also mitotic recombination in RDN1.
131                            The prevalence of mitotic recombination in regions proximal to Aprt did no
132                     Most previous studies of mitotic recombination in Saccharomyces cerevisiae have f
133                           The suppression of mitotic recombination in some F1 hybrids in which meioti
134           Meiotic recombination differs from mitotic recombination in that DSBs are repaired using ho
135                        Although interhomolog mitotic recombination in the CUP1 locus is elevated rela
136 ng distance from the centromere, implicating mitotic recombination in the process.
137  loss of heterozygosity (LOH) resulting from mitotic recombination in vivo constitutes the underlying
138 d model for studying somatic mutagenesis and mitotic recombination in vivo.
139  dominant-negative RAD51 protein to suppress mitotic recombination in WRN and control cells: the supp
140  that MutSbeta is required for the increased mitotic recombination induced by RS or FANCM loss at CFS
141           These data suggest that homologous mitotic recombination, induced by O6-MeG in a subpopulat
142 o examine recognition of these mismatches in mitotic recombination intermediates.
143 usion of the yeast-derived FRT/FLP system of mitotic recombination into Drosophila.
144 a recombination reporter, we have shown that mitotic recombination is dependent on the RAD52 gene, bu
145 ation reporter we have previously shown that mitotic recombination is dependent on the RAD52 gene.
146 g positive selection and/or that the rate of mitotic recombination is elevated in individuals with th
147                          Here we report that mitotic recombination is suppressed, to various degrees
148 ernal alleles were retained, indicating that mitotic recombination is the most likely mechanism of LO
149 itu hybridization analysis demonstrated that mitotic recombination is the predicted mechanism of LOH
150 frequency of Aprt LOH, mediated primarily by mitotic recombination, is fully consistent with our prev
151 rait has mild SCD resulting from postzygotic mitotic recombination leading to UPD.
152                    LOH generally occurred by mitotic recombination, leaving two identical alleles, ea
153 nomena, including allodiploid hybridization, mitotic recombination, loss-of-heterozygosity, and aneup
154 itous recombinase Rad51, suggesting that the mitotic recombination machinery is reactivated following
155 n to be required for spontaneous and induced mitotic recombination, meiotic recombination, and mating
156 endent probe amplification (MLPA) identified mitotic recombination (MR) as the cause of LOH in 14 out
157                                              Mitotic recombination (MR) between chromosome homologs i
158             Such cases appear to result from mitotic recombination occurring in early embryogenesis a
159 highly suppressed in G1 cells to ensure that mitotic recombination occurs solely between sister chrom
160                                              Mitotic recombination occurs with high frequency in huma
161           This observation demonstrates that mitotic recombination of chromosome 6 can also give rise
162   Through Cre/loxP-mediated interchromosomal mitotic recombination of two reciprocally chimeric fluor
163 e in evolution despite its susceptibility to mitotic recombination on inhibition of DNA replication.
164 ause of the observed tk deletions was either mitotic recombination or chromosomal loss accompanied by
165  polycythemia vera does not commonly involve mitotic recombination or chromosome loss and that micros
166 ome 3q to define the minimal region in which mitotic recombination or deletion results in LOH, which
167                                              Mitotic recombination org nondysjunction are common mech
168  2% sequence divergence were used to examine mitotic recombination outcome (crossover or noncrossover
169  of the copy number gain events occur by the mitotic recombination pathway.
170                  We suggest that meiotic and mitotic recombination pathways coexist within meiotic ce
171 are microsatellite repeat distributions with mitotic recombination patterns available from earlier ge
172 hts into the roles that both outcrossing and mitotic recombination play in shaping the genome archite
173 6 kbp convert efficiently during DSB-induced mitotic recombination, primarily by Msh2p- and Pms1p-dep
174  In a proportion of V617F-positive patients, mitotic recombination produces mutation-homozygous cells
175 from heterozygous to homozygous mutations by mitotic recombination provides a mechanism for relapse.
176                              FLP/FRT-induced mitotic recombination provides a powerful method for cre
177  of the plastic response in the interhomolog mitotic recombination rate, and we propose that this may
178 ants is not highly unusual in the sense that mitotic recombination rates are normal, implying that a
179 ved genetic escape mechanism was spontaneous mitotic recombination resulting in loss of heterozygosit
180  expansion of beta cells that have undergone mitotic recombination, resulting in paternal disomy of p
181 e frequency of somatic variants derived from mitotic recombination should be diminished in progeny fr
182  of gamma-ray damage, but exhibits decreased mitotic recombination similar to rad52 null strains, was
183 agents and established another site-specific mitotic recombination system.
184 l-cell communication via Notch (N) receptor, mitotic recombination that removes the N ligand Delta (D
185  cases, suggesting negative interference for mitotic recombination, the opposite of what is usually o
186 oth Sgs1 and Srs2 limit the overall level of mitotic recombination, there are distinct differences in
187 r integration, for deletion and for enhanced mitotic recombination, thereby increasing the repertoire
188                                 We have used mitotic recombination to generate patches of homozygous
189 t for syntaxin1 expression were generated by mitotic recombination to reduce the maternal contributio
190 levels of RNA polymerase II transcription on mitotic recombination was examined using lys2 recombinat
191 cy of clones with point mutations, such that mitotic recombination was still the primary cause of APR
192           The results demonstrate that while mitotic recombination was the mechanism for homozygosis
193 estimated that the frequency of interhomolog mitotic recombination was under standard growth conditio
194 ng an assay to detect unselected products of mitotic recombination, we found a significant decrease i
195 tic system in yeast that allows detection of mitotic recombination, we found that the frequency of cr
196 XpF/Ercc1 endonuclease has a similar role in mitotic recombination, we targeted the APRT locus in Chi
197 for spontaneous and transcription-stimulated mitotic recombination were determined using a recombinat
198 rminal domain, but Rad51 focus formation and mitotic recombination were elevated above wild-type leve
199 vious studies involving hetDNA formed during mitotic recombination were restricted to one locus.
200          This implies that mutation precedes mitotic recombination which acts as a "second hit" respo
201 etween homologous chromatids, in contrast to mitotic recombination, which occurs primarily between si
202    The assembled YAC was further extended by mitotic recombination with a YAC containing a 280-kb reg
203 ed DNA breaks (DSBs) that can be repaired by mitotic recombination with the homolog.
204  from silent chromatin regions and increased mitotic recombination within tandem repeats of rDNA.

 
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