ライフサイエンスコーパス: mixed cellularity

1 lularity subtypes but were much stronger for mixed cellularity.

2 In all cases of nodular sclerosis (132), mixed cellularity (34), lymphocyte depletion (2), and un

3 hocyte-predominant, 25 nodular sclerosis, 16 mixed cellularity, 5 lymphocyte depletion, and 5 unclass

4 D cases (27%) and was mainly confined to the mixed cellularity and nodular sclerosis subtypes.

5 %) of 551 nodular sclerosis, 67 (47%) of 143 mixed cellularity, and all 5 lymphocyte depletion.

6 ant, 26 of 39 nodular, sclerosis, two of two mixed cellularity, and two of four lymphocyte depletion)

7 lerosis subtype cases and only a minority of mixed cellularity cases, suggesting that B2M deficiency

8 Older patients with mixed cellularity had significantly worse 5-year event-f

9 Mixed cellularity histology and mantle field technique a

10 In patients with non-mixed cellularity histology, older patients had a signif

11 requent, explaining the higher proportion of mixed cellularity HL found in PWAs.

12 larity, thereby increasing the proportion of mixed cellularity HL seen in PWAs.

13 HL (OR = 3.18; 95% CI: 1.23, 8.17), and with mixed-cellularity HL (OR = 4.25; 95% CI: 1.66, 10.90).

14 9081, HLA-DPB1*03:01, Val86 in HLA-DRB1) and mixed cellularity Hodgkin lymphoma (rs1633096, rs1319632

15 nodular-sclerosis, lymphocyte-depletion, and mixed-cellularity Hodgkin's disease (HD).

16 four types of classic HL (nodular sclerosis, mixed cellularity, lymphocyte-depleted or lymphocyte-ric

17 pression of IP-10 and Mig was highest in the mixed cellularity (MC) subtype, whereas expression of eo

18 han patients with nodular-sclerosing (NS) or mixed-cellularity (MC) disease.

19 BV-positive HL with older age, male sex, and mixed-cellularity (MC) histological subtypes.

20 s (n = 64), lymphocyte predominant (n = 15), mixed cellularity (n = 3), and unclassified (n = 1).

21 nd in patients with mixed cellularity or non-mixed cellularity (nodular sclerosing and not-otherwise-

22 of 49 ALCL and 3 (2 nodular sclerosis and 1 mixed cellularity) of 72 HD showed the presence of NPM-A

23 nt factors both overall and in patients with mixed cellularity or non-mixed cellularity (nodular scle

24 creased age, EBV-encoded RNA positivity, and mixed cellularity subtype of CHL.

25 Mixed cellularity subtype predominated (five of eight ca

26 re significant for the nodular sclerosis and mixed cellularity subtypes but were much stronger for mi

27 iations were found for nodular sclerosis and mixed cellularity subtypes.

28 Nodular sclerosis and mixed-cellularity subtypes had similar frequency of CT45

29 sclerosing decreased more precipitously than mixed cellularity, thereby increasing the proportion of

30 In cases of mixed cellularity type, neoplastic cells generally appea