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1 matic valvular disease, or greater than mild mitral stenosis).
2 ion) is associated with a risk of functional mitral stenosis.
3  471 with aortic stenosis, and 193 with mild mitral stenosis.
4 treatment in selected patients with calcific mitral stenosis.
5 niques, particularly in regard to functional mitral stenosis.
6 missurotomy (PMC) remain debated in calcific mitral stenosis.
7 rly, and those with more than mild aortic or mitral stenosis.
8 sis, and treatment options for patients with mitral stenosis.
9 ell, and no patient had clinical evidence of mitral stenosis.
10 actical technique for clinical evaluation of mitral stenosis.
11 nly used exercise test for the evaluation of mitral stenosis.
12 illary basement membrane in diseases such as mitral stenosis.
13 ns in leaflet geometry seen in patients with mitral stenosis.
14 he application of balloon commissurotomy for mitral stenosis.
15  a further 1,262 (0.02%) were diagnosed with mitral stenosis.
16  may cause obstructive symptoms analogous to mitral stenosis.
17 atients with a larger initial MVA and milder mitral stenosis (0.12 vs. 0.06 vs. 0.03 cm2/year for mil
18 rtic regurgitation 0.67 (95% CI, 0.64-0.70), mitral stenosis 1.95 (95% CI, 1.76-2.20), and mitral reg
19 t prolapse (17), chordal shortening (1), and mitral stenosis (1).
20 or aortic stenosis 1.62 (95% CI, 1.59-1.65), mitral stenosis 2.28 (95% CI, 2.08-2.50), and excess ris
21  regurgitation 0.63 (95% CI, 0.43-0.94), and mitral stenosis 3.47 (95% CI, 1.37-8.84).
22 ated mitral valve replacement or evidence of mitral stenosis: A total of 504 (54%) had congenitally m
23 age, larger aortic root diameter, aortic and mitral stenosis and albuminuria.
24 uced hyperemic MBF were an HLHS subtype with mitral stenosis and aortic atresia (P=0.017), late gadol
25                                     Finally, mitral stenosis and mitral regurgitation studies evaluat
26 sing problem in elderly people, causing both mitral stenosis and regurgitation which are difficult to
27 tary effects of mitral balloon valvotomy for mitral stenosis and the development of a reasonable appr
28 alyzed long-term results of PMC for calcific mitral stenosis and the factors associated with late fun
29                                              Mitral stenosis and thromboembolism did not develop.
30 se with mitral regurgitation with or without mitral stenosis) and the multivalvular lesions (MVL) cat
31 creased after valvuloplasty in patients with mitral stenosis, and (2) whether the magnitude of the in
32 rable effect on the hemodynamic variables of mitral stenosis, and long-term follow-up data suggest th
33 their aortic stenosis, aortic regurgitation, mitral stenosis, and mitral regurgitation stage were def
34 rotid artery for aneurysm, John Abernethy on mitral stenosis, and Sir David Dundas on acute rheumatic
35 se with prosthetic heart valves, significant mitral stenosis, and valvular heart disease (VHD) requir
36 syndrome, double outlet right ventricle with mitral stenosis/atresia, or mitral valve dysplasia and R
37 modynamics were measured in 57 patients with mitral stenosis before and 20 to 30 min after undergoing
38  use of OMC in 312 consecutive patients with mitral stenosis between 1983 and the present.
39           This effect has been recognized in mitral stenosis but assumed to be absent in aortic steno
40                      Previously asymptomatic mitral stenosis can lead to remarkably sudden developmen
41 tral stenosis (DMS) is an important cause of mitral stenosis, developing secondary to severe mitral a
42                                 Degenerative mitral stenosis (DMS) is an important cause of mitral st
43                        Because patients with mitral stenosis frequently exhibit greater improvements
44 e was no association between SBP and risk of mitral stenosis (HR per 20 mmHg higher SBP 1.03; CI 0.93
45  aortic regurgitation and without associated mitral stenosis) in adults in the Western world has been
46                                              Mitral stenosis is a common disease that causes substant
47       Balloon mitral valvuloplasty (BMV) for mitral stenosis is a procedure that has evolved signific
48 rience with congenital interatrial shunts in mitral stenosis, it has been hypothesized that the creat
49 sults of PMC are less satisfying in calcific mitral stenosis, long-term functional outcome depends on
50 from 1993 to 2005 that was unassociated with mitral stenosis, mitral valve replacement, or a previous
51 ies typical of the spectrum in patients with mitral stenosis: mobile doming, intermediate conical, an
52 igher prevalences of aortic stenosis (AS) or mitral stenosis (MS) (p<0.001).
53  mostly in patients with moderate and severe mitral stenosis (MS) and AS.
54 D), aortic regurgitation (AR) in 279 (5.3%), mitral stenosis (MS) in 234 (4.5%), mitral regurgitation
55                       Prevalence of calcific mitral stenosis (MS) increases with age; however, its na
56                            Severe congenital mitral stenosis (MS) is a rare anomaly that is frequentl
57 ment (TAVR) in aortic stenosis patients with mitral stenosis (MS) suggested a poor short-term prognos
58 emporary patients with suspected significant mitral stenosis (MS) undergoing rest and treadmill stres
59                         Significant residual mitral stenosis (MS) was defined as mean gradient >=10 m
60                         Significant residual mitral stenosis (MS) was defined as mean gradient 10 mm
61 ant determinant of pulmonary hypertension in mitral stenosis (MS).
62 gitation (MR) and 16 with moderate to severe mitral stenosis (MS).
63                       Patients with isolated mitral stenosis often benefit from percutaneous balloon
64 ients with significant mitral valve disease (mitral stenosis or > or = moderate mitral regurgitation)
65             Patients with moderate to severe mitral stenosis or mechanical heart valves were excluded
66 farin excluded patients with moderate/severe mitral stenosis or mechanical heart valves, but variably
67  mitral valve replacement provides relief of mitral stenosis or mitral regurgitation.
68 aortic regurgitation) and without associated mitral stenosis or mitral valve replacement strongly sug
69 o-severe left-sided valvular disease (aortic/mitral stenosis or regurgitation), or severe left ventri
70  moderate or severe valvular disease (aortic/mitral stenosis or regurgitation, tricuspid regurgitatio
71            Patients operated on for isolated mitral stenosis or who had a prior mitral valve interven
72 history of atrial fibrillation, a pacemaker, mitral stenosis, or congenital heart disease.
73 utaneous approach is usually used to correct mitral stenosis, other valve lesions require surgical in
74 able on the echocardiographic progression of mitral stenosis, particularly on progressive changes in
75 , intensive care, right ventricular failure, mitral stenosis, prostacyclin, nitric oxide, sildenafil,
76 for mitral insufficiency, whereas congenital mitral stenosis remains extremely problematic in the you
77                                      In mild mitral stenosis, the half-time was approximately 100 mse
78 lidation n=121) of patients with symptomatic mitral stenosis undergoing PMV were studied.
79                      Seventeen patients with mitral stenosis underwent echocardiography and CMR.
80           It also examines the following: 1) mitral stenosis versus mitral regurgitation and the pres
81                     CBC for the treatment of mitral stenosis was performed in 132 patients from 1986
82              Thus, in selected patients with mitral stenosis who require an interventional procedure,
83  of 314 patients undergoing PMC for calcific mitral stenosis with 710 patients with noncalcified valv
84  mitral regurgitation might cause functional mitral stenosis, yet its clinical impact and underlying